Search results for "crisp"

showing 10 items of 104 documents

Modificazioni germinali del patrimonio genetico e biodiritto. I paradossi della de-differenziazione tra bioetica e biodiritto

2021

Il presente lavoro, incominciato nel novembre del 2017, è partito con l'ambizione di ricostruire la risposta che il sistema giuridico fornisce innanzi alle nuove tecniche di ingegneria genetica che, a fronte della loro applicabilità sugli esseri umani, hanno prodotto, negli ultimi anni, il sorgere di nuovi stakeholders e, ancor prima, di nuovi interessi meritevoli di tutela. Se fino a qualche anno fa pareva impensabile modificare il genoma umano e, men che meno, farlo in maniera precisa, efficiente ed economica, oggi grazie al sistema di modificazione genetica CRISPR/Cas9 è possibile, intervenendo sulla linea germinale degli embrioni umani, prevenire la contrazione di odiose malattie geneti…

Germ-line gene editingSettore IUS/20 - Filosofia Del Dirittogene editinghuman right:CIENCIAS JURÍDICAS Y DERECHO [UNESCO]philosophy of lawUNESCO::CIENCIAS JURÍDICAS Y DERECHObiolawsociology of lawdignitybioethicGeneticseugeniccrisprbioethicsGenome editing
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Translational adaptation to heat stress is mediated by RNA 5‐methylcytosine in Caenorhabditis elegans

2021

Abstract Methylation of carbon‐5 of cytosines (m5C) is a post‐transcriptional nucleotide modification of RNA found in all kingdoms of life. While individual m5C‐methyltransferases have been studied, the impact of the global cytosine‐5 methylome on development, homeostasis and stress remains unknown. Here, using Caenorhabditis elegans, we generated the first organism devoid of m5C in RNA, demonstrating that this modification is non‐essential. Using this genetic tool, we determine the localisation and enzymatic specificity of m5C sites in the RNome in vivo. We find that NSUN‐4 acts as a dual rRNA and tRNA methyltransferase in C. elegans mitochondria. In agreement with leucine and proline bein…

Hot TemperatureProlineRibosomeGeneral Biochemistry Genetics and Molecular BiologyArticle03 medical and health sciencesNSUNCytosine0302 clinical medicineRNA modificationsLeucinem5CAnimalsRNA Processing Post-TranscriptionalCaenorhabditis elegansMolecular BiologytRNACaenorhabditis elegansprotein translation030304 developmental biologyGene Editing0303 health sciencesGeneral Immunology and MicrobiologybiologyGeneral NeuroscienceTRNA MethyltransferaseRNATranslation (biology)MethylationArticlesMethyltransferasesRibosomal RNAbiology.organism_classificationRNA BiologyAdaptation Physiological5‐methylcytosineCell biologyMitochondriatranslation efficiencyProtein BiosynthesisTransfer RNA5-MethylcytosineRNACRISPR-Cas SystemsRibosomes030217 neurology & neurosurgeryHeat-Shock ResponseThe EMBO Journal
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Biochemical and Immunological implications of Lutein and Zeaxanthin

2021

Throughout history, nature has been acknowledged for being a primordial source of various bioactive molecules in which human macular carotenoids are gaining significant attention. Among 750 natural carotenoids, lutein, zeaxanthin and their oxidative metabolites are selectively accumulated in the macular region of living beings. Due to their vast applications in food, feed, pharmaceutical and nutraceuticals industries, the global market of lutein and zeaxanthin is continuously expanding but chemical synthesis, extraction and purification of these compounds from their natural repertoire e.g., plants, is somewhat costly and technically challenging. In this regard microbial as well as microalga…

LuteinOxidative degradationQH301-705.5Drug CompoundingBioactive moleculesReviewBiologyCatalysisInorganic ChemistryBiological Factorschemistry.chemical_compoundNutraceuticalDrug StabilityZeaxanthinsHumansMacula LuteaFood scienceBiology (General)Physical and Theoretical Chemistrymacular carotenoidsCRISPR/Cas9QD1-999Molecular BiologyCarotenoidSpectroscopyGene Editingchemistry.chemical_classificationgenetic engineeringEsterificationLuteinOrganic Chemistryfood and beveragesGeneral MedicineResearch needseye diseasesComputer Science ApplicationsZeaxanthinChemistryantioxidantschemistryXanthophyllbioavailabilitylutein binding proteinInternational Journal of Molecular Sciences
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Targeting PCSK9 for therapeutic gains: Have we addressed all the concerns?

