Search results for "drug interactions"

showing 10 items of 229 documents

Mechanism-based predictions of interactions.

1994

Abstract Exposure to more than one toxic compound is common in real life. The resulting toxic effects are often more than the simple sum of the effects of the individual compounds. It is unlikely that it will ever be possible to test all combinations. It is therefore highly desirable to improve or develop means for reasonably approximating predictions of interactions. In order to be valid and extrapolatable, these predictions are most promising if they are mechanism-based. Examples will be given for possibilities of mechanism-based predictions of interactions which exceed trivialities of simple increases by enzyme induction of enzymatic rates of a given biotransformation pathway leading to …

MaleSalmonella typhimuriumEndogenous FactorsHealth Toxicology and MutagenesisMetaboliteMechanism basedRats sprague dawleyXenobioticsRats Sprague-Dawleychemistry.chemical_compoundStilbenesBenzo(a)pyreneAnimalsIn real lifeDrug InteractionsPhosphorylationEpoxide HydrolasesMutagenicity TestsMechanism (biology)Public Health Environmental and Occupational HealthRatschemistryBiochemistryEnzyme InductionMicrosomes LiverBiochemical engineeringXenobioticMutagenicity TestResearch ArticleEnvironmental Health Perspectives
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Adolescent pre-exposure to ethanol or MDMA prolongs the conditioned rewarding effects of MDMA

2011

Adolescents often take ethanol (EtOH) in combination with MDMA (3,4-methylenedioxymethylamphetamine). In the present work we studied the effect of repeated intermittent adolescent pre-exposure to both drugs on the behavioral and neurochemical effects of MDMA in mice. Sixteen days after pre-treatment, the rewarding and reinstating effects of MDMA in the conditioned place preference (CPP) paradigm were evaluated, along with the levels of biogenic amines, basal motor activity and corticosterone response to different challenges. Pre-exposure to EtOH, MDMA or EtOH+MDMA did not affect the CPP induced by 10mg/kg of MDMA. However, adolescent exposure to EtOH or MDMA increased the duration of the co…

MaleSerotoninmedicine.medical_specialtyDopamineN-Methyl-34-methylenedioxyamphetaminePoison controlExperimental and Cognitive PsychologyStriatumMotor ActivityChoice BehaviorHippocampusDrug Administration ScheduleExtinction PsychologicalMiceBehavioral Neurosciencechemistry.chemical_compoundNeurochemicalRewardCorticosteroneInternal medicineConditioning Psychologicalmental disordersAnimals Outbred StrainsmedicineAnimalsDrug InteractionsCerebral CortexEthanolIllicit DrugsMDMAExtinction (psychology)Hydroxyindoleacetic AcidCorpus StriatumConditioned place preferenceMonoamine neurotransmitterEndocrinologychemistryAnesthesia34-Dihydroxyphenylacetic AcidCorticosteronePsychologypsychological phenomena and processesmedicine.drugPhysiology & Behavior
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Modification of depressant and disinhibitory action of flurazepam during short term treatment in the rat

1972

Employing a fixed-interval schedule of reinforcement (temporal discrimination), alternated punished (fixed-ratio) and unpunished (variable-ratio) schedules of reinforcement, a Conditioned Avoidance Response, and studying its interaction with Pentobarbital on general anaesthesia, it has been shown that flurazepam hydrochloride after a single treatment induces very intense depressant effects and slight disinhibitory effects. Short term treatment at longer than daily intervals reduces the depressant effect and unmasks the disinhibitory effect. The phenomenon is probably caused by selective tolerance concerning the depressant action. The results are discussed from the point of view of the signi…

MaleShort term treatmentPentobarbitalReinforcement ScheduleTime FactorsFlurazepammedicine.drug_classAvoidance responsePharmacologyFlurazepam HydrochlorideAvoidance LearningEthylaminesmedicineAnimalsHypnotics and SedativesDrug InteractionsReinforcementPentobarbitalPharmacologyDrug ToleranceFluorineBenzazepinesRatsAction (philosophy)DepressantPsychologymedicine.drugPsychopharmacologia
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Effects of the flavonol quercetin on the bioavailability of simvastatin in pigs

2009

The influence of the dietary flavonol quercetin on the pharmacokinetics of the HMG-CoA reductase inhibitor simvastatin was investigated in pigs. Simvastatin (0.25mg/kg body weight) was orally administered to six pigs either without or with quercetin (10mg/kg). In addition, simvastatin was administered to three pigs that had received a diet supplemented with the flavonol over a period of 1 week. Daily quercetin intake was 10mg/kg in these animals. Co-ingestion of quercetin with the statin did not alter area under the concentration time curve (AUC(0-->infinity)), time to achieve maximum plasma concentration (t(max)) or half-life (t(1/2)) of simvastatin. However, there was a trend towards a re…

