Search results for "estrogens"

showing 10 items of 160 documents

Sex steroids, carcinogenesis, and cancer progression

2004

The relationship between sex steroids and cancer has been studied for more than a century. Using an original intact cell analysis, we investigated sex steroid metabolism in a panel of human cancer cell lines, either hormone responsive or unresponsive, originating from human breast, endometrium, and prostate. We found that highly divergent patterns of steroid metabolism exist and that the catalytic preference (predominantly reductive or oxidative) is strictly associated with the steroid receptor status of cells. We explored intra-tissue concentrations and profiles of estrogens in a set of human breast tumors as compared to normal mammary tissues, also in relation to their estrogen receptor s…

Receptor StatusTime FactorsIntratumor estrogenCatecholsBreast cancer; Intratumor estrogens; Sex steroids; Adsorption; Androstenedione; Animals; Breast Neoplasms; Catalysis; Catechols; Cell Line Tumor; Chromatography High Pressure Liquid; Disease Progression; Estradiol; Estrogens; Humans; In Vitro Techniques; Ions; Kinetics; Models Biological; Neoplasms; Steroids; Time Factors; Biochemistry Genetics and Molecular Biology (all)Sex steroidmedicine.disease_causeEndometriumCatalysiBreast cancerNeoplasmsEstrogen Receptor StatusChromatography High Pressure LiquidEstradiolGeneral NeuroscienceSex hormone receptormedicine.anatomical_structureDisease ProgressionSteroidsBreast NeoplasmHumanmedicine.medical_specialtyTime FactorBreast NeoplasmsIn Vitro TechniquesBiologyModels BiologicalCatalysisGeneral Biochemistry Genetics and Molecular BiologyBreast cancerHistory and Philosophy of ScienceCell Line TumorInternal medicinemedicineAnimalsHumansIonSteroidKineticIonsBiochemistry Genetics and Molecular Biology (all)AnimalIn Vitro TechniqueAndrostenedioneCancerEstrogensmedicine.diseaseEstrogenKineticsEndocrinologySex steroidCatecholNeoplasmAdsorptionCarcinogenesis
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The risk for cardiovascular disease in women: from estrogens to selective estrogen receptor modulators.

2006

Cardiovascular disease, a generic denomination including coronary heart disease (CHD), stroke, and venous thromboembolic disease (VTED), has shown sensitivity to estrogens. The relative protection of women as compared with men has nourished a debate about a possible protective role for estrogens, but the prejudicial effects detected in clinical trials has created confusion on the risk/benefit ratio induced by hormone administration. The hypothesis that agonists distinct to estrogens might improve the effects associated with estrogens is at the base of the increasing interest on the role of selective estrogen receptor modulators (SERMs). There is a lack of definitive clearcut clinical data o…

Selective Estrogen Receptor ModulatorsCoronary DiseaseDiseaseBioinformaticsVenous thromboembolic diseaseSex FactorsRisk FactorsThromboembolismMedicineAnimalsHumansStrokeVenous Thrombosisbusiness.industryEstrogensmedicine.diseasePresent momentRatsClinical trialStrokeSelective estrogen receptor modulatorCardiovascular DiseasesWomen's HealthFemalebusinessVenous diseasehormones hormone substitutes and hormone antagonistsHormoneFrontiers in bioscience : a journal and virtual library
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Selective estrogen receptor modulators and risk for coronary heart disease.

2007

Coronary heart disease (CHD) is the leading cause of death in women in most countries. Atherosclerosis is the main biological process determining CHD. Clinical data support the notion that CHD is sensitive to estrogens, but debate exists concerning the effects of the hormone on atherosclerosis and its complications. Selective estrogen receptor modulators (SERMs) are compounds capable of binding the estrogen receptor to induce a functional profile distinct from estrogens. The possibility that SERMs may shift the estrogenic balance on cardiovascular risk towards a more beneficial profile has generated interest in recent years. There is considerable information on the effects of SERMs on disti…

Selective Estrogen Receptor Modulatorsmedicine.medical_specialtyEndotheliumEstrogen receptorCoronary DiseaseDiseaseCoronary Artery DiseaseRisk FactorsInternal medicinemedicineAnimalsHumansRaloxifenePlatelet activationCause of deathHemostasisbusiness.industryObstetrics and GynecologyEstrogensGeneral MedicineLipidsEndocrinologymedicine.anatomical_structureSelective estrogen receptor modulatorEndothelium Vascularbusinesshormones hormone substitutes and hormone antagonistsHormonemedicine.drugClimacteric : the journal of the International Menopause Society
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The adverse effects of estrogen and selective estrogen receptor modulators on hemostasis and thrombosis.

