Search results for "enzyme replacement therapy"

showing 10 items of 125 documents

Systemic therapies for mucopolysaccharidosis: ocular changes following haematopoietic stem cell transplantation or enzyme replacement therapy - a rev…

2010

The management of mucopolysaccharidosis (MPS) is focused on the multi-organ, sometimes life-threatening, clinical manifestations that occur over time. In the past, the limited, symptom-based treatment options led physicians to adopt a palliative approach towards individual disease-associated complications. The availability of systemic treatments such as haematopoietic stem cell transplantation (HSCT) and enzyme replacement therapy (ERT) has created a better prognosis for MPS patients, particularly when initiated early in life. As part of an integrated management approach, these therapies could be valuable in managing the ocular features that are present in many children with MPS. HSCT has b…

congenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyVisual acuitygenetic structuresbusiness.industryMucopolysaccharidosismedicine.medical_treatmentEye diseasenutritional and metabolic diseasesEnzyme replacement therapyHematopoietic stem cell transplantationmedicine.diseaseeye diseasesSurgeryTransplantationOphthalmologymedicineOptic nervesense organsStem cellmedicine.symptomIntensive care medicinebusinessClinical & Experimental Ophthalmology
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219 Incidence Testing of Hunter Syndrome in A Population at Risk - First Results of A Binational Screening Programme

2012

Background Hunter syndrome (Mucopolysaccharidosis type II; X-linked inheritance; prevalence rate in Europe approximately 1:77000 male newborns) is a rare, progressive, multisystemic disease, caused by deficiency of the lysosomal enzyme Iduronate-2-sulfatase. Due to the very heterogeneous phenotype Hunter syndrome is often not diagnosed before pre-school age. This is unfortunate, because patients would significantly benefit from the earliest possible start of treatment containing enzyme replacement therapy. Early screening methods are possible, but due to the rarity of this disease they are too expensive to be performed in all newborns. An at-risk patient population screening provides opport…

education.field_of_studyPediatricsmedicine.medical_specialtybusiness.industryIncidence (epidemiology)PopulationPrevalenceHunter syndromeEnzyme replacement therapymedicine.diseaseUmbilical herniaPediatrics Perinatology and Child Healthmedicinemedia_common.cataloged_instanceEuropean unionMucopolysaccharidosis type IIbusinesseducationmedia_commonArchives of Disease in Childhood
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Fabry Disease, a Complex Pathology Not Easy to Diagnose

2015

Fabry disease is a multisystemic lysosomal storage disorder, inherited in an X-linked manner. It is a defect of metabolism of the glycosphingolipids, due to the reduction or absence of the activity of lysosomal enzyme α-galactosidase A. This reduction of activity causes the storage of globotriaosylceramide and derivatives in the lysosomes, triggering a cascade of cellular events, mainly in vascular endothelium. These events are the responsible for the systemic clinical manifestations and the renal, cardiac and cerebrovascular complications, or a combination of them. The symptomatology can lead to the premature death of patient between the fourth or fifth decade of life. The first symptoms c…

lcsh:Diseases of the circulatory (Cardiovascular) systemPathologymedicine.medical_specialtyα-galactosidase Abusiness.industryGlobotriaosylceramideDiagnostic testDiseaseEnzyme replacement therapyAnderson-Fabry diseasemedicine.diseaseFabry diseaseVascular endotheliumchemistry.chemical_compoundPremature deathchemistrylcsh:RC666-701Clinical diagnosismedicineGeneral Earth and Planetary SciencesbusinessGLA gene.General Environmental ScienceCardiogenetics
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The Mainz Severity Score Index: a new instrument for quantifying the Anderson-Fabry disease phenotype, and the response of patients to enzyme replace…

2004

Anderson-Fabry disease (AFD) is an X-linked disorder caused by deficient activity of the lysosomal enzyme alpha-galactosidase A. The availability of enzyme replacement therapy (ERT) for this debilitating condition has led to the need for a convenient and sensitive instrument to monitor clinical effects in an individual patient. This study aimed to develop a scoring system--the Mainz Severity Score Index (MSSI)--to measure the severity of AFD and to monitor the clinical course of the disease in response to ERT. Thirty-nine patients (24 males and 15 females) with AFD were assessed using the MSSI immediately before and 1 year after commencing agalsidase alfa ERT. Control data were obtained fro…

medicine.medical_specialtyAlpha-galactosidasebiologyVascular diseasebusiness.industryCase-control studyEnzyme replacement therapyDiseasemedicine.diseaseFabry diseaseEl NiñoInternal medicineSeverity of illnessGeneticsmedicinebiology.proteinbusinessGenetics (clinical)Clinical Genetics
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Characteristics of patients with mucopolysaccharidosis type II who have received a bone marrow transplant: Data from the Hunter Outcome Survey

