Search results for "ethanol"

showing 10 items of 2041 documents

Patterns of Innate or Acquired Resistance to Anticancer Drugs: Our Experience to Overcome It

2021

Drug resistance, which is often of a multiple type, can be defined as the ability of cancer cells to obtain resistance to both conventional and novel chemotherapy agents. It remains a major problem to solve in cancer therapy. The mechanisms of resistance are multifactorial, and in our cellular models of acute myeloid leukemia, hepatocellular carcinoma, and triple-negative breast cancer, it involves the NF-κB pathway. In our opinion, multitarget molecules can be considered as privileged compounds capable of attacking and reversing the resistant phenotype. In the phenomena of both innate and acquired drug resistance that we have been studying since 1998 to today and up to 2016 under the guida…

Cancer ResearchAntineoplastic AgentsApoptosisPhosphatidylethanolamine Binding ProteinDrug resistanceMetastasisBreast cancerdrug resistance P-glycoprotein IAP NF-κBNeoplasmsHumansMedicineATP Binding Cassette Transporter Subfamily B Member 1Transcription factorYY1 Transcription FactorP-glycoproteinbiologybusiness.industryKinaseNF-kappa BMyeloid leukemiamedicine.diseaseDrug Resistance NeoplasmCancer cellSettore BIO/14 - Farmacologiabiology.proteinCancer researchbusinessCritical Reviews™ in Oncogenesis
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Autophagy and mitochondrial alterations in human retinal pigment epithelial cells induced by ethanol: implications of 4-hydroxy-nonenal

2014

Retinal pigment epithelium has a crucial role in the physiology and pathophysiology of the retina due to its location and metabolism. Oxidative damage has been demonstrated as a pathogenic mechanism in several retinal diseases, and reactive oxygen species are certainly important by-products of ethanol (EtOH) metabolism. Autophagy has been shown to exert a protective effect in different cellular and animal models. Thus, in our model, EtOH treatment increases autophagy flux, in a concentration-dependent manner. Mitochondrial morphology seems to be clearly altered under EtOH exposure, leading to an apparent increase in mitochondrial fission. An increase in 2′,7′-dichlorofluorescein fluorescenc…

Cancer ResearchImmunologyApoptosisRetinal Pigment EpitheliumMitochondrionBiologymedicine.disease_causeCell LineLipid peroxidationCellular and Molecular Neurosciencechemistry.chemical_compoundRetinal DiseasesmedicineAutophagyHumanschemistry.chemical_classificationReactive oxygen speciesAldehydesRetinal pigment epitheliumEthanolAutophagyRetinalEpithelial CellsCell BiologyCell biologyMitochondriaOxidative Stressmedicine.anatomical_structurechemistryBiochemistryMitochondrial fissionOriginal ArticleReactive Oxygen SpeciesOxidative stressCell Death & Disease
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Ethanol-Mediated Stress Promotes Autophagic Survival and Aggressiveness of Colon Cancer Cells via Activation of Nrf2/HO-1 Pathway

2019

Epidemiological studies suggest that chronic alcohol consumption is a lifestyle risk factor strongly associated with colorectal cancer development and progression. The aim of the present study was to examine the effect of ethanol (EtOH) on survival and progression of three different colon cancer cell lines (HCT116, HT29, and Caco-2). Our data showed that EtOH induces oxidative and endoplasmic reticulum (ER) stress, as demonstrated by reactive oxygen species (ROS) and ER stress markers Grp78, ATF6, PERK and, CHOP increase. Moreover, EtOH triggers an autophagic response which is accompanied by the upregulation of beclin, LC3-II, ATG7, and p62 proteins. The addition of the antioxidant N-acetyl…

Cancer Researchendocrine systemautophagyHO-1Colon cancer cellmedicine.disease_causelcsh:RC254-282ArticleNrf2Downregulation and upregulationSettore BIO/10 - Biochimicamedicinechemistry.chemical_classificationReactive oxygen speciesATF6Endoplasmic reticulumAutophagylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensHeme oxygenaseOncologychemistryCancer researchUnfolded protein responseER strecolon cancer cellsethanolMMPsER stressOxidative stressCancers
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Analysis of Possible Mechanisms Accounting for Raf-1 Kinase Inhibitor Protein Downregulation in Hepatocellular Carcinoma

2012

Abstract Raf-1 kinase inhibitor protein (RKIP) is a tumor and metastasis suppressor that promotes drug-induced apoptosis in cancer cells. It is frequently downregulated, both at the mRNA and protein level, in hepatocellular carcinoma (HCC), but the mechanisms leading to this reduction are obscure. We sequenced the whole RKIP gene in three human HCC cell lines (HA22T/VGH, HepG2, and Hep3B), and in five clinical HCC samples, but could not find any gene variant that might account for their low RKIP levels. We also examined whether gene methylation may be responsible for the altered RKIP expression. No methylation of the RKIP gene was found in the tumor samples, while among the cell lines only …

