Search results for "death"

showing 10 items of 1744 documents

Anandamide-induced apoptosis in Chang liver cells involves ceramide and JNK/AP-1 pathway

2006

In the present study we demonstrate that anandamide, the most important endogenous cannabinoid, markedly induced apoptosis in Chang liver cells, an immortalized non-tumor cell line derived from normal liver tissue, while it induced only modest effects in a number of hepatoma cell lines. The apoptotic effect was reduced by methyl-beta-cyclodextrin, a membrane cholesterol depletor, suggesting an interaction between anandamide and the membrane microdomains named lipid rafts. Anandamide effects were mediated by the production of ceramide, as demonstrated by experiments performed with the sphingomyelinase inhibitor, desipramine, or with the sphingomyelinase activator, melittin. This conclusion w…

CeramideProgrammed cell deathFas Ligand ProteinCell SurvivalPolyunsaturated AlkamidesLiver cytologyp38 mitogen-activated protein kinasesBlotting WesternApoptosisArachidonic AcidsBiologyCeramidesCell LineMembrane Potentialschemistry.chemical_compoundCell Line TumorProto-Oncogene ProteinsGeneticsHumansEnzyme InhibitorsMembrane GlycoproteinsBcl-2-Like Protein 11Dose-Response Relationship DrugDesipramineJNK Mitogen-Activated Protein KinasesMembrane ProteinsFree Radical ScavengersGeneral MedicineAnandamideEndocannabinoid systemAcetylcysteineCell biologyEnzyme ActivationTranscription Factor AP-1cannabinoids apoptosis tumor cells JNK/AP1LiverchemistryApoptosisCaspasesMitochondrial MembranesTumor Necrosis FactorsApoptosis Regulatory ProteinsSphingomyelinEndocannabinoidsSignal TransductionInternational Journal of Molecular Medicine
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Effectiveness of progestogens to improve perinatal outcome in twin pregnancies : an individual participant data meta-analysis

2015

Background In twin pregnancies, the rates of adverse perinatal outcome and subsequent long-term morbidity are substantial, and mainly result from preterm birth (PTB). Objectives To assess the effectiveness of progestogen treatment in the prevention of neonatal morbidity or PTB in twin pregnancies using individual participant data meta-analysis (IPDMA). Search strategy We searched international scientific databases, trial registration websites, and references of identified articles. Selection criteria Randomised clinical trials (RCTs) of 17–hydroxyprogesterone caproate (17Pc) or vaginally administered natural progesterone, compared with placebo or no treatment. Data collection and analysis I…

Cervical pessaryAdultmedicine.medical_specialtymedicine.medical_treatmentPerinatal DeathDiseasesCervix UteriInfant Newborn DiseasesEnterocolitis NecrotizingPregnancy17 alpha-Hydroxyprogesterone CaproatemedicineHydroxyprogesteronesHumansTwin PregnancyProgesteroneBronchopulmonary DysplasiaCerebral HemorrhagePregnancyRespiratory Distress SyndromeRespiratory Distress Syndrome NewbornProgestogenIntravaginalObstetricsbusiness.industryEnterocolitisInfant NewbornObstetrics and GynecologyInfantTwinmedicine.diseaseNewbornCervical Length MeasurementClinical trialAdministration IntravaginalTreatment OutcomePremature birthCervical Length MeasurementRelative riskAdministrationPregnancy TwinPremature BirthFemaleProgestinsbusinessNecrotizing
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Mother's Death: A Turning Point in the Lives of Charles Dickens's David Copperfield and Lev Tolstoy's Nikolay Irtenev

2022

Although it is not possible to establish exactly when Lev Tolstoy started reading Charles Dickens, it was in the 1850s, he was reading David Copperfield, which was one of his most favourite novels by the English writer. David Copperfield is probabiy the most popular of all Dickens’s novels, and it is certainly the most autobiographical. The tendency to combine personal with general in an ambiguous generic package was also present from the beginning of Tolstoy’s career. His Childhood can be described as a pseudo-autobiography that imitates autobiography in all aspects, except one: its author and narrator are not the same person. A strong mother-son relationship is clearly visible in David Co…

Charles Dickensmother's death in literatureLev TolstoyDavid CopperfieldEnglish literatureliterature of the 19th centuryChildhoodRussian literatureSlavia. Casopis pro slovanskou filologii / Slavia. The Journal for Slavic Philology
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Immunotherapy With Human Gamma Delta T Cells—Synergistic Potential of Epigenetic Drugs?