2019

Mucopolysaccharidosis type II (MPS II Hunter syndrome) is a rare, life-limiting, X-linked lysosomal storage disease. The Hunter Outcome Survey (HOS) is a Shire-sponsored, global, observational registry initiated in 2005 that collects real-world data on the natural history of MPS II and long-term treatment with enzyme replacement therapy (ERT) with idursulfase. Patients receiving other forms of pharmacological ERT are excluded from HOS but individuals who have received a bone marrow transplant (BMT) may be enrolled. This analysis examined the characteristics of 36 male patients in HOS from Europe and North America who had received a BMT (March 2018 data). In total, 22 patients (61.1%) were E…

medicine.medical_specialtyBone marrow transplantIdursulfasebusiness.industryEndocrinology Diabetes and MetabolismHunter syndromeEnzyme replacement therapymedicine.diseaseBiochemistryNatural historyEndocrinologyGraft-versus-host diseaseRespiratory failureInternal medicineGeneticsmedicineMucopolysaccharidosis type IIbusinessMolecular Biologymedicine.drugMolecular Genetics and Metabolism
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A 15-Year Perspective of the Fabry Outcome Survey

2016

Abstract The Fabry Outcome Survey (FOS) is an international long-term observational registry sponsored by Shire for patients diagnosed with Fabry disease who are receiving or are candidates for therapy with agalsidase alfa (agalα). Established in 2001, FOS provides long-term data on agalα safety/efficacy and collects data on the natural history of Fabry disease, with the aim of improving clinical management. The FOS publications have helped establish prognostic and severity scores, defined the incidence of specific disease variants and implications for clinical management, described clinical manifestations in special populations, confirmed the high prevalence of cardiac morbidity, and demon…

medicine.medical_specialtyEndocrinology Diabetes and MetabolismDisease030204 cardiovascular system & hematologyoutcomesFabry Outcome Survey03 medical and health sciences0302 clinical medicineInternal medicinemedicineGenetics (clinical)lcsh:R5-920agalsidase alfaFabry diseasebusiness.industryIncidence (epidemiology)Therapeutic effectEnzyme replacement therapymedicine.diseaseFabry diseaseNatural historyPediatrics Perinatology and Child HealthPhysical therapyObservational studybusinesslcsh:Medicine (General)Agalsidase alfa030217 neurology & neurosurgeryenzyme replacement therapy
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Long-term effectiveness of agalsidase alfa enzyme replacement in Fabry disease: A Fabry Outcome Survey analysis

2015

Outcomes from 5 years of treatment with agalsidase alfa enzyme replacement therapy (ERT) for Fabry disease in patients enrolled in the Fabry Outcome Survey (FOS) were compared with published findings for untreated patients with Fabry disease. Data were extracted from FOS, a Shire-sponsored database, for comparison with data from three published studies. Outcomes evaluated were the annualized rate of change in estimated glomerular filtration rate (eGFR) and left ventricular mass indexed to height (LVMI) as well as time to and ages at a composite morbidity endpoint and at death. FOS data were extracted for 740 treated patients who were followed for a median of ~ 5 years. Compared with no trea…

medicine.medical_specialtyEndocrinology Diabetes and MetabolismUrologyCardiomyopathyRenal functionSE Standard errorLeft ventricular hypertrophyBiochemistryLVH Left ventricular hypertrophyLong-term effectivenessEndocrinologyGeneticsMedicineMDRD Modification of Diet in Renal Diseaselcsh:QH301-705.5Molecular BiologyAgalsidase alfaeGFR Estimated glomerular filtration rateFabry diseaselcsh:R5-920CI Confidence intervalbusiness.industryEnzyme replacement therapymedicine.diseaseEgfr Estimated glomerular filtration rateFabry diseaseSurgeryARB Angiotensin receptor blockerSEM Standard error of the meanStandard errorlcsh:Biology (General)SI:TherapyEnzyme replacement therapyCohortFOS Fabry Outcome SurveyLVMI Left ventricular mass indexed to heightlcsh:Medicine (General)businessACEI Angiotensin-converting enzyme inhibitorAgalsidase alfaERT Enzyme replacement therapyMolecular Genetics and Metabolism Reports
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A multinational, multidisciplinary consensus for the diagnosis and management of spinal cord compression among patients with mucopolysaccharidosis VI.

