Search results for "dose–response"

showing 10 items of 85 documents

Effects of CGS 10746B on hyperactivity and place preference induced by morphine

2001

The effects of CGS 10746B, a dopamine release inhibitor, on spontaneous locomotor activity, morphine-induced hyperactivity, acquisition of conditioned place paradigm and morphine-induced conditioned place preference (CPP) was evaluated in male mice. In experiment 1, animals treated with CGS 10746B (0.5, 1, 2, 4, 8, 16, 24 and 32 mg/kg), morphine (40 mg/kg) or morphine (40 mg/kg) plus CGS 10746B (0.5, 1, 2, 4, 8, 16, 24 and 32 mg/kg) were placed in an actimeter during a period of 90 min. In experiment 2, animals treated with CGS 10746B (0.5, 1, 3 and 10 mg/kg), morphine (40 mg/kg) or morphine (40 mg/kg) plus CGS 10746B (0.5, 1, 3 and 10 mg/kg) were conditioned following a procedure unbiased …

Malemedicine.medical_specialtyThiazepinesDopamineConditioning ClassicalMale miceMice Inbred StrainsSocial EnvironmentChoice BehaviorMiceBehavioral Neurosciencechemistry.chemical_compoundDopamineInternal medicinemedicineAnimalsDrug InteractionsNeurotransmitterDose-Response Relationship DrugMorphineChemistryAssociation LearningBrainConditioned place preferenceDose–response relationshipEndocrinologyMorphineCatecholamineConditioningNeuroscienceAntipsychotic Agentsmedicine.drugBehavioural Brain Research
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LACTATE, NOT GLUCOSE, UP-REGULATES MITOCHONDRIAL OXYGEN CONSUMPTION BOTHIN SHAM AND LATERAL FLUID PERCUSSED RAT BRAINS

2006

OBJECTIVE: Failure of energy metabolism after traumatic brain injury may be a major factor limiting outcome. Although glucose is the primary metabolic substrate in the healthy brain, the well documented surge in tissue lactate after traumatic brain injury suggests that lactate may provide an energy need that cannot be met by glucose. We hypothesized, therefore, that administration of lactate or the combination of lactate and supraphysiological oxygen may improve mitochondrial oxidative respiration in the brain after rat fluid percussion injury. We measured oxygen consumption (VO2) to determine what effects glucose, lactate, oxygen, and the combination of lactate and oxygen have on mitochond…

Malemedicine.medical_specialtyTraumatic brain injuryCell RespirationOxidative phosphorylationMitochondrionRats Sprague-Dawleychemistry.chemical_compoundOxygen ConsumptionFraction of inspired oxygenInternal medicineRespirationmedicineAnimalsLactic AcidDose-Response Relationship Drugbusiness.industryBrainmedicine.diseaseMitochondriaRatsUp-RegulationCartesian diverLactic acidOxygenDose–response relationshipGlucoseEndocrinologychemistryBrain InjuriesAnesthesiaSurgeryNeurology (clinical)businessNeurosurgery
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Beta-adrenergic blocking activity and haemodynamic effects in man of K� 1313, a new beta-adrenergic antagonist

1971

The beta-adrenergic blocking activity and haemodynamic effects of o-[2-hydroxy-3-(isopropylamino)-propoxy]-benzonitril (Ko 1313) have been studied in 22 patients. Antagonism of isoproterenol-induced tachycardia was used as a measure of the beta-adrenergic blocking activity. Ko 1313 1.0 mg had its maximum beta-adrenoceptor blocking effect 5–30 min after intravenous injection. Ko 1313 10.0 mg produced maximum betablockade 1–4 h after oral administration. 1.0 mg Ko 1313 injected intravenously had approximately the same beta-adrenergic blocking effect as 1.0 mg propranolol also given intravenously. After intravenous administration Ko 1313 was 3–4 times as potent as the same dose given orally. A…

AdultTachycardiamedicine.medical_specialtyCardiac outputAdolescentAdrenergic receptorCardiac VolumeAdrenergic beta-AntagonistsAdministration OralBlood Pressure1-PropanolPropranololPharmacologyElectrocardiographyHeart RateOral administrationInternal medicineNitrilesHeart ratemedicineHumansPharmacology (medical)Cardiac OutputPharmacologyBeta-adrenergic blocking agentPropylaminesbusiness.industryHemodynamicsIsoproterenolGeneral MedicineMiddle AgedAmino AlcoholsPropranololDose–response relationshipEndocrinologyInjections IntravenousSympatholyticsVascular Resistancemedicine.symptombusinessmedicine.drugEuropean Journal of Clinical Pharmacology
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Food intake in lean and obese mice after peripheral administration of glucagon-like peptide 2

