Search results for "digestive system"

showing 10 items of 1747 documents

SGLT2 Inhibitors as the Most Promising Influencers on the Outcome of Non-Alcoholic Fatty Liver Disease

2022

Non-alcoholic fatty liver disease (NAFLD), the most frequent liver disease in the Western world, is a common hepatic manifestation of metabolic syndrome (MetS). A specific cure has not yet been identified, and its treatment is currently based on risk factor therapy. Given that the initial accumulation of triglycerides in the liver parenchyma, in the presence of inflammatory processes, mitochondrial dysfunction, lipotoxicity, glucotoxicity, and oxidative stress, can evolve into non-alcoholic steatohepatitis (NASH). The main goal is to identify the factors contributing to this evolution because, once established, untreated NASH can progress through fibrosis to cirrhosis and, ultimately, be co…

Carcinoma HepatocellularSettore MED/09 - Medicina Internatype 2 diabetes mellituSGLT2metabolic syndromeCatalysisInorganic ChemistryNon-alcoholic Fatty Liver DiseaseNAFLDAnimalsHumansHypoglycemic AgentsPhysical and Theoretical ChemistrySodium-Glucose Transporter 2 InhibitorsMolecular BiologySpectroscopyDipeptidyl-Peptidase IV InhibitorsHypoglycemic AgentLiver NeoplasmsOrganic Chemistrynutritional and metabolic diseasesGeneral Medicinedigestive system diseasesComputer Science ApplicationsGlucoseLiverDiabetes Mellitus Type 2HumanInternational Journal of Molecular Sciences
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Expression of WISPs and of their novel alternative variants in human hepatocellular carcinoma cells

2005

WISPs (Wnt-induced secreted proteins) are members of the CCN (CTGF/Cyr61/Nov) family involved in fibrotic disorders and tumorigenesis. They have a typical structure composed of four conserved cysteine-rich modular domains, but variants of CCN members lacking one or more modules, generated by alternative splicing or gene mutations, have been described in various pathological conditions. WISP genes were first described as downstream targets of the Wnt signaling pathway, which is frequently altered in human hepatocellular carcinoma (HCC). In the present study, WISP mRNA expression was analyzed by RT-PCR in four human HCC cell lines (HepG2, HuH-6, HuH-7, HA22T/VGH). Our results show for the fir…

Carcinoma HepatocellularWISPHepatocellular carcinomaApoptosisGene mutationBiologymedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyCCN Intercellular Signaling ProteinsWntalternative splicingHistory and Philosophy of ScienceCell Line TumorProto-Oncogene ProteinsCCN Intercellular Signaling ProteinsmedicineHumansRNA MessengerGeneDNA PrimersOncogene ProteinsGeneticsCCNModels GeneticReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceLiver NeoplasmsAlternative splicingIntracellular Signaling Peptides and ProteinsWnt signaling pathwaydigestive system diseasesNeoplasm ProteinsInsulin-Like Growth Factor Binding ProteinsRepressor ProteinsCTGFCYR61Cancer researchIntercellular Signaling Peptides and ProteinsRNACarcinogenesisWISPWntTranscription Factors
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Loss of response of carnitine palmitoyltransferase I to okadaic acid in transformed hepatic cells

1998

The specific activity of carnitine palmitoyltransferase I (CPT-I) was similar in mitochondria isolated from rat Fao and human HepG2 hepatoma cells and from rat hepatocytes, but almost twofold higher in permeabilized hepatoma cells than in permeabilized hepatocytes. Short-term exposure to okadaic acid induced a ca. 80% stimulation of CPT-I in hepatocytes, whereas no significant response of the enzyme from hepatoma cells was evident. Thus, the high CPT-I activity displayed by hepatoma cells may be reached by hepatocytes upon challenge to okadaic acid. Reconstitution experiments with purified mitochondrial and cytoskeletal fractions showed that the cytoskeleton of hepatocytes produced a more r…

Carcinoma Hepatocellularendocrine system diseasesMitochondria LiverMitochondrionBiologyBiochemistrychemistry.chemical_compoundLiver Neoplasms ExperimentalOkadaic AcidTumor Cells CulturedmedicineAnimalsHumansheterocyclic compoundsCarnitine O-palmitoyltransferaseCytoskeletonneoplasmsCell Line TransformedPharmacologyCarnitine O-PalmitoyltransferaseLiver NeoplasmsOkadaic aciddigestive system diseasesMitochondriaRatsCell biologyKineticsmedicine.anatomical_structureBiochemistrychemistryCell cultureHepatocyteHepatic stellate cellCarnitine palmitoyltransferase IBiochemical Pharmacology
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Nonalcoholic Fatty Liver Disease, Cardiovascular Risk, and Carotid Inflammation.

