Search results for "coli"

showing 10 items of 1981 documents

Natural history of cytomegalovirus infection in a series of patients diagnosed with moderate-severe ulcerative colitis

2011

AIM: To evaluate the natural history of human cytomegalovirus (HCMV) infection in a series of 28 ulcerative colitis patients in whom the search for HCMV was positive. METHODS: A series of 85 patients with moderate-severe ulcerative colitis flare-up were evaluated for a HCMV search by performing a haematoxylin and eosin stain, immunohistochemical assay and nested polymerase chain reaction on rectal biopsies. Among 85 screened patients (19 of whom were steroid resistant/dependant), 28 were positive for HCMV; after remission the patients were followed up clinically and histologically. RESULTS: Among the 22 patients with complete follow-up, in 8 (36%) patients HCMV-DNA persisted in the intestin…

AdultMaleHuman cytomegalovirusmedicine.medical_specialtyBrief ArticlevirusesCongenital cytomegalovirus infectionCytomegalovirusGastroenterologyRecurrenceInternal medicinemedicineHumansProspective StudiesColitisProspective cohort studyAntigens ViralAcute colitisAgedbusiness.industryRemission InductionGastroenterologyvirus diseasesGeneral MedicinePouchitisMiddle Agedbiochemical phenomena metabolism and nutritionmedicine.diseaseUlcerative colitisCytomegalovirus InfectionsDNA ViralImmunologyColitis UlcerativeFemalebusinessNested polymerase chain reactionWorld Journal of Gastroenterology
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Replication of interleukin 23 receptor and autophagy-related 16-like 1 association in adult- and pediatric-onset inflammatory bowel disease in Italy.

2008

AIM: To investigate gene variants in a large Italian inflammatory bowel disease (IBD) cohort, and to analyze the correlation of sub-phenotypes (including age at diagnosis) and epistatic interaction with other IBD genes. METHODS: Total of 763 patients with Crohn's disease (CD, 189 diagnosed at age < 19 years), 843 with ulcerative colitis (UC, 179 diagnosed < 19 years), 749 healthy controls, and 546 healthy parents (273 trios) were included in the study. The rs2241880 [autophagy-related 16-like 1 (ATG16L1)], rs11209026 and rs7517847 [interleukin 23 receptor (IL23R)], rs2066844, rs2066845, rs2066847 (CARD15), rs1050152 (OCTN1), and rs2631367 (OCTN2) gene variants were genotyped. RESULTS: The f…

AdultMaleInterleukin-23 receptorAdolescentGenotypeOrganic Cation Transport ProteinsIBDNod2 Signaling Adaptor ProteinAutophagy-Related Proteinsdigestive systemPolymorphism Single NucleotideInflammatory bowel diseaseYoung AdultSettore MED/38 - Pediatria Generale E SpecialisticaCrohn DiseaseIL23RClinical ResearchmedicineGenetic predispositionHumansGenetic Predisposition to DiseaseAge of OnsetYoung adultChildSolute Carrier Family 22 Member 5ReceptorAgedCrohn's diseaseSymportersbusiness.industryGastroenterologyInfantReceptors InterleukinGeneral MedicineMiddle AgedInflammatory Bowel Diseasesmedicine.diseaseUlcerative colitisdigestive system diseasesLogistic ModelsItalyCase-Control StudiesChild PreschoolImmunologyFemaleAge of onsetCarrier Proteinsbusiness
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DLG5 variants do not influence susceptibility to inflammatory bowel disease in the Scottish population

2005

Introduction: Recent data have suggested that specific haplotypic variants of the DLG5 gene on chromosome 10q23 may be associated with susceptibility to inflammatory bowel disease (IBD) in Germany. Haplotype D, notably characterised by the presence of a G→A substitution at nucleotide 113, was associated with susceptibility to Crohn’s disease (CD) whereas an extended haplotype A conferred protection. Aims: Association of DLG5 haplotypic variants with disease susceptibility, genotype-phenotype relationships, and epistasis with CARD15 was investigated in the Scottish population. Patients and methods: A total of 374 CD, 305 ulcerative colitis (UC), and 294 healthy controls (HC) were studied. Ge…

AdultMaleLetterGenotypePopulationNod2 Signaling Adaptor ProteinSingle-nucleotide polymorphismBiologyInflammatory bowel diseaseLoss of heterozygosityCrohn DiseaseGene FrequencyGenotypemedicineHumansGenetic Predisposition to DiseaseeducationGenotypingAllele frequencyGeneticseducation.field_of_studyTumor Suppressor ProteinsInflammatory Bowel DiseaseHaplotypeGastroenterologyIntracellular Signaling Peptides and ProteinsMembrane ProteinsEpistasis GeneticMiddle Agedmedicine.diseaseInflammatory Bowel Diseasesdigestive system diseasesPhenotypeHaplotypesScotlandImmunologyColitis UlcerativeFemale
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Reducing Body Myopathy with Cytoplasmic Bodies and Rigid Spine Syndrome: A Mixed Congenital Myopathy

