Search results for "degeneration"

showing 10 items of 601 documents

Serum neurofilament light chain is a biomarker of acute and chronic neuronal damage in early multiple sclerosis.

2018

Background: Monitoring neuronal injury remains one key challenge in early relapsing-remitting multiple sclerosis (RRMS) patients. Upon axonal damage, neurofilament – a major component of the neuro-axonal cytoskeleton – is released into the cerebrospinal fluid (CSF) and subsequently peripheral blood. Objective: To investigate the relevance of serum neurofilament light chain (sNfL) for acute and chronic axonal damage in early RRMS. Methods: sNfL levels were determined in 74 patients (63 therapy-naive) with recently diagnosed clinically isolated syndrome (CIS) or RRMS using Single Molecule Array technology. Standardized 3 T magnetic resonance imaging (MRI) was performed at baseline and 1–3 con…

AdultMalePathologymedicine.medical_specialtyNeurofilamentMultiple SclerosisNeurofilament lightIntermediate FilamentsSeverity of Illness IndexDisease activity03 medical and health sciencesYoung Adult0302 clinical medicineNeuronal damageNeurofilament ProteinsMedicineHumans030212 general & internal medicineNeuronsbusiness.industryMultiple sclerosisNeurodegenerationBrainMiddle Agedmedicine.diseaseNeurologyBiomarker (medicine)FemaleNeurology (clinical)Atrophybusiness030217 neurology & neurosurgeryClinical progressionBiomarkersMultiple sclerosis (Houndmills, Basingstoke, England)
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Degenerative alterations of the spine in an Early Mediaeval population from Mannheim-Seckenheim, Germany

2012

Abstract Palaeopathological and palaeoepidemiological analyses of human skeletal remains are some of the most important bases for the reconstruction of life of past populations. The assessment of frequency and degree of pathological alterations contributes to conclusions of a population's health status, labour conditions, and environmental influences. Degenerative diseases of the spine are among the most common lesions observed in archaeological human remains. The large number of excavated Early Mediaeval cemeteries in Germany enables the comparison of contemporary populations increasing the reliability of conclusions regarding their living conditions. In this study, 112 adult individuals w…

AdultMalePathologymedicine.medical_specialtyPaleopathologyPopulationDegeneration (medical)DiseaseGermanyHumansMedicineeducationPathologicaleducation.field_of_studyFossilsbusiness.industryAnatomySpondylarthrosisHistory MedievalSpine (zoology)AnthropologyFemaleSpinal DiseasesSpondylosisbusinessSpondylosis DeformansHOMO
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The temporal dynamics of postanoxic burst-suppression EEG.

2002

Burst-suppression EEG (BS-EEG) after cardiopulmonary resuscitation implies a bad prognosis, but little is known of the temporal dynamics of postanoxic BS-EEG. The authors studied 24 consecutive patients who developed BS-EEG within 24 hours after cardiopulmonary resuscitation, and followed 20 of these patients with serial EEGs. Except for one patient, BS-EEG was followed by another EEG pattern within 1 day, mainly areactive alpha EEG (n = 6), isoelectric EEG (n = 5), generalized continuous epileptiform discharges (n = 4), or theta; EEG (n = 3). The coexistence of different EEG patterns in the same recording was seen in 10 patients. Serial recordings disclosed a variety of EEG sequences with …

AdultMalePhysiologyElectroencephalographyEEG-fMRIBrain diagnosisEeg patternsEpilepsyPhysiology (medical)medicineHumansIn patientHypoxia BrainEvoked PotentialsAgedCerebral CortexNeuronsmedicine.diagnostic_testElectroencephalographyMiddle Agedmedicine.diseasePrognosisCardiopulmonary ResuscitationBurst suppressionNeurologyAnesthesiaNerve DegenerationBrain Damage ChronicEpilepsy GeneralizedFemaleNeurology (clinical)Anoxic encephalopathyPsychologyNeuroscienceJournal of clinical neurophysiology : official publication of the American Electroencephalographic Society
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Magnetic resonance imaging findings and their relationships in the thoracic and lumbar spine. Insights into the etiopathogenesis of spinal degenerati…

1995

Study design Descriptive epidemiologic study about magnetic resonance imaging findings in the spine. Objectives To describe the prevalence of magnetic resonance imaging findings in a general population at spinal levels T6-S1, and to examine the relationships of these findings within each spinal level and between levels. Summary of background data The prevalence of specific findings and the associations between findings and spinal levels can provide general insights into the etiopathogenesis of spinal degeneration. Methods Subjects consisted of 232 men from a population sample (mean age 49.3 years). Signal intensity, disc bulging, disc herniation, and endplate irregularities were among 11 fi…

AdultMalePopulationDegeneration (medical)Thoracic VertebraeLumbarmedicinePrevalenceHumansOrthopedics and Sports MedicineeducationAgededucation.field_of_studyLumbar Vertebraemedicine.diagnostic_testbusiness.industryAge FactorsReproducibility of ResultsMagnetic resonance imagingIntervertebral discAnatomyMiddle Agedmedicine.diseaseMagnetic Resonance ImagingSpondylolisthesisSpineIntervertebral diskmedicine.anatomical_structureLumbar spineNeurology (clinical)SpondylolisthesisbusinessIntervertebral Disc DisplacementSpine
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Translational value of choroid plexus imaging for tracking neuroinflammation in mice and humans.

