Search results for "cytoskeleton"

showing 10 items of 272 documents

The regulation mechanism for the auto-inhibition of binding of human filamin A to integrin.

2009

The ability of adhesion receptors to transmit biochemical signals and mechanical force across cell membranes depends on interactions with the actin cytoskeleton. Human filamins are large actin cross-linking proteins that connect integrins to the cytoskeleton. Filamin binding to the cytoplasmic tail of beta integrins has been shown to prevent integrin activation in cells, which is important for controlling cell adhesion and migration. The molecular-level mechanism for filamin binding to integrin has been unclear, however, as it was recently demonstrated that filamin undergoes intramolecular auto-inhibition of integrin binding. In this study, using steered molecular dynamics simulations, we f…

Models MolecularProtein Foldinganimal structuresIntegrin beta ChainsFilaminsmacromolecular substancesBiologyFilaminCD49cCollagen receptorFilamin bindingPhosphoserineContractile ProteinsStructural BiologyHumansPhosphorylationMolecular BiologyIntegrin bindingBinding SitesMicrofilament ProteinsActin cytoskeletonCell biologybody regionsIntegrin alpha Mbiology.proteinIntegrin beta 6Stress MechanicalPeptidesProtein BindingJournal of molecular biology
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Small-angle X-ray scattering reveals compact domain-domain interactions in the N-terminal region of filamin C

2014

Filamins are multi-domain, actin cross-linking, and scaffolding proteins. In addition to the actin cross-linking function, filamins have a role in mechanosensor signaling. The mechanosensor function is mediated by domain-domain interaction in the C-terminal region of filamins. Recently, we have shown that there is a three-domain interaction module in the Nterminal region of filamins, where the neighboring domains stabilize the structure of the middle domain and thereby regulate its interaction with ligands. In this study, we have used small-angle X-ray scattering as a tool to screen for potential domain-domain interactions in the N-terminal region. We found evidence of four domain-domain in…

Models MolecularScaffold proteinProtein StructureProtein ConformationFilaminslcsh:Medicinemacromolecular substancesBiologyFilaminBiochemistryProtein–protein interactionProtein structureX-Ray Diffractioncompact domain-domain interactionsScattering Small AngleMacromolecular Structure AnalysisProtein InteractionsCytoskeletonlcsh:ScienceMolecular BiologyActinMultidisciplinarySmall-angle X-ray scatteringlcsh:Rta1182Biology and Life SciencesProteinsComputational BiologyRecombinant ProteinsProtein Structure TertiaryCell biologyCytoskeletal Proteinssmall-angle X-ray scatteringDomain (ring theory)Biophysicslcsh:QGlobular ProteinsStructural ProteinsResearch Articlefilamin CPloS One
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The molecular basis of filamin binding to integrins and competition with talin.

2006

The ability of adhesion receptors to transmit biochemical signals and mechanical force across cell membranes depends on interactions with the actin cytoskeleton. Filamins are large, actin-crosslinking proteins that connect multiple transmembrane and signaling proteins to the cytoskeleton. Here, we describe the high-resolution structure of an interface between filamin A and an integrin adhesion receptor. When bound, the integrin beta cytoplasmic tail forms an extended beta strand that interacts with beta strands C and D of the filamin immunoglobulin-like domain (IgFLN) 21. This interface is common to many integrins, and we suggest it is a prototype for other IgFLN domain interactions. Notabl…

Models MolecularTalinanimal structuresIntegrin beta ChainsProtein ConformationFilaminsRecombinant Fusion ProteinsIntegrinMolecular Sequence Datamacromolecular substancesPlasma protein bindingFilaminCrystallography X-RayFilamin bindingMiceContractile ProteinsFLNAAnimalsAmino Acid SequenceMolecular BiologyNuclear Magnetic Resonance BiomolecularBinding SitesbiologySequence Homology Amino AcidCalpainMicrofilament ProteinsReproducibility of ResultsCell BiologyActin cytoskeletonCell biologyProtein Structure Tertiarybody regionsIntegrin alpha Mbiology.proteinNIH 3T3 CellsIntegrin beta 6Protein BindingMolecular cell
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Identification and relevance of the CD95-binding domain in the N-terminal region of ezrin.

