Search results for "fibrinolysi"

showing 10 items of 97 documents

Effect of gemfibrozil treatment on fibrinolysis system in patients with hypertriglyceridemia

1992

Abstract The effect of gemfibrozil on lipidic, coagulative, and fibrinolytic pattern was studied in 20 patients with primary type IV hyperlipoproteinemia. After a 4-week stabilization period during which administration of lipid-lowering drugs was stopped and an isocaloric diet (20% protein, 30% fat, and 50% carbohydrates) was prescribed, 20 patients (12 men and 8 women; mean age, 38 ± 4 years; body mass index, 23.4 ± 1.5) suffering from primary hypertriglyceridemia were included in this study and treated for a 12-week period with gemfibrozil (600 mg BID). Every 4 weeks the following parameters were checked: glycemia, triglycerides, total cholesterol, high density lipoprotein-cholesterol, to…

Pharmacologymedicine.medical_specialtyFactor VIIbusiness.industrymedicine.medical_treatmentHypertriglyceridemiaAntithrombinmedicine.diseaseFibrinogenchemistry.chemical_compoundEndocrinologychemistryHemostasisInternal medicineFibrinolysismedicineGemfibrozillipids (amino acids peptides and proteins)Pharmacology (medical)businessPlasminogen activatormedicine.drugCurrent Therapeutic Research
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Vascular atherosclerotic disease: Evaluation of white blood cell rheology and metabolism after acute intravenous administration of defibrotide

1994

Defibrotide is a single-strand polydeoxyribonucleotide, obtained by controlled depolymerization of DNA extracted from mammalian organs. It stimulates vascular prostacyclin production and secretion (1-3) and enhances fibrinolysis (1, 4-6). Several clinical trials have evidentiated that defibrotide is effective in the prophylaxis of deep vein thrombosis and in the treatment of peripheral obliterative arterial disease (for a review see ref. 7). There are several reports concerning, in vitro and in vivo, the dose-dependent activity of defibrotide on the polymorphonuclear leucocytes (PMN). From these results, the molecule influences the PMN adhesivity (8, 9) and especially PMN activation, with s…

PhysiologyVascular diseaseSuperoxidebusiness.industrymedicine.medical_treatmentProstacyclinHematologyDefibrotidePharmacologymedicine.diseaseThrombosischemistry.chemical_compoundmedicine.anatomical_structurechemistryIn vivoPhysiology (medical)White blood cellFibrinolysisImmunologymedicineCardiology and Cardiovascular Medicinebusinessmedicine.drug
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Activation of the first component of complement, C1: comparison of the effect of sixteen different enzymes on serum C1.

1983

In this study, the effect of sixteen different enzymes on serum C1 and its subcomponents was investigated. The sixteen enzymes could be divided into three groups. First, enzymes which activate native C1: trypsin (optimal concentration 2.4 x 10(-4) mM); alpha-chymotrypsin (2.3 x 10(3) mM); thrombin (1.0 x 10(-5) mM); plasmin (1.9 x 10(-5) mM); elastase (5.8 x 10(-5) mM); pronase (3.0 x 10(-6) mM). All these enzymes are serine esterase and activate native serum C1 bound to EAC4 at the given concentration within 10 min at 30 degrees C. Furthermore, native C1 inhibited by a pentosanpolysulfoester, Sp54, is unable to undergo the internal activation but can be externally activated by the serine e…

PlasminComplement Activating EnzymesImmunologyGuinea PigsDose-Response Relationship ImmunologicPronaseSerinechemistry.chemical_compoundComplement C1medicineImmunology and AllergyAnimalsHumansTrypsinFibrinolysinComplement Activationchemistry.chemical_classificationPentosan Sulfuric PolyesterbiologyHematologyTrypsinCarboxypeptidaseKineticsEnzymeBiochemistrychemistrybiology.proteinCollagenaseCattleRabbitsLysozymemedicine.drugPeptide HydrolasesImmunobiology
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Contribution of sinusoidal endothelial liver cells to liver fibrosis: expression of transforming growth factor-beta 1 receptors and modulation of pla…

1993

Transforming growth factor-beta 1 is an important cytokine in the pathophysiology of liver fibrosis, stimulating the production of extracellular matrix. Whether this cytokine can also control the degradation of matrix proteins in liver cells has not been investigated. Because plasmin is an important protease for the degradation of matrix glycoproteins, we investigated whether sinusoidal endothelial liver cells could contribute to fibrosing liver disease through the modulation of plasmin-generating enzymes in response to transforming growth factor-beta 1. Sinusoidal endothelial cells from guinea pig liver were investigated in pure monolayer culture. Using 125I-labelled transforming growth fa…