2021

Neuroinflammation is a pathophysiological hallmark of multiple sclerosis and has a close mechanistic link to neurodegeneration. Although this link is potentially targetable, robust translatable models to reliably quantify and track neuroinflammation in both mice and humans are lacking. The choroid plexus (ChP) plays a pivotal role in regulating the trafficking of immune cells from the brain parenchyma into the cerebrospinal fluid (CSF) and has recently attracted attention as a key structure in the initiation of inflammatory brain responses. In a translational framework, we here address the integrity and multidimensional characteristics of the ChP under inflammatory conditions and question w…

AdultMaleProteomicsEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisMiceNatalizumabCerebrospinal fluidImmune systemmedicineAnimalsHumansNeuroinflammationMultidisciplinarybusiness.industryMultiple sclerosisExperimental autoimmune encephalomyelitisNeurodegenerationBrainmedicine.diseaseMagnetic Resonance ImagingMice Inbred C57BLDisease Models AnimalBlood-Brain BarrierChoroid PlexusNeuroinflammatory DiseasesChoroid plexusFemalebusinessNeurosciencemedicine.drugProceedings of the National Academy of Sciences of the United States of America
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Mutations in ARL2BP, Encoding ADP-Ribosylation-Factor-Like 2 Binding Protein, Cause Autosomal-Recessive Retinitis Pigmentosa

2013

Retinitis pigmentosa (RP) is a genetically heterogeneous retinal degeneration characterized by photoreceptor death, which results in visual failure. Here, we used a combination of homozygosity mapping and exome sequencing to identify mutations in ARL2BP, which encodes an effector protein of the small GTPases ARL2 and ARL3, as causative for autosomal-recessive RP (RP66). In a family affected by RP and situs inversus, a homozygous, splice-acceptor mutation, c.101−1G>C, which alters pre-mRNA splicing of ARLBP2 in blood RNA, was identified. In another family, a homozygous c.134T>G (p.Met45Arg) mutation was identified. In the mouse retina, ARL2BP localized to the basal body and cilium-associated…

AdultMaleRetinal degenerationCentrioleMolecular Sequence DataGenes RecessiveBiologymedicine.disease_causeMice03 medical and health sciences0302 clinical medicineBardet–Biedl syndromeGTP-Binding ProteinsReportRetinitis pigmentosaGeneticsmedicineAnimalsHumansBasal bodyGenetics(clinical)Photoreceptor CellsGenetics (clinical)030304 developmental biologyPrimary ciliary dyskinesiaGenetics0303 health sciencesMutationBase SequenceADP-Ribosylation FactorsCiliumHomozygoteMembrane Transport ProteinsEpithelial Cellsmedicine.diseasePedigreeCell biologyMutationFemalesense organsCarrier ProteinsRetinitis Pigmentosa030217 neurology & neurosurgeryProtein BindingTranscription FactorsThe American Journal of Human Genetics
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Effect of focal cerebellar lesions on procedural learning in the serial reaction time task

1998

Prior studies have shown that procedural learning is severely impaired in patients with diffuse cerebellar damage (cortical degeneration) as measured by the serial reaction time task (SRTT). We hypothesize that focal cerebellar lesions can also have lateralized effects on procedural learning. Our objective was to assess the effects of focal cerebellar lesions in procedural learning as measured by the SRTT. We studied 14 patients with single, unilateral vascular lesions in the territory of the posterior-inferior or superior cerebellar artery, who were compared with ten age- and sex-matched controls in a one-handed version of the SRTT. Patients with lesions at any other level of the brain or …

AdultMaleSerial reaction timemedicine.medical_specialtyCerebellumNeurologyCentral nervous systemProcedural memoryLesionCerebellar Diseasesmedicine.arteryReaction TimemedicineHumansLearningSuperior cerebellar arteryAgedAnalysis of Variancemedicine.diagnostic_testGeneral NeuroscienceReproducibility of ResultsMagnetic resonance imagingMiddle Agedmedicine.anatomical_structureCase-Control StudiesChronic DiseaseNerve DegenerationFemaleRadiologymedicine.symptomPsychologyNeurosciencePsychomotor PerformanceExperimental Brain Research
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Allelic variants of IL1R1gene associate with severe hand osteoarthritis