MaleSimvastatinStatinFlavonolsSwinemedicine.drug_classBiological AvailabilityPharmaceutical SciencePharmacologyFood-Drug Interactionschemistry.chemical_compoundPharmacokineticsBlood plasmapolycyclic compoundsmedicineAnimalsIngestionheterocyclic compoundscardiovascular diseasesCross-Over StudiesbiologyChemistrynutritional and metabolic diseasesBioavailabilitySimvastatinHMG-CoA reductasebiology.proteinQuercetinlipids (amino acids peptides and proteins)Quercetinmedicine.drugEuropean Journal of Pharmaceutical Sciences
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Mouse photoreceptor synaptic ribbons lose and regain material in response to illumination changes

2004

Abstract Chemical synapses equipped with ribbons are tonically active, high-output synapses. The ribbons may play a role in the trafficking of synaptic vesicles. Recent findings in retinal rod cells of BALB/c mice indicate that ribbons are large and smooth in the dark phase, and, due to the formation and release of protrusions, small during the light phase. As a consequence of these changes, ribbons may traffick fewer vesicles in the light than in the dark phases. The aim of the present study was to find out whether the above ribbon changes in this mouse strain are strictly illumination-dependent and which signalling processes may be involved. Here, we show that ribbons form protrusions and…

MaleTime FactorsLightRibbon diagramDark AdaptationBiologyRibbon synapseModels BiologicalSynaptic vesicleRetinaPhotoreceptor cellCalcium ChlorideMiceOrgan Culture TechniquesmedicineAnimalsDrug InteractionsPhotoreceptor CellsCyclic GMPEgtazic AcidCalcimycinLightingChelating AgentsMelatoninSynaptic ribbonMice Inbred BALB CRetinaIonophoresGeneral NeurosciencefungiDarknessThionucleotidesCircadian Rhythmbody regionsMicroscopy Electronmedicine.anatomical_structurenervous systemSynapsesSynaptic plasticityBiophysicssense organsNeurosciencePhotic StimulationVisual phototransductionEuropean Journal of Neuroscience
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Inhibitory effects of N-valproyl-L-tryptophan on high potassium, low calcium and low magnesium-induced CA1 hippocampal epileptiform bursting activity…

2012

N-valproyl-l-tryptophan (VPA-Tryp), new antiepileptic drug, was tested on CA1 hippocampal epileptiform bursting activity obtained by increasing potassium and lowering calcium and magnesium concentrations in the fluid perfusing rat brain slices. Each slice was treated with a single concentration (0.2, 0.5, 1 or 2 mM) of Valproate (VPA) or VPA-Tryp. Both burst duration and interburst frequency during and after treatment were off-line compared with baseline values. For both parameters, the latency and the length of statistically significant response periods as well as the magnitude of drug-induced responses were calculated. VPA-Tryp evoked fewer and weaker early excitatory effects than VPA on …

Maleantiepileptic drug valproic acidPotassiumchemistry.chemical_elementAction PotentialsCalciumHippocampal formationPharmacologyIn Vitro TechniquesInhibitory postsynaptic potentialSettore BIO/09 - Fisiologiaamino-acidic derivativeBurstingmedicineReaction Timehippocampal epilepsyAnimalsDrug InteractionsMagnesiumRats WistarCA1 Region HippocampalBiological PsychiatryValproic AcidAnalysis of VarianceDose-Response Relationship DrugMagnesiumDipeptidesElectric StimulationRatsPsychiatry and Mental healthNeurologychemistrySettore CHIM/09 - Farmaceutico Tecnologico ApplicativoExcitatory postsynaptic potentialPotassiuminterictal burstslipids (amino acids peptides and proteins)AnticonvulsantsNeurology (clinical)medicine.drugJournal of neural transmission (Vienna, Austria : 1996)
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The effects of low doses of pregabalin on morphine analgesia in advanced cancer patients

2012

Abstract OBJECTIVES: The aim of this study was to evaluate the opioid response in patients receiving morphine and pregabalin, independently from the presumed pain mechanisms, in comparison with patients receiving morphine treatment only. METHODS: A multicenter prospective randomized controlled study was carried out in a sample of 70 advanced cancer patients with pain requiring strong opioids. Thirty-five patients (group MO) were randomized to receive sustained-release morphine using initial doses of 60 mg/day. Thirty-five patients (group MO-PR) were randomized to start the same morphine doses and pregabalin in increasing doses, starting with 25 mg/day up to 150 mg/day in one week. The follo…