PlasminGuinea PigsBiologyLiver Cirrhosis Experimental03 medical and health sciencesPlasminogen Activators0302 clinical medicineCell surface receptorTransforming Growth Factor betaPlasminogen Activator Inhibitor 1medicineAnimalsFibrinolysinCells Cultured030304 developmental biology0303 health sciencesHepatology3. Good healthCell biologyFibronectinEndothelial stem cellBiochemistryLiverTransforming growth factor beta 3Cell culturebiology.protein030211 gastroenterology & hepatologyFemaleEndothelium VascularPlasminogen activatorReceptors Transforming Growth Factor betamedicine.drugTransforming growth factorHepatology (Baltimore, Md.)
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The Internal Dynamics of Fibrinogen and Its Implications for Coagulation and Adsorption

2015

Fibrinogen is a serum multi-chain protein which, when activated, aggregates to form fibrin, one of the main components of a blood clot. Fibrinolysis controls blood clot dissolution through the action of the enzyme plasmin, which cleaves fibrin at specific locations. Although the main biochemical factors involved in fibrin formation and lysis have been identified, a clear mechanistic picture of how these processes take place is not available yet. This picture would be instrumental, for example, for the design of improved thrombolytic or anti-haemorrhagic strategies, as well as, materials with improved biocompatibility. Here, we present extensive molecular dynamics simulations of fibrinogen w…

Plasminmedicine.medical_treatmentAllosteric regulationPlasma protein bindingMolecular Dynamics SimulationFibrinogenFibrinCellular and Molecular NeuroscienceFibrinolysisGeneticsmedicineHumanslcsh:QH301-705.5Molecular BiologyBlood CoagulationEcology Evolution Behavior and SystematicsIntegrin bindingEcologybiologyChemistryComputational BiologyFibrinogenlcsh:Biology (General)Computational Theory and MathematicsCoagulationBiochemistryModeling and Simulationbiology.proteinAdsorptionmedicine.drugResearch ArticleProtein BindingPLoS Computational Biology
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Hypercoagulability during pregnancy: evidences for a thrombophilic state

2006

The development of thrombotic disorders is a major threat for young women during pregnancy. It is one of the main causes of pregnancy-related disorders, which may also result in harm for the conceptus. Successful pregnancies require an even balance of coagulation and fibrinolysis, in order to secure stabilization of the basal plate as well as adequate placental perfusion. Broad spectrum assays which measure a range of thrombin/fibrin formation in serum have become an established means of identifying activation of blood coagulation and/or fibrinolysis. There is considerable interest in the application of these assays to the diagnosis of other hypercoagulable states, such as thrombophilia dur…

PregnancyFibrinolysisPregnancy Complications HematologicFibrinolysiHumansThrombophiliaFemaleBlood CoagulationHuman
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High-Dose ϵ-Aminocaproic Acid Versus Aprotinin: Antifibrinolytic Efficacy in First-Time Coronary Operations

1998

The antifibrinolytic efficacy of a high-dose regimen of epsilon-aminocaproic acid (epsilon-ACA) was compared with aprotinin in first-time coronary operations.In a prospective, double-blinded, randomized study, 20 patients received high-dose epsilon-ACA (10 g both as a loading and cardiopulmonary bypass priming dose, 2.5 g/h until 4 hours after protamine), and another 20 patients received aprotinin (2 x 10(6) KIU [280 mg] for loading and priming, 0.5 x 10(6) KIU/h [70 mg/h]). Ten untreated patients served as controls.Both agents reduced postoperative levels of thrombin/antithrombin III complexes, D-dimers, fibrin degradation products, free plasma hemoglobin (epsilon-ACA versus aprotinin, p =…

Pulmonary and Respiratory MedicineAntifibrinolyticmedicine.drug_classHematocritHemostaticsFibrinlaw.inventionBlood Transfusion AutologousAprotininThrombinDouble-Blind MethodlawMyocardial RevascularizationmedicineCardiopulmonary bypassHumansBlood TransfusionAprotininProspective StudiesBlood CoagulationCardiopulmonary Bypassbiologymedicine.diagnostic_testbusiness.industryFibrinolysisAntithrombinAntifibrinolytic AgentsHematocritAnesthesiaAminocaproic Acidbiology.proteinSurgeryAminocaproic acidCardiology and Cardiovascular Medicinebusinessmedicine.drugThe Annals of Thoracic Surgery
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Reoperation and the centrifugal pump?

