Search results for "fisiologia"

showing 10 items of 672 documents

Hippocampal hyperexcitability is modulated by microtubule-active agent: evidence from in vivo and in vitro epilepsy models in the rat

2016

The involvement of microtubule dynamics on bioelectric activity of neurons and neurotransmission represents a fascinating target of research in the context of neural excitability. It has been reported that alteration of microtubule cytoskeleton can lead to profound modifications of neural functioning, with a putative impact on hyperexcitability phenomena. Altogether, in the present study we pointed at exploring the outcomes of modulating the degree of microtubule polymerization in two electrophysiological epileptiform activity in the rat hippocampus. To this aim, we used in vivo Maximal Dentate Activation (MDA) and in vitro hippocampal epileptiform bursting activity (HEBA) paradigms to asse…

0301 basic medicinehippocampusPaclitaxel.HippocampusContext (language use)BiologyNeurotransmissionHippocampal formationSettore BIO/09 - Fisiologialcsh:RC321-571Microtubule polymerization03 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compoundpaclitaxel0302 clinical medicineMicrotubulemedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchNeurotoxicitymedicine.diseaseelectrophysiologyNocodazole030104 developmental biologynocodazolechemistryepilepsyhippocampus epilepsy maximal dentate activation microtubule electrophysiology nocodazole paclitaxel.maximal dentate activationNeuroscience030217 neurology & neurosurgeryNeurosciencemicrotubule
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Current disease modifying approaches to treat Parkinson's disease

2015

Parkinson's disease (PD is a progressive neurological disorder characterized by the degeneration and death of midbrain dopamine and non-dopamine neurons in the brain leading to motor dysfunctions and other symptoms, which seriously influence the quality of life of PD patients. The drug L-dopa can alleviate the motor symptoms in PD, but so far there are no rational therapies targeting the underlying neurodegenerative processes. Despite intensive research, the molecular mechanisms causing neuronal loss are not fully understood which has hampered the development of new drugs and disease-modifying therapies. Neurotrophic factors are by virtue of their survival promoting activities attract candi…

0301 basic medicinemedicine.medical_specialtyParkinson's diseaseNeurturinNeurotrophic factorBiologySettore BIO/09 - Fisiologia03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineNeuroinflammationDopamineNeurotrophic factorsInternal medicineα-SynucleinmedicineGlial cell line-derived neurotrophic factorMolecular BiologyCerebral dopamine neurotrophic factorDopamine neuronPharmacologyDopaminergicCell Biologymedicine.diseaseDopamine neurons; ER stress; Mitochondria; Neuroinflammation; Neuropeptides; Neurotrophic factors; Protein aggregation; α-Synuclein; Molecular Medicine; Molecular Biology; Pharmacology; Cellular and Molecular Neuroscience; Cell Biology3. Good healthMitochondriaNeuropeptide030104 developmental biologyNerve growth factorEndocrinologybiology.proteinER streMolecular MedicineProtein aggregationNeuroscience030217 neurology & neurosurgerymedicine.drug
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Evaluation of an Antioxidant and Anti-inflammatory Cocktail Against Human Hypoactivity-Induced Skeletal Muscle Deconditioning

2020

International audience; Understanding the molecular pathways involved in the loss of skeletal muscle mass and function induced by muscle disuse is a crucial issue in the context of spaceflight as well as in the clinical field, and development of efficient countermeasures is needed. Recent studies have reported the importance of redox balance dysregulation as a major mechanism leading to muscle wasting. Our study aimed to evaluate the effects of an antioxidant/anti-inflammatory cocktail (741 mg of polyphenols, 138 mg of vitamin E, 80 mu g of selenium, and 2.1 g of omega-3) in the prevention of muscle deconditioning induced by long-term inactivity. The study consisted of 60 days of hypoactivi…

0301 basic medicinemedicine.medical_specialtyPhysiologymedicine.medical_treatment[SDV]Life Sciences [q-bio]Context (language use)Sciences du Vivant [q-bio]/Médecine humaine et pathologieBed restmedicine.disease_causelcsh:Physiology03 medical and health sciencesSciences du Vivant [q-bio]/Autre [q-bio.OT]0302 clinical medicineAtrophyDeconditioningInternal medicinePhysiology (medical)medicineoxidative stresscell signalingWastingOriginal Research[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologylcsh:QP1-981business.industrySkeletal musclemuscle wastingmedicine.disease3. Good health[SDV] Life Sciences [q-bio]030104 developmental biologyEndocrinologymedicine.anatomical_structureantioxidantsinactivityFisiologia humanamedicine.symptombusinessHypoactivity030217 neurology & neurosurgeryOxidative stress[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Obesogenic Diets Cause Alterations on Proteins and Theirs Post-Translational Modifications in Mouse Brains

