Search results for "g factor"

showing 10 items of 514 documents

Granulocyte-macrophage colony-stimulating factor-cultured bone marrow-derived macrophages reveal accessory cell function and synthesis of MHC class I…

1988

The antigen-mediated activation of a number of T cell clones by bone marrow (BM) cells cultivated in the presence of various colony-stimulating factor (CSF) preparations was investigated. BM macrophages (BMM phi) grown in L929 cell supernatant as a crude source of macrophage colony-stimulating factor (M-CSF) as well as BM cells propagated in the presence of recombinant M-CSF exhibited transient antigen presentation potential to some T cell clones, being maximal on day 7 and having declined to a low level by day 19 of in vitro culture. Treatment of these long-term-cultivated BMM phi populations with recombinant interferon-gamma (IFN-gamma) resulted in predominant antigen presentation capacit…

medicine.medical_specialtyT cellT-LymphocytesImmunologyAntigen presentationAntigen-Presenting CellsBone Marrow CellsMajor histocompatibility complexLymphocyte ActivationCell LineInterferon-gammaMiceAntigenColony-Stimulating FactorsInternal medicinemedicineImmunology and AllergyCytotoxic T cellAnimalsAntigensAntigen-presenting cellGrowth SubstancesMHC class IIHybridomasbiologyMonocyteMacrophagesHistocompatibility Antigens Class IIGranulocyte-Macrophage Colony-Stimulating FactorMolecular biologyCulture Mediamedicine.anatomical_structureEndocrinologybiology.proteinEuropean journal of immunology
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Zoledronate, ibandronate and clodronate enhance osteoblast differentiation in a dose dependent manner – A quantitative in vitro gene expression analy…

2010

Bisphosphonates are widely used in the clinical treatment of bone diseases with increased bone resorption. In terms of side effects, they are known to be associated with osteonecrosis of the jaw (BONJ). There are two groups of bisphosphonates: the nitrogen-containing bisphosphonates, e.g. zoledronate and ibandronate, and the non-nitrogen-containing bisphosphonates, e.g. clodronate. Their impact on bone metabolism seems to differ. The objective of this study was to compare the osteogenic differentiation potency of these two pharmacologic groups. Human osteoblasts were stimulated with zoledronate and ibandronate at concentrations of 5×10(-5) M, 5×10(-6) M and 5×10(-7) M over the experimental …

medicine.medical_specialtyTime Factorsmedicine.medical_treatmentOsteocalcinCell Culture TechniquesCore Binding Factor Alpha 1 SubunitZoledronic AcidIbandronic acidBone remodelingInternal medicineHumansMedicineIbandronic AcidHomeodomain ProteinsMSX1 Transcription FactorOsteoblastsBone Density Conservation AgentsDiphosphonatesDose-Response Relationship DrugbiologyReverse Transcriptase Polymerase Chain Reactionbusiness.industryImidazolesCell DifferentiationOsteoblastDLX5BisphosphonateRUNX2Zoledronic acidmedicine.anatomical_structureEndocrinologyGene Expression RegulationOtorhinolaryngologyOsteocalcinbiology.proteinSurgeryBone RemodelingClodronic AcidOral SurgerybusinessBiomarkersTranscription Factorsmedicine.drugJournal of Cranio-Maxillofacial Surgery
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Fibromyalgia and arthritides

2012

Fibromyalgia (FM) is a chronic pain syndrome that affects at least 2% of the adult population. It is characterised by widespread pain, fatigue, sleep alterations and distress, and emerging evidence suggests a central nervous system (CNS) malfunction that increases pain transmission and perception. FM is often associated with other diseases that act as confounding and aggravating factors, such as rheumatoid arthritis (RA), spondyloarthritides (SpA), osteoarthritis (OA) and thyroid disease. Mechanism-based FM management should consider both peripheral and central pain, including effects due to cerebral input and that come from the descending inhibitory pathways. Rheumatologists should be able…

medicine.medical_specialtylcsh:Internal medicineCentral nervous systemlcsh:MedicineOsteoarthritisSeverity of Illness IndexArthritis RheumatoidDiagnosis DifferentialRheumatologyQuality of lifeFibromyalgiaOsteoarthritisSpondylarthritismedicineHumanspainAggravating Factorlcsh:RC31-1245FatiguePain Measurementbusiness.industryArthritisThyroid diseaselcsh:Rmedicine.diseaseThyroid DiseasesArthritides pain fibromyalgia rheumatic diseasesarthritidesDistressmedicine.anatomical_structurearthritides; fibromyalgia; pain; rheumatic diseasesRheumatoid arthritisPhysical therapyrheumatic diseasesfibromyalgiaChronic Painbusiness
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Bafetinib inhibits functional responses of human eosinophils in vitro