1992

Postperfusion syndrome is still a problem in long cardiac operations using extracorporeal circulation (ECC). To evaluate whether or not centrifugal blood pumping during open heart surgery is beneficial, a randomized, prospective study was undertaken of 50 consecutive patients undergoing elective coronary artery bypass grafting. The patients were divided into two groups of 25 each. In group 1 a centrifugal pump (Biomedicus) was used as arterial blood pump, while in group 2 a roller pump (Stockert) was used. The two groups did not differ significantly and the number of variables during surgery was kept low (identical perfusion set, two surgeons, minimal cardiotomy suction). The parameters stu…

Pulmonary and Respiratory Medicinebusiness.industrymedicine.medical_treatmentExtracorporeal circulationPeristaltic pumpPostperfusion syndromeGeneral Medicinelaw.inventionmedicine.anatomical_structurelawAnesthesiaFibrinolysisCardiopulmonary bypassMedicineArterial bloodSurgeryCardiology and Cardiovascular MedicinebusinessPerfusionArteryEuropean Journal of Cardio-Thoracic Surgery
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Systemic Thrombolytic Therapy for Acute Pulmonary Embolism: Who Is a Candidate?

2017

Pulmonary embolism (PE) is a major cause of both acute and long-term morbidity for a large number of patients worldwide, and massive PE is frequently fatal. Right ventricular (RV) dysfunction is a key determinant of prognosis in the acute phase of PE. Patients with clinically overt RV failure, that is, with cardiogenic shock or persistent hypotension at presentation (acute high-risk PE), are clearly in need of immediate reperfusion treatment with systemic thrombolysis or, alternatively, surgical or catheter-directed techniques. On the other hand, within the large group of patients presenting without hemodynamic instability, the bleeding risk of full-dose intravenous thrombolytic treatment h…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyVentricular Dysfunction Rightmedicine.medical_treatmentHemodynamicsSalvage therapy030204 cardiovascular system & hematologyCritical Care and Intensive Care Medicine03 medical and health sciences0302 clinical medicineFibrinolytic AgentsInternal medicineFibrinolysismedicineHumansThrombolytic TherapyDecompensation030212 general & internal medicineWatchful WaitingIntensive care medicineSalvage Therapybusiness.industryCardiogenic shockAnticoagulantsThrombolysisPrognosismedicine.diseasePulmonary embolismAcute DiseaseCardiologyPulmonary EmbolismbusinessWatchful waitingSeminars in Respiratory and Critical Care Medicine
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Peripheral artery disease: potential role of ACE-inhibitor therapy

2008

Giuseppe Coppola, Giuseppe Romano, Egle Corrado, Rosa Maria Grisanti, Salvatore NovoDepartment of Internal Medicine, Cardiovascular and Nephro-Urological Diseases, Chair of Cardiovascular Disease, University of Palermo, Palermo, ItalyAbstract: Subjects with peripheral arterial disease (PAD) of the lower limbs are at high risk for cardiovascular and cerebrovascular events and the prevalence of coronary artery disease in such patients is elevated. Recent studies have shown that regular use of cardiovascular medications, such as therapeutic and preventive agents for PAD patients, seems to be promising in reducing long-term mortality and morbidity. The angiotensin-converting-enzyme (ACE) system…

Ramiprillcsh:Diseases of the circulatory (Cardiovascular) systemmedicine.medical_specialtyPathologyACE inhibitorsEndocrinology Diabetes and MetabolismBradykininAngiotensin-Converting Enzyme InhibitorsReviewDiseaseendothelial dysfunctionCoronary artery diseasechemistry.chemical_compoundperipheral arterial diseaseInternal medicineHumansMedicinePharmacology (medical)Endothelial dysfunctionCell ProliferationSubclinical infectionPeripheral Vascular Diseasesbusiness.industryFibrinolysisPublic Health Environmental and Occupational HealthCardiovascular AgentsPeripheral artery disease ACE-inhibitorintermittent claudicationHematologyGeneral Medicinemedicine.diseaseSettore MED/11 - Malattie Dell'Apparato Cardiovascolareperipheral arterial disease.Oxidative StressTreatment OutcomeLower ExtremitychemistryCardiovascular Diseaseslcsh:RC666-701ACE-inhibitorsACE inhibitorCardiovascular agentCardiologyEndothelium VascularatherosclerosisCardiology and Cardiovascular Medicinebusinessmedicine.drugVascular Health and Risk Management
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