2010

Background In search for genes predisposing to osteoarthritis (OA), several genome wide scans have provided evidence for linkage on 2q. In this study we targeted a 470 kb region on 2q11.2 presenting the locus with most evidence for linkage to severe OA of distal interphalangeal joints (DIP) in our genome wide scan families. Methods We genotyped 32 single nucleotide polymorphisms (SNPs) in this 470 kb region comprising six genes belonging to the interleukin 1 superfamily and monitored for association with individual SNPs and SNP haplotypes among severe familial hand OA cases (material extended from our previous linkage study; n = 134), unrelated end-stage bilateral primary knee OA cases (n =…

AdultMalelcsh:Internal medicineLinkage disequilibriumLINKAGE DISEQUILIBRIUMlcsh:QH426-470Hand JointsNF-KAPPA-BSingle-nucleotide polymorphismLocus (genetics)KNEE OSTEOARTHRITISBiologyRADIOGRAPHIC SIGNSPolymorphism Single NucleotideSeverity of Illness IndexGenomeCHROMOSOME 2QINTERLEUKIN-1 RECEPTOR ANTAGONIST03 medical and health sciences0302 clinical medicineOsteoarthritisGeneticsHumansSNPGenetic Predisposition to DiseaseGenetics(clinical)Allelelcsh:RC31-1245GeneAllelesPOLYMORPHISMSGenetics (clinical)AgedHAPLOTYPE RECONSTRUCTION030304 developmental biologyReceptors Interleukin-1 Type I030203 arthritis & rheumatologyGenetics0303 health sciencesHaplotypeCLUSTERMiddle Aged314 Health sciences3. Good healthlcsh:GeneticsCase-Control StudiesDISC DEGENERATIONFemaleResearch ArticleBMC Medical Genetics
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Angiotensin converting enzyme has an inhibitory role in CGRP metabolism in human skin

2005

The neutral endopeptidase (NEP) is important for calcitonin gene related peptide (CGRP) degradation, while the role of angiotensin converting enzyme (ACE) remains unclear. By using dermal microdialysis we explored the effect of phosphoramidon (NEP blocker), captopril (ACE blocker) and a mixture of both drugs on the intensity of electrically-induced CGRP-mediated neurogenic flare. The results reveal that phosphoramidon elevated flare intensity, but that this was not further increased by adding captopril. In contrast, neurogenic flare was decreased when the drug mixture was applied in compared to NEP only. Electrically released CGRP levels could be measured directly in perfusates containing p…

AdultMalemedicine.medical_specialtyCaptoprilPhysiologyCalcitonin Gene-Related PeptideNeuropeptideAngiotensin-Converting Enzyme InhibitorsPeptidyl-Dipeptidase ACalcitonin gene-related peptideBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundEndocrinologyInternal medicineRetrograde DegenerationLaser-Doppler FlowmetrymedicineHumansNeprilysinSkinintegumentary systembiologyChemistryCatabolismfungiPhosphoramidonGlycopeptidesAngiotensin-converting enzymeCaptoprilMetabolismrespiratory systemVasodilationEndocrinologybiology.proteinFemalemedicine.drugPeptides
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Prevalence and associations of diabetic retinopathy in a large cohort of prediabetic subjects: The Gutenberg Health Study

2014

Abstract Aims To evaluate the prevalence of diabetic retinopathy/maculopathy (DR/DMac) and its associations with cardiovascular risk factors (CRF) in participants with prediabetes (PwPD) in a large European cohort within the population-based Gutenberg Health Study (GHS). Methods The study was based on a sub-cohort of the GHS (n=5,000, age: 35–74 y). Prediabetes was diagnosed according to HbA 1c levels (5.7–6.4%). DR/DMac was graded from fundus photographs. Blood samples and comprehensive questionnaires served for evaluation of laboratory results and CRF. Results The prevalence of prediabetes was 22.4%, and of DR/DMac 8.1%/0.2%, respectively. The majority of participants had mild DR (7.2%). …

AdultMalemedicine.medical_specialtyDiabetic CardiomyopathiesEndocrinology Diabetes and MetabolismPopulationSeverity of Illness IndexRetinaCohort StudiesPrediabetic StateMacular DegenerationEndocrinologyRisk FactorsGermanySurveys and QuestionnairesInternal medicineDiabetes mellitusPrevalenceInternal MedicineHumansMedicinePrediabeteseducationAgedGlycated Hemoglobineducation.field_of_studyDiabetic Retinopathybusiness.industryDiabetic retinopathyMiddle Agedmedicine.diseaseCardiovascular DiseasesCohortPhysical therapyFemalebusinessDiabetic AngiopathiesCohort studyRetinopathyKidney diseaseJournal of Diabetes and its Complications
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