Carcinoma HepatocellularLeupeptinsAntineoplastic AgentsPhosphatidylethanolamine Binding ProteinRKIP (Raf-1 kinase inhibitor protein) hepatocellular carcinomaBiologyBiochemistryDownregulation and upregulationRNA interferenceCell Line TumorGeneticsHumansMetastasis suppressorPromoter Regions GeneticMolecular BiologyRegulation of gene expressionKinaseLiver NeoplasmsHep G2 CellsMethylationDNA Methylationdigestive system diseasesGene Expression Regulation NeoplasticMicroRNAsMutationCancer cellDNA methylationAzacitidineSettore BIO/14 - FarmacologiaCancer researchMolecular MedicineRNA InterferenceBiotechnologyOMICS: A Journal of Integrative Biology
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Identification and quantification of carotenoids including geometrical isomers in fruit and vegetable juices by liquid chromatography with ultraviole…

2004

A method was established for the identification and quantification of carotenoids including geometrical isomers in fruit and vegetable juices by liquid chromatography with an ultraviolet−diode array detector, using a C18 Vydac 201TP54 column. The mobile phase used was the ternary methanol mixture (0.1 M ammonium acetate), tert-butyl methyl ether and water, in a concentration gradient, and a temperature gradient was applied. Retinol palmitate was added as an internal standard. An extraction process (ethanol/hexane, 4:3, v/v) was performed, followed by saponification with diethyl ether/methanolic KOH (0.1%, w/v, BHT) (1:1, v/v) for 0.5 h at room temperature. Seventeen different (cis and trans…

Carrot juiceCitrusChromatographyExtraction (chemistry)food and beveragesGeneral ChemistryHigh-performance liquid chromatographyCarotenoidsDaucus carotaHexaneBeverageschemistry.chemical_compoundchemistryFruitVegetablesMethanolDiethyl etherGeneral Agricultural and Biological SciencesAmmonium acetateSaponificationChromatography High Pressure LiquidJournal of agricultural and food chemistry
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"Partial oxidation of methane and methanol on FeOx-, MoOx- and FeMoOx -SiO2 catalysts prepared by sol-gel method: a comparative study"

2020

[EN] FeOx-, MoOx and FeMoOx-SiO2 materials prepared by a sol-gel procedure have been evaluated as catalysts for the partial oxidation of methane and methanol. The effect of decreasing the pH of the synthesis gel on the chemical nature of FeOx and MoOx species has been investigated. Characterization results show that low pH improves the dispersion of metal oxide species present in SiO2 matrix. For FeOx/SiO2 materials, the presence of dispersed FeOx species (rather than bulk Fe2O3) improves the selectivity to formaldehyde in the partial oxidation of methane and methanol. For FeMoOx/SiOx catalysts, dispersed species favor the selectivity to formaldehyde only for methane oxidation. In contrast,…

CatalizadorFormaldehydeOxideEstudio comparativo010402 general chemistry01 natural sciencesAldehydeCatalysisMethaneCatalysischemistry.chemical_compoundFormaldehydeIron oxidePartial oxidationPhysical and Theoretical Chemistrychemistry.chemical_classification010405 organic chemistryProcess Chemistry and TechnologyMethanolFormaldehídoMolybdenum oxide0104 chemical scienceschemistryChemical engineeringAnaerobic oxidation of methaneMethanolMethane
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Sphingomyelin inhibition of Ciona intestinalis (Tunicata) cytotoxic hemocytes assayed against sheep erythrocytes

1995

Hemocytes from the ascidian, Ciona intestinalis, are capable of lysing erythrocytes in vitro following cell membrane contact. With the aim of examining the mechanism of cytotoxicity, we performed inhibition experiments with lipid components of erythrocyte membranes. Cholesterol is not an inhibitor, whereas, among the phospholipids tested, (sphingomyelin, phosphatidylcholine, phosphatidylserine, phosphatidylethanolamine) sphingomyelin inhibits the hemolytic activity of hemocytes. However, thin layer chromatography showed that sphingomyelinase activity was not contained in the chloroform-methanol extracts from hemocyte debris. The inhibition capacity of the components ceramide and phosphorylc…