2012

Agonists of the estrogen receptor include estrogens and selective estrogen receptor modulators (SERMs). Both types of compounds increase the risk for thrombosis in the arterial and the venous tree. The magnitude of the effect is influenced by potency, which depends on the type of compound and the dose. The particulars of the process change in each territory. Atherosclerosis, which creates local inflammatory conditions, may favor thrombogenesis in arteries. A direct effect of estrogen agonists is also well endorsed at both arteries, as suggested from data with high-estrogenic contraceptives, and veins. Dose reduction has been proved to be an effective strategy, but there is debate on whether…

Selective Estrogen Receptor Modulatorsmedicine.medical_specialtymedicine.drug_classmedicine.medical_treatmentEstrogen receptorPharmacologyInternal medicineMedicineHumansStrokeHemostasisbusiness.industryEstrogensThrombosisHematologymedicine.diseaseThrombosisVenous thrombosisEndocrinologySelective estrogen receptor modulatorEstrogenHemostasisFemaleHormone therapyCardiology and Cardiovascular Medicinebusinesshormones hormone substitutes and hormone antagonistsSeminars in thrombosis and hemostasis
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Circulating miR-21, miR-146a and Fas ligand respond to postmenopausal estrogen-based hormone replacement therapy--a study with monozygotic twin pairs.

2014

Biological aging is associated with physiological deteriorations and its’ remodeling, which are partly due to changes in the hormonal profile. MicroRNAs are known to post-transcriptionally regulate various cellular processes associated with cell senescence; differentiation, replication and apoptosis. Measured from the serum, microRNAs have the potential to serve as noninvasive markers for diagnostics/prognostics and therapeutic targets. We analysed the association of estrogen-based hormone replacement therapy (HRT) with selected microRNAs and inflammation markers from the serum, leukocytes and muscle tissue biopsy samples obtained from 54-62 year-old postmenopausal monozygotic twins (n=11 p…

SenescenceAdultmedicine.medical_specialtyAgingFas Ligand Proteinmedicine.drug_classmedicine.medical_treatmentMonozygotic twinInflammationApoptosisBiologyta3111Fas ligand“Inflamm-aging”Internal medicinemicroRNAmedicineestrogenHumansmicrornasMuscle SkeletalHormone therapyCellular SenescenceInflammationmicroRNAEstrogen Replacement TherapyapoptosisHormone replacement therapy (menopause)ta3141Cell DifferentiationEstrogenstwinsTwins MonozygoticMiddle AgedPostmenopauseAgeinghormone replacement therapyMicroRNAsEndocrinologyEstrogenFemalemedicine.symptomBiomarkersDevelopmental BiologyHormoneMechanisms of ageing and development
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Gathering of aging and estrogen withdrawal in vascular dysfunction of senescent accelerated mice.

2010

The aim of this work was to characterize a mouse model of experimental menopause and cardiovascular aging that closely reflects menopause in women. Senescence accelerated mouse (SAM)-Resistant type 1 (SAMR1, n=30) and SAM-Prone type 8 (SAMP8, n=30) were separated at 5months of age into three groups: 1) sham-operated (Sham); 2) ovariectomized (Ovx); and 3) ovariectomized chronically-treated with estrogen (Ovx+E2). Contractile responses to KCl (60mM) and thromboxane A(2) were greater in aorta from SAMP8 mice compared with SAMR1 in all groups. Neither ovariectomy nor estrogen replacement modified the contractile responses from SAMR1 mice. Conversely, in Ovx SAMP8 the increased maximal contract…

SenescenceAgingmedicine.medical_specialtyEndotheliummedicine.drug_classThromboxaneOvariectomyVasodilator AgentsDown-RegulationIn Vitro TechniquesNitric OxideBiochemistryReceptors Thromboxane A2 Prostaglandin H2Thromboxane A2chemistry.chemical_compoundMiceEndocrinologyInternal medicineGeneticsmedicineAnimalsVasoconstrictor AgentsEnzyme InhibitorsMolecular BiologyAortaEstradiolChemistryEstrogen Replacement TherapyAging PrematureEstrogensCell Biologymedicine.diseaseAcetylcholineMenopauseVasodilationEndocrinologymedicine.anatomical_structureNG-Nitroarginine Methyl EsterEstrogenVasoconstriction15-Hydroxy-11 alpha9 alpha-(epoxymethano)prosta-513-dienoic AcidOvariectomized ratBlood VesselsFemalehormones hormone substitutes and hormone antagonistsAcetylcholinemedicine.drugExperimental gerontology
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Postmenopausal hormone replacement therapy modifies skeletal muscle composition and function: a study with monozygotic twin pairs

2009

We investigated whether long-term hormone replacement therapy (HRT) is associated with mobility and lower limb muscle performance and composition in postmenopausal women. Fifteen 54- to 62-yr-old monozygotic female twin pairs discordant for HRT were recruited from the Finnish Twin Cohort. Habitual (HWS) and maximal (MWS) walking speeds over 10 m, thigh muscle composition, lower body muscle power assessed as vertical jumping height, and maximal isometric hand grip and knee extension strengths were measured. Intrapair differences (IPD%) with 95% confidence intervals (CI) were calculated. The mean duration of HRT use was 6.9 ± 4.1 yr. MWS was on average 7% (0.9 to 13.1%, P = 0.019) and muscle…

