Search results for "endothelial cells"

showing 10 items of 441 documents

Hypoxia-induced reduction of sVEGFR-2 levels in human colonic microvascular endothelial cells in vitro: Comparative study with HUVEC.

2008

The functionality of large-vessel endothelial cells, such as human umbilical vein endothelial cells (HUVEC), may differ significantly from that in the microvasculature. We established a method for the isolation of human colonic microvascular endothelial cells (HCMEC). Since colonic diseases are often accompanied by hypoxia we examined its effects on HCMEC of five individuals in comparison with HUVEC, with respect to the secretion of the soluble form of the two important vascular endothelial growth factor (VEGF) receptors, VEGFR-1 and 2. After dissociation by dispase/collagenase of mucosal and submucosal tissue obtained from normal adult colon, HCMEC were isolated using CD31-coated magnetic …

ColonBiologyUmbilical veinAndrologychemistry.chemical_compoundDispaseGeneticsmedicineHumansReceptorCells CulturedVascular Endothelial Growth Factor Receptor-1Endothelial CellsKinase insert domain receptorGeneral MedicineHypoxia (medical)Vascular Endothelial Growth Factor Receptor-2In vitroCell HypoxiaCell biologyVascular endothelial growth factorchemistryGene Expression RegulationApoptosiscardiovascular systemmedicine.symptomInternational journal of molecular medicine
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Lovastatin protects human endothelial cells from the genotoxic and cytotoxic effects of the anticancer drugs doxorubicin and etoposide

2006

Background and purpose: 3-Hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reductase inhibitors (statins) are frequently used lipid-lowering drugs. Moreover, they exert pleiotropic effects on cellular stress responses and death. Here, we analysed whether lovastatin affects the sensitivity of primary human endothelial cells (HUVEC) to the anticancer drug doxorubicin. Experimental approach: We investigated whether pretreatment of HUVEC with low dose of lovastatin influences the cellular sensitivity to doxorubicin. To this end, cell viability, proliferation and apoptosis as well as DNA damage-triggered stress response were analysed. Key results: Lovastatin reduced the cytotoxic potency of doxorub…

DNA ReplicationCell SurvivalDNA damageApoptosisBiologyPharmacologypolycyclic compoundsmedicineHumansTopoisomerase II InhibitorsDoxorubicinLovastatinEtoposideEtoposideFluorescent DyesPharmacologyAntibiotics AntineoplasticReverse Transcriptase Polymerase Chain ReactionTopoisomeraseCell CycleEndothelial Cellsnutritional and metabolic diseasesAntimutagenic AgentsFibroblastsCell cycleResearch PapersAntineoplastic Agents PhytogenicDoxorubicinDrug Resistance NeoplasmHMG-CoA reductasebiology.proteinlipids (amino acids peptides and proteins)LovastatinHydroxymethylglutaryl-CoA Reductase InhibitorsTopoisomerase-II InhibitorReactive Oxygen SpeciesFluorescein-5-isothiocyanateDNA Damagemedicine.drugBritish Journal of Pharmacology
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A Polyphenylene Dendrimer Drug Transporter with Precisely Positioned Amphiphilic Surface Patches

2014

The design and synthesis of a polyphenylene dendrimer (PPD 3) with discrete binding sites for lipophilic guest molecules and characteristic surface patterns is presented. Its semi-rigidity in combination with a precise positioning of hydrophilic and hydrophobic groups at the periphery yields a refined architecture with lipophilic binding pockets that accommodate defined numbers of biologically relevant guest molecules such as fatty acids or the drug doxorubicin. The size, architecture, and surface textures allow to even penetrate brain endothelial cells that are a major component of the extremely tight blood-brain barrier. In addition, low to no toxicity is observed in in vivo studies using…

DendrimersScaffoldEmbryo NonmammalianMaterials sciencePolymersStereochemistryBiomedical EngineeringPharmaceutical ScienceChemistry Techniques SyntheticBlood–brain barrierCell LineBiomaterialsMiceIn vivoDendrimerAmphiphilemedicineAnimalsHumansMoleculeTissue DistributionBinding siteZebrafishDrug CarriersBrainEndothelial CellsTransportermedicine.anatomical_structureDoxorubicinDrug DesignBiophysicsHydrophobic and Hydrophilic InteractionsAdvanced Healthcare Materials
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Functional feature of a novel model of blood brain barrier: Studies on permeation of test compounds

2001

Drug delivery to the central nervous system (CNS) is subject to the permeability limitations imposed by the blood-brain barrier (BBB). Several systems in vitro have been described to reproduce the physical and biochemical behavior of intact BBB, most of which lack the feature of the in vivo barrier. We developed a fully formed monolayer of RBE4.B immortalized rat brain microvessel endothelial cells (ECs), grown on top of polycarbonate filter inserts with cortical neuronal cells grown on the outside. Neurons induce ECs to synthesize and sort occludin to the cell periphery. Occludin localization is regulated by both compositions of the substratum and soluble signals released by cortical co-cu…

