Search results for "fragmentation"

showing 10 items of 798 documents

Macrophage-mediated clearance of cells undergoing caspase-3-independent death

2003

Little is known of the functions of caspases in mediating the surface changes required for phagocytosis of dying cells. Here we investigate the role played by the effector caspase, caspase-3 in this process using the caspase-3-defective MCF-7 breast carcinoma line and derived caspase-3-expressing transfectants. Our results indicate that, while certain typical features of apoptosis induced by etoposide - namely classical morphological changes and the ability to degrade DNA into oligonucleosomal fragments - are caspase-3-dependent, loss of cell adhesion to plastic and the capacity to interact with, and to be phagocytosed by, human monocyte-derived macrophages - both by CD14-dependent and CD14…

Programmed cell deathTime FactorsBlotting WesternGreen Fluorescent ProteinsLipopolysaccharide ReceptorsApoptosisCaspase 3PhosphatidylserinesDNA FragmentationTransfectionCaspase 7Proinflammatory cytokinePhagocytosisCell Line TumorSettore BIO/10 - BiochimicaHumansMacrophageAnnexin A5Cell adhesionCytokineMolecular BiologyCells CulturedCaspaseEtoposideCaspase 7InflammationCell DeathbiologyCaspase 3MacrophagesDNACell BiologyCaspaseCell biologyEnzyme ActivationLuminescent ProteinsApoptosisCaspasesbiology.proteinCytokinesElectrophoresis Polyacrylamide GelCell Death & Differentiation
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α-Tocopherol impairs 7-ketocholesterol-induced caspase-3-dependent apoptosis involving GSK-3 activation and Mcl-1 degradation on 158N murine oligoden…

2011

Abstract In important and severe neurodegenerative pathologies, 7-ketocholesterol, mainly resulting from cholesterol autoxidation, may contribute to dys- or demyelination processes. On various cell types, 7-ketocholesterol has often been shown to induce a complex mode of cell death by apoptosis associated with phospholipidosis. On 158N murine oligodendrocytes treated with 7-ketocholesterol (20 μg/mL corresponding to 50 μM, 24–48 h), the induction of a mode of cell death by apoptosis characterised by the occurrence of cells with condensed and/or fragmented nuclei, caspase activation (including caspase-3) and internucleosomal DNA fragmentation was observed. It was associated with a loss of tr…

Programmed cell deathTime FactorsCell Survivalalpha-TocopherolApoptosisCaspase 3BiochemistryDephosphorylationGlycogen Synthase Kinase 3MiceMembrane MicrodomainsGSK-3AnimalsKetocholesterolsMolecular BiologyProtein kinase BCell ProliferationMembrane Potential MitochondrialPhospholipidosisGlycogen Synthase Kinase 3 betaCaspase 3ChemistryOrganic ChemistryCytochromes cCell BiologyCell biologyEnzyme ActivationOligodendrogliaProtein TransportProto-Oncogene Proteins c-bcl-2ApoptosisMyeloid Cell Leukemia Sequence 1 ProteinDNA fragmentationChemistry and Physics of Lipids
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Effect of flupirtine on cell death of human umbilical vein endothelial cells induced by reactive oxygen species.

1999

Abstract Flupirtine (KATADOLON®), known as a nonopiate centrally acting analgesic drug, was tested as to its potential to prevent apoptosis of human endothelial cells induced by reactive oxygen species (ROS). It was found that Flupirtine displayed no effect on viability and cell proliferation of human umbilical vein endothelial cells (HUVEC) up to a concentration of 10 μg/mL. Apoptosis, induced by ROS and generated by hypoxanthine/xanthine oxidase (EC 1.1.3.22) (HX/XOD) or t-butyl hydroperoxide, was reduced after preincubation with Flupirtine for 3 hr by 35% and 41%, respectively. The maximal cytoprotective effect against apoptosis was observed at a drug concentration of 1 to 3 μg/mL. Flow …

Programmed cell deathUmbilical VeinsXanthine OxidaseAminopyridinesDNA FragmentationPharmacologyBiochemistryXanthineUmbilical veinchemistry.chemical_compoundNecrosismedicineHumansXanthine oxidaseHypoxanthineCells CulturedPharmacologychemistry.chemical_classificationReactive oxygen speciesCell DeathDose-Response Relationship DrugCell growthchemistryBiochemistryApoptosisCalciumEndothelium VascularFlupirtineReactive Oxygen Speciesmedicine.drugBiochemical pharmacology
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Evidence for an instructive role of apoptosis during the metamorphosis of Hydractinia echinata (Hydrozoa)

2011

Apoptosis is a highly conserved mechanism of cell deletion that destroys redundant, dysfunctional, damaged, and diseased cells. Furthermore, apoptotic cell death is essential during the development of multicellular organisms. However, there are only a few examples where the occurrence of apoptosis has been shown to be a direct prerequisite for developmental processes. As described previously by our group, the degradation of larval tissue during the first half of the metamorphosis of Hydractinia echinata involves extensive cell death. A large number of cells are removed, and we observed several cellular features of apoptotic cell death in the dying tissue, e.g., nucleosomal DNA fragmentation…