2018

Epigenetics has emerged as one of the fastest growing concepts, adding more than 45 new publications every day, spreading through various fields ( 1). Conrad Waddington coined the term “epigenetics” in 1942; however, a multitude of definitions has been endorsed by different researchers. In essence, Waddington’s definition of “epigenetics” and its redefinition by Holiday is at the heart of cellular function. Hence, it is obvious that epigenetic regulation plays a central role also in the specification, differentiation, and functional plasticity of T lymphocytes ( 2). T-cell fate decision in progenitor cells, functional CD4 T-cell plasticity, CD8 T-cell differentiation, but also T-cell memory…

Checkpoint Inhibitorslcsh:Immunologic diseases. Allergy0301 basic medicineDeltaOpinionmedicine.medical_treatmentImmune checkpoint inhibitorsCell PlasticityImmunologyNatural-killer Group 2 Member DBiologyEpigenesis Genetic03 medical and health sciencesCell Plasticitymedicineddc:6AnimalsHumansgamma delta T cellsImmunology and Allergyddc:610EpigeneticsIntraepithelial Lymphocytesprogrammed death 1DNA methylationnatural-killer group 2 member DProgrammed Death 1articlehistone acetylationGamma Delta T CellsImmunotherapy030104 developmental biologyHistone acetylationDNA methylationCancer researchIntraepithelial lymphocyteBromodomain And Extraterminal DomainBromodomain and ExtraTerminal domainImmunotherapyimmunotherapyProgrammed death 1lcsh:RC581-607checkpoint inhibitorsFrontiers in Immunology
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ATRIP protects progenitor cells against DNA damage in vivo

2020

AbstractThe maintenance of genomic stability during the cell cycle of progenitor cells is essential for the faithful transmission of genetic information. Mutations in genes that ensure genome stability lead to human developmental syndromes. Mutations in Ataxia Telangiectasia and Rad3-related (ATR) or in ATR-interacting protein (ATRIP) lead to Seckel syndrome, which is characterized by developmental malformations and short life expectancy. While the roles of ATR in replicative stress response and chromosomal segregation are well established, it is unknown how ATRIP contributes to maintaining genomic stability in progenitor cells in vivo. Here, we generated the first mouse model to investigat…

CheckpointsProgrammed cell deathDNA damage[SDV]Life Sciences [q-bio]610 MedizinBiologyDNA replicationDNA damage responseArticle03 medical and health sciences0302 clinical medicine610 Medical sciencesmedicineProgenitor celllcsh:QH573-671GeneMitosisComputingMilieux_MISCELLANEOUSCell proliferation030304 developmental biology0303 health scienceslcsh:CytologyDisease modelCell cyclemedicine.diseaseCell biologyApoptosis030220 oncology & carcinogenesisAtaxia-telangiectasiaCell Death & Disease
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Inhibitory effects of N-acetylcysteine on the functional responses of human eosinophils in vitro

2007

Background Oxidative stress appears to be relevant in the pathogenesis of inflammation in allergic diseases like bronchial asthma. Eosinophils are oxidant-sensitive cells considered as key effectors in allergic inflammation. Objective The aim of this work was to study the effects of the clinically used antioxidant N-acetyl-L-cysteine (NAC) on the functional responses of human-isolated eosinophils. Methods Human eosinophils were purified from the blood of healthy donors by a magnetic bead separation system. The effects of NAC were investigated on the generation of reactive oxygen species (chemiluminescence and flow cytometry), Ca2+ signal (fluorimetry), intracellular glutathione (GSH; flow c…

Chemokine CCL11EotaxinLuminescenceImmunologyhuman eosinophilsBiologyPharmacologymedicine.disease_causeTranslocation GeneticAllergic inflammationAcetylcysteinechemistry.chemical_compoundmedicineHumansImmunology and Allergychemistry.chemical_classificationreactive oxygen speciesEosinophil cationic proteinReactive oxygen speciesCell DeathEosinophil Cationic ProteinNADPH OxidasesFree Radical ScavengersGlutathioneEosinophilPhosphoproteinsGlutathioneN-acetylcysteineAcetylcysteineEosinophilsN-Formylmethionine Leucyl-Phenylalaninemedicine.anatomical_structurechemistryBiochemistryChemokines CCeosinophil cationic proteinCalciumReactive Oxygen SpeciesOxidative stressp47(phox)medicine.drugp67(phox)
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Calcium signatures and signaling in cytosol and organelles of tobacco cells induced by plant defense elicitors

2011

Calcium signatures induced by two elicitors of plant defense reactions, namely cryptogein and oligogalacturonides, were monitored at the subcellular level, using apoaequorin-transformed Nicotiana tabacum var Xanthi cells, in which the apoaequorin calcium sensor was targeted either to cytosol, mitochondria or chloroplasts. Our study showed that both elicitors induced specific Ca2+ signatures in each compartment, with the most striking difference relying on duration. Common properties also emerged from the analysis of Ca2+ signatures: both elicitors induced a biphasic cytosolic [Ca2+] elevation together with a single mitochondrial [Ca2+] elevation concomitant with the first cytosolic [Ca2+] p…

