Search results for "encephalomyopathy"

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Mitochondrial Neurogastrointestinal Encephalomyopathy: Into the Fourth Decade, What We Have Learned So Far

2018

Mitochondrial neurogastrointestinal encephalomyopathy (MNGIE) is an ultra-rare metabolic autosomal recessive disease, caused by mutations in the nuclear gene TYMP which encodes the enzyme thymidine phosphorylase. The resulting enzyme deficiency leads to a systemic accumulation of the deoxyribonucleosides thymidine and deoxyuridine, and ultimately mitochondrial failure due to a progressive acquisition of secondary mitochondrial DNA (mtDNA) mutations and mtDNA depletion. Clinically, MNGIE is characterized by gastrointestinal and neurological manifestations, including cachexia, gastrointestinal dysmotility, peripheral neuropathy, leukoencephalopathy, ophthalmoplegia and ptosis. The disease is …

0301 basic medicinedeoxyribonucleosidelcsh:QH426-470Mitochondrial diseaseTYMPrare diseaseReviewDiseasemitochondrial DNABioinformaticsthymidine phosphorylaseCachexiaLeukoencephalopathy03 medical and health sciences0302 clinical medicineGeneticsmedicineThymidine phosphorylaseGenetics (clinical)Gastrointestinal dysmotilitymitochondrial neurogastrointestinal encephalomyopathybusiness.industrymedicine.diseaselcsh:Geneticsmitochondrial disease030104 developmental biologyPeripheral neuropathyMNGIEMolecular Medicinebusiness030217 neurology & neurosurgeryRare disease
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The Mitochondrial tRNASer(UCN) Gene: A Novel m.7484A>G Mutation Associated with Mitochondrial Encephalomyopathy and Literature Review

2023

Mitochondrial tRNASer(UCN) is considered a hot-spot for non-syndromic and aminoglycoside-induced hearing loss. However, many patients have been described with more extensive neurological diseases, mainly including epilepsy, myoclonus, ataxia, and myopathy. We describe a novel homoplasmic m.7484A>G mutation in the tRNASer(UCN) gene affecting the third base of the anticodon triplet in a girl with profound intellectual disability, spastic tetraplegia, sensorineural hearing loss, a clinical history of epilepsia partialis continua and vomiting, typical of MELAS syndrome, leading to a myoclonic epilepticus status, and myopathy with severe COX deficiency at muscle biopsy. The mutation was also …

Space and Planetary SciencePaleontologyencephalomyopathy tRNASer(UCN) homoplasmic mutation mitochondrial DNAGeneral Biochemistry Genetics and Molecular BiologyEcology Evolution Behavior and Systematics
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