2012

Eosinophils play a prominent role in the process of allergic inflammation. Non-receptor associated Lyn tyrosine kinases generate key initial signals in eosinophils. Bafetinib, a specific Abl/Lyn tyrosine kinase inhibitor has shown a potent antiproliferative activity in leukemic cells, but its effects on eosinophils have not been reported. Therefore, we studied the effects of bafetinib on functional and mechanistic responses of isolated human eosinophils. Bafetinib was more potent than non-specific tyrosin kinase comparators genistein and tyrphostin inhibiting superoxide anion triggered by N-formyl-Met-Leu-Phe (fMLF; 100 nM) (−log IC50=7.25±0.04 M; 6.1±0.04 M; and 6.55±0.03 M, respectively).…

medicine.medical_specialtymedicine.drug_classFarmacologíaGenisteinApoptosisPharmacologyBiologyTyrosine-kinase inhibitorAllergic inflammationchemistry.chemical_compoundCell MovementSuperoxidesLYNInternal medicinemedicineHumansProtein Kinase InhibitorsPeroxidasePharmacologyKinaseEosinophil Cationic ProteinGranulocyte-Macrophage Colony-Stimulating FactorEosinophilLeukotriene C4Respiratory burstEosinophilsN-Formylmethionine Leucyl-PhenylalaninePyrimidinesmedicine.anatomical_structureEndocrinologychemistryCalciumInterleukin-5Tyrosine kinaseEuropean Journal of Pharmacology
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Tutorial in oral antithrombotic therapy: biology and dental implications.

2013

Objectives: Recent developments of new direct oral anticoagulants that target specific clotting factors necessitate understanding of coagulation biology. The objective of this tutorial is to offer dental professionals a review of coagulation mechanisms and the pharmacodynamics of the conventional and new oral anticoagulants. Also, we summarized the dental implications of the conventional and new anticoagulants. Method: We searched Medline using search terms “antithrombotic”, “antihemostasis” or “anticoagulation” and combined them with the search results of “dental”, “oral surgery” or “periodontal”. We restricted the results to “human” and “English”. Results: The early coagulation cascade, t…

medicine.medical_specialtymedicine.drug_mechanism_of_actionFactor Xa InhibitorOral Surgical ProceduresAdministration OralOdontologíaHemorrhage030204 cardiovascular system & hematologyOral Surgical Procedures03 medical and health sciences0302 clinical medicineFibrinolytic AgentsRisk FactorsAntithromboticmedicineHumansIntensive care medicineGeneral DentistryClotting factorbusiness.industryWarfarinReview-Article030206 dentistryPerioperative:CIENCIAS MÉDICAS [UNESCO]Ciencias de la salud3. Good healthSurgerystomatognathic diseasesOtorhinolaryngologyDirect thrombin inhibitorUNESCO::CIENCIAS MÉDICASSurgeryOral SurgerybusinessFibrinolytic agentmedicine.drugMedicina oral, patologia oral y cirugia bucal
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Polypeptides controlling hematopoietic blood cell development and activation

1989

Colony-stimulating factors (CSFs) have entered the clinical arena. Several investigators have explored, in first clinical phase I studies, different routes of administration to define the optimum biological dose, maximum tolerated dose, toxicity, and pharmacokinetics of these reagents. It has been demonstrated that recombinant human (rh) granulocyte-macrophage CSF (GM-CSF) and granulocyte CSF (G-CSF) can be safely administered over a broad dose range to increase number of circulating granulocytes in man. More recently, GM-CSF and G-CSF have been involved in phase Ib/II studies to assess the granulopoietic responses of patients with granulocytopenia due to various underlying disease states i…

medicine.medical_specialtymedicine.medical_treatmentGranulocyteCyclic neutropeniaColony-Stimulating FactorsBone MarrowInternal medicinemedicineHumansAplastic anemiaChemotherapyHematologybusiness.industryHematologyGeneral MedicineHematopoietic Stem Cellsmedicine.diseaseHematopoiesisGranulocyte colony-stimulating factorHaematopoiesisGranulocyte macrophage colony-stimulating factormedicine.anatomical_structureImmunologyDrug EvaluationPeptidesbusinessmedicine.drugBlut
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Granulocyte-macrophage colony-stimulating factor (CSF) and multilineage CSF recruit human monocytes to express granulocyte CSF