2016

Proprotein Convertase Subtilisin/Kexin Type 9 (PCSK9) regulates the expression of low-density lipoprotein (LDL)-receptors, through reducing their recycling by binding to the receptor along with LDL and targeting it for lysosomal destruction. PCSK9 also enhances the degradation of very-low-density-lipoprotein receptor (VLDLR) and lipoprotein receptor-related protein 1 (LRP-1) in a LDL-receptor independent manner. This role in lipid homeostasis presents PCSK9 as an attractive target for the therapeutic management of familial hypercholesterolemia as well as other refractory dyslipidaemias. However, PCSK9 mediates multifarious functions independent of its role in lipid homeostasis, which can be…

Male0301 basic medicineCell signalingHIPERCOLESTEROLEMIALow-density lipoprotein receptor gene familyHypercholesterolemiaMice TransgenicFamilial hypercholesterolemiaBiologyAntiviral AgentsPermeabilityMice03 medical and health sciencesAlzheimer DiseasemedicineAnimalsHomeostasisHumansGlucose homeostasisRNA Small InterferingEpithelial Sodium ChannelsGlycoproteinsNeuronsPCSK9PCSK9 InhibitorsAntibodies MonoclonalCell DifferentiationOligonucleotides Antisensemedicine.diseaseProprotein convertaseLipidsCircadian RhythmLiver RegenerationCell biology030104 developmental biologyReceptors LDLBiochemistryLDL receptorKexinFemalelipids (amino acids peptides and proteins)CRISPR-Cas SystemsProprotein Convertase 9Cardiology and Cardiovascular MedicineAtherosclerosis
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Infant developmental profile of Crisponi syndrome due to compound heterozygosity for CRLF1 deletion.

2020

Crisponi syndrome/CISS1, is an autosomal recessive ciliary neurotrophic factor receptor (CNTFR)-related genodermatosis caused in 95% of cases by mutations in CRLF1 on chromosome 19p13. The CNTFR pathway is important for CNS development. Crisponi syndrome/ CISS1 can be suspected in the presence of the following clinical triad: camptodactyly with fisted hands, intermittent hyperthermia and muscular contractions with feeding difficulties.

MalePathologymedicine.medical_specialtyCrisponi syndromeCompound heterozygosityPathology and Forensic MedicineCamptodactylyDeath SuddenPeriodic feverMedicineHumansHyperhidrosisReceptors CytokineGenetics (clinical)Sequence DeletionDevelopmental profiledevelopmental delay thin corpus callosum clinical profilebusiness.industryInfant NewbornFaciesInfantCold-induced sweating syndromeGeneral MedicineThin corpus callosumPediatrics Perinatology and Child HealthTrismusAnatomymedicine.symptomDevelopmental DelayCold-induced sweating syndrome CamptodactylyThin corpus callosum Periodic feverbusinessHand Deformities CongenitalClinical dysmorphology
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Lysosomal Signaling Licenses Embryonic Stem Cell Differentiation via Inactivation of Tfe3

2019

International audience; Self-renewal and differentiation of pluripotent murine embryonic stem cells (ESCs) is regulated by extrinsic signaling pathways. It is less clear whether cellular metabolism instructs developmental progression. In an unbiased genome-wide CRISPR/Cas9 screen, we identified components of a conserved amino-acid-sensing pathway as critical drivers of ESC differentiation. Functional analysis revealed that lysosome activity, the Ragulator protein complex, and the tumor-suppressor protein Folliculin enable the Rag GTPases C and D to bind and seclude the bHLH transcription factor Tfe3 in the cytoplasm. In contrast, ectopic nuclear Tfe3 represses specific developmental and met…

MaleTranscription GeneticGTPaseGTP PhosphohydrolasesPATHWAYMice0302 clinical medicineNeural Stem CellsCRISPRTUMOR-SUPPRESSORCell Self RenewalPhosphorylationSPECIFICATIONdevelopmental disorder0303 health sciencesGenomeBasic Helix-Loop-Helix Leucine Zipper Transcription FactorsCell DifferentiationMouse Embryonic Stem CellsFlcndifferentiationCell biologymedicine.anatomical_structuremTORMolecular MedicineFemaleSignal transductionProtein BindingSignal TransductionRECRUITMENTBiology03 medical and health sciencesRag GTPasesLysosomeGeneticsmedicineAnimalsHumansPoint MutationNAIVE PLURIPOTENCYAMINO-ACID LEVELSTranscription factorAllelesPI3K/AKT/mTOR pathway030304 developmental biologyCOMPLEXFOLLICULINRagulatorCell Biologypluripotencyembryonic stem cellEmbryonic stem cellTfe3[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsCytoplasmLysosomes030217 neurology & neurosurgeryCell Stem Cell
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Can a magic recipe foster university spin-off creation?

2015

This study examines factors that explain the creation of university spin-offs. The study focuses on mechanisms that technology transfer offices (TTOs) and universities employ to foster spin-offs. These mechanisms include technology transfer activities that support spin-offs, normative frameworks, support infrastructures (i.e., business incubators and science parks), and TTO staff's specialist technical skills. The analysis also differentiates between public and private universities. Spin-offs belong to one or more of the following groups: spin-offs with support from the university's TTO, spin-offs operating under a license agreement, and spin-offs in which the TTO or university holds equity…

MarketingFuzzy-set QCAEntrepreneurshipFuzzy setsMagic (illusion)Conjunts borrososUniversity spin-offbusiness.industryQualitative comparative analysisEquity (finance)Crisp-setQCATransferència de tecnologiaAcademic spin-outsPublic relationsUniversity spin-offsAntecedent (grammar):Economia i organització d'empreses [Àrees temàtiques de la UPC]NormativeEmpreses spin-offSociologyMarketingbusinessLicenseTechnology transfer
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Between a rock and a soft place: the role of viruses in lithification of modern microbial mats.