Programmed cell deathmedia_common.quotation_subjectMolecular Sequence DataCellApoptosisContext (language use)Gene Expression Regulation EnzymologicHydractinia echinatamedicineAnimalsAmino Acid SequenceMetamorphosisConserved SequencePhylogenyCaspasemedia_commonbiologyGene Expression ProfilingMetamorphosis Biologicalbiology.organism_classificationCell biologyHydrozoamedicine.anatomical_structureApoptosisCaspasesGene Knockdown Techniquesbiology.proteinDNA fragmentationAnimal Science and ZoologySequence AlignmentZoology
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Peroxisome proliferator-activated receptor δ (PPARδ) activation protects H9c2 cardiomyoblasts from oxidative stress-induced apoptosis

2005

Activation of peroxisome proliferator-activated receptor alpha (PPARalpha) and PPARgamma plays beneficial roles in cardiovascular disorders such as atherosclerosis and heart reperfusion. Although PPARalpha and gamma have been documented to reduce oxidative stress in the vasculature and the heart, the role of PPARdelta remains poorly studied.We focused on PPARdelta function in the regulation of oxidative stress-induced apoptosis in the rat cardiomyoblast cell line H9c2. Using semi-quantitative reverse transcriptase-polymerase chain reaction (RT-PCR), we showed that PPARdelta is the predominantly expressed isotype whereas PPARalpha was weakly detected. By performing cell viability assays, we …

Programmed cell deathmedicine.medical_specialtyPhysiologyBlotting WesternPeroxisome proliferator-activated receptorApoptosisCaspase 3DNA FragmentationBiologyTransfectionmedicine.disease_causeCell LineGW501516Physiology (medical)Internal medicineIn Situ Nick-End LabelingmedicineAnimalsPPAR deltaViability assayReceptorchemistry.chemical_classificationCaspase 3Reverse Transcriptase Polymerase Chain ReactionHydrogen PeroxideCatalasemedicine.diseaseRatsUp-RegulationCell biologyOxidative StressThiazolesEndocrinologychemistryApoptosisCaspasesCardiology and Cardiovascular MedicineMyoblasts CardiacOxidative stressCardiovascular Research
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Fatty acids liberated from low-density lipoprotein trigger endothelial apoptosis via mitogen-activated protein kinases.

2005

Enzymatic modification of low-density lipoprotein (LDL) as it probably occurs in the arterial intima drastically increases its cytotoxicity, which could be relevant for the progression of atherosclerotic lesions. LDL was treated with a protease and cholesterylesterase to generate a derivative similar to lesional LDL, with a high content of free cholesterol and fatty acids. Exposure of endothelial cells to the enzymatically modified lipoprotein (E-LDL), but not to native or oxidized LDL, resulted in programmed cell death. Apoptosis was triggered by apoptosis signal-regulating kinase 1 dependent phosphorylation of p38. Depletion and reconstitution experiments identified free fatty acids (FFA)…

Programmed cell deathp38 mitogen-activated protein kinasesBlotting WesternApoptosisDNA FragmentationBiologyFatty Acids NonesterifiedMAP Kinase Kinase Kinase 5p38 Mitogen-Activated Protein Kinaseschemistry.chemical_compoundHumansPhosphorylationMolecular BiologyCells CulturedCaspase 7Cell growthKinaseCaspase 3Cell BiologyCell biologyLipoproteins LDLchemistryBiochemistryApoptosisLow-density lipoproteinCaspasesPhosphorylationlipids (amino acids peptides and proteins)Endothelium VascularLipoproteinOleic AcidCell death and differentiation
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Antiproton over proton and K$^-$ over K$^+$ multiplicity ratios at high $z$ in DIS

2020

The $\bar{\rm p} $ over p multiplicity ratio is measured in deep-inelastic scattering for the first time using (anti-) protons carrying a large fraction of the virtual-photon energy, $z>0.5$. The data were obtained by the COMPASS Collaboration using a 160 GeV muon beam impinging on an isoscalar $^6$LiD target. The regime of deep-inelastic scattering is ensured by requiring $Q^2$ > 1 (GeV/$c$)$^2$ for the photon virtuality and $W > 5$ GeV/$c^2$ for the invariant mass of the produced hadronic system. The range in Bjorken-$x$ is restricted to $0.01 < x < 0.40$. Protons and antiprotons are identified in the momentum range $20 ��60$ GeV/$c$. In the whole studied $z$-region, the $\…