2021

Obesity constitutes a major global health threat and is associated with a variety of diseases ranging from metabolic and cardiovascular disease, cancer to neurodegeneration. The hallmarks of neurodegeneration include oxidative stress, proteasome impairment, mitochondrial dysfunction and accumulation of abnormal protein aggregates as well as metabolic alterations. As an example, in post-mortem brain of patients with Alzheimer’s disease (AD), several studies have reported reduction of insulin, insulin-like growth factor 1 and insulin receptor and an increase in tau protein and glycogen-synthase kinase-3β compared to healthy controls suggesting an impairment of metabolism in the AD patient’s …

0301 basic medicinemedicine.medical_specialtyRC620-627Endocrinology Diabetes and Metabolismmedicine.medical_treatmentTau proteinObesity nutrition brain impairment proteomics post-translational modificationsBrain damageMitochondrionProteomicsmedicine.disease_causeSettore BIO/09 - Fisiologia03 medical and health sciencesproteomics0302 clinical medicineInternal medicinepost-translational modificationsmedicineTX341-641ObesityNutritional diseases. Deficiency diseasesOriginal ResearchSettore MED/04 - Patologia GeneraleNutrition and DieteticsbiologyNutrition. Foods and food supplyInsulinNeurodegenerationmedicine.diseasebrain impairmentInsulin receptornutrition030104 developmental biologyEndocrinologybiology.proteinmedicine.symptom030217 neurology & neurosurgeryOxidative stressFood ScienceNutrition and Metabolic Insights
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Nutrition, obesity and hormones

2018

Obesity is a chronic pathological condition with a multifactorial aetiology, characterised by an excessive body fat accumulation with multiple organ-specific consequences. Emerging evidence highlights that obesity appears to be associated with multiple alterations in the endocrine system. However, the mechanisms underlying the interactions between obesity and this system remain still controversial. This review discusses the impact of obesity on various endocrine systems and, in particular, would provide a general overview on the biochemical changes that may occur in each of these axes in association with obesity.

0301 basic medicinemedicine.medical_specialtyobesityPlant ScienceGrowth hormoneSettore BIO/09 - FisiologiaGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesInternal medicinemedicineEndocrine systemlcsh:QH301-705.5thyroid hormonesbusiness.industryBiochemistry (medical)medicine.diseaseObesityThyroid hormone030104 developmental biologyEndocrinologyendocrine systemnutritionlcsh:Biology (General)Thyroid hormonesgrowth hormoneSettore BIO/14 - FarmacologiabusinessHormoneJournal of Biological Research
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Redox modulation of muscle mass and function

2020

Muscle mass and strength are very important for exercise performance. Training-induced musculoskeletal injuries usually require periods of complete immobilization to prevent any muscle contraction of the affected muscle groups. Disuse muscle wasting will likely affect every sport practitioner in his or her lifetime. Even short periods of disuse results in significant declines in muscle size, fiber cross sectional area, and strength. To understand the molecular signaling pathways involved in disuse muscle atrophy is of the utmost importance to develop more effective countermeasures in sport science research. We have divided our review in four different sections. In the first one we discuss t…

0301 basic medicinemuscle[SDV]Life Sciences [q-bio]Clinical BiochemistryPhysiologyFisiologiaBiochemistryArticleAntioxidants03 medical and health sciences0302 clinical medicineAtrophymedicineAnimalsHumansMuscle Skeletallcsh:QH301-705.5Wastinglcsh:R5-920Mechanism (biology)business.industryOrganic Chemistrymedicine.diseaseMuscle atrophy3. Good healthProtein catabolismMuscular Atrophy030104 developmental biologylcsh:Biology (General)Fisiologia humanamedicine.symptomSignal transductionlcsh:Medicine (General)businessReactive Oxygen SpeciesOxidation-Reduction030217 neurology & neurosurgeryFunction (biology)Muscle contraction
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Non-invasive Brain Stimulation in Pediatric Migraine: A Perspective From Evidence in Adult Migraine