1989

Abstract We assessed the capacity of recombinant human granulocyte-macrophage colony-stimulating factor (GM-CSF) and multilineage (Multi)-CSF to induce release of granulocyte-CSF (G-CSF) by highly purified peripheral blood monocyte (Mo) preparations. Our results reveal that GM-CSF and Multi-CSF, either alone or in a synergistic concert, activate Mo to transcribe G-CSF messenger (m) RNA and release biologically active G- CSF protein into their culture supernatants. G-CSF had no regulatory effect on Mo expression of cytoplasmic G-CSF mRNA levels and G-CSF protein secretion by itself. These differential actions of CSFs provide further insight into self-regulatory mechanisms within the growth f…

medicine.medical_treatmentImmunologyGranulocyteBiologyBiochemistryMonocyteslaw.inventionColony-Stimulating FactorslawmedicineHumansRNA MessengerGrowth SubstancesCells CulturedCSF albuminCell-Free SystemGrowth factorGranulocyte-Macrophage Colony-Stimulating FactorRNABiological activityCell BiologyHematologyMolecular biologyRecombinant ProteinsDrug CombinationsGranulocyte macrophage colony-stimulating factorSecretory proteinmedicine.anatomical_structureImmunologyRecombinant DNAGranulocytesmedicine.drugBlood
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A MiR-142-3p/EGR2 Feedback Circuitry In Human CSF-1 Driven Differentiation of Monocytes Into Macrophages

2011

Abstract Abstract 2366 Colony-stimulating factor-1 (CSF-1 or M-CSF) triggers the differentiation of human peripheral blood monocytes into macrophages through and integrated cytokine/transcription factors circuitry. Using microarray profiling to explore the role of microRNAs (miRNAs) in this molecular circuitry, we identified the down-regulation of miR-142-3p in human macrophages obtained from CSF-1-treated monocytes. We show that miR-142-3p is a repressor of the transcription factor EGR2 (Early Growth Response 2) through direct 3'UTR interactions. Interestingly, EGR2 binds the promoter of the pre-miR-142-3p gene to negatively regulate its expression, identifying a self-regulatory feedback l…

medicine.medical_treatmentImmunologyRepressorChronic myelomonocytic leukemiaCell BiologyHematologyBiologyColony-stimulating factormedicine.diseaseBiochemistryCell biologyCytokinemicroRNAmedicineGeneTranscription factorProto-oncogene tyrosine-protein kinase SrcBlood
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Factor selection procedures in a Google Earthtm aided landslide susceptibility model: application to the Beiro river basin (Spain)

2011

A procedure to select the controlling factors connected to the slope instability has been defined. It allowed to assess the landslide susceptibility in the Rio Beiro basin (about 10 km2) over the north-eastern area of the city of Granada (Spain). Field and remote (Google EarthTM) recognition techniques allowed to generate a landslide inventory consisting in 127 phenomena. Univariate tests, using both association coefficients and validation results of single parameter susceptibility models, allowed to select among 15 controlling factors the ones that resulted as good predictor variables; these have been combined for unique conditions analysis and susceptibility maps were finally prepared. In…

multivariate landslide susceptibility models conditional analysis controlling factor selection model validation Google EarthTm.geography.geographical_feature_categorySettore GEO/04 - Geografia Fisica E GeomorfologiaUnivariateDrainage basinForecast skillLandslideLandslide susceptibilityField (geography)GeographyGoodness of fitApproximation errorStatisticsCartography
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Antitumor Cell-Complex Vaccines Employing Genetically Modified Tumor Cells and Fibroblasts

2014

The present study evaluates the immune response mediated by vaccination with cell complexes composed of irradiated B16 tumor cells and mouse fibroblasts genetically modified to produce GM-CSF. The animals were vaccinated with free B16 cells or cell complexes. We employed two gene plasmid constructions: one high producer (pMok) and a low producer (p2F). Tumor transplant was performed by injection of B16 tumor cells. Plasma levels of total IgG and its subtypes were measured by ELISA. Tumor volumes were measured and survival curves were obtained. The study resulted in a cell complex vaccine able to stimulate the immune system to produce specific anti-tumor membrane proteins (TMP) IgG. In the g…

non-viralHealth Toxicology and MutagenesisGenetic enhancementCellMelanoma Experimentallcsh:MedicineBiologyToxicologyArticleImmunoglobulin GMicePlasmidImmune systemCell Line TumormedicineAnimalsCells Culturedlcsh:RGranulocyte-Macrophage Colony-Stimulating FactorMembrane ProteinsTransfectionFibroblastsMolecular biologygene therapycell complexesTumor BurdenGenetically modified organismGranulocyte macrophage colony-stimulating factormedicine.anatomical_structureImmunoglobulin Gbiology.proteincancer vaccinesbystander cellsmedicine.drugToxins
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