Search results for "fibroma"

showing 10 items of 119 documents

NEUROFIBROMATOSI DI TIPO 1 ED IPOTIROIDISMO SUB-CLINICO DA TIROIDITE AUTOIMMUNE: DESCRIZIONE DI UN CASO IN ETA’ PEDIATRICA

2011

NEUROFIBROMATOSI DI TIPO 1IPOTIROIDISMO SUB-CLINICOTIROIDITE AUTOIMMUNE
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Identificazione di due nuove mutazioni del gene NF1 in pazienti affetti da neurofibromatosi

2009

NF1 gene neurofibromatosi
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Neuroendocrine neoplasms in the context of inherited tumor syndromes: a reappraisal focused on targeted therapies

2022

Purpose Neuroendocrine neoplasms can occur as part of inherited disorders, usually in the form of well-differentiated, slow-growing tumors (NET). The main predisposing syndromes include: multiple endocrine neoplasias type 1 (MEN1), associated with a large spectrum of gastroenteropancreatic and thoracic NETs, and type 4 (MEN4), associated with a wide tumour spectrum similar to that of MEN1; von Hippel-Lindau syndrome (VHL), tuberous sclerosis (TSC), and neurofibromatosis 1 (NF-1), associated with pancreatic NETs. In the present review, we propose a reappraisal of the genetic basis and clinical features of gastroenteropancreatic and thoracic NETs in the setting of inherited syndromes with a s…

Neuroendocrine neoplasmMEN4EndocrinologyMEN1Endocrinology Diabetes and MetabolismVon Hippel–Lindau (VHL) syndromeTargeted therapieVon Hippel–Lindau (VHL) syndrome.Neurofibromatosis 1 (NF-1)Tuberous sclerosis (TSC)
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Current standing and frontiers of gene therapy for meningiomas

2013

Meningiomas are among the most common intracranial tumors. The treatment of choice for these lesions is complete resection, but in 50% of cases it is not achieved due to tumor location and/or surgical morbidities. Moreover, benign meningiomas have high recurrence rates of up to 32% in long-term follow-up. Molecular analyses have begun to uncover the genetics behind meningiomas, giving rise to potential genetics-based treatments, including gene therapy. The authors performed a literature review on the most relevant genes associated with meningiomas and both current and potential gene therapy strategies to treat these tumors. Wild-type NF2 gene insertion, oncolytic viruses, and transfer of si…

Neurofibromatosis 2medicine.medical_specialtyAngiogenesisGenetic enhancementGenetic TherapyGeneral MedicineBiologymedicine.diseaseOncolytic virusMeningiomaClinical trialBenign MeningiomaMeningeal Neoplasmsotorhinolaryngologic diseasesmedicineCancer researchHumansSurgeryNeurology (clinical)NeurosurgeryMeningiomaGeneSignal TransductionNeurosurgical Focus
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Oral manifestations of Type I Neurofibromatosis in a family

2011

Neurofibroma is a benign peripheral nerve sheath tumor. It is one of the most frequent tumors of neural origin and its presence is one of the clinical criteria for the diagnosis of neurofibromatosis type I (NF-I). Neurofibromatosis type I is an autosomal dominantly inherited disease due to an alteration in the long arm of chromosome 17. About 50% of NF-I patients have no family history of the disease. NF-I patients have skin lesions (café au lait spots and neurofibromas) as well as bone malformations and central nervous system tumors. Diagnosis is based on a series of clinical criteria. NF-I presents with certain definite oral manifestations which confers to the dentists a major responsibil…

Neurofibromatosis type IPathologymedicine.medical_specialtybusiness.industryOdontologíaDisease:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseCiencias de la saludChromosome 17 (human)Café au lait spotUNESCO::CIENCIAS MÉDICASmedicineNeurofibromaFamily historyNeurofibromatosismedicine.symptombusinessGeneral DentistryPeripheral Nerve SheathJournal of Clinical and Experimental Dentistry
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Clinical, pathologic, and genetic features of massive soft tissue neurofibromas in a Sicilian patient

2008

Abstract CONTEXT: Lipase maturation factor 1 (LMF1) gene is a novel candidate gene in severe hypertriglyceridemia. Lmf1 is involved in the maturation of lipoprotein lipase (LPL) and hepatic lipase in endoplasmic reticulum. To date only one patient with severe hypertriglyceridemia and related disorders was found to be homozygous for a nonsense mutation in LMF1 gene (Y439X). OBJECTIVE: The objective of the study was to investigate LMF1 gene in hypertriglyceridemic patients in whom mutations in LPL, APOC2, and APOA5 genes had been excluded. RESULTS: The resequencing of LMF1 gene led to the discovery of a novel homozygous nonsense mutation in one patient with severe hypertriglyceridemia and rec…