DopamineL-DOPAPharmaceutical ScienceBrain capillaries endothelial cells (ECs)OccludinBlood–brain barrierDopamine agonistPermeabilityLevodopaRats Sprague-DawleyDopamineIn vivoSettore BIO/10 - BiochimicamedicineAnimalsCells CulturedChemistryTryptophanPermeationRatsEndothelial stem cellBlood-brain barrier (BBB)medicine.anatomical_structureBiochemistryPermeability (electromagnetism)Blood-Brain BarrierSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoBiophysicsSettore MED/26 - Neurologiamedicine.drugL-Tryptophan
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EGFL7 ligates αvβ3 integrin to enhance vessel formation

2013

Angiogenesis, defined as blood vessel formation from a preexisting vasculature, is governed by multiple signal cascades including integrin receptors, in particular integrin αVβ3. Here we identify the endothelial cell (EC)-secreted factor epidermal growth factor-like protein 7 (EGFL7) as a novel specific ligand of integrin αVβ3, thus providing mechanistic insight into its proangiogenic actions in vitro and in vivo. Specifically, EGFL7 attaches to the extracellular matrix and by its interaction with integrin αVβ3 increases the motility of EC, which allows EC to move on a sticky underground during vessel remodeling. We provide evidence that the deregulation of EGFL7 in zebrafish embryos leads …

EGF Family of ProteinsEmbryo NonmammalianAngiogenesisAmino Acid MotifsImmunologyIntegrinGene ExpressionMice NudeEndothelial Growth FactorsBiochemistryCollagen receptorMiceCell MovementCell AdhesionHuman Umbilical Vein Endothelial CellsmedicineAnimalsHumansImmunoprecipitationPhosphorylationZebrafishbiologyReverse Transcriptase Polymerase Chain ReactionChemistryCalcium-Binding ProteinsInfarction Middle Cerebral ArteryVenous plexusCell BiologyHematologyIntegrin alphaVbeta3ImmunohistochemistryExtracellular MatrixCell biologyEndothelial stem cellHEK293 Cellsmedicine.anatomical_structureIntegrin alpha MImmunologybiology.proteinBlood VesselsRNA InterferenceIntegrin beta 6Protein BindingBlood vesselBlood
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Flotillin-involved uptake of silica nanoparticles and responses of an alveolar-capillary barrier in vitro

2013

AbstractDrug and gene delivery via nanoparticles across biological barriers such as the alveolar-capillary barrier of the lung constitutes an interesting and increasingly relevant field in nanomedicine. Nevertheless, potential hazardous effects of nanoparticles (NPs) as well as their cellular and systemic fate should be thoroughly examined. Hence, this study was designed to evaluate the effects of amorphous silica NPs (Sicastar) and (poly)organosiloxane NPs (AmOrSil) on the viability and the inflammatory response as well as on the cellular uptake mechanisms and fate in cells of the alveolar barrier. For this purpose, the alveolar epithelial cell line (NCI H441) and microvascular endothelial…

EndosomeCell SurvivalLipid BilayersPharmaceutical ScienceGene deliverysilica nanoparticlesEndocytosisClathrinNP transportCell LineDrug Delivery SystemsAlveolar-capillary barrierAlveolar capillary barrierElectric ImpedanceHumansColoring AgentsInflammationFlotillin-1/-2-dependent uptake/traffickingbiologyChemistryRhodaminesVesicleMicrocirculationEndothelial CellsMembrane ProteinsGeneral Medicinerespiratory systemSilicon DioxideNP uptakeIn vitroCoculture TechniquesEndocytosisCapillariesEndothelial stem cellPulmonary AlveoliNP-transportNanomedicineCell cultureImmunologybiology.proteinBiophysicsNanoparticlesBiotechnologyEuropean Journal of Pharmaceutics and Biopharmaceutics
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An antihypertensive lactoferrin hydrolysate inhibits angiotensin I-converting enzyme, modifies expression of hypertension-related genes and enhances …

2015

This study was aimed to explore whether an antihypertensive lactoferrin hydrolysate (LFH) can inhibit angiotensin I-converting enzyme (ACE) activity and modify the expression of genes related to hypertension in human umbilical vein endothelial cells (HUVEC). LFH induced significant inhibition of ACE activity but it did not affect ACE mRNA levels after 24 h of exposure. LFH treatment significantly affected the expression of genes encoding for proteins involved in nitric oxide pathway such as soluble guanylate cyclase 1 α3 subunit (GUCY1A3; 4.42-fold increase) and nitric oxide synthase trafficking (NOSTRIN; 2.45-fold decrease). Furthermore, expression of the PTGS2/COX-2 gene encoding prostagl…

