Search results for "comparative"

showing 10 items of 1371 documents

Exploring by whole exome sequencing patients with initial diagnosis of Rubinstein-Taybi syndrome: the interconnections of epigenetic machinery disord…

2019

Rubinstein–Taybi syndrome (RSTS) is an autosomal-dominant neurodevelopmental disease affecting 1:125,000 newborns characterized by intellectual disability, growth retardation, facial dysmorphisms and skeletal abnormalities. RSTS is caused by mutations in genes encoding for writers of the epigenetic machinery: CREBBP (~ 60%) or its homologous EP300 (~ 10%). No causative mutation is identified in up to 30% of patients. We performed whole-exome sequencing (WES) on eight RSTS-like individuals who had normal high-resolution array CGH testing and were CREBBP- and EP300-mutation -negative, to identify the molecular cause. In four cases, we identified putatively causal variants in three genes (ASXL…

MaleGenetic Association StudieCompound heterozygosityWhole Exome SequencingArticleEpigenesis Genetic03 medical and health scienceswhole exome sequencing Rubinstein–Taybi syndrome epigenetic mutationsExome SequencingGeneticsmedicineHumansEpigeneticsEP300ChildGenetics (clinical)Exome sequencingGenetic Association Studies030304 developmental biologyGeneticsRubinstein-Taybi Syndrome0303 health sciencesComparative Genomic HybridizationbiologyRubinstein–Taybi syndrome030305 genetics & heredityInfant NewbornFaciesInfantmedicine.diseaseFacieCREB-Binding ProteinHuman geneticsRSTSKMT2APhenotypeChild PreschoolMutationbiology.proteinNeurodegenerative disordersFemaleHaploinsufficiencyE1A-Associated p300 ProteinHumanHuman genetics
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A novel DFNB1 deletion allele supports the existence of a distant cis-regulatory region that controls GJB2 and GJB6 expression

2010

Contains fulltext : 87760_1.pdf (author's version ) (Open Access) Contains fulltext : 87760_2.pdf (Publisher’s version ) (Closed access) Eleven affected members of a large German-American family segregating recessively inherited, congenital, non-syndromic sensorineural hearing loss (SNHL) were found to be homozygous for the common 35delG mutation of GJB2, the gene encoding the gap junction protein Connexin 26. Surprisingly, four additional family members with bilateral profound SNHL carried only a single 35delG mutation. Previously, we demonstrated reduced expression of both GJB2 and GJB6 mRNA from the allele carried in trans with that bearing the 35delG mutation in these four persons. Usin…

MaleGenetics and epigenetic pathways of disease [NCMLS 6][SDV]Life Sciences [q-bio]PenetranceMESH: Base SequenceRegulatory Sequences Nucleic Acidsensorineural hearing lossConnexinsMESH: GenotypeMESH: Hearing Loss Sensorineural/diagnosisMESH: PenetranceGenotypeCopy-number variationGenetics (clinical)Sequence DeletionGeneticsComparative Genomic Hybridization0303 health sciencesMESH: Genetic TestingMESH: Gene Expression Regulation*030305 genetics & heredityPenetranceGJB2PedigreeConnexin 26MESH: Sequence Deletion*MESH: Hearing Loss Sensorineural/geneticsFemaleChromosome DeletionFunctional Neurogenomics [DCN 2]GJB6GenotypeMESH: PedigreeMESH: Chromosome DeletionHearing Loss SensorineuralMolecular Sequence Dataconnexin 26connexin 30DFNB1gene expression regulationGJB2GJB6sensorineural hearing losssequence deletionBiologyMESH: Connexin 30MESH: Connexins/genetics*MESH: Sequence Homology Nucleic AcidArticleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesMonoallelic MutationGJB6MESH: Connexin 26Sequence Homology Nucleic AcidConnexin 30otorhinolaryngologic diseasesGeneticsHumansGenetic TestingAlleleGeneMESH: Regulatory Sequences Nucleic Acid/genetics*AllelesDFNB1030304 developmental biologyFamily HealthMESH: HumansMESH: Molecular Sequence DataBase SequenceChromosomes Human Pair 13MESH: AllelesBreakpointMESH: MaleMESH: Comparative Genomic HybridizationGene Expression RegulationMESH: Family Healthbiology.proteinHuman medicineMESH: Chromosomes Human Pair 13/geneticsMESH: FemaleClinical Genetics
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Interstitial deletion of chromosome 2p15-16.1: Report of two patients and critical review of current genotype–phenotype correlation