Nonsynonymous substitutionMalecongenital hereditary and neonatal diseases and abnormalitiesPathologymedicine.medical_specialtyHeterozygoteNeurofibromatosis 1BiopsyDNA Mutational AnalysisMutation MissenseSoft Tissue NeoplasmsDermatologymassive soft tissue neurofibromas NeurofibromatosisBiologymedicine.disease_causeFrameshift mutationExonGenes Neurofibromatosis 1medicineSettore MED/35 - Malattie Cutanee E VenereeMissense mutationHumansNeurofibromatosisFrameshift MutationGeneSicilyGeneticsMutationHeterozygote advantageGeneral MedicineExonsMiddle Agedmedicine.diseasenervous system diseasesGene Expression Regulation NeoplasticButtocks
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Frequent chromosomal gains in recurrent juvenile nasopharyngeal angiofibroma.

2007

Juvenile nasopharyngeal angiofibroma (JNA) is a rare benign tumor, mostly affecting adolescent males. Some patients develop recurrences after surgery independently of completeness of removal. Only very limited data concerning underlying chromosomal changes are available. We therefore analyzed samples of 22 JNAs, including six recurrences, with comparative genomic hybridization (CGH). Additionally, quantitative image cytometry was used for measurement of DNA aneuploidy in representative samples. Of the 13 primary JNAs without later recurrence, DNA gains were identified on autosomes in only two samples. Four patients with one or two recurrences were included in the study; for one of these, no…

OncologyAdultMaleCancer Researchmedicine.medical_specialtyAdolescentJuvenile nasopharyngeal angiofibromaBiologyBioinformaticsAngiofibromaBenign tumorInternal medicineGeneticsmedicineHumansChildMolecular BiologyGeneChromosome AberrationsAutosomeNucleic Acid HybridizationNasopharyngeal NeoplasmsGenomicsDna amplificationmedicine.diseasePrimary tumorChromosome 4Neoplasm Recurrence LocalComparative genomic hybridizationCancer genetics and cytogenetics
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Patterns of Prior and Subsequent Neoplasms in Children and Adolescents With Soft Tissue Sarcomas.

2020

Background The occurrence of prior, concurrent and subsequent neoplasms (SN) represents a serious problem in children and adolescents with soft tissue sarcomas. Pathogenic germline variants contribute to the diagnosis of multiple neoplasms in sarcoma survivors. Materials and methods The records of 748 children and adolescents, diagnosed with soft tissue sarcomas and registered in trials/registries by the cooperative soft tissue sarcoma (Cooperative Weichteilsarkom Studie) group, were reviewed for the occurrence of SNs. Reference histology review was available for all cases; the presence of oncogenic fusions known at the time of diagnosis was confirmed for fusion-positive (F+) entities. Resu…

OncologyMalemedicine.medical_specialtyAdolescentOncogene Proteins FusionGermline03 medical and health sciences0302 clinical medicineCancer SurvivorsRisk FactorsInternal medicineGermanyBiomarkers TumorMedicineHumansRegistriesNeurofibromatosisChildGerm-Line MutationClinical Trials as Topicbusiness.industryIncidence (epidemiology)Soft tissue sarcomaIncidenceSoft tissueInfantHistologyNeoplasms Second PrimarySarcomaHematologymedicine.diseasePrognosisCombined Modality TherapySurvival RateOncology030220 oncology & carcinogenesisChild PreschoolPediatrics Perinatology and Child HealthFemaleSarcomabusiness030215 immunologyFollow-Up StudiesJournal of pediatric hematology/oncology
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Neoformazione esofitica mandibolare in una giovane paziente

2022

Una donna caucasica di 18 anni si presentava all’osservazione presso il Settore di Medicina Orale (Università degli Studi di Palermo) data la comparsa di una neoformazione localizzata in zona trigono retromolare destro. L’anamnesi patologica non evidenziava la presenza di patologie sistemiche attuali o pregresse. La paziente era in buone condizioni di salute, riferiva di non assumere farmaci e di non avere subito alcun intervento chirurgico; dichiarava di non avere mai fumato. La paziente riferiva un lento e progressivo accrescimento della lesione, in assenza di sintomatologia algica. L’esame obiettivo extra-orale non evidenziava linfoadenopatie latero-cervicali o altre anomalie. All’esame …

OrthodonticsOral SurgeryPeripheral ossifying fibroma Oral medicine Oral surgery Reactive lesion Benign neoplasm
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A Cemento-ossifying fibroma of the mandible in an 18 years old female patient: a case report

2019

OrthodonticsPhysiologybusiness.industryCemento-ossifying fibroma mandiblePhysiology (medical)Female patientMandibleMedicineCemento-ossifying fibromabusinessFrontiers in Physiology
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