Endothelial cellsMedicine (miscellaneous)PharmacologyLactoferrin hydrolysateTranscriptomic analysisUmbilical veinNitric oxidechemistry.chemical_compoundDownregulation and upregulationTX341-641Nutrition and DieteticsAngiotensin II receptor type 1biologyNutrition. Foods and food supplyLactoferrinGUCY1A3Nitric oxideACE inhibitionNOSTRINMolecular biologyNitric oxide synthasechemistryNitric Oxide Pathwaybiology.proteinFood Science
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Licofelone, a novel 5-LOX/COX-inhibitor, attenuates leukocyte rolling and adhesion on endothelium under flow

2005

The main mechanism of action of non-steroidal anti-inflammatory drugs (NSAIDs) is the inhibition of cycloxygenases COX-1 and COX-2. During recent years, combined 5-LOX/COX-inhibition, interfering with the biosynthesis of both prostaglandins and leukotrienes (LTs), has emerged as a possibility to avoid side effects related to COX-inhibition. The aim of the present study was to investigate if there is a contribution of mechanisms other than the reduction of inflammatory prostaglandins and leukotrienes to the anti-inflammatory effect of the LOX/COX inhibitor licofelone. In a flow chamber assay, licofelone (10-30 microM) dose-dependently decreased both the rolling and adhesion of leukocytes on …

EndotheliumAcetatesPharmacologyBiochemistrychemistry.chemical_compoundCell MovementIn vivoCell AdhesionLeukocytesmedicineHumansCyclooxygenase InhibitorsPyrrolesLipoxygenase InhibitorsRNA MessengerCells CulturedPharmacologybiologyChemistryEndothelial Cellsmedicine.anatomical_structureMechanism of actionImmunologyArachidonate 5-lipoxygenaseCelecoxibbiology.proteinCyclooxygenasemedicine.symptomLicofeloneCell Adhesion MoleculesSelectinmedicine.drugBiochemical Pharmacology
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Lactate adversely affects the in vitro formation of endothelial cell tubular structures through the action of TGF-beta1.

2006

When lactate accumulation in a tumor microenvironment reaches an average concentration of 10-20 mM, it tends to reflect a high degree of malignancy. However, the hypothesis that tumor-derived lactate has a number of partially adverse biological effects on malignant and tumor-associated host cells requires further evidence. The present study attempted to evaluate the impact of lactate on the process of angiogenesis, in particular on the formation of tubular structures. The endothelial cell (EC) network in desmoplastic breast tumors is primarily located in areas of reactive fibroblastic stroma. We employed a fibroblast-endothelial cell co-culture model as in vitro angiogenesis system normally…

EndotheliumAngiogenesisCell SurvivalCellPopulationNeovascularization PhysiologicBiologyTransforming Growth Factor beta1Cell MovementNeutralization TestsmedicineHumansLactic AcidRNA MessengerFibroblasteducationCell ProliferationTumor microenvironmenteducation.field_of_studyCell growthEndothelial CellsCell BiologyFibroblastsActinsCoculture TechniquesCell biologyEndothelial stem cellmedicine.anatomical_structureGene Expression RegulationImmunologyExperimental cell research
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Tumour vascularization via endothelial differentiation of glioblastoma stem-like cells

2010

Glioblastoma is a highly angiogenetic malignancy, the neoformed vessels of which are thought to arise by sprouting of pre-existing brain capillaries. The recent demonstration that a population of glioblastoma stem-like cells (GSCs) maintains glioblastomas indicates that the progeny of these cells may not be confined to the neural lineage. Normal neural stem cells are able to differentiate into functional endothelial cells. The connection between neural stem cells and the endothelial compartment seems to be critical in glioblastoma, where cancer stem cells closely interact with the vascular niche and promote angiogenesis through the release of vascular endothelial growth factor(VEGF) and str…

EndotheliumAngiogenesisTransplantation HeterologousSettore MED/27 - NEUROCHIRURGIAMice TransgenicMice SCIDBiologyModels BiologicalMiceVasculogenesisNeural Stem CellsMice Inbred NODCell Line TumormedicineAnimalsHumansCell LineageVasculogenic mimicryglioblastoma tumor vascularizationIn Situ Hybridization FluorescenceChromosome AberrationsMultidisciplinaryNeovascularization PathologicEndothelial CellsCell DifferentiationVascular endothelial growth factor BEndothelial stem cellVascular endothelial growth factor Amedicine.anatomical_structureVascular endothelial growth factor CTumor Markers BiologicalImmunologyCancer researchEndothelium VascularGlioblastomaNeoplasm TransplantationNature
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