Search results for "contractility"

showing 10 items of 87 documents

Relaxation induced by N-terminal fragments of chromogranin A in mouse gastric preparations.

2007

Abstract A definitive role for chromogranin A (CGA)-derived fragments in the control of the gastrointestinal smooth muscle contractility has not been yet established. The purpose of the present study was to evaluate, in vitro , the effects of the recombinant vasostatin 1–78 (VS-1), CGA 7–57 and CGA 47–66 on the mouse gastric mechanical activity, recording the changes of intraluminal pressure. VS-1, CGA 7–57 and CGA 47–66 produced concentration-dependent relaxations. Mouse anti-vasostatin-1 monoclonal antibody 5A8, recognising the region 53–57, abolished the relaxation induced by VS-1, indicating the specificity of the effect. The relaxation was significantly reduced by tetrodotoxin (TTX), b…

endocrine systemmedicine.medical_specialtyPhysiologyMuscle RelaxationClinical BiochemistryBiologyIn Vitro TechniquesApaminInhibitory postsynaptic potentialBiochemistrySettore BIO/09 - FisiologiaNitric oxideContractilityGastric relaxationCellular and Molecular Neurosciencechemistry.chemical_compoundMiceEndocrinologyInternal medicinemedicineAnimalsGastrointestinal tractCGA-derived peptideDose-Response Relationship DrugStomachChromogranin ANitric oxideMuscle SmoothMolecular biologyIn vitroPeptide FragmentsRecombinant ProteinsMice Inbred C57BLEndocrinologychemistryTetrodotoxinbiology.proteinVasostatinChromogranin ACalreticulinRegulatory peptides
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Assessment of the cardiostimulant action of propionyl-L-carnitine on chronically volume-overloaded rat hearts.

1993

Chronic volume overload was induced in young rats of Wistar strain by surgical opening of the aorto-caval fistula. Three months later, during in vitro perfusion with exogenous palmitate, left ventricular function and energy turnover (QO2) of hypertrophied hearts were severely depressed. This seemed to be related to impaired long-chain fatty acid utilization, as reflected by decreased 14CO2 production from U-14C-palmitate and decreased tissue levels of L-carnitine. Another group of rats exposed to chronic volume overload was pretreated for 2 weeks before sacrifice with propionyl-L-carnitine (250 mg/kg/day), and the hearts were perfused with 1.2 mM palmitate and 10 mM propionyl-L-carnitine. I…

medicine.medical_specialtyCardiotonic AgentsVolume overloadPalmitatesCardiomegalyMitochondria HeartVentricular Function LeftMuscle hypertrophyContractilityOxygen ConsumptionInternal medicineCarnitinemedicineAnimalsPharmacology (medical)Rats WistarPharmacologychemistry.chemical_classificationVentricular functionbusiness.industryMyocardiumFatty acidGeneral MedicinePropionyl l carnitineCarbon DioxideMyocardial ContractionRatsEndocrinologychemistryVolume (thermodynamics)Cardiac hypertrophyCardiology and Cardiovascular MedicinebusinessCardiovascular drugs and therapy
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D1 receptors play a major role in the dopamine modulation of mouse ileum contractility

2010

Since the role of dopamine in the bowel motility is far from being clear, our aim was to analyse pharmacologically the effects of dopamine on mouse ileum contractility. Contractile activity of mouse ileum was examined in vitro as changes in isometric tension. Dopamine caused a concentration-dependent reduction of the spontaneous contraction amplitude of ileal muscle up to their complete disappearance. SCH-23390, D1 receptor antagonist, which per se increased basal tone and amplitude of spontaneous contractions, antagonized the responses to dopamine, whilst sulpiride or domperidone, D2 receptor antagonists, were without effects. The application of both D1 and D2 antagonists had additive effe…

medicine.medical_specialtyDopamineMouse ileumD1 receptorIn Vitro TechniquesSettore BIO/09 - FisiologiaEnteric Nervous SystemPotassium channelsContractilityMicechemistry.chemical_compoundDopamine receptor D1IleumDopamineInternal medicineDopamine receptor D2medicineAnimalsPharmacologySCH-23390Dose-Response Relationship DrugReceptors Dopamine D1BenzazepinesAdenosine receptorContractile activityD2 receptorDopamine D2 Receptor AntagonistsEndocrinologychemistryDopamine receptorDopamine AntagonistsEndogenous agonistAdenylyl CyclasesMuscle Contractionmedicine.drugPharmacological Research
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Normalwerte für die Dobutamin-Streßechokardiographie*