2003

The CD95 (Fas/APO-1) linkage to the actin cytoskeleton through ezrin is an essential requirement for susceptibility to the CD95-mediated apoptosis in CD4+ T cells. We have previously shown that moesin was not involved in the binding to CD95. Here we further support the specificity of the ezrin/CD95 binding, showing that radixin did not bind CD95. The ezrin region specifically and directly involved in the binding to CD95 was located in the middle lobe of the ezrin FERM domain, between amino acids 149 and 168. In this region, ezrin, radixin, and moesin show 60-65% identity, as compared with the 86% identity in the whole FERM domain. Transfection of two different human cell lines with a green …

Moesinchemical and pharmacologic phenomenaApoptosismacromolecular substancesBiologyBiochemistryEzrinRadixinhemic and lymphatic diseasesHumansfas ReceptorMolecular BiologyActinBinding SitesFERM domainhemic and immune systemsCell BiologyTransfectionActin cytoskeletonPhosphoproteinsActinsCell biologyProtein Structure TertiaryCytoskeletal ProteinsMutationbiological phenomena cell phenomena and immunityBinding domainHeLa CellsProtein BindingSignal TransductionThe Journal of biological chemistry
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Papillote and Piopio:DrosophilaZP-domain proteins required for cell adhesion to the apical extracellular matrix and microtubule organization

2005

Adhesion between epithelial cells and extracellular substrates is normally mediated through basal adhesion complexes. However, some cells also possess comparable junctions on their apical surface. Here, we describe two new Drosophila proteins, Piopio and Papillote, that are required for the link between the apical epithelial surface and the overlying apical extracellular matrix (aECM). The two proteins share a zona pellucida (ZP) domain with mammalian aECM components, including the tectorins found in the vertebrate inner ear. Tagged versions of both proteins localized to the apical epithelial surface. Mutations in piopio, papillote and dumpy (another gene encoding a ZP-domain protein) cause…

Molecular Sequence DataBiologyMicrotubulesEpitheliumExtracellular matrixMicrotubuleCell AdhesionmedicineExtracellularAnimalsDrosophila ProteinsWings AnimalAmino Acid SequenceCell adhesionCytoskeletonZona pellucidaMicrotubule nucleationExtracellular Matrix ProteinsSequence Homology Amino AcidMembrane ProteinsEpithelial CellsCell BiologyExtracellular MatrixCell biologyMicroscopy ElectronDrosophila melanogasterPhenotypemedicine.anatomical_structureMicroscopy FluorescenceMutationCarrier ProteinsDrosophila ProteinJournal of Cell Science
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MyRIP, a novel Rab effector, enables myosin VIIa recruitment to retinal melanosomes

2002

Defects of the myosin VIIa motor protein cause deafness and retinal anomalies in humans and mice. We report on the identification of a novel myosin-VIIa-interacting protein that we have named MyRIP (myosin-VIIa- and Rab-interacting protein), since it also binds to Rab27A in a GTP-dependent manner. In the retinal pigment epithelium cells, MyRIP, myosin VIIa and Rab27A are associated with melanosomes. In transfected PC12 cells, overexpression of MyRIP was shown to interfere with the myosin VIIa tail localization. We propose that a molecular complex composed of Rab27A, MyRIP and myosin VIIa bridges retinal melanosomes to the actin cytoskeleton and thereby mediates the local trafficking of thes…

Molecular Sequence Datamacromolecular substancesMyosinsBiologyBiochemistryRetinarab27 GTP-Binding ProteinsMotor proteinMicechemistry.chemical_compoundTwo-Hybrid System Techniquesotorhinolaryngologic diseasesGeneticsmedicineAnimalsHumansAmino Acid SequenceRAB27Molecular BiologyGene LibraryMelanosomesRetinal pigment epitheliumScientific ReportsDyneinsRetinalActin cytoskeletonCell biologymedicine.anatomical_structurechemistryOrgan Specificityrab GTP-Binding ProteinsMelanosome transportMyosin VIIaMelanophilinsense organsRabSequence Alignmentcirculatory and respiratory physiologyEMBO reports
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Tension causes structural unfolding of intracellular intermediate filaments