2019

Pediatric migraine remains still a challenge for the headache specialists as concerns both diagnostic and therapeutic aspects. The less ability of children to describe the exact features of their migraines and the lack of reliable biomarker for migraine contribute to complicate the diagnostic process. Therefore, there's need for new effective tools for supporting diagnostic and therapeutic approach in children with migraine. Recently, promising results have been obtained in adult headache by means of application of neurostimulation techniques both for investigating pathophysiological mechanisms and also for therapeutical applications. Non-invasive brain stimulation (NIBS) techniques like tr…

0301 basic medicinenon-invasive brain stimulationmedicine.medical_specialtyTMS tDCS migraine pediatric populationMini Reviewmedicine.medical_treatmentSettore BIO/09 - Fisiologialcsh:RC346-42903 medical and health sciencesTherapeutic approach0302 clinical medicinetranscranial magnetic stimulationtherapeuticsMedicineIntensive care medicineNeurostimulationlcsh:Neurology. Diseases of the nervous systemTranscranial direct-current stimulationbusiness.industrypediatric migrainemedicine.diseaseSettore MED/39 - Neuropsichiatria InfantileBiomarker (cell)Transcranial magnetic stimulation030104 developmental biologyNeurologyMigraineBrain stimulationSettore MED/26 - NeurologiaNeurology (clinical)transcranial direct current stimulationHeadachesmedicine.symptombusiness030217 neurology & neurosurgeryFrontiers in Neurology
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Lateral Habenula 5-HT2C Receptor Function Is Altered by Acute and Chronic Nicotine Exposures

2021

Serotonin (5-HT) is important in some nicotine actions in the CNS. Among all the 5-HT receptors (5-HTRs), the 5-HT2CR has emerged as a promising drug target for smoking cessation. The 5-HT2CRs within the lateral habenula (LHb) may be crucial for nicotine addiction. Here we showed that after acute nicotine tartrate (2 mg/kg, i.p.) exposure, the 5-HT2CR agonist Ro 60-0175 (5–640 µg/kg, i.v.) increased the electrical activity of 42% of the LHb recorded neurons in vivo in rats. Conversely, after chronic nicotine treatment (6 mg/kg/day, i.p., for 14 days), Ro 60-0175 was incapable of affecting the LHb neuronal discharge. Moreover, acute nicotine exposure increased the 5-HT2CR-immunoreactive (IR)…

0301 basic medicinenucleus accumbensdorsal raphe nucleusmedicine.medical_treatmentstriatumStriatumSmoking cessationSettore BIO/09 - FisiologiaSerotonin -- ReceptorsNicotineNicotine addiction -- Treatment0302 clinical medicinesingle cell-extracellular recordingMedicinesubstantia nigra pars compactadentate gyrusBiology (General)SpectroscopyGeneral MedicineDorsal raphe nucleuComputer Science ApplicationsVentral tegmental area5-HT2C receptorChemistrymedicine.anatomical_structuredepressionaddictionmedicine.drugAgonistmedicine.medical_specialtyendocrine systemQH301-705.5medicine.drug_classRo 60-0175ventral tegmental areaNucleus accumbensDentate gyruCatalysisInorganic Chemistry03 medical and health sciencesInternal medicineNucleus accumbenPhysical and Theoretical ChemistryQD1-999Molecular BiologyHabenulabusiness.industryOrganic Chemistry030104 developmental biologyEndocrinologySmoking cessationSerotoninbusinessSerotonin -- Agonists030217 neurology & neurosurgerymedial prefrontal cortexInternational Journal of Molecular Sciences
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RADIOLOGIA DE LA DEGLUCIÓ

1995

Descripción de la deglución animal y representación de las funciones complejas.

2401.13 Ciències de la VidaBiologia Zoologia Deglució animal Fisiologia Facultat de Biologia
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3,5-DIIODO-L-THYRONINE-INDUCED MODIFICATION IN PITUITARY-THYROID AXIS IN RATS FED HIGH-FAT DIET. A PRELIMINARY REPORT

2014

Experimental observations highlight that the administration of 3,5-diiodo-L-thyronine (T2) may decrease the body weight and the plasma levels of cholesterol and triglycerides and may prevent the onset of hepatic steatosis in rats fed diets rich in lipids (HFD). On the basis of these findings we have carried out some in vivo studies to evaluate the effects of increased levels of T2 on pituitary thyroid axis function in HFD rats. Fifteeen Wistar male rats were divided in 3 groups. The first group (N) was fed with a standard diet. The second group (G) was fed with a diet high in fat (HDF), while the third group (GT2) was additionally administered intraperitoneally with T2 (70 ug/100g body weig…

35-DIIODO-L-THYRONINE HIGH-FAT DIETSettore BIO/09 - Fisiologia
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