2008

UNLABELLED Dobutamine stress echocardiography having established itself as a sensitive method for diagnosing coronary heart disease, even in the absence of normative values, the physiological haemodynamics as well as the physiological values for global and regional left ventricular myocardial function were measured. TEST PERSONS AND METHODS 14 healthy subjects (ten men, four women; median age 25 [range 21-32] years) underwent dobutamine stress echocardiography according to an internationally practised dosage steps protocol (5-40 micrograms/kg/min with additional 0.5 mg atropine at 40 micrograms). RESULTS Maximal infusion rate achieved a serum dobutamine level of 1.67 micrograms/ml with mini…

medicine.medical_specialtyEjection fractionbusiness.industryDiastoleHemodynamicsGeneral MedicineContractilityAtropineBlood pressureInternal medicineHeart ratemedicineCardiologyDobutaminebusinessmedicine.drugDMW - Deutsche Medizinische Wochenschrift
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Effect of the Alterations in Contractility and Morphology Produced by Atrial Fibrillation on the Thrombosis Potential of the Left Atrial Appendage

2021

Atrial fibrillation (AF) is a common arrhythmia mainly affecting the elderly population, which can lead to serious complications such as stroke, ischaemic attack and vascular dementia. These problems are caused by thrombi which mostly originate in the left atrial appendage (LAA), a small muscular sac protruding from left atrium. The abnormal heart rhythm associated with AF results in alterations in the heart muscle contractions and in some reshaping of the cardiac chambers. This study aims to verify if and how these physiological changes can establish hemodynamic conditions in the LAA promoting thrombus formation, by means of computational fluid dynamic (CFD) analyses. In particular, sinus …

medicine.medical_specialtyHistologythromboembolic eventleft atrial appendagelcsh:Biotechnology0206 medical engineeringBiomedical EngineeringThrombogenicityHemodynamicsBioengineering02 engineering and technology030204 cardiovascular system & hematologyContractilitySettore ING-IND/14 - Progettazione Meccanica E Costruzione Di Macchine03 medical and health sciencescomputational fluid dynamic0302 clinical medicinecardiac wall motionInternal medicinelcsh:TP248.13-248.65Medicineatrial fibrillationThrombusStrokeOriginal ResearchFibrillationbusiness.industrySettore ING-IND/34 - Bioingegneria IndustrialeBioengineering and BiotechnologyAtrial fibrillationmedicine.disease020601 biomedical engineeringThrombosisCardiologymedicine.symptombusinessBiotechnologyFrontiers in Bioengineering and Biotechnology
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Damage-associated molecular pattern activated Toll-like receptor 4 signalling modulates blood pressure in L-NAME-induced hypertension

2013

Aims Recent publications have shed new light on the role of the adaptive and innate immune system in the pathogenesis of hypertension. However, there are limited data whether receptors of the innate immune system may influence blood pressure. Toll-like receptor 4 (TLR4), a pattern recognition receptor, is a key component of the innate immune system, which is activated by exogenous and endogenous ligands. Hypertension is associated with end-organ damage and thus might lead to the release of damage-associated molecular patterns (DAMPs), which are endogenous activators of TLR4 receptors. The present study aimed to elucidate whether TLR4 signalling is able to modulate vascular contractility in …

medicine.medical_specialtyPhysiologyMedizinInflammationBiologyContractilityMicePhysiology (medical)Internal medicinemedicineAnimalsReceptorCyclic GMPInflammationToll-like receptorInnate immune systemPattern recognition receptorDamage-associated molecular patternCell biologyMice Inbred C57BLToll-Like Receptor 4EndocrinologyNG-Nitroarginine Methyl EsterHypertensionTLR4Blood Vesselsmedicine.symptomCardiology and Cardiovascular MedicineSignal Transduction
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Modulatory role of a constitutively active population of α1D-adrenoceptors in conductance arteries

2002

A constitutively active population of α1D-adrenoceptors in iliac and proximal, distal, and small mesenteric rat arteries was studied. The increase in resting tone (IRT) that evidences it was observed only in iliac and proximal mesenteric and was inhibited by prazosin (pIC50 = 9.57), 5-methylurapidil (pIC50 = 7.61), and BMY 7378 (pIC50 = 8.77). Chloroethylchlonidine (100 μmol/l) did not affect IRT, but when added before the other antagonists it blocked their effect. The potency shown by BMY 7378 confirms the α1D-subtype as responsible for IRT. BMY 7378 displayed greater inhibition of adrenergic responses in iliac (pIC50 = 7.57 ± 0.11) and proximal mesenteric arteries (pIC50 = 8.05 ± 0.2) th…