2020

AbstractIntermediate filament (IF) proteins are a class of proteins that constitute different filamentous structures in mammalian cells. As such, IF proteins are part of the load-bearing cytoskeleton and support the nuclear envelope. Molecular dynamics simulations have shown that IF proteins undergo secondary structural changes to compensate mechanical loads, which has been confirmed by experimental in vitro studies on IF hydrogels. However, the structural response of intracellular IF to mechanical load has yet to be elucidated in cellulo. Here, we use in situ nonlinear Raman imaging combined with multivariate data analysis to quantify the intracellular secondary structure of the IF cytoske…

Molecular dynamicsMechanical loadbiologyChemistrySelf-healing hydrogelsBiophysicsbiology.proteinVimentinCytoskeletonIntermediate filamentProtein secondary structureIntracellular
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Topical Review: Progress in Desmin-Related Myopathies

2000

Desmin-related myopathies are sporadic and familial neuromuscular conditions of considerable clinical heterogeneity uniformly marked by the pathologic accretion of desmin, often in a filamentous fashion. A large variety of other proteins, some of them cytoskeletal, also accrue. Morphologically, two types may be distinguished, one characterized by inclusions such as cytoplasmic and spheroid bodies or desmin-dystrophin plaques and another marked by granulofilamentous material. The genetic spectrum of desmin-related myopathies is quite diverse in that missense mutations and deletions in the desmin gene and a missense mutation in the α-B crystallin gene have been detected and several genes on o…

MutationMutantmacromolecular substancesBiologymedicine.disease_causeMolecular biology03 medical and health sciences0302 clinical medicineCrystallinCytoplasm030225 pediatricsPediatrics Perinatology and Child HealthmedicineMissense mutationDesminNeurology (clinical)CytoskeletonGene030217 neurology & neurosurgeryJournal of Child Neurology
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Are dendrites in Drosophila homologous to vertebrate dendrites?

2005

AbstractDendrites represent arborising neurites in both vertebrates and invertebrates. However, in vertebrates, dendrites develop on neuronal cell bodies, whereas in higher invertebrates, they arise from very different neuronal structures, the primary neurites, which also form the axons. Is this anatomical difference paralleled by principal developmental and/or physiological differences? We address this question by focussing on one cellular model, motorneurons of Drosophila and characterise the compartmentalisation of these cells. We find that motorneuronal dendrites of Drosophila share with typical vertebrate dendrites that they lack presynaptic but harbour postsynaptic proteins, display c…

NeuriteCompartmentalisationDendriteDendriteAnimals Genetically ModifiedMicePostsynaptic potentialbiology.animalmedicineAnimalsUrbilaterianMolecular BiologyMosaic analysisCytoskeletonCells CulturedMotor NeuronsDendritic spikeTransmitter receptorsbiologyVertebrateCell PolarityCell DifferentiationCell BiologyAnatomyDendritesbiology.organism_classificationBiological EvolutionCell biologyRatsmedicine.anatomical_structureDrosophila melanogasterDrosophilaSomaCalciumRabbitsCellular modelDevelopmental BiologyDevelopmental biology
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Microtubule Dynamics and Neuronal Excitability: Advances on Cytoskeletal Components Implicated in Epileptic Phenomena

2020

AbstractExtensive researches have deepened knowledge on the role of synaptic components in epileptogenesis, but limited attention has been devoted to the potential implication of the cytoskeleton. The study of the development of epilepsy and hyperexcitability states involves molecular, synaptic, and structural alterations of neuronal bioelectric activity. In this paper we aim to explore the neurobiological targets involved in microtubule functioning and cytoskeletal transport, i.e. how dynamic scaffolding of microtubules can influence neuronal morphology and excitability, in order to suggest a potential role for microtubule dynamics in the processes turning a normal neuronal network in a hy…

NeurodevelopmentHyperexcitabilityMicrotubuleBiologyMicrotubulesSettore BIO/09 - FisiologiaEpileptogenesisNeuroprotectionMicrotubule polymerizationCellular and Molecular NeuroscienceNeurotrophic factorsMicrotubulemedicineHumansPremovement neuronal activityCannabinoidCytoskeletonNeuronsEpilepsyNeurodegenerationCell BiologyGeneral Medicinemedicine.diseaseNeuroprotectionMicrotubule-Associated ProteinsNeuroscienceNeural developmentCellular and Molecular Neurobiology
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