Search results for "epilepsy"

showing 10 items of 420 documents

Anophthalmia-Waardenburg syndrome with expanding phenotype: does neural crest play a role?

2007

We describe a child with bilateral anophthalmia, limb anomalies, skin lesions, cerebral malformations, epilepsy, and mental retardation. This patient, according to eponymous classification, should fit into the Anophthalmia-Waardenburg syndrome, although he also presents cutaneous and cerebral manifestations never reported in this syndrome until now. These clinical findings could be explained by the new classification of brain malformations, which takes into account the role of neural crest in Waardenburg syndrome.

MaleAnophthalmia Waardenburg syndromeElectroencephalography03 medical and health sciencesEpilepsy0302 clinical medicine030225 pediatricsAnophthalmosmedicineHumansWaardenburg SyndromeChildAnophthalmiamedicine.diagnostic_testWaardenburg syndromeNeural crestAnophthalmosElectroencephalographyAnatomymedicine.diseasePhenotypePhenotypeNeural CrestPediatrics Perinatology and Child HealthNeurology (clinical)Psychology030217 neurology & neurosurgeryJournal of child neurology
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Anticonvulsant effects of carbenoxolone in genetically epilepsy prone rats (GEPRs).

2004

Carbenoxolone (CBX), the succinyl ester of glycyrrhetinic acid, is an inhibitor of gap junctional intercellular communication. Systemic administration of CBX was able to decrease the seizure severity score and to increase the latency time of seizure onset in genetically epilepsy prone rats (GEPRs). In particular, intravenous or intraperitoneal administration of carbenoxolone (5-30 mg/kg) produced a dose-dependent and significant reduction in the clonic and tonic phases of the audiogenic seizures in GEPRs. The anticonvulsant doses were not associated with an impairment of motor coordination. The bilateral microinjection of CBX (0.001-0.50 microg/0.5 microl) into the inferior colliculi, the s…

MaleAudiogenic seizuremedicine.medical_treatmentGap junctionGEPR-9sCarbenoxoloneSubstantia nigraPharmacologyConnexinConnexinsRats Sprague-DawleyCellular and Molecular NeuroscienceEpilepsyMedicineAnimalsMicroinjectionPharmacologyEpilepsybusiness.industrymedicine.diseaseMotor coordinationRatsAnticonvulsantAnesthesiaSystemic administrationCarbenoxoloneAnticonvulsantsFemalebusinessPars reticulataGEPR-3medicine.drugNeuropharmacology
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Essential thalamic contribution to slow waves of natural sleep

2013

Slow waves represent one of the prominent EEG signatures of non-rapid eye movement (non-REM) sleep and are thought to play an important role in the cellular and network plasticity that occurs during this behavioral state. These slow waves of natural sleep are currently considered to be exclusively generated by intrinsic and synaptic mechanisms within neocortical territories, although a role for the thalamus in this key physiological rhythm has been suggested but never demonstrated. Combining neuronal ensemble recordings, microdialysis, and optogenetics, here we show that the block of the thalamic output to the neocortex markedly (up to 50%) decreases the frequency of slow waves recorded dur…

MaleCalcium channels T-typeepilepsy cns.ThalamusRapid eye movement sleepAction PotentialsSleep spindleOptogeneticsElectroencephalographyQ1Settore BIO/09 - Fisiologia03 medical and health sciencesCalcium Channels T-Type0302 clinical medicineThalamusSlow wave sleepmedicineAnimalsAnesthesiaRats Wistar030304 developmental biologySlow-wave sleepCerebral CortexNeurons0303 health sciencesNeocortexmedicine.diagnostic_testGeneral NeuroscienceElectroencephalographyArticlesSleep in non-human animalsRatsmedicine.anatomical_structureRapid eye movement sleep[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]PsychologySleepNeuroscience030217 neurology & neurosurgery
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The DYRK1A gene is a cause of syndromic intellectual disability with severe microcephaly and epilepsy.

2012

Background DYRK1A plays different functions during development, with an important role in controlling brain growth through neuronal proliferation and neurogenesis. It is expressed in a gene dosage dependent manner since dyrk1a haploinsufficiency induces a reduced brain size in mice, and DYRK1A overexpression is the candidate gene for intellectual disability (ID) and microcephaly in Down syndrome. We have identified a 69 kb deletion including the 5′ region of the DYRK1A gene in a patient with growth retardation, primary microcephaly, facial dysmorphism, seizures, ataxic gait, absent speech and ID. Because four patients previously reported with intragenic DYRK1A rearrangements or 21q22 microd…

