Search results for "dendritic spines"

showing 4 items of 24 documents

Synaptopodin regulates denervation-induced homeostatic synaptic plasticity

2013

Synaptopodin (SP) is a marker and essential component of the spine apparatus (SA), an enigmatic cellular organelle composed of stacked smooth endoplasmic reticulum that has been linked to synaptic plasticity. However, SP/SA-mediated synaptic plasticity remains incompletely understood. To study the role of SP/SA in homeostatic synaptic plasticity we here used denervation-induced synaptic scaling of mouse dentate granule cells as a model system. This form of plasticity is of considerable interest in the context of neurological diseases that are associated with the loss of neurons and subsequent denervation of connected brain regions. In entorhino-hippocampal slice cultures prepared from SP-de…

Patch-Clamp TechniquesDendritic SpinesGreen Fluorescent ProteinsNonsynaptic plasticityMice TransgenicTetrodotoxinBiologyIn Vitro TechniquesHippocampusReceptors N-Methyl-D-AspartateMiceHomeostatic plasticitySynaptic augmentationMetaplasticityAnimalsEntorhinal CortexHomeostasisPromoter Regions GeneticMultidisciplinarySynaptic scalingNeuronal PlasticityMicrofilament ProteinsRyanodine Receptor Calcium Release ChannelBiological SciencesDenervationSpine apparatusMice Inbred C57BLSynaptic fatigueSynaptic plasticityDentate GyrusSynapsesCalcium ChannelsNeuroscience
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Amyloid Beta-Mediated Changes in Synaptic Function and Spine Number of Neocortical Neurons Depend on NMDA Receptors

2021

Onset and progression of Alzheimer’s disease (AD) pathophysiology differs between brain regions. The neocortex, for example, is a brain region that is affected very early during AD. NMDA receptors (NMDARs) are involved in mediating amyloid beta (Aβ) toxicity. NMDAR expression, on the other hand, can be affected by Aβ. We tested whether the high vulnerability of neocortical neurons for Aβ-toxicity may result from specific NMDAR expression profiles or from a particular regulation of NMDAR expression by Aβ. Electrophysiological analyses suggested that pyramidal cells of 6-months-old wildtype mice express mostly GluN1/GluN2A NMDARs. While synaptic NMDAR-mediated currents are unaltered in 5xFAD …

QH301-705.5Amyloid betasomatosensory cortexDendritic SpinesMice TransgenicNeocortexSomatosensory systemReceptors N-Methyl-D-AspartateCatalysisArticleInorganic ChemistryAlzheimer Diseasemental disordersmedicineAnimalsBiology (General)Physical and Theoretical ChemistryQD1-999Molecular BiologySpectroscopyNeuronsNeocortexAmyloid beta-PeptidesbiologyPyramidal Cellsmusculoskeletal neural and ocular physiologyOrganic ChemistryWild typeAmyloid betaExcitatory Postsynaptic PotentialsGeneral Medicine5xFADPathophysiologyComputer Science ApplicationsNMDARChemistryElectrophysiologyProtein Subunitsmedicine.anatomical_structurenervous systemKnockout mouseSynapsesbiology.proteinNMDA receptorbiological phenomena cell phenomena and immunityNeuroscienceAlzheimer’s diseasepsychological phenomena and processesInternational Journal of Molecular Sciences
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Differential evolution of PSA-NCAM expression during aging of the rat telencephalon

2007

Changes in the ability of neuronal networks to undergo structural remodeling may be involved in the age-associated cognitive decline. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) declines dramatically during postnatal development, but persists in several regions of the young-adult rat telencephalon, where it participates, through its anti-adhesive properties, in neuronal structural plasticity. However, PSA-NCAM expression during aging has only been studied in the dentate gyrus and the piriform cortex layer II, where it is strongly downregulated in adult (middle-aged) individuals. Using immunohistochemistry, we have observed that in most of the telencephalic areas …

TelencephalonAgingDendritic SpinesDown-RegulationHippocampusCell CountNeural Cell Adhesion Molecule L1BiologyPiriform cortexCell AdhesionLimbic SystemmedicineNeuropilAnimalsCognitive declineCerebral CortexNeuronsNeuronal PlasticityNeocortexGeneral NeuroscienceDentate gyrusAmygdalaImmunohistochemistryRats Inbred F344RatsDisease Models Animalmedicine.anatomical_structurenervous systemSialic AcidsFemaleNeural cell adhesion moleculeNeurology (clinical)Geriatrics and GerontologyNeuroscienceBiomarkersDevelopmental BiologyStratum lucidumNeurobiology of Aging
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Induction of neuronal plasticity during adulthood. Role of cortical interneurons and plasticity-related molecules.

2019

La complejidad del sistema nervioso de los vertebrados representa un reto a la hora de conocer su funcionamiento. Hace ya más de cien años que se postuló la neurona como unidad básica de procesamiento, generación y transmisión de la información a través de este sistema, y, con el paso de los años, ha evolucionado el conocimiento sobre esta unidad funcional. La idea clásica del sistema nervioso central (SNC) adulto es la de un tejido asombrosamente interconectado y complejo, pero prácticamente inmutable y muy poco adaptable a los cambios. No obstante, esta visión se ha ido desplazando, formándose una nueva idea del SNC adulto que, aunque de manera más limitada que durante el desarrollo, tamb…

perineuronal netsinterneuronsfluoxetineUNESCO::CIENCIAS DE LA VIDAneuronal plasticitydendritic spineschondroitinaseABC:CIENCIAS DE LA VIDA [UNESCO]dark exposure
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