medicine.medical_specialtyPhysiologyPopulationConstitutively activeIliac ArteryClonidinePiperazinesContractilityNorepinephrineNorepinephrineReceptors Adrenergic alpha-1Physiology (medical)Internal medicinemedicineAnimalsRats WistareducationAdrenergic alpha-AntagonistsAortaeducation.field_of_studyDose-Response Relationship DrugChemistryConductanceArteriesPrazosinMesenteric ArteriesRatsmedicine.anatomical_structureEndocrinologyCirculatory systemCatecholamineCalciumFemaleVascular ResistanceCardiology and Cardiovascular MedicineAdrenergic alpha-AgonistsBlood vesselmedicine.drugAmerican Journal of Physiology-Heart and Circulatory Physiology
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Septic cardiomyopathy: evidence for a reduced force-generating capacity of human atrial myocardium in acute infective endocarditis

2017

AbstractBackground:This study analyzes the myocardial force-generating capacity in infective endocarditis (IE) using an experimental model of isolated human atrial myocardium. In vivo, it is difficult to decide whether or not alterations in myocardial contractile behavior are due to secondary effects associated with infection such as an altered heart rate, alterations of preload and afterload resulting from valvular defects, and altered humoral processes. Our in vitro model using isolated human myocardium, in contrast, guarantees exactly defined experimental conditions with respect to preload, afterload, and contraction frequency, thus not only preventing confounding by in vivo determinants…

medicine.medical_specialtyRD1-811medicine.medical_treatmentHemodynamicsmyocardial contractilityIsometric exercise030204 cardiovascular system & hematologyContractility03 medical and health sciences0302 clinical medicineAfterloadInternal medicinemedicineEndocarditiscardiovascular diseasesacute infective endocarditisbusiness.industryMitral valve replacement030208 emergency & critical care medicineOriginal Articlesmedicine.diseasePreloadInfective endocarditisseptic cardiomyopathyCardiologycardiovascular systemSurgerybusinesshuman atrial myocardiumInnovative Surgical Science
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Kinin receptors in human vascular tissue: their role in atheromatous disease

1997

Using samples of many human blood vessels, obtained at autopsy and specific antibodies directed to peptide sequences of the kinin B1 and B2 receptors, we demonstrate the localisation of these receptors within the human vascular system using standard immunolabelling techniques. In large elastic arteries and veins, kinin receptors are present only in the endothelial cells whereas in all muscular arteries and arterioles, these receptors are present in both the endothelial and smooth muscle cells. The identification of kinin receptors in human blood vessels confirms that kinins may modulate both vascular permeability and contractility. The incidental finding at histology, of patchy atheromatous…

medicine.medical_specialtyReceptor Bradykinin B2EndotheliumArteriosclerosisMolecular Sequence DataImmunocytochemistryEnzyme-Linked Immunosorbent AssayVascular permeabilityBiologyReceptor Bradykinin B1Muscle Smooth VascularVeinsCapillary PermeabilityContractilityAntibody SpecificityInternal medicinemedicineHumansVasoconstrictor AgentsAmino Acid SequenceReceptorVascular tissuePharmacologyStaining and LabelingReceptors BradykininArteriesKininImmunohistochemistryMolecular WeightArteriolesmedicine.anatomical_structureEndocrinologycardiovascular systemImmunohistochemistryKallikreinsAutopsyEndothelium VascularTissue KallikreinsMuscle ContractionImmunopharmacology
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Alterations of Neuromuscular Function After Prolonged Running, Cycling and Skiing Exercises

2004

It is well known that impairment of performance resulting from muscle fatigue differs according to the types of contraction involved, the muscular groups tested and the exercise duration/intensity. Depending on these variables, strength loss with fatigue can originate from several sites from the motor cortex through to contractile elements. This has been termed 'task dependency of muscle fatigue'. Only recently have studies focused on the origin of muscle fatigue after prolonged exercise lasting 30 minutes to several hours. Central fatigue has been shown to contribute to muscle fatigue during long-distance running by using different methods such as the twitch interpolation technique, the ra…

medicine.medical_specialtyTime FactorsContraction (grammar)Neuromuscular transmissionPhysical Therapy Sports Therapy and RehabilitationPhysical exerciseElectromyographyNervous SystemRunningContractilityPhysical medicine and rehabilitationSkiingmedicineHumansOrthopedics and Sports MedicineMuscle fatiguemedicine.diagnostic_testElectromyographybusiness.industryBicyclingBiomechanical PhenomenaPeripheralmedicine.anatomical_structureMuscle FatiguePhysical therapybusinessMotor cortexSports Medicine
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