Search results for "enzyme inhibitors"

showing 10 items of 559 documents

Effects Of COOH-terminal tripeptide alpha-MSH (11-13) on corneal epithelial wound healing:role of nitric oxide

2006

It is known that alpha-melanocyte stimulating hormone (alpha-MSH) may exert anti-inflammatory effects and facilitate reparative processes in different tissues. The effective message sequence of alpha-MSH resides in the COOH-terminal tripeptide alpha-MSH(11-13). This study was undertaken to investigate the effects of topical administration of the COOH-terminal tripeptide sequence of alpha-MSH (alpha-MSH(11-13), KPV) on corneal epithelial wound healing in rabbits and the possible role of nitric oxide (NO) in these effects. The whole corneal epithelium was denuded in both eyes by mechanical abrasion. The area of the corneal epithelial defect was stained with fluorescein, photographed, and then…

MaleNitroprussideMelanocyte-Stimulating Hormonemedicine.medical_treatmentCorneal abrasionRabbitPharmacologyKPVNitric OxideNitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundPeptide Fragmentα-MSHmedicineAnimalsEnzyme InhibitorNitric Oxide DonorsMelanocyte-Stimulating HormonesFluoresceinEnzyme InhibitorsSalineCells CulturedCorneal epitheliumCell ProliferationEpithelial CellWound HealingbiologyDose-Response Relationship DrugAnimalcorneal wound healingEpithelium CornealEpithelial CellsNitric Oxide Donormedicine.diseaseSensory SystemsPeptide FragmentsNitric oxide synthaseOphthalmologymedicine.anatomical_structureNG-Nitroarginine Methyl EsterchemistryBiochemistrybiology.proteinSodium nitroprussideRabbitsWound healingmedicine.drug
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Nitrergic and purinergic interplay in inhibitory transmission in rat gastric fundus.

2007

Summary 1  This study was undertaken to analyse the involvement of ATP in non-adrenergic non- cholinergic (NANC) relaxation and possible interplay between nitrergic and purinergic systems in rat gastric fundus. 2  Experiments were performed in vitro on strips of longitudinal muscle from rat gastric fundus, recording the mechanical activity as changes in isometric force. In addition, NO release induced by different experimental conditions was assayed. 3  Under NANC conditions in serotonin-precontracted strips, electrical field stimulation (EFS) elicited a tetrodotoxin (TTX)-sensitive relaxation accompanied by nitric oxide (NO) release. This effect was antagonized by pretreatment with the NO …

MaleNitroprussidemedicine.medical_specialtyAdenosineMuscle RelaxationStimulationTetrodotoxinIn Vitro TechniquesInhibitory postsynaptic potentialApaminNitric OxideNitroargininechemistry.chemical_compoundAdenosine TriphosphateDesensitization (telecommunications)Internal medicineNitrergic NeuronsmedicineAnimalsNitric Oxide DonorsGastric FundusEnzyme InhibitorsRats WistarPharmacologyPurinergic receptorReceptors PurinergicMuscle SmoothAdenosine MonophosphateElectric StimulationRatsAdenosine DiphosphateEndocrinologychemistryApaminTetrodotoxinCholinergicFemaleSodium nitroprussideNitric Oxide Synthasemedicine.drugAutonomicautacoid pharmacology
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Diabetes potentiates acetylcholine-induced relaxation in rabbit renal arteries.

2001

Abstract The response of rabbit renal arteries to acetylcholine and its endothelial modulation in diabetes were investigated. Acetylcholine induced concentration-related endothelium-dependent relaxation of renal arteries that was significantly more potent in diabetic rabbits than in control rabbits. Pretreatment with NG-nitro- l -arginine ( l -NOArg), indomethacin, or l -NOArg plus indomethacin induced partial inhibition of acetylcholine-induced relaxation. Inhibition induced by l -NOArg plus indomethacin was significantly higher in arteries from diabetic rabbits than in arteries from control rabbits. In renal arteries depolarised with KCl 30 mM and incubated with l -NOArg plus indomethacin…

MaleNitroprussidemedicine.medical_specialtyArginineEndotheliumVasodilator AgentsIndomethacinProstacyclinNitric OxideNitroarginineNitric oxideDiabetes Mellitus Experimentalchemistry.chemical_compoundRenal Arterymedicine.arteryInternal medicineMedicineAnimalsRenal arteryEnzyme InhibitorsPharmacologybusiness.industryAnti-Inflammatory Agents Non-SteroidalAcetylcholineVasodilationEndocrinologymedicine.anatomical_structurechemistrycardiovascular systemSodium nitroprussideEndothelium VascularRabbitsbusinessAcetylcholinemedicine.drugArteryEuropean journal of pharmacology
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Contribution of nitric oxide synthase isoforms to cholinergic vasodilation in murine retinal arterioles.