2021

10 pages; International audience; Stromatolites are geobiological systems formed by complex microbial communities, and fossilized stromatolites provide a record of some of the oldest life on Earth. Microbial mats are precursors of extant stromatolites; however, the mechanisms of transition from mat to stromatolite are controversial and are still not well understood. To fully recognize the profound impact that these ecosystems have had on the evolution of the biosphere requires an understanding of modern lithification mechanisms and how they relate to the geological record. We propose here viral mechanisms in carbonate precipitation, leading to stromatolite formation, whereby viruses directl…

Microbiology (medical)Geologic SedimentsBiogeochemical cycleviral lifestyleEarth sciencevirus–host interactionsGeologic recordMicrobiologyMESH: Host-Parasite InteractionsHost-Parasite InteractionsMESH: Viruses03 medical and health sciencesGeologic time scalebacteriophageVirologylytic/lysogenic cyclevirusesMicrobial matstromatoliteLithification030304 developmental biologyearly Earth0303 health sciencesBacteriabiology030306 microbiologyMESH: Virus Physiological PhenomenamicrobialitesBiosphereexopolymeric substances (EPS)MESH: Geologic Sedimentsbiology.organism_classificationEarly Earthmicrobial matMESH: BacteriaInfectious Diseases[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyStromatolite13. Climate actionCRISPRbiosignaturesVirus Physiological Phenomena
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Beta-Lactam Sensitive Bacteria Can Acquire ESBL-Resistance via Conjugation after Long-Term Exposure to Lethal Antibiotic Concentration

2020

Beta-lactams are commonly used antibiotics that prevent cell-wall biosynthesis. Beta-lactam sensitive bacteria can acquire conjugative resistance elements and hence become resistant even after being exposed to lethal (above minimum inhibitory) antibiotic concentrations. Here we show that neither the length of antibiotic exposure (1 to 16 h) nor the beta-lactam type (penam or cephem) have a major impact on the rescue of sensitive bacteria. We demonstrate that an evolutionary rescue can occur between different clinically relevant bacterial species (Klebsiella pneumoniae and Escherichia coli) by plasmids that are commonly associated with extended-spectrum beta-lactamase (ESBL) positive hospita…

Microbiology (medical)antibiotic resistancemedicine.drug_classKlebsiella pneumoniaeAntibioticsextended-spectrum beta-lactamaseBiologymedicine.disease_causeBiochemistryMicrobiologyArticleMicrobiologybakteerit03 medical and health sciencesplasmiditPlasmidAntibiotic resistancemedicineCRISPRPharmacology (medical)Klebsiella-bakteeritGeneral Pharmacology Toxicology and PharmaceuticsEscherichia coli030304 developmental biology0303 health sciencesCephemconjugative plasmid.030306 microbiologylcsh:RM1-950antibiootitbiology.organism_classification3. Good healthExtended-spectrum beta-lactamaseInfectious Diseaseslcsh:Therapeutics. Pharmacologyconjugative plasmidevolutionary rescuehorisontaalinen geeninsiirtoBacteriakolibakteeritantibioottiresistenssi
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Adapting a Phage to Combat Phage Resistance

2020

Phage therapy is becoming a widely recognized alternative for fighting pathogenic bacteria due to increasing antibiotic resistance problems. However, one of the common concerns related to the use of phages is the evolution of bacterial resistance against the phages, putatively disabling the treatment. Experimental adaptation of the phage (phage training) to infect a resistant host has been used to combat this problem. Yet, there is very little information on the trade-offs of phage infectivity and host range. Here we co-cultured a myophage FCV-1 with its host, the fish pathogen Flavobacterium columnare, in lake water and monitored the interaction for a one-month period. Phage resistance was…

Microbiology (medical)phage therapyGLIDING MOTILITYPhage therapyvirusesmedicine.medical_treatmentevoluutioVirulencefish pathogenmedicine.disease_causeBiochemistryMicrobiologyGenomebakteriofagitArticleMicrobiologyBacteriophage03 medical and health sciencesAntibiotic resistancemedicineCRISPRPharmacology (medical)General Pharmacology Toxicology and Pharmaceutics030304 developmental biology11832 Microbiology and virologyInfectivitylääkeresistenssi0303 health sciencesPREDATIONPRODUCTIVITYbiology030306 microbiologylcsh:RM1-950ARMS-RACEPathogenic bacteriakalatauditbiology.organism_classificationEVOLUTIONfagiterapialcsh:Therapeutics. PharmacologyInfectious Diseasesphage resistancecoevolution1182 Biochemistry cell and molecular biologyVIRULENCEHOST-RANGEBACTERIOPHAGEAntibiotics
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