Search results for "dam"

showing 10 items of 3828 documents

Lovastatin causes sensitization of HeLa cells to ionizing radiation‐induced apoptosis by the abrogation of G2 blockage

2003

To investigate the effect of inhibition of Ras/Rho-regulated signalling by 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors (statins) on radiation-induced cell killing and apoptosis.Different human cell lines were pretreated or not with lovastatin before exposure to gamma-rays. Afterwards, radiation-induced cell killing, formation and repair of double-strand breaks, activation of radiation-inducible signal mechanisms (i.e. p53, p21, extracellular-signal-related kinase (ERK), NF-kappaB), changes in cell cycle progression and apoptosis were analysed.As shown by a colony formation assay, lovastatin sensitized HeLa cells to gamma-radiation-induced cell killing. The lovastati…

Cyclin-Dependent Kinase Inhibitor p21G2 PhaseMAPK/ERK pathwayApoptosisBiologyHeLaCyclinspolycyclic compoundsmedicineHumansRadiology Nuclear Medicine and imagingLovastatinSensitizationRadiological and Ultrasound TechnologyKinaseNF-kappa Bnutritional and metabolic diseasesCell cyclebiology.organism_classificationCell biologyCell killingmedicine.anatomical_structureGamma RaysApoptosislipids (amino acids peptides and proteins)LovastatinHydroxymethylglutaryl-CoA Reductase InhibitorsMitogen-Activated Protein KinasesTumor Suppressor Protein p53DNA DamageHeLa Cellsmedicine.drugInternational Journal of Radiation Biology
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Benzo[a]pyrene represses DNA repair through altered E2F1/E2F4 function marking an early event in DNA damage-induced cellular senescence

2020

AbstractTranscriptional regulation of DNA repair is of outmost importance for the restoration of DNA integrity upon genotoxic stress. Here we report that the potent environmental carcinogen benzo[a]pyrene (B[a]P) activates a cellular DNA damage response resulting in transcriptional repression of mismatch repair (MMR) genes (MSH2, MSH6, EXO1) and of RAD51, the central homologous recombination repair (HR) component, ultimately leading to downregulation of MMR and HR. B[a]P-induced gene repression is caused by abrogated E2F1 signalling. This occurs through proteasomal degradation of E2F1 in G2-arrested cells and downregulation of E2F1 mRNA expression in G1-arrested cells. Repression of E2F1-me…

Cyclin-Dependent Kinase Inhibitor p21SenescenceAcademicSubjects/SCI00010DNA repairDNA damageRAD51E2F4 Transcription FactorBiologyDNA Mismatch Repair03 medical and health sciences0302 clinical medicineCell Line TumorBenzo(a)pyreneGeneticsHumansCellular SenescenceCell Line Transformed030304 developmental biology0303 health sciencesGene regulation Chromatin and EpigeneticsRecombinational DNA RepairEpithelial CellsKv Channel-Interacting ProteinsCell Cycle CheckpointsDNAFibroblastsCell biologyDNA-Binding ProteinsRepressor ProteinsMSH6DNA Repair EnzymesExodeoxyribonucleasesMutS Homolog 2 ProteinGamma RaysMSH2030220 oncology & carcinogenesisCarcinogensMCF-7 CellsDNA mismatch repairRad51 RecombinaseCell agingE2F1 Transcription FactorDNA DamageSignal TransductionNucleic Acids Research
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The role of reactive oxygen species and subsequent DNA-damage response in the emergence of resistance towards resveratrol in colon cancer models

2014

AbstractIn spite of the novel strategies to treat colon cancer, mortality rates associated with this disease remain consistently high. Tumour recurrence has been linked to the induction of resistance towards chemotherapy that involves cellular events that enable cancer cells to escape cell death. Treatment of colon cancer mainly implicates direct or indirect DNA-damaging agents and increased repair or tolerances towards subsequent lesions contribute to generate resistant populations. Resveratrol (RSV), a potent chemosensitising polyphenol, might share common properties with chemotherapeutic drugs through its indirect DNA-damaging effects reported in vitro. In this study, we investigated how…