2015

Nonalcoholic fatty liver disease (NAFLD) is defined by excessive triglycerides (TGs) accumulation in the liver (>5% of hepatocytes histologically) in the absence of alcohol excess. The NAFLD ranges from simple steatosis to steatohepatitis and cirrhosis. The NAFLD and nonalcoholic steatohepatitis (NASH) are now the number one cause of liver disease in Western countries. The prevalence of NAFLD is increasing but is underreported, and the epidemiology and demographic characteristics vary worldwide. The prevalence is increasing because of the rising occurrence of obesity and type 2 diabetes (T2DM); in fact, NAFLD is considered as the hepatic manifestation of metabolic syndrome (MetS). Nonalcoho…

Carotid Artery Diseasesmedicine.medical_specialtyVery low-density lipoproteinLipoproteins//purl.org/becyt/ford/3.3 [https]Risk FactorsInternal medicineNonalcoholic fatty liver diseasemedicinePrevalenceHumansInflammationAdiponectinbusiness.industryFatty liverNon Alcoholic Fatty Liver Diseasenutritional and metabolic diseasesmedicine.diseasedigestive system diseasesFatty LiverOxidative StressEndocrinologyCardiovascular DiseasesDisease Progression//purl.org/becyt/ford/3 [https]Hepatic lipaseMetabolic syndromeSteatohepatitisInsulin ResistanceCardiology and Cardiovascular MedicinebusinessBiomarkersLipoproteinAngiology
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Mammalian intestinal alkaline phosphatase acts as highly active exopolyphosphatase.

2001

Recent results revealed that inorganic polyphosphates (polyP), being energy-rich linear polymers of orthophosphate residues known from bacteria and yeast, also exist in higher eukaryotes. However, the enzymatic basis of their metabolism especially in mammalian cells is still uncertain. Here we demonstrate for the first time that alkaline phosphatase from calf intestine (CIAP) is able to cleave polyP molecules up to a chain length of about 800. The enzyme acts as an exopolyphosphatase degrading polyP in a processive manner. The pH optimum is in the alkaline range. Divalent cations are not required for catalytic activity but inhibit the degradation of polyP. The rate of hydrolysis of short-ch…

Cations DivalentBiophysicsBiologymedicine.disease_causeBiochemistryDivalentSubstrate SpecificityNitrophenolschemistry.chemical_compoundOrganophosphorus CompoundsStructural BiologyNickelPolyphosphatesmedicineAnimalsMagnesiumneoplasmsMolecular BiologyEscherichia coliEdetic AcidExopolyphosphatasechemistry.chemical_classificationPolyphosphateSubstrate (chemistry)MetabolismCobaltHydrogen-Ion ConcentrationAlkaline PhosphataseMolecular biologydigestive system diseasesAcid Anhydride HydrolasesIntestinesKineticsEnzymechemistryBiochemistryAlkaline phosphataseCattleBiochimica et biophysica acta
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Gluten Free Diet for the Management of Non Celiac Diseases: The Two Sides of the Coin

2020

A lifelong adherence to a gluten-free (GF) diet is currently the only treatment for Celiac disease (CD), an autoimmune disorder that arises after gluten ingestion in individuals who are genetically predisposed. The gluten intake exerts toxic effects through several pathways involving gut barrier integrity, intestinal microbiota composition and immune system stimulation. However, despite the great benefit of GF diet for CD patients, its use has been debated. Indeed, individuals who adopt this diet regime may be at risk of nutrient deficiencies. Emerging evidence supports a beneficial effect of a GF diet also for other pathological conditions, including gluten-related disorders (GRD) often as…

Celiac diseasesLeadership and Managementlcsh:MedicineHealth InformaticsReviewDisease03 medical and health sciences0302 clinical medicineImmune systemHealth Information Managementgluten-free dietSettore BIO/10 - BiochimicaDiabetes mellitusMedicineIngestionIrritable bowel syndrome030304 developmental biologychemistry.chemical_classification0303 health sciencesbusiness.industryHealth Policylcsh:Rnutritional and metabolic diseasesnon celiac diseasemedicine.diseaseGlutendigestive system diseaseschemistryglutenImmunology030211 gastroenterology & hepatologyGluten freebusinessHealthcare
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Altered morphological and electrophysiological properties of Cajal-Retzius cells in cerebral cortex of embryonic Presenilin-1 knockout mice

2004

Mutations of Presenilin-1 are the major cause of familial Alzheimer's disease. Presenilin-1 knockout (PS1-/-) mice develop severe cortical dysplasia related to human type 2 lissencephaly. This overmigration syndrome has been attributed to the premature loss of Cajal-Retzius cells (CRcs), pioneer neurons required for the termination of radial neuronal migration. To elucidate the potential cellular mechanisms responsible for this premature neuronal loss, we investigated the morphological and electrophysiological properties of visually identified CRcs of wild-type (WT) and PS1-/- mouse brains at embryonic day 16.5. The density of CRcs was substantially reduced in the cerebral cortex of PS1-/-.…