2012

We investigated the potential anorectic action of peripherally administered glucagon-like peptide 2 (GLP2) in lean and diet-induced obese (DIO) mice. Mice, fasted for 16 h, were injected i.p. with native GLP2 or [Gly2]GLP2, stable analog of GLP2, before or after GLP2 (3–33), a GLP2 receptor (GLP2R) antagonist, or exendin (9–39), a GLP1R antagonist. Food intake was measured at intervals 1, 2, 4, 8, and 24 h postinjection. In addition, we tested in lean mice the influence of [Gly2]GLP2 on gastric emptying and the effects of GLP1 alone or in combination with [Gly2]GLP2 on food intake. [Gly2]GLP2 dose dependently and significantly inhibited food intake in lean and DIO mice. The reduction of foo…

Malemedicine.medical_specialtyTime FactorsEndocrinology Diabetes and MetabolismPeptideDiet High-FatSettore BIO/09 - FisiologiaGlucagon-Like Peptide-1 ReceptorEatingMiceEndocrinologyGLP-2 food intake diet induced obesityGlucagon-Like Peptide 1Internal medicineAppetite DepressantsGlucagon-Like Peptide 2Receptors GlucagonmedicineAnimalsObesityReceptorchemistry.chemical_classificationDose-Response Relationship DrugGastric emptyingAntagonistReceptor Cross-TalkGlucagon-like peptide-2Peptide FragmentsMice Inbred C57BLDose–response relationshipEndocrinologyGastric EmptyingchemistryGlucagon-Like Peptide-2 ReceptorAnorecticGlucagon-Like Peptide-2 Receptor
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Reinstatement of Morphine-Induced Conditioned Place Preference in Mice by Priming Injections

2004

To construct a model of relapse of drug abuse in mice, the induction, we evaluated the extinction and reinstatement of morphine-induced place preference. In Experiment 1, we examined the effects of morphine (0, 2, 3, 5, 10, 20 and 40 mg/kg) in the conditioned place preference (CPP) paradigm. Mice showed CPP with 5, 10, 20 and 40 mg/kg. In Experiment 2, we evaluated the effects of two different extinction procedures. After conditioning with 40 mg/kg of morphine, the mice underwent daily extinction sessions of 60 or 15 min of duration. CPP was extinguished after seven and nine sessions, respectively. In Experiment 3, we tested the reinstating effects of several priming doses of morphine. Mice…

MaleNarcoticsReinforcement SchedulePharmacologyArticleExtinction Psychologicallcsh:RC321-571MiceRewardmedicineAnimalslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryDose-Response Relationship DrugMorphineExtinction (psychology)Conditioned place preferenceDose–response relationshipNeurologyAnesthesiaMorphineConditioning OperantConditioningNeurology (clinical)PsychologyReinforcement PsychologyPriming (psychology)Injections Intraperitonealmedicine.drugNeural Plasticity
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Developmental Changes and Daily Rhythm in Melatonin-Induced Inhibition of 3′,5′-Cyclic AMP Accumulation in the Rat Pituitary

1990

Melatonin's transduction mechanisms were investigated using in vitro cultured anterior hemipituitaries. Melatonin inhibited cAMP and 3',5'-cyclic GMP accumulation in neonatal rat anterior pituitary stimulated with LHRH. Maximal inhibitory effect was reached within 25 min and persisted for at least 20 min. Inhibition of cAMP accumulation is specific for melatonin because its analogs N-acetylserotonin and 5-methoxytryptamine are 1000 times less potent. Melatonin effect is age- and time-dependent. Marked inhibition was observed in 5-, 10-, and 14-day-old rats but not in 29-day-old ones. Melatonin was significantly more potent when applied at the end of the light period as compared with the fir…

Agingendocrine systemmedicine.medical_specialtyPituitary glandTime FactorsGonadotropin-releasing hormoneIn Vitro TechniquesBiologyGonadotropin-Releasing HormoneMelatoninEndocrinologyAnterior pituitaryInternal medicineCyclic AMPmedicineAnimalsCircadian rhythmCyclic GMPMelatoninDose-Response Relationship DrugRats Inbred StrainsCircadian RhythmRatsDose–response relationshipEndocrinologymedicine.anatomical_structurePituitary GlandSecond messenger systemhormones hormone substitutes and hormone antagonistsEndocrine glandmedicine.drugEndocrinology
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Sevoflurane affects neurogenesis after forebrain ischemia in rats.

2007

BACKGROUND: The effect of sevoflurane on the neuroregenerative potential after neuronal injury is unclear. We investigated the effect of low and high concentrations of sevoflurane on endogenous neurogenesis after cerebral ischemia. METHODS: Anesthetized and ventilated rats were randomized to four different treatment groups. Groups 1 and 2: 1.4% sevoflurane; Groups 3 and 4: 2.8% sevoflurane. In Groups 1 and 3, no cerebral ischemia was induced (sham-operated). In Groups 2 and 4, 10 min of forebrain ischemia was induced by bilateral carotid artery occlusion plus hemorrhagic hypotension. Physiological variables were maintained constant. Bromodeoxyuridine was given as a marker of neurogenesis. A…

MaleMethyl EthersIschemiaH&E stainHippocampusHippocampusSevofluraneBrain IschemiaRats Sprague-Dawleychemistry.chemical_compoundRandom AllocationSevofluraneProsencephalonmedicineAnimalsCell ProliferationNeuronsDose-Response Relationship Drugbusiness.industryDentate gyrusNeurogenesismedicine.diseaseNerve RegenerationRatsDose–response relationshipDisease Models AnimalAnesthesiology and Pain MedicinechemistryAnesthesiaAnesthetics InhalationDentate GyrusbusinessBromodeoxyuridinemedicine.drugAnesthesia and analgesia
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Characteristics of histamine tachyphylaxis in rat uterine smooth muscle.

