Search results for "dyskinesia"

showing 10 items of 60 documents

PRRT2 mutations are the major cause of benign familial infantile seizures.

2012

Mutations in PRRT2 have been described in paroxysmal kinesigenic dyskinesia (PKD) and infantile convulsions with choreoathetosis (PKD with infantile seizures), and recently also in some families with benign familial infantile seizures (BFIS) alone. We analyzed PRRT2 in 49 families and three sporadic cases with BFIS only of Italian, German, Turkish, and Japanese origin and identified the previously described mutation c.649dupC in an unstable series of nine cytosines to occur in 39 of our families and one sporadic case (77% of index cases). Furthermore, three novel mutations were found in three other families, whereas 17% of our index cases did not show PRRT2 mutations, including a large fami…

AdultMaleAdolescentChoreoathetosisNerve Tissue ProteinsBiologymedicine.disease_causeSeizures FebrileInfantile seizures03 medical and health sciencesEpilepsy0302 clinical medicineGeneticsmedicineHumansChildGenetics (clinical)030304 developmental biologyAgedGenetics0303 health sciencesMutationBenign familial infantile epilepsyEpilepsyPRRT2; EpilepsyInfantMembrane ProteinsParoxysmal dyskinesiaMiddle Agedmedicine.diseaseMajor genePedigreeChild PreschoolMutationPRRT2medicine.symptomSpasms Infantile030217 neurology & neurosurgeryPRRT2Human mutation
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Development and validation of a method of cilia motility analysis for the early diagnosis of primary ciliary dyskinesia.

2011

Background: Primary ciliary dyskinesia (PCD) is a clinically uniform entity, but cilia motility and structure can vary between patients, making the diagnosis difficult. The aim of this study was to evaluate the sensitivity and specificity in diagnosing PCD of a system of high-resolution digital high-speed video analysis with proprietary software that we developed for analysis of ciliary motility (Desinsoft-Bio 200). The secondary aim was to correlate nasal ciliary activity with clinical and structural abnormalities in PCD. Material and methods: We analysed nasal mucociliary transport, cilia ultrastructure, nasal ciliary beat frequency and beat pattern studied by high-resolution digital high…

AdultMalePathologymedicine.medical_specialtyAdolescentDyneinCiliary dyskinesiaSensitivity and SpecificityYoung Adultotorhinolaryngologic diseasesmedicineHumansSinusitisChildPrimary ciliary dyskinesiaAgedBronchiectasisbusiness.industryKartagener SyndromeCiliumKartagener SyndromeInfantGeneral MedicineMiddle Agedmedicine.diseaseSitus inversusEarly DiagnosisChild PreschoolFemalebusinessActa otorrinolaringologica espanola
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Ultrastructural patterns of primary ciliar dyskinesia syndrome.

2005

Clinical presentation, ciliary ultrastructure, and nasal mucociliary transport by a radioisotopic technique were analyzed in 14 Kartagener syndrome patients. In this study the most common pattern was the absence of outer and inner dynein arms in 57% of cases. Also reported are 14% patients with short inner dynein arms. A total of 29% of the patients showed normal dynein arms. Mucociliary stasis was observed in 13 cases. Primary ciliary dyskinesia syndrome and Kartagener syndrome are clinically homogeneous and morphologically heterogeneous. The authors conclude that a typical clinical presentation with an altered mucociliary transport obtained by radioisotopic technique is diagnostic althoug…

AdultMalePathologymedicine.medical_specialtyAdolescentMucociliary clearanceBiologyPathology and Forensic MedicineDiagnosis DifferentialMicroscopy Electron TransmissionStructural BiologymedicineHumansCiliaChildPrimary ciliary dyskinesiaKartagener SyndromeKartagener SyndromeDyneinsInfantAnatomyMiddle Agedmedicine.diseaseSitus inversusNasal MucosaDyskinesiaHomogeneousMucociliary ClearanceUltrastructureFemalemedicine.symptomCiliary ultrastructureUltrastructural pathology
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Mutations in ARL2BP, Encoding ADP-Ribosylation-Factor-Like 2 Binding Protein, Cause Autosomal-Recessive Retinitis Pigmentosa

2013

Retinitis pigmentosa (RP) is a genetically heterogeneous retinal degeneration characterized by photoreceptor death, which results in visual failure. Here, we used a combination of homozygosity mapping and exome sequencing to identify mutations in ARL2BP, which encodes an effector protein of the small GTPases ARL2 and ARL3, as causative for autosomal-recessive RP (RP66). In a family affected by RP and situs inversus, a homozygous, splice-acceptor mutation, c.101−1G>C, which alters pre-mRNA splicing of ARLBP2 in blood RNA, was identified. In another family, a homozygous c.134T>G (p.Met45Arg) mutation was identified. In the mouse retina, ARL2BP localized to the basal body and cilium-associated…