SenescenceAgingmedicine.medical_specialtyvaihdevuodetPhysiologyMonozygotic twinWalkingIsometric ContractionPhysiology (medical)Internal medicineIkääntymienmedicineHumansMuscle Skeletalmuscle powersukupuolihormonitHand Strengthbusiness.industryEstrogen Replacement TherapySkeletal muscleEstrogenslihaksen voimantuottotehoTwins MonozygoticMiddle Agedmedicine.diseaseTwin studyTwin Studies as TopicMenopauseEndocrinologymedicine.anatomical_structureTransgender hormone therapyAgeingTwin Studies as TopicFemaleMenopauseTomography X-Ray ComputedbusinessMuscle ContractionlihasvoimaJournal of Applied Physiology
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Nutritional prevention of cognitive decline and dementia.

2018

Cognitive impairment results from a complex interplay of many factors. The most important independent predictor of cognitive decline is age but other contributing factors include demographic, genetic, socio-economic, and environmental parameters, including nutrition. The number of persons with cognitive decline and dementia will increase in the next decades in parallel with aging of the world population. Effective pharmaceutical treatments for age-related cognitive decline are lacking, emphasizing the importance of prevention strategies. There is extensive evidence supporting a relationship between diet and cognitive functions. Thus, nutritional approaches to prevent or slow cognitive decli…

Settore MED/09 - Medicina InternaCurcuminPhytoestrogensAntioxidantsCatechinHealthy AgingCaffeineHealthy Aging - ReviewFatty Acids Omega-3AutophagyHumansoxidative stressCognitive DysfunctionMagnesiumChocolateGarlicInflammationTeaagingGinkgo bilobaVitaminscognitive declineDietnutritionResveratrolAlzheimerCentral Nervous System StimulantsDementiaAlzheimer cognitive decline aging diet nutrition inflammation oxidative stressPhytotherapyActa bio-medica : Atenei Parmensis
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Genotoxic and endocrine activities of bis(hydroxyphenyl)methane (bisphenol F) and its derivatives in the HepG2 cell line

2008

International audience; Human can be exposed to bis(hydroxyphenyl)methane (bisphenol F or BPF) and its derivatives as environment and food's contaminants. This study was investigated to identify and to compare toxic potency of BPF, BFDGE, and two of BPF metabolites using in vitro methods. BPF did not induce any genic mutation in bacteria when the Ames test was performed according to the OECD guideline. In contrast, using Human cell lines and Comet assay, we demonstrated that BPF and Bisphenol F Diglycidyl Ether (BFDGE) were effective on HepG2 cell DNA fragmentation at non-cytotoxic concentrations. DHB was also positive but at higher concentrations, near its limit of solubility. Neither BPF,…

StereochemistryCell SurvivalEndocrine activitiesOxidative BPF metabolitesBisphenol F Diglycidyl Ether (BFDGE)[SDV.TOX.TCA]Life Sciences [q-bio]/Toxicology/Toxicology and food chain010501 environmental sciencesEndocrine DisruptorsToxicologymedicine.disease_causeTransfection01 natural sciencesAmes testCell Line03 medical and health scienceschemistry.chemical_compoundHuman cell linesmedicineHumansEstrogens Non-SteroidalBenzhydryl CompoundsBisphenol F (BPF)Bisphenol A diglycidyl ether030304 developmental biology0105 earth and related environmental sciences0303 health sciencesMicronucleus TestsMutagenicity TestsAndrogen AntagonistsMolecular biologyIn vitro3. Good healthComet assaychemistryCell cultureMicronucleus testDNA fragmentationComet AssayGenotoxicityGenotoxicityMutagens
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Management of urinary incontinence in postmenopausal women: An EMAS clinical guide.

2020

INTRODUCTION: The prevalence of urinary incontinence and of other lower urinary tract symptoms increases after the menopause and affects between 38 % and 55 % of women aged over 60 years. While urinary incontinence has a profound impact on quality of life, few affected women seek care. AIM: The aim of this clinical guide is to provide an evidence-based approach to the management of urinary incontinence in postmenopausal women. MATERIALS AND METHODS: Literature review and consensus of expert opinion. SUMMARY RECOMMENDATIONS: Healthcare professionals should consider urinary incontinence a clinical priority and develop appropriate diagnostic skills. They should be able to identify and manage a…

Stress incontinencemedicine.medical_specialtyAgingUrinary incontinenceUrinary incontinenceGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineQuality of lifeLower urinary tract symptomsmedicineHumans030212 general & internal medicineMidurethral SlingsAging; Estrogens; Menopause; Midurethral slings; Urinary incontinence030219 obstetrics & reproductive medicinePostmenopausal womenbusiness.industryObstetrics and GynecologyEstrogensmedicine.diseasePostmenopauseMenopauseUrinary IncontinenceSacral nerve stimulationPhysical therapyFemaleMidurethral slingsmedicine.symptomMenopausebusiness
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