Cyclin-Dependent Kinase Inhibitor p21SenescenceCancer ResearchProgrammed cell deathColonDNA damageColorectal cancerImmunologyApoptosisBiologyResveratrolS PhaseHistonesPolyploidyCellular and Molecular Neurosciencechemistry.chemical_compoundCell Line TumorStilbenesmedicineAnimalsHumansCHEK1Cyclin-Dependent Kinase Inhibitor p16Cell Biologymedicine.diseaseAntineoplastic Agents PhytogenicRatsGene Expression Regulation NeoplasticCheckpoint Kinase 2chemistryDrug Resistance NeoplasmResveratrolApoptosisCheckpoint Kinase 1Cancer cellImmunologyCancer researchOriginal ArticleTumor Suppressor Protein p53Reactive Oxygen SpeciesProtein KinasesDNA DamageSignal TransductionCell Death & Disease
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Catalytic Oxidation of Alkanes and Alkenes by H 2 O 2 with a μ‐Oxido Diiron(III) Complex as Catalyst/Catalyst Precursor

2015

A new mu-oxo diiron(III) complex of the lithium salt of the pyridine-based unsymmetrical ligand 3-[(3-{[bis(pyridin-2-ylmethyl)amino]methyl}-2-hydroxy-5-methylbenzyl)(pyridin2-ylmethyl)amino] propanoate (LiDPCPMPP), [Fe-2(mu-O)(LiDPCPMPP)(2)](ClO4)(2), has been synthesized and characterized. The ability of the complex to catalyze oxidation of several alkanes and alkenes has been investigated by using CH3COOH/H2O2 (1:1) as an oxidative system. Moderate activity in cyclohexane oxidation (TOF = 33 h(-1)) and good activity in cyclohexene oxidation (TOF = 72 h(-1)) were detected. Partial retention of configuration (RC = 53%) in cis- and trans-1,2-dimethylcyclohexane oxidation, moderate 3 degrees…

Cyclohexane010405 organic chemistryAdamantaneCyclohexeneHomogeneous catalysis010402 general chemistryPhotochemistry01 natural sciencesMedicinal chemistry0104 chemical sciencesCatalysisInorganic Chemistrychemistry.chemical_compoundchemistryCatalytic oxidationlithiumPyridineSelectivityta116European Journal of Inorganic Chemistry
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Ultrastructural and histochemical analysis reveals ethylene-induced responses underlying reduced peel collapse in detached citrus fruit

2010

Fruits from many citrus cultivars develop depressed areas in the flavedo (outer part of the peel) and albedo (inner part) following detachment. Although ultrastructural analysis may provide important information about multiple plant responses to stresses and external stimuli at the cell and tissue levels, and despite the proved efficacy of ethylene in reducing peel damage in citrus fruit, cytological responses of this horticultural crop to protective ethylene concentrations have not yet been reported. We show that applying high ethylene levels (2 mu L L(-1) for 14 days) causes sublethal stress as it favored the alteration of cuticle, vacuole, middle lamella and primary wall, especially in t…

CyclopropanesCitrusHistologyEthylenefood.ingredientPectinStarchCuticleBOTANICAVacuoleBiologyPolysaccharideElectron Microscopy Service of the UPVchemistry.chemical_compoundfoodMicroscopy Electron TransmissionPolysaccharidesBotanyInstrumentationMiddle lamellachemistry.chemical_classificationBIOLOGIA VEGETALfood and beveragesStarchEthylenesCell ultrastructurePectinMedical Laboratory TechnologyHorticulturechemistryFruitPeel damageUltrastructureAnatomyCross-protection
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Nuclear Translocation of Mismatch Repair Proteins MSH2 and MSH6 as a Response of Cells to Alkylating Agents

