Search results for "coli"

showing 10 items of 1981 documents

Retrospective review of L3 myelomeningocele in three age groups: should posterolateral iliopsoas transfer still be indicated to stabilize the hip?

2005

The data from 58 hips in 29 myelodysplastic children with L3 paralysis has been reviewed retrospectively. Posterolateral transfer of the iliopsoas (Sharrard technique) was performed on all hips to correct muscle imbalance between 1975 and 1992. The average length of follow-up was 21 years. Preoperatively, 58.6% of the hips were either subluxated or dislocated (these were surgically reduced at the same stage). Radiological assessment revealed that 91.4% of the hips were stable at preschool age and 84.5% in adolescence. Of the patients 86% were functional walkers at preschool age and 75.8% in adolescence. Downward transitions in seven patients were related to the presence of scoliosis, spasti…

AdultJoint InstabilityMalemusculoskeletal diseasesmedicine.medical_specialtyMeningomyeloceleAdolescentWalkingScoliosisImmobilizationJoint capsuleParalysismedicineHip DislocationHumansParalysisOrthopedics and Sports MedicineSpasticityStage (cooking)ChildMuscle SkeletalRetrospective Studiesbusiness.industryRetrospective cohort studymedicine.diseaseSurgeryTreatment Outcomemedicine.anatomical_structureChild PreschoolRadiological weaponPediatrics Perinatology and Child HealthFemaleHip Jointmedicine.symptomIliopsoasbusinessJoint CapsuleFollow-Up StudiesJournal of Pediatric Orthopaedics B
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Role of TLR4 polymorphisms in inflammatory responses: implications for unsuccessful aging.

2007

The total burden of infection at various sites may affect the progression of atherosclerosis and Alzheimer's disease (AD), the risk being modulated by host genotype. The role of lipopolysaccharide (LPS) receptor TLR4 is paradigmatic. It initiates the innate immune response against gram-negative bacteria, and TLR4 single nucleotide polymorphisms (SNPs), such as +896A/G, known to attenuate receptor signaling, have been described. This SNP shows a significantly lower frequency in patients affected by myocardial infarction or AD. Thus, people genetically predisposed to developing lower inflammatory activity seem to have less chance of developing cardiovascular disease (CVD) or AD. In the presen…

AdultLipopolysaccharidesMaleAgingTime FactorsLipopolysaccharideGenotypeLeukotriene B4Myocardial InfarctionInflammationSingle-nucleotide polymorphismBiologyLeukotriene B4Polymorphism Single NucleotideGeneral Biochemistry Genetics and Molecular BiologyDinoprostoneProinflammatory cytokinechemistry.chemical_compoundHistory and Philosophy of ScienceAlzheimer DiseaseGenotypemedicineTLR4 SNPAgeing related disease longevityEscherichia coliHumansCells CulturedEscherichia coli InfectionsSettore MED/04 - Patologia GeneraleInflammationInnate immune systemBlood CellsGeneral NeuroscienceMiddle AgedImmunity InnateToll-Like Receptor 4chemistryImmunologyTLR4lipids (amino acids peptides and proteins)Femalemedicine.symptomAnnals of the New York Academy of Sciences
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Elevated levels of anti-endotoxin antibodies in patients with bilateral idiopathic acute anterior uveitis

2010

. Purpose:  Endotoxins have been proved to be responsible for acute anterior uveitis (AAU) in animals in a well-established experimental model of endotoxin-induced uveitis (EIU). The purpose of our study was the detection of antibodies against endotoxins of selected enterobacteria in the serum of patients with idiopathic AAU and searching for correlations between the levels of these antibodies and the presence of HLA-B27 antigen as well as characteristic signs of EIU such as bilaterality and the absence of spontaneous recurrences of the disease. Methods:  Reactions of serum IgG antibodies with lipopolysaccharides (LPSs) of Escherichia coli O1, E. coli O10, E. coli O111, E. coli J5, and Kleb…

AdultLipopolysaccharidesMaleKlebsiella pneumoniaeEnzyme-Linked Immunosorbent AssayInflammationDiseaseEye Infections BacterialAntigenGram-Negative BacteriaEscherichia coliHumansMedicineHLA-B27 Antigenbiologybusiness.industryIncidence (epidemiology)General MedicineMiddle Agedmedicine.diseasebiology.organism_classificationAntibodies BacterialUveitis AnteriorKlebsiella pneumoniaeOphthalmologyImmunoglobulin GAcute DiseaseImmunologybiology.proteinEtiologyFemaleAntibodymedicine.symptomGram-Negative Bacterial InfectionsbusinessUveitisActa Ophthalmologica
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Induction of antitoxin responses in Clostridium-difficile-infected patients compared to healthy blood donors