AdultMaleRetinal degenerationCentrioleMolecular Sequence DataGenes RecessiveBiologymedicine.disease_causeMice03 medical and health sciences0302 clinical medicineBardet–Biedl syndromeGTP-Binding ProteinsReportRetinitis pigmentosaGeneticsmedicineAnimalsHumansBasal bodyGenetics(clinical)Photoreceptor CellsGenetics (clinical)030304 developmental biologyPrimary ciliary dyskinesiaGenetics0303 health sciencesMutationBase SequenceADP-Ribosylation FactorsCiliumHomozygoteMembrane Transport ProteinsEpithelial Cellsmedicine.diseasePedigreeCell biologyMutationFemalesense organsCarrier ProteinsRetinitis Pigmentosa030217 neurology & neurosurgeryProtein BindingTranscription FactorsThe American Journal of Human Genetics
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Improvement of wound healing after hemorrhoidectomy: a double-blind, randomized study of botulinum toxin injection.

2005

PURPOSE: Hemorrhoidectomy is usually associated with significant pain during the postoperative period. The spasm of the internal sphincter seems to play in important role in the origin of pain. This study was designed to evaluate the effectiveness of intrasphincter injection of botulinum toxin after hemaorrhoidectomy in reducing the maximum testing pressure of the anal canal, accelerating wound healing, and decreasing postoperative pain when resting and during defecation. METHODS: Thirty patients with hemorrhoids of third and fourth degree were included in the study and randomized in two groups. Anorectal manometry was performed preoperatively and 5 and 30 days afterward ill all patients un…

AdultMalemedicine.medical_specialtyBotulinum ToxinsManometrymedicine.medical_treatmentAnal CanalPainSodium ChlorideHemorrhoidsInternal anal sphincterHemorrhoidsDouble-Blind MethodPressureMedicineHumansbotulinum toxinDefecationSalineWound Healingposthemorrhoidectomy painbusiness.industryAnti-Dyskinesia AgentshemorrhoidectomyUrethral sphincterAnorectal manometryGastroenterologyGeneral MedicineAnal canalMiddle Agedmedicine.diseaseBotulinum toxinSurgerymedicine.anatomical_structureTreatment OutcomeAnesthesiaDefecationFemalebusinessmedicine.drugDiseases of the colon and rectum
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A functional study of the esophagus in patients with non-cardiac chest pain and dysphagia.

2015

Background/Aims: Nutcracker esophagus and non-specific motility disorders are the main causes of non-cardiac chest pain (NCCP), with gastroesophageal reflux in 60% of cases. Achalasia and diffuse esophageal spasm are the most frequent anomalies described in patients with dysphagia. The goal of this study was to evaluate the occurrence of esophageal body and lower esophageal sphincter motor abnormalities in patients with dysphagia, NCCP, or both. Materials and Methods: This study is a retrospective analysis of 716 patients with NCCP and/or dysphagia tested between January 1994 and December 2010. 1023 functional studies were performed, 707 of which were esophageal manometries, 225 esophageal …

AdultMalemedicine.medical_specialtyChest PainManometryAchalasiaChest painEsophaguGastroenterologyEsophageal Sphincter LowerEsophagusRetrospective StudieInternal medicineotorhinolaryngologic diseasesmedicineHumansIn patientEsophagusDeglutition DisorderNon-cardiac chest painAgedRetrospective StudiesSettore MED/12 - Gastroenterologiabusiness.industryMedicine (all)Esophageal dyskinesiaDysphagia; Esophageal dyskinesia; Gastroesophageal reflux; Non-cardiac chest pain; Adult; Aged; Chest Pain; Deglutition Disorders; Esophageal Achalasia; Esophageal Sphincter Lower; Esophageal Sphincter Upper; Esophagus; Female; Gastroesophageal Reflux; Humans; Hydrogen-Ion Concentration; Male; Manometry; Middle Aged; Retrospective Studies; Gastroenterology; Medicine (all)RefluxGastroenterologyNutcracker esophagusDysphagiaHydrogen-Ion ConcentrationMiddle Agedmedicine.diseaseEsophageal Sphincter UpperDysphagiaSurgeryEsophageal AchalasiaSettore MED/18 - Chirurgia Generalemedicine.anatomical_structureGastroesophageal RefluxEsophageal spasmFemalemedicine.symptombusinessDeglutition DisordersHumanThe Turkish journal of gastroenterology : the official journal of Turkish Society of Gastroenterology
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Therapeutic drug monitoring for optimizing amisulpride therapy in patients with schizophrenia.