2000

Mammalian mismatch repair has been implicated in mismatch correction, the prevention of mutagenesis and cancer, and the induction of genotoxicity and apoptosis. Here, we show that treatment of cells specifically with agents inducing O(6)-methylguanine in DNA, such as N-methyl-N'-nitro-N-nitrosoguanidine and N-methyl-N-nitrosourea, elevates the level of MSH2 and MSH6 and increases GT mismatch binding activity in the nucleus. This inducible response occurs immediately after alkylation, is long-lasting and dose-dependent, and results from translocation of the preformed MutSalpha complex (composed of MSH2 and MSH6) from the cytoplasm into the nucleus. It is not caused by an increase in MSH2 gen…

CytoplasmDNA RepairBase Pair MismatchRNA StabilityChromosomal translocationmedicine.disease_causeBiochemistrychemistry.chemical_compoundMismatch Repair Endonuclease PMS2Adenosine TriphosphatasesNuclear ProteinsMethylnitrosoureaNeoplasm ProteinsDNA-Binding ProteinsMutS Homolog 2 ProteinDNA mismatch repairMutL Protein Homolog 1Protein BindingAlkylating AgentsMethylnitronitrosoguanidinecongenital hereditary and neonatal diseases and abnormalitiesGuanineActive Transport Cell NucleusBiologyCell LineO(6)-Methylguanine-DNA MethyltransferaseProto-Oncogene ProteinsDNA Repair ProteinmedicineHumansRNA MessengerneoplasmsMolecular BiologyAdaptor Proteins Signal TransducingCell NucleusMutagenesisnutritional and metabolic diseasesDNACell BiologyDNA MethylationMolecular biologydigestive system diseasesMSH6DNA Repair EnzymesGene Expression RegulationchemistryMSH2Carrier ProteinsGenotoxicityDNADNA DamageHeLa CellsJournal of Biological Chemistry
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Ectodomain shedding of L1 adhesion molecule promotes cell migration by autocrine binding to integrins.

2001

The L1 adhesion molecule plays an important role in axon guidance and cell migration in the nervous system. L1 is also expressed by many human carcinomas. In addition to cell surface expression, the L1 ectodomain can be released by a metalloproteinase, but the biological function of this process is unknown. Here we demonstrate that membrane-proximal cleavage of L1 can be detected in tumors and in the developing mouse brain. The shedding of L1 involved a disintegrin and metalloproteinase (ADAM)10, as transfection with dominant-negative ADAM10 completely abolishes L1 release. L1-transfected CHO cells (L1-CHO) showed enhanced haptotactic migration on fibronectin and laminin, which was blocked …

CytoplasmIntegrinsL1; shedding; ADAM10; cell migration; integrinsADAM10IntegrinGene ExpressionCHO CellsBiologyArticle03 medical and health sciencesParacrine signallingMice0302 clinical medicineCell MovementCricetinaeEndopeptidasesTumor Cells CulturedAnimalsAspartic Acid EndopeptidasesHumansReceptors VitronectinFibrinolysinNeural Cell Adhesion Molecules030304 developmental biology0303 health sciencesBinding SitesMembrane GlycoproteinsCell adhesion moleculeCell MembraneAntibodies MonoclonalBrainCell migrationBiological TransportCell BiologyMolecular biologyPeptide FragmentsCell biologyFibronectinAutocrine CommunicationEctodomainSolubility030220 oncology & carcinogenesisbiology.proteinNeural cell adhesion moleculeAmyloid Precursor Protein SecretasesLeukocyte L1 Antigen ComplexOligopeptidesThe Journal of cell biology
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Electron-interacting dark matter: Implications from DAMA/LIBRA-phase2 and prospects for liquid xenon detectors and NaI detectors