2013

Abstract Nitric oxide synthases (NOSs) are critically involved in regulation of ocular perfusion. However, the contribution of the individual NOS isoforms to vascular responses is unknown in the retina. Because some previous findings suggested an involvement of inducible nitric oxide synthase (iNOS) in the regulation of retinal vascular tone, a major goal of the present study was to examine the hypothesis that iNOS is involved in mediating cholinergic vasodilation responses of murine retinal arterioles. Another subject of this study was to test the contribution of the other two NOS isoforms, neuronal (nNOS) and endothelial NOS (eNOS), to cholinergic retinal arteriole responses. Expression o…

MaleNitroprussidemedicine.medical_specialtyNitric Oxide Synthase Type IIIVasodilator AgentsNitric Oxide Synthase Type IIVasodilationNitric Oxide Synthase Type IBiologyEndothelial NOSReal-Time Polymerase Chain ReactionGene Expression Regulation EnzymologicNitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundMiceInternal medicinemedicineAnimalsRNA MessengerEnzyme InhibitorsMice KnockoutBrainRetinal VesselsRetinalSensory SystemsAcetylcholineNitric oxide synthaseMice Inbred C57BLVasodilationOphthalmologyArteriolesEndocrinologyNG-Nitroarginine Methyl Esterchemistrybiology.proteinCholinergicmedicine.symptomVasoconstrictionAcetylcholinemedicine.drugExperimental eye research
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Ca2+-activated K+ channels mediate relaxation of forearm veins in chronic renal failure

2003

In arteries, agonists such as acetylcholine release an endothelium-derived hyperpolarizing factor (EDHF) that is neither nitric oxide nor prostacyclin.To examine the responses to acetylcholine in segments of forearm veins from patients with chronic renal failure who either had never received dialysis or had undergone long-term dialysis, and to determine the contribution of nitric oxide and EDHF to endothelium-dependent relaxation in veins from patients with chronic renal failure.Isometric tension was recorded in rings of forearm vein from 34 non-dialysed patients, 27 dialysed patients and 14 multiorgan donors (controls).Relaxation in response to acetylcholine was reduced in veins of non-dia…

MaleNitroprussidemedicine.medical_specialtyPhysiologyVasodilator AgentsVasodilationIn Vitro TechniquesNitric OxideVeinsNitric oxideBiological FactorsPotassium Channels Calcium-Activatedchemistry.chemical_compoundForearmQuinoxalinesInternal medicineInternal MedicinemedicineHumansEnzyme InhibitorsVeinOxadiazolesomega-N-MethylarginineVascular diseasebusiness.industryMiddle Agedmedicine.diseaseAcetylcholinePotassium channelVasodilationForearmEndocrinologymedicine.anatomical_structurechemistrycardiovascular systemKidney Failure ChronicFemaleNitric Oxide SynthaseCardiology and Cardiovascular MedicinebusinessAcetylcholineKidney diseasemedicine.drugJournal of Hypertension
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Nitric oxide as neuromodulator of sympathetic transmission in rat vas deferens.

1998

Summary Electrical field stimulation (EFS) of muscle strips in vitro elicited a tetrodotoxin (TTX)-sensitive biphasic contractile response consisting of a phasic component followed by a tonic one. The amplitude of both components of the response was impaired by Nω-nitro-L-arginine and potentiated by sodium nitroprusside. Cystamine caused a reduction in amplitude of both phasic and tonic componets of the response to EFS. Neither Nω-nitro-L-arginine, sodium nitroprusside, nor cystamine induced changes in the resting muscle tone, or in the contractile response to exogenous agonists ATP and noradrenaline (NA). The nitric oxide scavenger, 2-phenyl-4,4,5,5-tetramethylimidazoline-1-oxyl-3-oxide, i…

MaleNitroprussidemedicine.medical_specialtySympathetic Nervous SystemCystamineNeurotransmissionNitric OxideNitroarginineTonic (physiology)Nitric oxideCyclic N-Oxideschemistry.chemical_compoundVas DeferensCystamineInternal medicinemedicineExtracellularAnimalsEnzyme InhibitorsRats WistarPharmacologyGeneral NeuroscienceVas deferensImidazolesElectric StimulationRatsmedicine.anatomical_structureEndocrinologychemistryGuanylate CyclaseTetrodotoxinSodium nitroprussideNitric Oxide Synthasemedicine.drugMuscle ContractionJournal of autonomic pharmacology
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Comparative impact of multiple biomarkers and N-terminal pro-brain natriuretic peptide in the context of conventional risk factors for the prediction…

2006

Background— Individual markers of inflammation may add incremental predictive value in the context of conventionally available risk factors. We evaluated the ability of 9 inflammatory biomarkers, microalbuminuria, and N-terminal pro-brain natriuretic peptide (Nt-proBNP) to improve cardiovascular risk prediction beyond that obtained from traditional risk factors in a secondary-prevention population. Methods and Results— We measured biomarkers representing the acute-phase reaction (C-reactive protein, fibrinogen, and interleukin-6), proinflammatory pathways (soluble tumor necrosis factor receptor-1 and -2, soluble interleukin-1 receptor antagonist, and interleukin-18), endothelial activation…

