Search results for "complex"

showing 10 items of 5889 documents

The function of the soluble IL-6 receptor in vivo.

1996

Interleukin-6 (IL-6) is considered an important mediator of acute inflammatory responses. Moreover, IL-6 functions as a differentiation and growth factor of hematopoietic precursor cells, B-cells, T-cells, keratinocytes, neuronal cells, osteoclasts and endothelial cells. IL-6 exhibits its action via a receptor complex consisting of a specific IL-6 receptor (IL-6R) and a signal-transducing subunit (gp130). Soluble forms of both receptor components are generated by shedding and are found in patients with various diseases such as AIDS, rheumatoid arthritis and others. The function of the soluble IL-6R in vivo is unknown. To discriminate between the biologic function of hIL-6 alone and that of …

Genetically modified mousemedicine.medical_specialtyReceptor complexTransgenemedicine.medical_treatmentImmunologyMice TransgenicBiologyMiceAntigens CDInternal medicinemedicineHypersensitivityImmunology and AllergyAnimalsHumansReceptorInterleukin-6Growth factorReceptors InterleukinGlycoprotein 130Hematopoietic Stem CellsReceptors Interleukin-6Cell biologyHaematopoiesisEndocrinologySolubilityInterleukin-6 receptorCarrier ProteinsAcute-Phase ProteinsImmunology letters
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Recent Advances in Complex Networks Theories with Applications

2014

Genetics and Molecular Biology (all)Dynamic network analysisArticle SubjectComputer sciencelcsh:MedicineNetwork sciencelcsh:TechnologyBiochemistryGeneral Biochemistry Genetics and Molecular BiologyTheoreticalModelsHuman dynamicsHumanslcsh:ScienceGeneral Environmental ScienceCognitive science2300lcsh:TInterdependent networksbusiness.industryMedicine (all)lcsh:RGeneral MedicineModels TheoreticalNeural Networks (Computer)Complex networkNetwork dynamicsEditorialEvolving networksHumans; Models Theoretical; Neural Networks (Computer); Medicine (all); Biochemistry Genetics and Molecular Biology (all); 2300lcsh:QNeural Networks ComputerArtificial intelligenceHierarchical network modelbusinessThe Scientific World Journal
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Usher syndrome: molecular links of pathogenesis, proteins and pathways.

2006

Contains fulltext : 50437.pdf (Publisher’s version ) (Closed access) Usher syndrome is the most common form of deaf-blindness. The syndrome is both clinically and genetically heterogeneous, and to date, eight causative genes have been identified. The proteins encoded by these genes are part of a dynamic protein complex that is present in hair cells of the inner ear and in photoreceptor cells of the retina. The localization of the Usher proteins and the phenotype in animal models indicate that the Usher protein complex is essential in the morphogenesis of the stereocilia bundle in hair cells and in the calycal processes of photoreceptor cells. In addition, the Usher proteins are important in…

Genetics and epigenetic pathways of disease [NCMLS 6]Usher syndromeCell Cycle ProteinsNerve Tissue ProteinsBiologyRetinaAdherens junctionMiceHair Cells AuditoryCell polarityGeneticsmedicineotorhinolaryngologic diseasesNeurosensory disorders [UMCN 3.3]AnimalsHumansProtein IsoformsCell Cycle ProteinMolecular BiologyGenetics (clinical)Renal disorder [IGMD 9]Adaptor Proteins Signal TransducingStereociliumMembrane ProteinsSignal transducing adaptor proteinGeneral MedicineActin cytoskeletonmedicine.diseaseeye diseasesCell biologyCytoskeletal ProteinsGenetic defects of metabolism [UMCN 5.1]Ear InnerMultiprotein ComplexesCateninSynapsessense organsUsher SyndromesPhotoreceptor Cells Vertebrate
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2015

Joubert syndrome (JBTS) and related disorders are defined by cerebellar malformation (molar tooth sign), together with neurological symptoms of variable expressivity. The ciliary basis of Joubert syndrome related disorders frequently extends the phenotype to tissues such as the eye, kidney, skeleton and craniofacial structures. Using autozygome and exome analyses, we identified a null mutation in KIAA0556 in a multiplex consanguineous family with hallmark features of mild Joubert syndrome. Patient-derived fibroblasts displayed reduced ciliogenesis potential and abnormally elongated cilia. Investigation of disease pathophysiology revealed that Kiaa0556 -/- null mice possess a Joubert syndrom…

Genetics0303 health sciencesEnzyme complexCiliumCiliary basal bodyBiologymedicine.diseaseJoubert syndromeCell biology03 medical and health sciences0302 clinical medicineMicrotubuleCiliogenesismedicineBasal bodyCiliary base030217 neurology & neurosurgery030304 developmental biologyGenome Biology
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Gene expression models based on a reference laboratory strain are bad predictors of Mycobacterium tuberculosis complex transcriptional diversity

2016

ABSTRACTSpecies of the Mycobacterium tuberculosis complex (MTBC) kill more people every year than any other infectious disease. As a consequence of its global distribution and parallel evolution with the human host the bacteria is not genetically homogeneous. The observed genetic heterogeneity has relevance at different phenotypic levels, from gene expression to epidemiological dynamics. However current systems biology datasets have focused in the laboratory reference strain H37Rv. By using large expression datasets testing the role of almost two hundred transcription factors, we have constructed computational models to grab the expression dynamics of Mycobacterium tuberculosis H37Rv genes.…