2005

Amisulpride is a clinically effective antipsychotic drug in a broad dose range with low propensity for extrapyramidal symptoms (EPS). Daily doses and plasma levels of amisulpride were analyzed within a large-scale therapeutic drug monitoring (TDM) survey to find plasma level ranges for optimized treatment under naturalistic conditions. Data of 378 schizophrenic patients treated with amisulpride (100-1550 mg) were included (40% female). Amisulpride plasma levels were analyzed at steady state; assessment comprised improvement (CGI-I) and side-effects, particularly EPS. For detection of cut-off values regarding non-response or EPS, receiver operating characteristics (ROC) curves were applied a…

AdultMalemedicine.medical_specialtyDyskinesia Drug-InducedAdolescentmedicine.drug_classStatistics as TopicAtypical antipsychoticPharmacologyGastroenterologyExtrapyramidal symptomsInternal medicineGermanymedicineHumansAmisulprideBiological PsychiatryAgedRetrospective StudiesAged 80 and overNeurologic ExaminationPsychiatric Status Rating ScalesReceiver operating characteristicmedicine.diagnostic_testDose-Response Relationship Drugbusiness.industryDopamine antagonistMiddle Agedmedicine.diseasePsychiatry and Mental healthDose–response relationshipROC CurveSchizophreniaTherapeutic drug monitoringSchizophreniaFemalemedicine.symptomAmisulprideDrug MonitoringSulpiridebusinessmedicine.drugAntipsychotic AgentsJournal of psychiatric research
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Quality of life in Parkinson's disease patients with motor fluctuations and dyskinesias in five European countries.

2013

Little is known about the relationship between specific subtypes of treatment-associated motor complications and different domains of health-related Quality of Life (QoL) in patients with Parkinson's disease (PD). Larger studies that investigate these aspects within a cross-cultural setting are scarce.To assess QoL and its association with on-off fluctuations, peak-dose dyskinesias, biphasic dyskinesias, and off-dystonias in PD patients from five European countries.Data from 817 PD patients were collected cross-sectionally in France, Germany, Italy, Spain, and the UK. QoL was measured with the generic EuroQoL 5-Dimension questionnaire (EQ-5D) and the disease-specific Parkinson's Disease Que…

AdultMalemedicine.medical_specialtyParkinson's diseaseNeurologyDiseaseAntiparkinson AgentsQuality of life (healthcare)Surveys and QuestionnairesActivities of Daily LivingMedicineHumansIn patientPsychiatryAgedAged 80 and overDyskinesiasbusiness.industryParkinson DiseaseMiddle Agedmedicine.diseaseEuropeNeurologyDyskinesiaQuality of LifeFemaleNeurology (clinical)Geriatrics and Gerontologymedicine.symptombusinessParkinsonismrelated disorders
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New adenylate kinase 7 (AK7) mutation in primary ciliary dyskinesia.

2012

Background Primary ciliary dyskinesia (PCD) is a congenital hereditary disease affecting 1/20,000–60,000 people that causes chronic sinusitis, bronchiectasis, sinus hypoplasia, secretory otitis media, and low fertility. The complexity and heterogeneity of the disease make diagnosis difficult. Although the genetic origin of PCD is clear, mutations in only five genes have been associated with the disease, and, to date, no disease-causing gene has been identified. Recently, low levels of AK7 gene expression have been linked to PCD. This study was designed to determine the mutational status of the AK7 gene in 31 PCD (17 PCD and 14 Kartagener syndrome diagnosed) patients compared with 40 healthy…

AdultPathologymedicine.medical_specialtySingle-nucleotide polymorphismBiologyReal-Time Polymerase Chain ReactionPolymorphism Single NucleotideExonCiliogenesisGene expressionotorhinolaryngologic diseasesmedicineImmunology and AllergyHumansChildGenePrimary ciliary dyskinesiaKartagener SyndromeAdenylate KinaseKartagener SyndromeGeneral MedicineMiddle Agedmedicine.diseaseReal-time polymerase chain reactionOtorhinolaryngologyMutationAmerican journal of rhinologyallergy
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Segmental dyskinesia in Wolff–Parkinson–White syndrome: A possible cause of dilatative cardiomyopathy

2006

Wolff-Parkinson-White (WPW) is a syndrome characterized by the presence of an accessory pathway that skipping A-V node may lead the electrical stimulus from the atrium directly to the ventricle. Some studies reported the finding of myocardial dyskinesia in the segments precociously activated by the accessory pathway, at echocardiogram and at nuclear cardiac study. Soria et al. reported, in 1985, an increased incidence of dilative cardiomyopathy in patients with WPW. The pathophysiological pathway that leads to ventricular dilation may be due to the increase of end-diastolic pressure secondary to a tachycardia-induced cardiomyopathy. Tachycardia-induced cardiomyopathy is usually secondary to…

Cardiomyopathy DilatedTachycardiamedicine.medical_specialtyCardiomyopathyHemodynamicsAccessory pathwayAneurysmInternal medicinemedicineHumanscardiovascular diseasesChildbusiness.industryInfantArrhythmias Cardiacmedicine.diseasePathophysiologymedicine.anatomical_structureDyskinesiaVentriclecardiovascular systemCardiologyWolff-Parkinson-White Syndromemedicine.symptomCardiology and Cardiovascular MedicinebusinessFollow-Up StudiesInternational Journal of Cardiology
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