2019

We investigate the possibility for the direct detection of low-mass (GeV scale) weakly interacting massive particles (WIMP) dark matter in scintillation experiments. Such WIMPs are typically too light to leave appreciable nuclear recoils but may be detected via their scattering off atomic electrons. In particular, the DAMA Collaboration [R. Bernabei et al., Nucl. Phys. At. Energy 19, 307 (2018)] has recently presented strong evidence of an annual modulation in the scintillation rate observed at energies as low as 1 keV. Despite a strong enhancement in the calculated event rate at low energies, we find that an interpretation in terms of electron-interacting WIMPs cannot be consistent with ex…

DAMA/LIBRACosmology and Nongalactic Astrophysics (astro-ph.CO)detector: performancePhysics::Instrumentation and DetectorsDark matterFOS: Physical scienceschemistry.chemical_elementElectron01 natural sciencesWIMP: dark matterNuclear physicsHigh Energy Physics - Phenomenology (hep-ph)XenonWIMP0103 physical sciences010306 general physicsenhancementscintillation counterenergy: lowPhysicsScintillationxenon: liquid010308 nuclear & particles physicsatom: wave functionDAMAmodulationHigh Energy Physics - Phenomenologychemistryelectron: scatteringWeakly interacting massive particles[PHYS.HPHE]Physics [physics]/High Energy Physics - Phenomenology [hep-ph]direct detection[PHYS.ASTR]Physics [physics]/Astrophysics [astro-ph]Event (particle physics)Astrophysics and astroparticle physicsAstrophysics - Cosmology and Nongalactic AstrophysicsPhysical Review
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Simultaneous measurement of temperature and strain in glass fiber/epoxy composites by embedded fiber optic sensors: II. Post-cure testing

2007

In this paper the use of fiber optic sensors embedded into GFRP laminates for structural health monitoring of thermo-mechanical loads is presented. The proposed sensing system, consisting of two coupled fiber Bragg grating (FBG) sensors, allows the simultaneous measurement of both temperature and strain by decoupling the change in reflected wavelength induced by temperature variations from that induced by either mechanical or apparent strain, with significant advantages either for monitoring the composite cure kinetics, as described in the accompanying paper (Part I), or for assessing the structural response to thermo-mechanical loads. Both thermal ramp and mechanical static/dynamic bending…

DAMAGE DETECTIONQuasi-static bending testsTRANSVERSE CRACKSFiber Bragg grating (FBG) sensorsDynamic testsCoefficient of thermal expansion (CTE)Condensed Matter PhysicsAtomic and Molecular Physics and OpticsCONCRETE STRUCTURESMechanics of MaterialsGFRP composite materialsSignal ProcessingGeneral Materials ScienceElectrical and Electronic EngineeringBRAGG GRATING SENSORSFiber Bragg grating (FBG) sensors; GFRP composite materials; Quasi-static bending tests; Dynamic tests; Coefficient of thermal expansion (CTE)Civil and Structural EngineeringFBG SENSORSCURE
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Numerical simulation of fatigue-driven delamination using interface elements

2005

This paper presents a computational technique for the prediction of fatigue-driven delamination growth in composite materials. The interface element, which has been extensively applied to predict delamination growth due to static loading, has been modified to incorporate the effects of cyclic loading. Using a damage mechanics formulation, the constitutive law for the interface element has been extended by incorporating a modified version of a continuum fatigue damage model. The paper presents details of the fatigue degradation strategy and examples of the predicted fatigue delamination growth in mode I, mode II and mixed mode I/II are presented to demonstrate that the numerical model mimics…

DAMAGEinterface elementNumerical AnalysisEngineeringComputer simulationbusiness.industryApplied Mathematicscomposite materialsConstitutive equationGeneral EngineeringFracture mechanicsFatigue damageStructural engineeringdelaminationFinite element methodComputational TechniqueINTERLAMINAR FRACTURE SPECIMENS DAMAGE COMPOSITES FATIGUE GROWTHFATIGUE GROWTHDamage mechanicsMode couplingCOMPOSITESfatigueINTERLAMINAR FRACTURE SPECIMENSbusinessInternational Journal for Numerical Methods in Engineering
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