MaleOncologymedicine.medical_specialtyHeart Diseasesmedicine.drug_classPopulationMyocardial InfarctionAngiotensin-Converting Enzyme InhibitorsContext (language use)Coronary Artery DiseaseProinflammatory cytokineEndothelial activationRamiprilPredictive Value of TestsRisk FactorsPhysiology (medical)Internal medicineNatriuretic Peptide BrainNatriuretic peptideHumansVitamin EMedicineRisk factoreducationAgededucation.field_of_studyInterleukin-6business.industryFibrinogenMiddle Agedmedicine.diseaseBrain natriuretic peptideC-Reactive ProteinEndocrinologyFemaleMicroalbuminuriaCardiology and Cardiovascular MedicinebusinessBiomarkers
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Modulatory effects of nitric oxide-active drugs on the anticonvulsant activity of lamotrigine in an experimental model of partial complex epilepsy in…

2007

Abstract Background The effects induced by administering the anticonvulsant lamotrigine, the preferential inhibitor of neuronal nitric oxide synthase 7-nitroindazole and the precursor of NO synthesis L-arginine, alone or in combination, on an experimental model of partial complex seizures (maximal dentate gyrus activation) were studied in urethane anaesthetized rats. The epileptic activity of the dentate gyrus was obtained through the repetitive stimulation of the angular bundle and maximal dentate gyrus activation latency, duration and post-stimulus afterdischarge duration were evaluated. Results Either Lamotrigine (10 mg kg-1) or 7-nitroindazole (75 mg kg-1) i.p. administration had an ant…

MalePARTIAL COMPLEX EPILEPSYIndazolesArgininemedicine.medical_treatmentLamotriginePharmacologyArginineLamotrigineNitric OxideSettore BIO/09 - Fisiologialcsh:RC321-571Nitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundEpilepsy Complex PartialmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats Wistarlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNitric oxide Lamotrigine epilepsy controlbiologyTriazinesExperimental modelGeneral NeuroscienceDentate gyruslcsh:QP351-495BrainElectric StimulationRatsNitric oxide synthaseDisease Models Animallcsh:Neurophysiology and neuropsychologyAnticonvulsantnervous systemchemistryDentate Gyrusbiology.proteinAnticonvulsantsNitric Oxide SynthaseResearch Articlemedicine.drugBMC Neuroscience
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Glutathione protects metastatic melanoma cells against oxidative stress in the murine hepatic microvasculature.

1998

Calcein-labeled B16 melanoma (B16M) cells were injected intraportally, and in vivo video microscopy was used to study the distribution and damage of cancer cells arrested in the liver microvasculature over a period of 4 hours. The contribution of glutathione (GSH)-dependent antioxidant machinery to the possible oxidative stress-resistance mechanism of B16M cell was determined by in vitro incubation with the selective inhibitor of GSH synthesis L-buthionine (S,R)-sulphoximine (BSO) before B16M cell injection in untreated and 0.5-mg/kg lipopolysaccharide (LPS)-treated mice. In addition, untreated and LPS-treated isolated syngeneic hepatic sinusoidal endothelial cells (HSE) were used to determ…

MalePathologymedicine.medical_specialtyCellMelanoma ExperimentalVideo RecordingVideo microscopyBiologymedicine.disease_causeAndrologychemistry.chemical_compoundMiceIn vivomedicineCell AdhesionTumor Cells CulturedAnimalsEnzyme InhibitorsNeoplasm MetastasisCell damageButhionine SulfoximineHepatologyMicrocirculationLiver NeoplasmsCell PolarityGlutathionemedicine.diseaseGlutathioneMice Inbred C57BLOxidative Stressmedicine.anatomical_structurechemistryLiverCancer cellOxidative stressIntracellularHepatology (Baltimore, Md.)
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Disease and patient characteristics in NP-C patients: findings from an international disease registry.

2013

Abstract Background Niemann-Pick disease type C (NP-C) is a rare neurovisceral disease characterized by progressive neurodegeneration and premature death. We report data recorded at enrolment in an ongoing international NP-C registry initiated in September 2009 to describe disease natural history, clinical course and treatment experience of NP-C patients in clinical practice settings. Methods The NPC Registry is a prospective observational cohort study. Participating sites are encouraged to evaluate all consecutive patients with a confirmed diagnosis of NP-C, regardless of their treatment status. All patients undergo clinical assessments and medical care as determined by their physicians. D…

MalePediatricslcsh:Medicine[SDV.GEN] Life Sciences [q-bio]/Genetics0302 clinical medicineMiglustatDiagnosisGenetics(clinical)Pharmacology (medical)Prospective StudiesRegistriesAge of OnsetEnzyme InhibitorsChildProspective cohort studyGenetics (clinical)Medicine(all)0303 health sciencesCholestasisNiemann-Pick disease type CNiemann-Pick Disease Type CGeneral MedicineDysphagia3. Good healthChild PreschoolCohortNeurologicalFemalemedicine.symptomCohort studymedicine.drugHepatomegalymedicine.medical_specialty1-DeoxynojirimycinAtaxiaAdolescent03 medical and health sciencesDisease registrymedicineHumansDisabled PersonsVertical supranuclear palsy030304 developmental biology[SDV.GEN]Life Sciences [q-bio]/Geneticsbusiness.industryResearchlcsh:RInfantMutationSymptomsSplenomegalyAge of onsetbusiness030217 neurology & neurosurgery
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