2016

According to the literature Clostridium difficile antitoxins are present in up to 66% of humans. In a survey of ∼400 plasma samples from healthy blood donors we found that less than 6% were positive for anti-TcdA or anti-TcdB antitoxins. Using the same standard immunoassay protocol, we looked for IgG and IgA antitoxins in the blood and stool samples from 25 patients with C. difficile infection (CDI). Some patients with CDI had no antitoxin detected at all, while others had high levels of specific IgG- and IgA-antitoxins against both TcdA and TcdB in blood and IgA-anti-TcdA and -anti-TcdB antibodies in stool. Systemic responses to TcdB and mucosal responses to TcdA predominated. Among patien…

AdultMale0301 basic medicineAdolescentBacterial ToxinsClostridium difficile toxin ABlood DonorsBiologyMicrobiologyMicrobiologyYoung Adult03 medical and health sciences0302 clinical medicineImmune systemmedicineHumans030212 general & internal medicineEnterocolitis PseudomembranousAgedAntigens Bacterialmedicine.diagnostic_testClostridioides difficileCase-control studyMiddle AgedClostridium difficileAntibodies BacterialMolecular TypingTreatment Outcome030104 developmental biologyInfectious DiseasesCase-Control StudiesImmunoassayImmunologyHumoral immunitybiology.proteinFemaleAntitoxinAntibodyAnaerobe
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Characterization of γδ T cells in intestinal mucosa from patients with early onset or long standing inflammatory bowel disease and their correlation …

2019

Abstract Background and Aims Inflammatory bowel disease [IBD] is a complex chronic inflammatory disease of the human gut with no clear aetiology. Traditionally, dysregulated adaptive immune responses play an important role even though accumulating evidence suggests a role also for innate immunity. Because of the well-known plasticity of γδ T cells, we investigated their percentage occurrence, phenotypic features and effector functions in the intestinal mucosa of early-onset and long-standing IBD patients, as compared to healthy subjects. Methods Fresh biopsies from 30 Crohn’s disease and ulcerative colitis patients were obtained and digested, and cells were analysed by flow cytometry. Resul…

AdultMale0301 basic medicineNecrosisAdolescentBiopsyT-LymphocytesInflammatory bowel disease03 medical and health sciences0302 clinical medicineImmune systemIntestinal mucosainflammatory bowel diseasemedicineHumansgamma delta T cellsIntestinal MucosaAgedAged 80 and overInnate immune systembusiness.industryGastroenterologyGeneral MedicineMiddle AgedFlow CytometryInflammatory Bowel Diseasesmedicine.diseaseUlcerative colitisPhenotype030104 developmental biologyImmunologyFemaleTumor necrosis factor alphaInterleukin 17medicine.symptombusinessBiomarkers030215 immunology
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Elevated levels of Bcl-3 inhibits Treg development and function resulting in spontaneous colitis

2017

Bcl-3 is an atypical NF-κB family member that regulates NF-κB-dependent gene expression in effector T cells, but a cell-intrinsic function in regulatory T (Treg) cells and colitis is not clear. Here we show that Bcl-3 expression levels in colonic T cells correlate with disease manifestation in patients with inflammatory bowel disease. Mice with T-cell-specific overexpression of Bcl-3 develop severe colitis that can be attributed to defective Treg cell development and function, leading to the infiltration of immune cells such as pro-inflammatory γδT cells, but not αβ T cells. In Treg cells, Bcl-3 associates directly with NF-κB p50 to inhibit DNA binding of p50/p50 and p50/p65 NF-κB dimers, t…

AdultMale0301 basic medicineP50ScienceGeneral Physics and AstronomyBiologyT-Lymphocytes RegulatoryInflammatory bowel diseaseArticleGeneral Biochemistry Genetics and Molecular BiologyYoung Adult03 medical and health sciences0302 clinical medicineImmune systemB-Cell Lymphoma 3 ProteinProto-Oncogene ProteinsGene expressionmedicineAnimalsHumansColitisMice KnockoutRegulation of gene expressionMultidisciplinaryEffectorHEK 293 cellsQNF-kappa BTranscription Factor RelANF-kappa B p50 SubunitGeneral ChemistryMiddle AgedColitismedicine.diseaseMice Inbred C57BLHEK293 Cells030104 developmental biologyGene Expression Regulation030220 oncology & carcinogenesisImmunologyFemaleProtein BindingTranscription FactorsNature Communications
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Kosaki overgrowth syndrome: A novel pathogenic variant in PDGFRB and expansion of the phenotype including cerebrovascular complications

2020

Heterozygous activating variants in platelet-derived growth factor, beta (PDGFRB) are associated with phenotypes including Kosaki overgrowth syndrome (KOGS), Penttinen syndrome and infantile myofibromatosis (IM). Here, we present three new cases of KOGS, including a patient with a novel de novo variant c.1477A > T p.(Ser493Cys), and the oldest known individual age 53 years. The KOGS phenotype includes characteristic facial features, tall stature, scoliosis, hyperelastic thin skin, lipodystrophy, variable intellectual and neurological deterioration, and abnormalities on brain imaging. Long-term outcome is unknown. Our cases confirm the phenotypic spectrum includes progressive flexion contrac…