2001

At the age of five years a male child started to develop a progressive rigid spine, torsion scoliosis, and flexion contractures of his elbows, knees, hips, and ankles owing to severe proximal and distal muscle weakness. He had three muscle biopsies from three different muscles at ages 7, 11, and 14 years, respectively. Myopathologically, these muscle tissues contained numerous inclusions which, at the ultrastructural level, turned out to be reducing bodies and cytoplasmic bodies, often in close spatial proximity. Similar histological inclusions, although not further identified by histochemistry and electron microscopy, were seen in his maternal grandmother's biopsied muscle tissue who had d…

AdultMaleMuscle tissuePathologymedicine.medical_specialtyWeaknessScoliosisSpinal Muscular Atrophies of ChildhoodSarcomereMyositis Inclusion BodymedicineHumansGenetic Predisposition to DiseaseMuscle SkeletalMyopathyMyositisAgedInclusion Bodiesbusiness.industrySyndromeGeneral MedicineAnatomymedicine.diseasePenetrancePedigreemedicine.anatomical_structureChild PreschoolPediatrics Perinatology and Child HealthDisease ProgressionLordosisFemaleDesminNeurology (clinical)medicine.symptombusinessMyopathies Structural CongenitalNeuropediatrics
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Variants of CARD15 are associated with an aggressive clinical course of Crohn's Disease. An IG-IBD Study

2005

Three major variants of the CARD15 gene confer susceptibility to Crohn's disease (CD). Whether or not these variants correlate with specific clinical features of the disease is under evaluation.We investigated the possible association of CARD15 variants with specific clinical characteristics, including the occurrence of anti-Saccharomyces cerevisiae antibodies (ASCA) and antineutrophil cytoplasmic antibodies (ANCA), in a large cohort of inflammatory bowel disease (IBD) patients and their unaffected relatives.Three hundred and sixteen CD patients (156 with positive family history), 408 ulcerative colitis (UC) patients (206 with positive family history), 588 unaffected relatives, and 205 unre…

AdultMaleNod2 Signaling Adaptor Proteindigestive systemCrohn DiseaseGene FrequencyHumansMedicineSettore MED/12 - GastroenterologiaCrohn's diseaseHepatologybiologyCrohn diseasebusiness.industryIntracellular Signaling Peptides and ProteinsGastroenterologyClinical courseMiddle Agedmedicine.diseasedigestive system diseasesPhenotypeItalyCase-Control StudiesMutationImmunologybiology.proteinColitis UlcerativeFemaleAntibodybusinessFollow-Up Studies
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Confocal Endomicroscopy Identifies Loss of Local Barrier Function in the Duodenum of Patients with Crohnʼs Disease and Ulcerative Colitis

2014

Background: Increased cell shedding with gap formation and local barrier dysfunction can be identified endomicroscopically in the terminal ileum of patients with inflammatory bowel disease. We aim to evaluate whether these changes are also present in the duodenum of patients with inflammatory bowel disease. Methods: Fifteen patients with Crohn's disease (CD), 10 patients with ulcerative colitis (UC), and 10 controls underwent fluorescein-aided confocal laser endomicroscopy (CLE). CLE was performed on macroscopically normal antral and duodenal (D1, D2, D3, D4) mucosa. Representative CLE images were prospectively analyzed. Images were scored for the number of epithelial gaps, cell shedding, a…

AdultMalePathologymedicine.medical_specialtyDuodenumInflammatory bowel diseaseGastroenterologyEndoscopy GastrointestinalEpithelial DamageYoung AdultCrohn DiseaseDuodenitisRisk FactorsInternal medicinemedicineEndomicroscopyHumansImmunology and AllergyProspective StudiesColitisAgedFluorescent DyesCrohn's diseaseMicroscopy ConfocalDuodenitisbusiness.industryGastroenterologyMiddle AgedPrognosismedicine.diseaseUlcerative colitismedicine.anatomical_structureCase-Control StudiesDuodenumColitis UlcerativeFemaleFluoresceinbusinessFollow-Up StudiesInflammatory Bowel Diseases
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HETEROGENEITY OF METATROPIC DYSPLASIA

1983

Metatropic dysplasia is a neonatally manifest entity that is characterized clinically by a rapidly progressing kyphoscoliosis leading to severe shortening of the originally long trunk ("metatropism"). Major radiographic features include flattening and defective ossification of the vertebral bodies, a narrow thorax and a marked hypoplasia of the basilar portions of the ilia with crescent-shaped iliac crests. There is some evidence of genetic heterogeneity. From five personal observations and from a review of the literature we conclude that metatropic dysplasia comprises at least three genetic entities: (1) a nonlethal type with autosomal recessive transmission; (2) a nonlethal dominant type …

AdultMalePathologymedicine.medical_specialtyDwarfismDwarfismShort staturemedicineHumansKyphosisChildKyphoscoliosisBone Diseases DevelopmentalGenetic heterogeneityOssificationbusiness.industryAnatomymedicine.diseaseOsteochondrodysplasiaTrunkHypoplasiaScoliosisPediatrics Perinatology and Child HealthFemalemedicine.symptombusiness
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Stromal SPARC contributes to the detrimental fibrotic changes associated with myeloproliferation whereas its deficiency favors myeloid cell expansion.