Malecancer painAnalgesicPregabalinPregabalinPainSettore MED/42 - Igiene Generale E Applicatalaw.inventionDose-Response Relationshipadvanced cancer patientDrug TherapyQuality of lifeRandomized controlled triallawassessment toolsNeoplasmsmedicineHumansDrug Interactionslow doses pregabalinBrief Pain Inventorygamma-Aminobutyric AcidPain Measurementneuropathic painAnalgesicsDose-Response Relationship DrugMorphinebusiness.industryassessment tools; cancer pain; neuropathic pain; opioid response; Analgesics; Dose-Response Relationship Drug; Drug Interactions; Drug Therapy Combination; Female; Humans; Italy; Male; Morphine; Neoplasms; Pain; Pain Measurement; Palliative Care; Pregabalin; Treatment Outcome; gamma-Aminobutyric Acid; Anesthesiology and Pain Medicine; Neurology (clinical)Palliative CareAdvanced cancerTreatment OutcomeAnesthesiology and Pain MedicineItalyOpioidlow doses pregabalin; advanced cancer patients; epidemiologic studyopioid responseAnesthesiaCombinationepidemiologic studyMorphineDrug Therapy CombinationFemaleNeurology (clinical)Drugbusinessmedicine.drug
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Fos-related antigen 2 (Fra-2) memorizes photoperiod in the rat pineal gland

2004

As the physiological role of fos-related antigen-2 (Fra-2) is largely unknown and since the pineal plays an important role in the photoperiodic control of the body, we have tested the hypothesis that Fra-2 expression is photoperiod-dependent and may be involved in imprinting photoperiod on the pineal gland and the body as a whole. To this end, we have investigated Fra-2 mRNA expression and Fra-2 protein expression under various light/dark (LD) cycles. A clear nocturnal increase occurs for both monitored parameters under all photoperiodic conditions studied. The level of Fra-2 protein expression clearly depends on photoperiod, because the amount of protein at dark onset and during the night …

Maleendocrine systemmedicine.medical_specialtyTime FactorsPhotoperiodBlotting WesternDeiodinaseFos-Related Antigen-2CREBPineal GlandRats Sprague-DawleyPhenylephrinePineal glandOrgan Culture TechniquesAcetyltransferasesInternal medicineCyclic AMPmedicineAnimalsDrug InteractionsProtein phosphorylationRNA MessengerCircadian rhythmCyclic GMPHeat-Shock ProteinsbiologyReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceIsoproterenolAdrenergic beta-AgonistsAdaptation PhysiologicalPeptide FragmentsRatsDNA-Binding ProteinsEndocrinologymedicine.anatomical_structureGene Expression RegulationIodothyronine deiodinasebiology.proteinArylalkylamineFemaleAdrenergic alpha-Agonistshormones hormone substitutes and hormone antagonistsTranscription FactorsEndocrine glandNeuroscience
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Benzodiazepine receptor binding: the interactions of some non-benzodiazepine drugs with specific [3H] diazepam binding to rat brain synaptosomal memb…

1978

The interaction of several non-benzodiazepine drugs with [3H] diazepam binding to benzodiazepine receptors in rat brain synaptosomal membranes was investigated. Baclofen, benzoctamine, hydroxyzine, chlorpromazine, haloperidol, imipramine, and amitriptyline displace specific [3H] diazepam binding, but the concentrations needed are too high to explain pharmacological effects of these drugs by an interaction with benzodiazepine receptors. The most potent non-benzodiazepine drug for inhibiting specific [3H] diazepam binding was methaqualone (IC50 value of 150 micrometer). It is suggested that interactions with benzodiazepine receptors may account for the anxiolytic and anticonvulsive side effec…

Malemedicine.drug_classReceptors DrugPharmacologyIn Vitro TechniquesAnxiolyticBinding Competitivechemistry.chemical_compoundmedicineAnimalsDrug InteractionsBenzodiazepine receptor bindingPharmacologyBenzodiazepineDiazepam bindingDiazepamMembranesGABAA receptorBrainGeneral MedicineRatsBaclofenAnalepticchemistryBenzoctaminemedicine.drugSynaptosomesNaunyn-Schmiedeberg's archives of pharmacology
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Modulation by nitric oxide of spontaneous mechanical activity in rat proximal colon.

1999

Summary 1 In order to examine the role of nitric oxide (NO) in the tonic neural inhibition in rat proximal colon, the effects of Nω-nitro- l-arginine methyl ester (L-NAME) were studied on the spontaneous contractions of circular muscle (monitored as intraluminal pressure changes) and of longitudinal muscle (detected as isometric tension changes). 2 L-NAME (3 × 10−−6–3 × 10−−4 m) caused a concentration-dependent increase in the amplitude of circular contractions, without affecting those of longitudinal muscle. This effect was prevented by l-arginine (1–5 × 10−−3 m), but not d-arginine. 3 In the presence of tetrodotoxin (10−−6 m), which per se induced increase of the pressure waves, L-NAME (1…

Malemedicine.medical_specialtyColonIsometric exerciseNeurotransmissionIn Vitro TechniquesInhibitory postsynaptic potentialNitric OxideNitric oxideTonic (physiology)chemistry.chemical_compoundInternal medicineIsometric ContractionmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats WistarGuanethidinePharmacologyDose-Response Relationship DrugGeneral NeuroscienceMuscle SmoothRatsEndocrinologyNG-Nitroarginine Methyl EsterchemistryTetrodotoxinHexamethoniummedicine.drugMuscle ContractionJournal of autonomic pharmacology
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