Cell ExtractsHemocytesCiona intestinaliCytotoxicityHemocyteTunicate;Cell membraneHemolysin Proteinschemistry.chemical_compoundSphingomyelin inhibition;InvertebratePhospholipidsCiona intestinalis;biologyInvertebrate;PhosphatidylserineCiona intestinalisSphingomyelinsCytotoxicity;Sheep erythrocytesCholesterolSphingomyelin Phosphodiesterasemedicine.anatomical_structureBiochemistrylipids (amino acids peptides and proteins)SphingomyelinHemolysis inhibitionSphingomyelin inhibitionCeramideHemolysis inhibition;ImmunologyTunicateHemolysisMembrane LipidsPhosphatidylcholinemedicineAnimalsCiona intestinalisPhosphatidylethanolamineSheepPhosphorylcholineCell MembraneOsmolar ConcentrationCytotoxicity Tests Immunologicbiology.organism_classificationCulture MediaHemocytes;chemistryChromatography Thin LayerDevelopmental Biology
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Mitogenic effects of phospholipase D and phosphatidic acid in transiently permeabilized astrocytes: effects of ethanol.

2003

Investigations of lipid-mediated signalling pathways are often limited by a lack of methods for the intracellular delivery of lipid messengers. We established a procedure for the transient permeabilization of astrocytes by an oxygen-insensitive mutant of streptolysin-O (SLO) to investigate the participation of the phospholipase D (PLD) signalling pathway in astroglial cell proliferation. Exogenous PLD, when incubated in the presence of SLO, caused an increase in DNA synthesis (measured by thymidine incorporation) which was completely suppressed by ethanol (0.3%, v/v). In parallel experiments, phosphatidic acid also induced a dose-dependent mitogenic response which, however, was not affected…

Cell Membrane PermeabilityIndolesmedicine.drug_classPhosphatidic AcidsBiologyBiochemistryDiglyceridesCellular and Molecular Neurosciencechemistry.chemical_compoundBacterial ProteinsmedicinePhospholipase DAnimalsEnzyme InhibitorsProtein kinase ACells CulturedDiacylglycerol kinaseDNA synthesisDose-Response Relationship DrugEthanolPhospholipase DPhosphatidic acidDNAProtein kinase inhibitorRatschemistryBiochemistryAstrocytesStreptolysinslipids (amino acids peptides and proteins)Signal transductionMitogensIntracellularCell DivisionSignal TransductionJournal of neurochemistry
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Endocytosis in cultured neurons is altered by chronic alcohol exposure.

2010

Endocytosis is required for many cellular pivotal processes, including membrane recycling, nutrient uptake, and signal transduction. This complex process is particularly relevant in polarized cells, such as neurons. Previous studies have demonstrated that alcohol alters intracellular traffic, including endocytosis, in several cell types. However, information on the effect of chronic alcohol exposure on this process in neurons is scarce. As an approach, we investigated the effect of alcohol exposure on the internalization of two widely used endocytic markers, albumin and transferrin, in developing hippocampal neurons in primary culture. The effect of this treatment on the levels of several r…

Cell signalingRHOAmedia_common.quotation_subjectEndocytic cycleNerve Tissue ProteinsEndosomesToxicologyEndocytosisClathrinHippocampusAlbuminsAnimalsInternalizationCells Culturedmedia_commonNeuronsbiologyEthanolTransferrinCentral Nervous System DepressantsBiological TransportActin cytoskeletonClathrinEndocytosisCell biologyRatsbiology.proteinFemaleIntracellularBiomarkersToxicological sciences : an official journal of the Society of Toxicology
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Comparison of the effects of chloral hydrate and trichlorethanol on the EEG of the isolated perfused rat brain.

1973

An isolated perfused rat brain preparation was used to compare the effects of chloral hydrate and its metabolite trichloroethanol on the EEG. The concentrations of chloral hydrate and trichloroethanol in the perfusion medium ranged from 1.5 to 5.5 mM. 5, 10, 15, and 30 min after the beginning of the perfusions EEG-recordings were taken. The recordings were evaluated both by a descriptive method and by a simple quantitative appraoch, counting the waves with an amplitude greater than 50 microvolts and averaging this value for a period of 1 sec. The following results were obtained: Both drugs exhibited CNS depressant activity. Between 5 and 10 min of perfusion the effect of trichloroethanol wa…

Central Nervous SystemMaleTime FactorsMetaboliteCns depressantPharmacology toxicologyChloral hydrateElectroencephalographyIn Vitro Techniqueschemistry.chemical_compoundmedicineAnimalsChloral HydratePharmacologymedicine.diagnostic_testEthanolChemistryHydrocarbons HalogenatedBrainElectroencephalographyGeneral MedicineRat brainRatsPerfusionAnesthesiaChlorinePerfusionmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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