MaleCandidate geneDown syndromeMicrocephalyAdolescentGenotypeBiologyProtein Serine-Threonine KinasesBioinformaticsFrameshift mutationEpilepsyAngelman syndromeIntellectual DisabilityGene OrderGeneticsmedicineHumansChildGenetics (clinical)GeneticsEpilepsyBase SequenceFaciesElectroencephalographySyndromeProtein-Tyrosine Kinasesmedicine.diseasePhenotypeChild PreschoolSpeech delayMutationMicrocephalyFemalemedicine.symptomHaploinsufficiencyJournal of medical genetics
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CB1 Cannabinoid Receptors and On-Demand Defense Against Excitotoxicity

2003

Abnormally high spiking activity can damage neurons. Signaling systems to protect neurons from the consequences of abnormal discharge activity have been postulated. We generated conditional mutant mice that lack expression of the cannabinoid receptor type 1 in principal forebrain neurons but not in adjacent inhibitory interneurons. In mutant mice,the excitotoxin kainic acid (KA) induced excessive seizures in vivo. The threshold to KA-induced neuronal excitation in vitro was severely reduced in hippocampal pyramidal neurons of mutants. KA administration rapidly raised hippocampal levels of anandamide and induced protective mechanisms in wild-type principal hippocampal neurons. These protecti…

MaleCannabinoid receptorReceptors Drugmedicine.medical_treatment2-ArachidonoylglycerolExcitotoxicityHippocampal formationmedicine.disease_causeHippocampusMicechemistry.chemical_compoundPiperidinesCannabinoid receptor type 1Excitatory Amino Acid AgonistsReceptors Cannabinoidgamma-Aminobutyric AcidMice KnockoutNeuronsKainic AcidMultidisciplinaryBrainEndocannabinoid systemNeuroprotective AgentsMitogen-Activated Protein KinasesRimonabantSignal Transductionmedicine.medical_specialtyKainic acidPolyunsaturated AlkamidesGlutamic AcidMice TransgenicArachidonic AcidsIn Vitro TechniquesBiologyGlyceridesProsencephalonInternal medicinemedicineAnimalsFuransGenes Immediate-EarlyEpilepsyCannabinoidsBrain-Derived Neurotrophic FactorExcitatory Postsynaptic PotentialsMice Inbred C57BLEndocrinologyGene Expression Regulationnervous systemchemistryMutationPyrazolesCannabinoidNeuroscienceEndocannabinoidsScience
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EEG-related Functional MRI in Benign Childhood Epilepsy with Centrotemporal Spikes

2003

The localization of epileptic foci is an important issue in children with extratemporal epilepsies. However, the value of noninvasive methods such as the EEG-assisted functional magnetic resonance imaging (fMRI) has not been sufficiently investigated in children. As a model of extratemporal epilepsies, we studied 7 patients aged 5 to 12 (median 10) years with benign childhood epilepsy and centrotemporal (rolandic) spikes. Interictal spikes were recorded during the fMRI acquisition on a MR-compatible battery-powered digital EEG system with 16 channels. The fMRI sequences were correlated off-line with the EEG spikes and analyzed with the software Statistical Parametrical Mapping SPM99. The fM…

MaleChildhood epilepsyElectroencephalographyEEG-fMRICentral nervous system diseaseEpilepsyOxygen ConsumptionImage Processing Computer-AssistedmedicineHumansIctalChildEvoked PotentialsMathematical ComputingBrain Mappingmedicine.diagnostic_testElectroencephalographySignal Processing Computer-AssistedImage Enhancementmedicine.diseaseEpilepsy RolandicMagnetic Resonance ImagingTemporal LobeFunctional imagingNeurologyChild PreschoolFemaleNeurology (clinical)Functional magnetic resonance imagingPsychologyNeuroscienceEpilepsia
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The first case of myoclonic epilepsy in a child with a de novo 22q11.2 microduplication

2011

Chromosome 22, particularly the q11.2 sub-band, has long been recognized as responsible for multiple congenital anomaly disorders. In particular, its susceptibility to subtle microdeletions or, more rarely, microduplications has been attributed to the presence of several low-copy repeats spanning the region as mediators of nonallelic homologous recombination that result in 22q11.2 rearrangements. While recent data suggest that the frequency of 22q11.2 microduplications could be approximately half of all deletions, now only 50 unrelated cases have been reported thus far. However, it is reasonable to suppose that microduplications of 22q11.2 may be largely undetected as a result of a less-dis…