2011

Abstract We report two individuals with developmental delay and dysmorphic features, in whom array-based comparative genomic hybridization (array CGH) led to the identification of a 2p15p16.1 de novo deletion. In the first patient (Patient 1) a familial deletion of 6q12, inherited from her father, was also detected. In the second patient (Patient 2) in addition to the 2p15p16.1 microdeletion a de novo deletion in Xq28 was detected. Both individuals shared dysmorphic features and developmental delay with the six reported patients with a 2p15p16.1 microdeletion described in medical literature. Conclusion: in the first patient a 642 kb 2p16.1 deletion (from 60.604 to 61.246 Mb), and a 930 kb 6…

MaleGenotypeDevelopmental delayDevelopmental DisabilitiesBioinformaticsContiguous gene syndromeGenotype phenotypeCorrelationGeneticsHumansChromosomal delectionMedicineAbnormalities MultipleClinical phenotypeGenetic Association StudiesIn Situ Hybridization FluorescenceSex Chromosome AberrationsGenetics (clinical)Sequence DeletionGeneticsChromosomes Human XComparative Genomic Hybridizationbusiness.industryInfantChromosomeSyndromeGeneral MedicineMicrodeletion syndromemedicine.diseaseXq28PhenotypeChild PreschoolChromosomes Human Pair 2FemaleChromosome DeletionbusinessComparative genomic hybridizationEuropean Journal of Medical Genetics
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Stability of pedalling mechanics during a prolonged cycling exercise performed at different cadences.

2005

The aim of this study was to analyse the effect of pedalling rate on the pattern of mechanical torque application and on neuromuscular fatigue during prolonged cycling exercise. Eleven well-trained individuals performed three 1-h pedalling sessions, at 50 rev.min-1, 110 rev.min-1 and a freely chosen cadence, at an intensity corresponding to 65% of their maximal aerobic power. The mechanical torque applied on the right pedal was recorded for 30 s every 5 min while pedalling. Contractile and neural properties of the quadriceps and hamstring muscles were analysed before and immediately after each of the three pedalling sessions. The post-exercise reduction in knee extensors maximal voluntary c…

MaleHamstring muscles[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]MESH: Muscle ContractionMuscle RelaxationMESH : Analysis of VarianceElectromyographyCohort Studies0302 clinical medicineVoluntary contractionMESH: Risk FactorsRisk FactorsOrthopedics and Sports MedicineMESH: Oxygen ConsumptionMESH : Oxygen ConsumptionMESH: Cohort StudiesKnee extensorsmedicine.diagnostic_test[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]BiomechanicsMESH: Comparative StudyMESH : AdultMESH : Risk FactorsMESH: Muscle FatigueMuscle FatigueMESH : ElectromyographyCyclingCadenceMESH: Physical EnduranceMESH : Physical EnduranceMuscle ContractionAdultmedicine.medical_specialtyMESH: ProbabilityMESH : ProbabilityMESH : MalePhysical ExertionMESH : ExertionMESH : Cohort StudiesPhysical Therapy Sports Therapy and RehabilitationSensitivity and SpecificityMESH : Muscle RelaxationMESH: BicyclingMESH: Electromyography03 medical and health sciencesPhysical medicine and rehabilitationOxygen ConsumptionMESH: Analysis of VariancemedicineMESH: Exertion[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HumansProbabilityAnalysis of VarianceMESH : SMESH: Humansbusiness.industryElectromyographyMESH : HumansMESH : Comparative StudyMESH: Adult030229 sport sciencesNegative workMESH : Muscle FatigueMESH: MaleBicyclingbody regionsMESH : Exercise TestTorqueMESH : BicyclingMESH: SMESH: Muscle RelaxationPhysical therapyExercise TestPhysical EnduranceMESH : Muscle ContractionbusinessMESH: Exercise Testhuman activities030217 neurology & neurosurgeryJournal of sports sciences
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A Comparative Study on Adolescents’ Health Literacy in Europe: Findings from the HBSC Study

2020

(1) Background: There is a need for studies on population-level health literacy (HL) to identify the current state of HL within and between countries. We report comparative findings from 10 European countries (Austria, Belgium (Fl), Czechia, England, Estonia, Finland, Germany, Macedonia, Poland, and Slovakia) on adolescents&rsquo