2002

Objective and design: To study both the desensitisation induced by short-term exposure to histamine and the mechanism responsible in the isolated rat uterus.¶Material: Precontracted isolated uterus (37 mM KCl) from oestrogenised Wistar rats were used.¶Treatment: Repetitive responses to histamine (10–6, 10–5, 10–4, 10–3 M), dimaprit and clonidine (10–4 M) were tested at 15, 30, 45 and 105 min., with their modifications by (5 mg/ kg, 24 h before sacrifice) reserpine, 10–7 M propranolol, 10–8 M atropine, and 10–6 M indomethacin. Dose-response curves for adrenaline were carried out as standard protocol.¶Methods: In vitro techniques (de Jalon's solution, 31°C, carbogen, isotonic registration, re…

medicine.medical_specialtyEpinephrineMuscle RelaxationImmunologyPropranololTachyphylaxisIn Vitro TechniquesClonidineHistamine Agonistschemistry.chemical_compoundUterine ContractionDimapritInternal medicinemedicineCyclic AMPAnimalsIsotonic ContractionRats WistarTachyphylaxisPharmacologyDose-Response Relationship Drugbusiness.industryUterusEstrogensReserpineDimapritClonidineRatsAtropineDose–response relationshipEndocrinologychemistryFemalebusinessAdrenergic alpha-AgonistsHistaminemedicine.drugHistamineInflammation research : official journal of the European Histamine Research Society ... [et al.]
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Investigations of the sensory blockade effect of perineurally injected ethanol on the tail nerve of the mouse.

1976

The effect of an alcohol block on the conduction of sensory stimuli in the tail nerve of the mouse was investigated using the perineural injection of solutions of ethanol (35, 40 and 45%). One hundred and fifty white mice of either sex were given 2 X 0.03 ml of the relevant alcohol solution into both sides of the tail. Before and after the injections repeated sensory conduction measurements were made using the rat tail method. Using 35% ethanol a temporary block of pain conduction could be achieved in both sexes. By increasing the concentration to 40 or 45%, a prolongation of the blocking effect and an increase in the accompanying increase of the pain threshold was observed in some animals.…

MaleTailTime Factorsmedicine.medical_treatmentNeural ConductionSensory systemAlcoholInjectionschemistry.chemical_compoundMiceThreshold of painParalysismedicineReaction TimeAnimalsParalysisNeural ConductionEthanolDose-Response Relationship DrugEthanolbusiness.industryNerve BlockDose–response relationshipAnesthesiology and Pain MedicinechemistryAnesthesiaNerve blockFemalemedicine.symptombusinessBritish journal of anaesthesia
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Cytokine profile of breast cell lines after different radiation doses

2017

Purpose: Ionizing radiation (IR) treatment activates inflammatory processes causing the release of a great amount of molecules able to affect the cell survival. The aim of this study was to analyze the cytokine signature of conditioned medium produced by non-tumorigenic mammary epithelial cell line MCF10A, as well as MCF7 and MDA-MB-231 breast cancer cell lines, after single high doses of IR in order to understand their role in high radiation response. Materials and methods: We performed a cytokine profile of irradiated conditioned media of MCF10A, MCF7 and MDA-MB-231 cell lines treated with 9 or 23 Gy, by Luminex and ELISA analyses. Results: Overall, our results show that both 9 Gy and 23 …

0301 basic medicineIonizing radiationRadiology Nuclear Medicine and ImagingCell SurvivalCytokine profileBreast NeoplasmsInflammationRadiationRadiation ToleranceIonizing radiation03 medical and health sciences0302 clinical medicineBreast cancerbreast cancerCell Line TumormedicinecytokineHumansskin and connective tissue diseasesCell survivalRadiological and Ultrasound TechnologyChemistrybreast cancer cytokines inflammation Ionizing radiation Breast Neoplasms Cell Line Tumor Cell Survival Culture Media Conditioned Dose-Response Relationship Radiation Humans Phenotype Radiation ToleranceDose-Response Relationship Radiationmedicine.diseasecytokinesDose–response relationship030104 developmental biologyPhenotypeCell cultureinflammation030220 oncology & carcinogenesisCulture Media ConditionedImmunologyCancer researchmedicine.symptomBreast NeoplasmHuman
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