Genetics0303 health sciencesGenetic diversityGenetic heterogeneitySystems biologyBiologybiology.organism_classificationPhenotype3. Good health03 medical and health sciences0302 clinical medicineMycobacterium tuberculosis complexInfectious disease (medical specialty)Gene expressionGene030217 neurology & neurosurgery030304 developmental biology
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Mycobacterium tuberculosiscomplex lineage 5 exhibits high levels of within-lineage genomic diversity and differing gene content compared to the type …

2020

AbstractPathogens of theMycobacterium tuberculosiscomplex (MTBC) are considered monomorphic, with little gene content variation between strains. Nevertheless, several genotypic and phenotypic factors separate the different MTBC lineages (L), especially L5 and L6 (traditionally termedMycobacterium africanum), from each other. However, genome variability and gene content especially of L5 and L6 strains have not been fully explored and may be potentially important for pathobiology and current approaches for genomic analysis of MTBC isolates, including transmission studies.We compared the genomes of 358 L5 clinical isolates (including 3 completed genomes and 355 Illumina WGS (whole genome seque…

Genetics0303 health sciencesLineage (genetic)030306 microbiologySequence assemblySingle-nucleotide polymorphismBiologybiology.organism_classificationGenome3. Good health03 medical and health sciencesMycobacterium tuberculosis complexGeneMycobacterium africanum030304 developmental biologyReference genome
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Immunogenetics of longevity. Is major histocompatibility complex polymorphism relevant to the control of human longevity? A review of literature data.

2001

Literature data suggest that human longevity may be directly correlated with optimal functioning of the immune system. Therefore, it is likely that one of the genetic determinants of longevity resides in those polymorphisms for the immune system genes that regulate immune responses. Accordingly, studies performed on mice have suggested that the Major Histocompatibility Complex (MHC), known to control a variety of immune functions, is associated with the life span of the strains. In the last 25 years, a fair number of cross-sectional studies that searched for the role of HLA (the human MHC) genes on human longevity by comparing HLA antigen frequencies between groups of young and elderly pers…

GeneticsAgingPolymorphism Geneticmedia_common.quotation_subjectHaplotypeLongevityLongevityHuman leukocyte antigenImmunogeneticsBiologyMajor histocompatibility complexHistocompatibilityMajor Histocompatibility ComplexMiceImmune systemAntigenHLA AntigensImmunologybiology.proteinImmunogeneticsAnimalsHumansGenetic Predisposition to DiseaseDevelopmental Biologymedia_commonMechanisms of ageing and development
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A Gene-Specific Requirement for FACT during Transcription Is Related to the Chromatin Organization of the Transcribed Region

2006

The FACT complex stimulates transcription elongation on nucleosomal templates. In vivo experiments also involve FACT in the reassembly of nucleosomes traversed by RNA polymerase II. Since several features of chromatin organization vary throughout the genome, we wondered whether FACT is equally required for all genes. We show in this study that the in vivo depletion of Spt16, one of the subunits of Saccharomyces cerevisiae FACT, strongly affects transcription of three genes, GAL1, PHO5, and Kluyveromyces lactis LAC4, which exhibit positioned nucleosomes at their transcribed regions. In contrast, showing a random nucleosome structure, YAT1 and Escherichia coli lacZ are only mildly influenced …

GeneticsChromatin ImmunoprecipitationSaccharomyces cerevisiae ProteinsTranscription GeneticbiologyHigh Mobility Group ProteinsRNA polymerase IIPromoterArticlesSaccharomyces cerevisiaeCell BiologyFACT complexChromatinChromatin remodelingChromatinDNA-Binding ProteinsHistone methylationProtein FACTEscherichia colibiology.proteinTranscriptional Elongation FactorsTranscription factor II DMolecular BiologyRNA polymerase II holoenzymePlasmidsMolecular and Cellular Biology
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Lack of linkage between gene(s) controlling the synthesis of the seventh component of complement and the HLA region on chromosome No. 6 in man.

1976

The family of an individual was studied who lacks the seventh component of complement in his serum (C7 homozygous deficiency). Both parents are C7 heterozygousdeficient. In this investigation, the following parameters were determined: complement components in functional and immunochemical tests; HLA-A,B antigens, HLA-D (MLC) determinants; the Bf system; glyoxalase I and B cell antigens. No evidence for linkage between the immunogenetic linkage group on chromosome 6 and gene(s) controlling the synthesis of the seventh component of complement was obtained. This is in accordance with the assumption that only genes controlling components of the initiating rather than the membrane attack unit of…

GeneticsChromosomes Human 6-12 and XMaleGenetic LinkageChromosomeHuman leukocyte antigenComplement System ProteinsBiologyComplement factor BHuman geneticsComplement C7Complement (complexity)medicine.anatomical_structureAntigenHLA AntigensHistocompatibility AntigensGeneticsmedicineHumansLymphocyte Culture Test MixedChildGeneGenetics (clinical)B cellHuman genetics
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Development of cytoplasmically incompatible and integrated (translocated incompatible) strains ofCulex pipiens fatigans for use in genetic control

1976

Crosses were made between strains of the Culex pipiens complex to select and develop strains with suitable cytoplasmic incompatibility properties for use in field experiments in genetic control of Culex pipiens fatigans near Delhi, India.

GeneticsCulex pipiens fatigansvirusesparasitic diseasesfungiBotanyGeneticsvirus diseasesCulex pipiens complexBiologygeographic locationsCytoplasmic incompatibilityJournal of Genetics
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