AdultMale0301 basic medicinePathologymedicine.medical_specialtyInfantile myofibromatosisPDGFRBScoliosis030105 genetics & heredityCraniosynostosisReceptor Platelet-Derived Growth Factor beta03 medical and health sciencesCamptodactylyGeneticsmedicineHumansJoint dislocationStrokeGrowth DisordersGenetics (clinical)business.industryGenetic VariationMiddle Agedmedicine.diseaseCerebrovascular DisordersPhenotype030104 developmental biologymedicine.symptomLipodystrophybusinessClinical Genetics
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The role of body muscle mass as an indicator of activity in inflammatory bowel disease patients

2020

Malnutrition is an objective disease activity parameter for patients with inflammatory bowel disease (IBD), particularly Crohn's Disease (CD), and is an indicator of lesion expansion or inflammatory activity. Active disease is correlated with the systemic response of the body's immune system, activating a hypermetabolic state and protein degradation (Argiles JM, 2015). These conditions lead to malnutrition, which significantly increases the risk of impaired clinical outcomes, such as delayed recovery or increased mortality (Landi F, 2019). Our aim was to identify malnutrition parameters associated with more pronounced metabolic status changes in IBD patients (i.e., classified as by low and …

AdultMale0301 basic medicinemedicine.medical_specialtyAdolescentEndocrinology Diabetes and MetabolismPilot Projects030209 endocrinology & metabolismProtein degradationInflammatory bowel diseaseEnteral administration03 medical and health sciences0302 clinical medicineWeight lossInternal medicinemedicineHumansMedical historyProspective StudiesNot evaluated030109 nutrition & dieteticsNutrition and Dieteticsbusiness.industryMusclesInflammatory Bowel Diseasesmedicine.diseaseMalnutritionParenteral nutritionColitis UlcerativeFemalemedicine.symptombusinessClinical Nutrition ESPEN
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Significance of serum Il-9 levels in inflammatory bowel disease

2015

IL-9, which may be an inflammatory or regulatory cytokine, can be experimentally produced in a Th17 or modified Th2 context in the presence of T cell receptor (TCR) stimulation. The primary aim of this study was to measure serum IL-9 levels in patients with inflammatory bowel disease (IBD), and evaluate their relationships with the patients’ clinical characteristics. The secondary aim was to determine the levels of interferon-γ (IFN (interferon)-γ), Th2 cytokines (IL-4, IL-5 and IL-13), and IL-6 in order to clarify the context of detectable peripheral cytokines in which IL-9 is produced. Venous blood samples of 43 IBD patients (20 with Crohn’s disease [CD] and 23 with ulcerative colitis [U…

AdultMaleAdolescentmedicine.medical_treatmentImmunologyPopulationContext (language use)lymphocyteInflammatory bowel diseaseTh17 CellTh2 CellsmedicineImmunology and AllergyHumansInterleukin 9educationInterleukin 6Pharmacologyeducation.field_of_studybiologyinflammation; inflammatory bowel disease; interleukin; lymphocyte homing; lymphocytes; mucosal immunity; Adolescent; Adult; Female; Humans; Inflammatory Bowel Diseases; Interleukin-6; Interleukin-9; Male; Middle Aged; Th17 Cells; Th2 Cells; Immunology and Allergy; Immunology; Pharmacologybusiness.industryinterleukinInterleukin-6Inflammatory Bowel DiseaseInterleukin-9Middle Agedmedicine.diseaseInflammatory Bowel DiseasesUlcerative colitisCytokineinflammationImmunologybiology.proteinTh17 Cellsmucosal immunityFemalelymphocyte homingAntibodybusinessHuman
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Quantification of the Fabry marker lysoGb3 in human plasma by tandem mass spectrometry

2011

Morbus Fabry is a hereditary metabolic disorder with low prevalence and late clinical manifestation. A defect in the α-galactosidase gene leads to lysosomal accumulation of the glycolipid globotriaosylceramide (Gb3). Gb3 may be used for monitoring of enzyme replacement therapy (ERT), but diagnostic sensitivity is limited. Recently, globotriaosylsphingosine (lysoGb3) was introduced as a promising new marker with significantly better sensitivity. For Fabry diagnosis, clinical studies and possible therapy monitoring, we established a fast and reliable LC-MS/MS assay for quantification of lysoGb3 in human plasma. Protein precipitation and glycolipid extraction from EDTA plasma was performed usi…

AdultMaleAnalyteMolecular Sequence DataClinical BiochemistryGlobotriaosylceramideChemical FractionationTandem mass spectrometryBiochemistryHigh-performance liquid chromatographyAnalytical Chemistrychemistry.chemical_compoundTandem Mass SpectrometrymedicineHumansProtein precipitationDerivatizationChromatography High Pressure LiquidSphingolipidsChromatographyElutionTrihexosylceramidesReproducibility of ResultsCell BiologyGeneral Medicinemedicine.diseaseFabry diseaseCarbohydrate SequencechemistryCase-Control StudiesLinear ModelsFabry DiseaseFemaleGlycolipidsBiomarkersJournal of Chromatography B
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