ProtonIsoscalarHadron0 [higher-order]Deep-inelastic scatteringtarget: isoscalar01 natural sciencesCOMPASSdeep inelastic scattering [muon+ nucleon]High Energy Physics - ExperimentHigh Energy Physics - Experiment (hep-ex)[PHYS.HEXP]Physics [physics]/High Energy Physics - Experiment [hep-ex]anti-p: multiplicityInvariant massisoscalar [target]Nuclear Experiment (nucl-ex)Nuclear ExperimentHadron multiplicitiesNuclear ExperimentQuantum chromodynamicsPhysicsmultiplicity [K+]quark: fragmentation functionhigher-order: 0K+: multiplicityphotonperturbation theory: higher-orderhigher-order: 1multiplicity [anti-p]lcsh:QC1-999Bjorken [scaling]beam [muon]factorization [cross section]1 [higher-order]Particle Physics - Experimentperturbation theory [quantum chromodynamics]Nuclear and High Energy PhysicsFOS: Physical sciencesratio [multiplicity]530pQCDfragmentation function [quark]scaling: Bjorkenx-dependenceNuclear physicsQuantum chromodynamics; pQCD; Deep-inelastic scattering; Hadron multiplicities; COMPASSphase space0103 physical sciencesddc:530quantum chromodynamics: perturbation theory010306 general physicsmuon+ nucleon: deep inelastic scatteringp: multiplicityMuonmultiplicity [K-]multiplicity: ratio010308 nuclear & particles physicshep-exmuon: beamcross section: factorizationCERN SPSDeep inelastic scatteringmultiplicity: measured [charged particle]higher-order [perturbation theory]K-: multiplicityAntiprotonHigh Energy Physics::Experimentlcsh:PhysicsQuantum chromodynamicscharged particle: multiplicity: measuredhadronizationmultiplicity [p]experimental results
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Biomechanical properties and histomorphometric features of aortic tissue in patients with or without bicuspid aortic valve

2020

Background We sought to investigate and compare biomechanical properties and histomorphometric findings of thoracic ascending aorta aneurysm (TAA) tissue from patients with bicuspid aortic valve (BAV) and tricuspid aortic valve (TAV) in order to clarify mechanisms underlying differences in the clinical course. Methods Circumferential sections of TAA tissue in patients with BAV (BAV-TAA) and TAV (TAV-TAA) were obtained during surgery and used for biomechanical tests and histomorphometrical analysis. Results In BAV-TAA, we observed biomechanical higher peak stress and lower Young modulus values compared with TAV-TAA wall. The right lateral longitudinal region seemed to be the most fragile zon…

Pulmonary and Respiratory MedicineAortic valveTunica mediamedicine.medical_specialtyaortopathyDissection (medical)030204 cardiovascular system & hematologycomplex mixtures030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineBicuspid aortic valvefluid dynamic analysisInternal medicineparasitic diseasesmedicineIn patientAortic dissectionbiologybusiness.industryaortic wallelastic tissue fragmentationmedicine.diseasedigestive system diseasesAortic wallSettore MED/23medicine.anatomical_structureBicuspid aortic valve (BAV)biology.proteinCardiologycardiovascular systemOriginal ArticlebusinessElastin
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Degrees of freedom effect on fragmentation in tandem mass spectrometry of singly charged supramolecular aggregates of sodium sulfonates

2013

The characteristic collision energy (CCE) to obtain 50% fragmentation of positively and negatively single charged non-covalent clusters has been measured. CCE was found to increase linearly with the degrees of freedom (DoF) of the precursor ion, analogously to that observed for synthetic polymers. This suggests that fragmentation behavior (e.g. energy randomization) in covalent molecules and clusters are similar. Analysis of the slope of CCE with molecular size (DoF) indicates that activation energy of fragmentation of these clusters (loss of a monomer unit) is similar to that of the lowest energy fragmentation of protonated leucine-enkephalin. Positively and negatively charged aggregates b…

Quantitative Biology::BiomoleculesPhysics::Instrumentation and DetectorsChemistryAnalytical chemistrySupramolecular chemistryProtonationActivation energyIonchemistry.chemical_compoundMonomerFragmentation (mass spectrometry)Covalent bondMoleculeSpectroscopyJournal of Mass Spectrometry
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Production of π0 and η mesons in Cu+Au collisions at sNN=200GeV

2018

Production of π0 and η mesons has been measured at midrapidity in Cu+Au collisions at sNN=200GeV. Measurements were performed in π0(η)→γγ decay channel in the 1(2)-20GeV/c transverse momentum range. A strong suppression is observed for π0 and η meson production at high transverse momentum in central Cu+Au collisions relative to the p+p results scaled by the number of nucleon-nucleon collisions. In central collisions the suppression is similar to Au+Au with comparable nuclear overlap. The η/π0 ratio measured as a function of transverse momentum is consistent with mT-scaling parametrization down to pT=2GeV/c, its asymptotic value is constant and consistent with Au+Au and p+p and does not show…

Quantum chromodynamicsPhysicsMapleMeson productionMeson010308 nuclear & particles physicsNuclear Theorychemistry.chemical_elementengineering.material16. Peace & justice01 natural sciencesCopperNuclear physicsFragmentation (mass spectrometry)chemistry0103 physical sciencesQuark–gluon plasmaengineeringHigh Energy Physics::ExperimentImpact parameterNuclear Experiment010306 general physicsPhysical Review C
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