2012

Cervical cord compression is a sequela of mucopolysaccharidosis VI, a rare lysosomal storage disorder, and has devastating consequences. An international panel of orthopedic surgeons, neurosurgeons, anesthesiologists, neuroradiologists, metabolic pediatricians, and geneticists pooled their clinical expertise to codify recommendations for diagnosing, monitoring, and managing cervical cord compression; for surgical intervention criteria; and for best airway management practices during imaging or anesthesia. The recommendations offer ideal best practices but also attempt to recognize the worldwide spectrum of resource availability. Functional assessments and clinical neurological examinations …

medicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentConsensus Development Conferences as TopicMucopolysaccharidosis type VIBiochemistryMyelopathyEndocrinologySpinal cord compressionGeneticsMedicineHumansIntensive care medicineMolecular BiologyMucopolysaccharidosis VIbusiness.industryStandard treatmentSequelaCervical cord compressionEnzyme replacement therapymedicine.diseaseSurgeryPractice Guidelines as TopicCervical VertebraeAirway managementbusinessSpinal Cord CompressionMolecular genetics and metabolism
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Downregulation of alpha-galactosidase A upregulates CD77: functional impact for Fabry nephropathy.

2009

Anderson-Fabry disease, an inherited deficiency in the lysosomal enzyme alpha-galactosidase A, is characterized by the progressive accumulation of globotriaosylceramide (Gb3), also known as CD77. We sought to clarify the pathogenesis of Fabry disease by establishing a cell model of this disorder. The expression of alpha-galactosidase A was transiently silenced by RNA interference in HK2 and primary human renal epithelial cells and stably silenced in HK2 cells by retroviral transfection with small hairpin RNA. All of the silenced cells had histological similarities to cells of patients with Fabry disease. The cells had reduced viability, significant accumulation of intracellular Gb3, and a m…

medicine.medical_specialtyGlobotriaosylceramideGb3Cell LineSmall hairpin RNAchemistry.chemical_compoundRNA interferenceDownregulation and upregulationInternal medicineMedicineGene silencingHumansGene SilencingRNA Small InterferingAnderson–Fabry diseaseGlobosidesbusiness.industryTrihexosylceramidesEpithelial CellsTransfectionEnzyme replacement therapymedicine.diseaseFabry diseaseα-galactosidaseEndocrinologychemistryGene Expression RegulationNephrologyCell culturealpha-GalactosidaseCancer researchFabry DiseaseCD77businessenzyme replacement therapyKidney international
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Effects of enzyme replacement therapy with agalsidase alfa on glomerular filtration rate in patients with Fabry disease: preliminary data

2007

Progressive deposition of globotriaosylceramide results in severe complications involving the kidney, heart and brain in both hemizygous male and heterozygous female patients with Fabry disease. Analysis of renal data from FOS - the Fabry Outcome Survey - suggests that enzyme replacement therapy with agalsidase alfa can significantly improve renal function in patients with Fabry disease, at least in those with a mild decrease in glomerular filtration rate, and may also be able to slow down the natural decline in renal function in patients with a moderate reduction in glomerular filtration rate. Conclusion: Initial results from the large cohort of patients within FOS indicate that treatment …

medicine.medical_specialtyGlobotriaosylceramideUrologyRenal functionchemistry.chemical_compoundInternal medicinemedicineHumansIn patientBeneficial effectsKidneybusiness.industryGeneral MedicineEnzyme replacement therapymedicine.diseaseFabry diseaseRecombinant ProteinsIsoenzymesEndocrinologymedicine.anatomical_structurechemistryalpha-GalactosidasePediatrics Perinatology and Child HealthFabry DiseasebusinessAgalsidase alfaGlomerular Filtration RateActa Paediatrica
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