MaleChromosomes Human Pair 22Non-allelic homologous recombinationEpilepsies MyoclonicMultiple congenital anomalyBiologyRAB36 genemyoclonic epilepsySettore MED/38 - Pediatria Generale E SpecialisticaChromosome DuplicationGene duplicationClinical heterogeneityGeneticsmedicineHumansChildIn Situ Hybridization FluorescenceGenetics (clinical)GeneticsComparative Genomic HybridizationFaciesmedicine.diseaseMild learning difficultiesdevelopmental delayPhenotypeSettore MED/03 - Genetica MedicaChild PreschoolMyoclonic epilepsynonallelic homologous recombinationChromosome 2222q11.2 microduplicationComparative genomic hybridizationAmerican Journal of Medical Genetics Part A
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Myocardial infarction following convulsive and nonconvulsive seizures

2009

Abstract Myocardial infarction (MI) has been rarely reported in association with seizures, and only of convulsive type. Methods We describe a series of five patients observed over a 4-year period, who presented MI immediately following seizures, either convulsive or nonconvulsive. Results Patient 1 had pre-existent coronary disease (CD) and presented multiple focal nonconvulsive seizures. Patient 2 had no CD, normal coronary angiography and presented secondary generalized convulsive seizures. Patient 3 had no history of CD, normal angiography and had a first single convulsive seizure. Patient 4 had severe CD and suffered from a single convulsive event. Patient 5 had a partial and a generali…

MaleCoronary angiographyGeneralized seizures; Myocardial infarction; Partial seizures; Sudden deathClinical NeurologySudden deathConvulsive seizureEpilepsySeizuresGeneralized seizuresmedicineHumansIn patientMyocardial infarctionAgedAged 80 and overPartial seizuresmedicine.diagnostic_testbusiness.industrySudden deathGeneral MedicineMiddle Agedmedicine.diseaseMyocardial infarctionConvulsive SeizuresNeurologyAnesthesiaAngiographyEpilepsy GeneralizedFemaleNeurology (clinical)businessSeizure
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Biallelic pathogenic variants in the lanosterol synthase gene LSS involved in the cholesterol biosynthesis cause alopecia with intellectual disabilit…

2019

International audience; Purpose Lanosterol synthase (LSS) gene was initially described in families with extensive congenital cataracts. Recently, a study has highlighted LSS associated with hypotrichosis simplex. We expanded the phenotypic spectrum of LSS to a recessive neuroectodermal syndrome formerly named alopecia with mental retardation (APMR) syndrome. It is a rare autosomal recessive condition characterized by hypotrichosis and intellectual disability (ID) or developmental delay (DD), frequently associated with early-onset epilepsy and other dermatological features. Methods Through a multicenter international collaborative study, we identified LSS pathogenic variants in APMR individu…

MaleDevelopmental DisabilitiesIntellectual disabilitycholesterol pathwayWhole Exome Sequencingchemistry.chemical_compoundMissense mutationAge of OnsetChildIntramolecular TransferasesGenetics (clinical)Exome sequencingGeneticsSanger sequencing0303 health sciencesbiologyLanosterol030305 genetics & heredityLSS3. Good healthPedigreeCholesterolPhenotypeintellectual disabilityChild PreschoolAllelic ImbalanceCongenital cataractssymbolsFemaleSqualeneearly-onset epileptic encephalopathy03 medical and health sciencessymbols.namesakeLanosterolCholesterol pathwayExome SequencingmedicineHumans030304 developmental biologyEpilepsyInfantAlopeciaalopeciamedicine.diseaseEarly-onset epileptic encephalopathychemistryMutationbiology.proteinHypotrichosis[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology[SDV.MHEP.DERM]Life Sciences [q-bio]/Human health and pathology/DermatologyLanosterol synthase
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Transcriptomic profile of epileptic children treated with ketogenic therapies.

2022

Background: Ketogenic dietary therapies (KDT) are used as a treatment in childhood epilepsy. However, their mechanism has not yet been established. The main objective of this study was to determine the changes in the transcriptomic profile induced by KDT in children with epilepsy in order to shed light on its possible mechanisms. Methods: Eight children with refractory epilepsy were enrolled in the study. Peripheral blood mononuclear cells were obtained before and after the children were treated with KDT for a minimum of 6 months. RNA was extracted and mRNA and miRNA profiling were performed and analyzed. Results: Our intervention with KDT significantly reduced the seizure number in seven o…

MaleDrug Resistant EpilepsyGeneral NeurosciencesynapsismirnomeInfantketogenic dietary therapiesNeurosciences. Biological psychiatry. NeuropsychiatryGeneral MedicineMicroRNAsketogenic dietChild PreschoolOutcome Assessment Health CareepilepsyHumansFemaleanticonvulsantChildDiet KetogenicTranscriptomeRC321-571Journal of integrative neuroscience
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