MaleHealth Toxicology and MutagenesiseducationPsychological interventionlcsh:MedicineHealth literacyterveysosaaminenArticlekoettu terveysself-rated health03 medical and health sciencesSex Factors0302 clinical medicinenuoretvertaileva tutkimusHumansMedicine030212 general & internal medicineChildcomparative studySelf-rated health030505 public healthbusiness.industrylcsh:RPublic Health Environmental and Occupational HealthEuropebody regionsterveyskäyttäytyminenadolescentFemaleHealth behavior0305 other medical sciencebusinesshealth literacyDemographyInternational Journal of Environmental Research and Public Health
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Novel LRPPRC compound heterozygous mutation in a child with early-onset Leigh syndrome French-Canadian type: Case report of an Italian patient

2020

Abstract Background Mitochondrial diseases, also known as oxidative phosphorylation (OXPHOS) disorders, with a prevalence rate of 1:5000, are the most frequent inherited metabolic diseases. Leigh Syndrome French Canadian type (LSFC), is caused by mutations in the nuclear gene (2p16) leucine-rich pentatricopeptide repeat-containing (LRPPRC). It is an autosomal recessive neurogenetic OXPHOS disorder, phenotypically distinct from other types of Leigh syndrome, with a carrier frequency up to 1:23 and an incidence of 1:2063 in the Saguenay-Lac-St Jean region of Quebec. Recently, LSFC has also been reported outside the French-Canadian population. Patient presentation We report a male Italian (Sic…

MaleHypotonia - developmental delayPediatricsmedicine.medical_specialtyPopulationEncephalopathyCytochrome-c Oxidase DeficiencyCase ReportHypotoniaCompound heterozygosityDiagnosis Differential03 medical and health sciences0302 clinical medicineWhole-genome-sequencingHypotonia; developmental delay; Mitochondrial disease; Whole-exome sequencing; CCT5030225 pediatricsmedicineMissense mutationHumansGlobal developmental delayeducationeducation.field_of_studyComparative Genomic Hybridizationbusiness.industrylcsh:RJ1-570Infant Newbornlcsh:Pediatricsmedicine.diseaseHypotoniaHypoplasiaMitochondrial diseaseNeoplasm Proteinsdevelopmental delayNeonatal hypotoniaPhenotypeItalyWhole-exome sequencingMutationLSFCmedicine.symptomLeigh DiseaseCCT5business030217 neurology & neurosurgeryInfant Premature
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Evolution of chromatin-remodeling complexes: comparative genomics reveals the ancient origin of "novel" compensasome genes.

2003

Dosage compensation in Drosophila is mediated by a complex, called compensasome, com- posed of at least five proteins and two noncoding RNAs. Genes encoding compensasome proteins have been collectively named male-specific lethals or msls. Recent work showed that three of the Drosophila msls (msl-3, mof, and mle) have an ancient origin. In this study, I describe likely orthologues of the two re- maining msls, msl-1 and msl-2, in several inverte- brates and vertebrates. The MSL-2 protein is the only one found in Drosophila and vertebrate genomes that contains both a RING finger and a peculiar type of CXC domain, related to the one present in Enhancer of Zeste proteins. MSL-1 also contains two…

MaleLeucine zipperAmino Acid MotifsMolecular Sequence DataBiologyGenomeChromatin remodelingEvolution MolecularDosage Compensation GeneticGeneticsRing fingermedicineAnimalsDrosophila ProteinsHumansAmino Acid SequenceEnhancerMolecular BiologyEcology Evolution Behavior and SystematicsCaenorhabditis elegansPhylogenyComparative genomicsGeneticsDosage compensationfungiNuclear ProteinsGenomicsbiology.organism_classificationChromatin Assembly and DisassemblyProtein Structure TertiaryDNA-Binding Proteinsmedicine.anatomical_structureVertebratesDrosophilaSequence AlignmentTranscription FactorsJournal of molecular evolution
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Breed, sex, and litter effects in 2-month old puppies’ behaviour in a standardised open-field test

2017

AbstractA considerable number of studies have reported differences among dog breeds with respect to their genetic profile, cognitive abilities or personality traits. Each dog breed is normally treated as a homogeneous group, however, researchers have recently questioned whether the behavioural profile of modern breeds still reflects their historical function or if the intense divergent selective pressures and geographical barriers have created a more fragmented picture. The majority of studies attempting to assess and compare modern breeds’ personality focused on the evaluation of adult dogs where the potential effects of environmental/human factors on the dogs’ behaviour are hard to discer…