2012

Abstract In myeloid malignancies, the neoplastic clone outgrows normal hematopoietic cells toward BM failure. This event is also sustained by detrimental stromal changes, such as BM fibrosis and osteosclerosis, whose occurrence is harbinger of a dismal prognosis. We show that the matricellular protein SPARC contributes to the BM stromal response to myeloproliferation. The degree of SPARC expression in BM stromal elements, including CD146+ mesenchymal stromal cells, correlates with the degree of stromal changes, and the severity of BM failure characterizing the prototypical myeloproliferative neoplasm primary myelofibrosis. Using Sparc−/− mice and BM chimeras, we demonstrate that SPARC contr…

AdultMalePathologymedicine.medical_specialtyMyeloidStromal cellImmunologyAdenomatous Polyposis Coli ProteinGene ExpressionCD146 AntigenBiologyBiochemistryMiceBone MarrowMyeloproliferationmedicineAnimalsHumansMyeloid CellsOsteonectinMyelofibrosisMyeloproliferative neoplasmCells CulturedAgedCell ProliferationAged 80 and overMice KnockoutMesenchymal stem cellMesenchymal Stem CellsPMF SPARC MYELOFIBROSISCell BiologyHematologyMiddle Agedmedicine.diseaseTransplantationHaematopoiesismedicine.anatomical_structureThrombopoietinLeukemia MyeloidPrimary MyelofibrosisFemaleSPARC stroma
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Reactivity of infiltrating T lymphocytes with microbial antigens in Crohn's disease.

1991

Intestinal T lymphocytes are normally unresponsive to microbial and recall antigens in vitro, whereas the same antigens induce strong immune responses in peripheral-blood-derived T cells. We obtained T lymphocytes from peripheral blood and from the non-inflamed and inflamed intestinal mucosa of 6 patients (3 male, 3 female; mean age 33 years) with Crohn's disease. The T cells were stimulated in vitro with a range of microbial antigens. Whereas T cells from normal mucosa were unresponsive, those from inflamed mucosa had a proliferative response comparable to that of the peripheral-blood-derived T cells. These findings suggest that physiologic unresponsiveness to luminal antigens is abrogated…

AdultMalePathologymedicine.medical_specialtyT-LymphocytesInflammationIn Vitro TechniquesLymphocyte ActivationImmune systemIntestinal mucosaAntigenCrohn DiseasemedicineEscherichia coliHumansIntestinal MucosaCrohn's diseaseAntigens BacterialbiologyGeneral MedicineT lymphocyteMycobacterium tuberculosismedicine.diseasebiology.organism_classificationIn vitroImmunologyFemalemedicine.symptomBacteriaLancet (London, England)
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Mean temperature and humidity variations, along with patient age, predict the number of visits for renal colic in a large urban Emergency Department:…

2012

Background: A marked geographic variability has been reported in stone disease, partially attributed to the Mean Annual Temperature (MAT), as well as to the seasonal fluctuations of climatic conditions. Accordingly, peaks in Emergency Department (ED) visits for renal colic are commonplace during the summer. Materials and methods: The aim of this study was to assess the influence of day-by-day climate changes on the number of visits as a result of renal colic in the ED (City of Parma, northern Italy, temperate continental climate). A total of 10,802 colic episodes were retrieved from the database during a period of 3286 days (January 2002 to December 2010). Results: The analysis of the data …

AdultMalePediatricsmedicine.medical_specialtyDatabases FactualEpidemiologyClimateRisk AssessmentSeverity of Illness IndexArticleOlder populationCohort StudiesYoung AdultAgeHospitals UrbanPatient agePredictive Value of TestsEpidemiologymedicineHumansRenal colicMean radiant temperatureRenal ColicStone diseaseAgedRetrospective StudiesAged 80 and overAnalysis of Variancebusiness.industrylcsh:Public aspects of medicineIncidenceTemperatureAge Factorslcsh:RA1-1270HumidityEmergency departmentMiddle AgedNorthern italyItalyLinear ModelsFemaleSeasonsmedicine.symptombusinessEmergency Service HospitalJournal of Epidemiology and Global Health
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