ChlorophyllChloroplastsTime FactorsPhysiology[SDV]Life Sciences [q-bio]Nicotiana tabacumAequorinMitochondrionMITOCHONDRIALAntiportersCA2+CytosolPlant defenseINTACT CHLOROPLASTSCation Transport ProteinsCalcium signalingRECOMBINANT AEQUORINDEATHfood and beveragesARABIDOPSISOligogalacturonidesMitochondriaChloroplastBiochemistry[SDE]Environmental SciencesCryptogeinPhytophthorachemistry.chemical_elementCalciumBiologyChloroplastFluorescenceFungal ProteinsPHOTOSYSTEM-IIPlant CellsTobaccoOrganelle[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyCalcium SignalingMolecular BiologyHYPERSENSITIVE RESPONSENITRIC-OXIDECell MembraneCell Biologybiology.organism_classificationSALICYLIC-ACIDOxygenCytosolchemistryBiophysicsbiology.proteinCalciumCell Calcium
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Plastid-nuclear complexes in the photosynthesizing cells from their mitosis up to programmed death

2013

Permanent plastid-nuclear complexes (PNCs) exist in tobacco cells from their mitosis up to programmed cell death (PCD). PNCs in senescing cells of tobacco leaves were typical by enclosure of peroxisomes and mitochondria among chloroplasts which were in contact with nucleus. Such a complex position provides simultaneous interaction of these organelles and direct regulation of metabolism and PCD avoiding the cytosol.

ChloroplastProgrammed cell deathCytosolPhysiologyOrganelleotorhinolaryngologic diseasesPlant SciencePeroxisomeBiologyPlastidMitochondrionMitosisCell biologyPhotosynthetica
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Paradoxical effect of increased diastolic Ca(2+) release and decreased sinoatrial node activity in a mouse model of catecholaminergic polymorphic ven…

2012

Background— Catecholaminergic polymorphic ventricular tachycardia is characterized by stress-triggered syncope and sudden death. Patients with catecholaminergic polymorphic ventricular tachycardia manifest sinoatrial node (SAN) dysfunction, the mechanisms of which remain unexplored. Methods and Results— We investigated SAN [Ca 2+ ] i handling in mice carrying the catecholaminergic polymorphic ventricular tachycardia–linked mutation of ryanodine receptor (RyR2 R4496C ) and their wild-type (WT) littermates. In vivo telemetric recordings showed impaired SAN automaticity in RyR2 R4496C mice after isoproterenol injection, analogous to what was observed in catecholaminergic polymorphic ventricul…

ChronotropicTachycardiaMalePatch-Clamp TechniquesAction Potentials030204 cardiovascular system & hematologyVentricular tachycardiaMice0302 clinical medicineSinoatrial NodeCatecholaminergic0303 health sciencesRyanodine receptorAdrenergic beta-AgonistsMiddle AgedSarcoplasmic Reticulummedicine.anatomical_structurecardiovascular systemCardiologyFemalemedicine.symptomCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtyIn Vitro TechniquesCatecholaminergic polymorphic ventricular tachycardiaSudden deathArticle03 medical and health sciencesPhysiology (medical)Internal medicinemedicineAnimalsHumansCalcium SignalingExercise030304 developmental biologyAgedbusiness.industrySinoatrial nodeIsoproterenolRyanodine Receptor Calcium Release Channelmedicine.diseaseMice Mutant StrainsMice Inbred C57BLDisease Models AnimalEndocrinologyMutationTachycardia VentricularCalciumbusinessCirculation
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Impact Of Hypoxic And Acidic Extracellular Conditions On Cytotoxicity Of Chemotherapeutic Drugs

2007

In the microenvironment of solid growing tumors, pronounced hypoxia or extracellular acidosis is commonly found The aim of this study was the analysis of the cytotoxic effect of different chemotherapeutic agents (cisplatin, daunorubicin, docetaxel) under these conditions in vitro Prostate carcinoma cells (R3327-AT1) were exposed to hypoxia (pO 2 05 mmHg) or extracellular acidosis (pH=66) for 6h After 3h, cytotoxic drugs were added The cytotoxic effect was assessed by measuring caspase 3-activity (apoptosis), LDH release (necrosis) and repopulation of the cells after chemotherapy (cell death) Compared to aerobic control conditions, severe hypoxia over 6h per se led to a slight increase in ap…

CisplatinChemotherapyProgrammed cell deathNecrosisDaunorubicinmedicine.medical_treatmentBiologyPharmacologyApoptosismedicineExtracellularmedicine.symptomCytotoxicitymedicine.drug
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