Cell Adhesion Molecules NeuronalNerve Tissue ProteinsBiologyBicucullineMembrane PotentialsGABA AntagonistsMicemental disordersExcitatory Amino Acid AgonistsPresenilin-1medicineAnimalsneoplasms6-Cyano-7-nitroquinoxaline-23-dioneCerebral CortexMice KnockoutNeuronsMembrane potentialExtracellular Matrix ProteinsGABAA receptorStem CellsGeneral NeuroscienceSerine EndopeptidasesExcitatory Postsynaptic PotentialsMembrane ProteinsCortical dysplasiaBicucullineEmbryo Mammalianmedicine.diseaseImmunohistochemistryElectric Stimulationdigestive system diseasesnervous system diseasesCell biologyReelin ProteinElectrophysiologymedicine.anatomical_structure2-Amino-5-phosphonovaleratenervous systemCerebral cortexKnockout mouseExcitatory postsynaptic potentialExcitatory Amino Acid AntagonistsNeurosciencemedicine.drugEuropean Journal of Neuroscience
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Bacillus thuringiensis Cry1Ac Toxin-Binding and Pore-Forming Activity in Brush Border Membrane Vesicles Prepared from Anterior and Posterior Midgut R…

2008

ABSTRACT It is generally accepted that Bacillus thuringiensis Cry toxins insert into the apical membrane of the larval midgut after binding to specific receptors, and there is evidence that the distribution of binding molecules along the midgut is not uniform. By use of the voltage-sensitive dye DiSC 3 (5) and 125 I-labeled Cry1Ac, we have measured the effect of Cry1Ac in terms of permeabilization capacity and of binding parameters on brush border membrane vesicles (BBMV) prepared from the anterior and the posterior regions of the larval midgut from two insect species, Manduca sexta and Helicoverpa armigera . The permeabilizing activity was significantly higher with BBMV from the posterior …

Cell Membrane PermeabilityBrush bordermedia_common.quotation_subjectBacterial ProteinInsectApplied Microbiology and BiotechnologyIodine RadioisotopeIodine RadioisotopesHemolysin ProteinsEndotoxinBacterial ProteinsManducaBacillus thuringiensisInvertebrate MicrobiologyAnimalsmedia_commonBacillus thuringiensis ToxinsMicrovilliEcologybiologyAnimalVesiclefungiMidgutHemolysin ProteinApical membraneAlkaline Phosphatasebiology.organism_classificationEndotoxinsEnzyme ActivationLepidopteraBiochemistryManduca sextaLarvaPotassiumBiophysicsManducaDigestive SystemProtein BindingFood ScienceBiotechnologyApplied and Environmental Microbiology
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Polyphosphate, the physiological metabolic fuel for corneal cells: a potential biomaterial for ocular surface repair

2019

The regeneration of the epithelium, covering the avascular cornea, involves the processes of differentiation, proliferation and migration of cells originating from the corneal epithelial stem cells. We ask the question if these energy-consuming processes can be fueled by the physiological, inorganic polyphosphate (polyP), the main energy storage/donor molecule in the extracellular space. The ex vivo results reveal that addition of polyP, in the form of soluble Na-polyP, to the culture medium elicits a strong stimulatory effect on cell viability/growth and migration of corneal epithelial cells. Microscopic analyses of partially denuded cornea specimens show that in the presence of polyP, but…

Cell SurvivalCell Culture TechniquesBiomedical Engineering02 engineering and technology010402 general chemistry01 natural sciencesCorneaCorneal limbusCell MovementPolyphosphatesCorneaotorhinolaryngologic diseasesmedicineHumansRegenerationGeneral Materials ScienceViability assayCells CulturedCell ProliferationCorneal epitheliumTissue ScaffoldsChemistryRegeneration (biology)Mucin-1Epithelial Cells021001 nanoscience & nanotechnologyeye diseasesdigestive system diseasesEpitheliumCulture Media0104 chemical sciencesCell biologysurgical procedures operativemedicine.anatomical_structureSolubilityCell cultureCalciumsense organsStem cell0210 nano-technologyBiomaterials Science
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Utilization of metabolic energy in treatment of ocular surface disorders: polyphosphate as an energy source for corneal epithelial cell proliferation.

2019

Impaired regeneration of the corneal epithelium, as found in many ocular surface diseases, is a major clinical problem in ophthalmology. We hypothesized that corneal epithelial regeneration can be promoted by the physiological, energy-delivering as well as “morphogenetically active” polymer, inorganic polyphosphate (polyP). Corneal limbal explants (diameter, 4 mm) were cultivated on collagen-coated well plates in the absence or presence of polyP (chain length, ∼40 Pi units; 50 μg ml−1) or human platelet lysate (hp-lysate; 5% v/v). Cell outgrowth and differentiation were analyzed after staining with DRAQ5 (nuclei) and rhodamine phalloidin (cytoskeleton), as well as by environmental scanning …

Cell growthChemistryGeneral Chemical EngineeringRegeneration (biology)CellCell migration02 engineering and technologyGeneral Chemistry010402 general chemistry021001 nanoscience & nanotechnologyMast cell01 natural sciencesCell junctiondigestive system diseases0104 chemical sciencesCell biologymedicine.anatomical_structuremedicine0210 nano-technologyEnergy sourceCorneal epitheliumRSC advances
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