MaleLitter (animal)Veterinary medicineLitter SizeSciencemedia_common.quotation_subjectBreedingBiologyArticleOpen fieldGenetic profileQuantitative Trait HeritableSex FactorsSpecies SpecificityJournal ArticleAnimalsCluster AnalysisPersonality0501 psychology and cognitive sciences050102 behavioral science & comparative psychologyBig Five personality traitsmedia_commonMultidisciplinaryBehavior AnimalQ05 social sciencesR0402 animal and dairy science04 agricultural and veterinary sciences040201 dairy & animal scienceBreedTest (assessment)Homogeneous groupMedicineFemaleDemographyScientific Reports
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MALT1 is deregulated by both chromosomal translocation and amplification in B-cell non-Hodgkin lymphoma

2003

The MALT1 gene was identified through its involvement in t(11;18)(q21;q21), seen in 30% of cases of mucosa-associated lymphoid tissue (MALT) lymphoma. Here, we show that deregulated MALT1 expression may occur in B-cell non-Hodgkin lymphoma (B-NHL) of various histologic subtypes either through translocation to the immunoglobulin heavy chain (IGH) locus or by genomic amplification. First, 2 cases, one case of MALT lymphoma and another of aggressive marginal zone lymphoma (MZL) with t(14;18)(q32;q21), cytogenetically identical to the translocation involving BCL2, were shown by fluorescence in situ hybridization (FISH) to involve MALT1, which lies about 5 Mb centromeric of BCL2. Molecular cloni…

MaleLymphoma B-CellImmunologyBiologyBiochemistryTranslocation Geneticimmune system diseaseshemic and lymphatic diseasesGene duplicationmedicineHumansRNA NeoplasmAgedChromosomes Human Pair 14medicine.diagnostic_testGene Expression ProfilingGene AmplificationMALT lymphomaLymphoma B-Cell Marginal ZoneCell BiologyHematologyMiddle Agedmedicine.diseaseMolecular biologyGenes bcl-2Neoplasm ProteinsGene Expression Regulation NeoplasticGene expression profilingMALT1Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 ProteinCaspasesB-Cell Non-Hodgkin LymphomaImmunoglobulin heavy chainFemaleChromosomes Human Pair 18Comparative genomic hybridizationFluorescence in situ hybridizationBlood
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Proportion of odorants impacts the configural versus elemental perception of a binary blending mixture in newborn rabbits.

2011

WOS: 000295167200002; International audience; Processing of odor mixtures by neonates is weakly understood. Previous studies showed that a binary mixture of ethyl isobutyrate/ethyl maltol (odorants A/B) blends in newborn rabbits at the 30/70 ratio: Pups would perceive a configural odor in addition to the components' odors. Here, we investigated whether the emergence of this additional odor in AB is determined by specific ratio(s) of A and B. To that goal, we tested whether pups discriminated between AB mixtures with lower (A(-)B, 8/92 ratio) or higher (A(+)B, 68/32) proportion of A. In Experiment 1, pups conditioned to A (or B) responded to A(-)B and A(+)B but not to AB. In Experiment 2, pu…

MaleMESH: Olfactory PerceptionPhysiology[SDV]Life Sciences [q-bio]pupMESH: RabbitsMESH: Animals NewbornDevelopmental psychologystimuliBehavioral Neurosciencechemistry.chemical_compound0302 clinical medicineDiscrimination Psychologicalemissionrabbit (Oryctolagus cuniculus)MESH : FemaleMESH: AnimalsMESH: Discrimination (Psychology)configural perceptionodorant proportionChemistryMESH : Animals Newborn05 social sciencesEthyl maltolmammary pheromoneMESH : OdorsSensory SystemsqualityFemaleRabbitsolfactionmedicine.medical_specialtyMESH : MaleOlfactioncomponents03 medical and health sciencesPhysiology (medical)Internal medicineMESH : Olfactory PerceptionmedicineAnimalsMESH : Rabbits0501 psychology and cognitive sciences050102 behavioral science & comparative psychologyodor mixtureMESH: Odors[ SDV ] Life Sciences [q-bio]Olfactory PerceptionMESH : Discrimination (Psychology)neonatesMESH: MaleEndocrinologyOdorAnimals NewbornOdorantsConditioningMESH : AnimalsMESH: Female030217 neurology & neurosurgery
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