Search results for "genetic disease"

showing 10 items of 64 documents

Translational readthrough inducing drugs: a study of toxicity in mice models and in vitro safety validation of the specific readthrough process.

2022

Objective Nonsense mutations are responsible for 15% of Cystic Fibrosis (CF) patients due to the introduction of a premature stop codon (PTC) in the mRNA and the production of a truncated CFTR (Cystic Fibrosis Transmembrane Conductance Regulator) protein1. A promising therapeutic approach for stop mutations is the suppression therapy by Translational Readthrough Inducing Drugs (TRIDs) to restore the expression of the protein2,3. Recently three new TRIDS (NV848, NV914, NV930) have been proposed and validated by several assays. Our work was focused on TRIDs NV848, NV914, NV930. Important aspects of TRIDs to be evaluated are their specificity towards PTC, to demonstrate that TRIDs do not inter…

Settore BIO/18 - GeneticaSettore BIO/11 - Biologia MolecolareSettore CHIM/06 - Chimica OrganicaNonsense mutations genetic diseases oxadizole target therapy TRIDs.Settore CHIM/08 - Chimica Farmaceutica
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Misdiagnosis of familial Mediterranean fever in patients with Anderson-Fabry disease

2013

Fabry disease (FD) is an underdiagnosed pathology due to its symptomatology that overlaps with various systemic and rheumatic disorders, including familial Mediterranean fever (FMF). We examined the Mediterranean fever (MEFV) and α-galactosidase A (GLA) genes, whose mutations are responsible for FMF and FD, respectively, in 42 unrelated patients diagnosed with FMF, which revealed significant ambiguity regarding some of the symptoms which are also present in FD. The objective of this study was to determine the spectrum of mutations present in these genes, in order to identify cases of mistaken diagnosis of FMF and/or missed diagnosis of FD. Ten out of 42 patients had one mutation in homozyg…

Settore MED/04 - Patologia GeneraleFabbry Diseasefamilial Mediterranean feverMEFVAnderson-Fabry diseaseMEFV; familial Mediterranean fever; Anderson-Fabry diseaseMediterranean Fever Genetic diseases
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Characterisation of patients with familial chylomicronaemia syndrome (FCS) and multifactorial chylomicronaemia syndrome (MCS): Establishment of an FC…

2018

Data presented in this article are supplementary material to our article entitled "Identification and diagnosis of patients with familial chylomicronaemia syndrome (FCS): expert panel recom mendations and proposal of an "FCS Score" (Moulin et al., 2018, in press). The data describe the genotypes of patients with familial chylomicronaemia syndrome (FCS) and multifactorial chylomicro naemia syndrome (MCS), from the validation and replication cohorts.

Settore MED/09 - Medicina InternadiagnosisMEDLINE030209 endocrinology & metabolism030204 cardiovascular system & hematologyBioinformaticslcsh:Computer applications to medicine. Medical informatics03 medical and health sciencesfamilial chylomicronaemia syndrome diagnostic score0302 clinical medicineDiagnòsticDiagnosisMalalties hereditàriesscoreMedicinelcsh:Science (General)Genetics Genomics and Molecular BiologyMultidisciplinarybusiness.industryfamilial chylomicronaemia syndrome (FCS) multifactorial chylomicronaemia syndrome (MCS) diagnosis scorefamilial chylomicronaemia syndrome (FCS)Rare diseaseslcsh:R858-859.7lipids (amino acids peptides and proteins)Malalties rareschylomicronaemia syndrome ; multifactorial chylomicronaemia syndromebusinessmultifactorial chylomicronaemia syndrome (MCS)Genetic diseaseslcsh:Q1-390Data in Brief
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SiRNA-mediated selective inhibition of mutant keratin mRNAs responsible for the skin disorder pachyonychia congenita.

2006

RNA interference offers a novel approach for treating genetic disorders including the rare monogenic skin disorder pachyonychia congenita (PC). PC is caused by mutations in keratin 6a (K6a), K6b, K16, and K17 genes, including small deletions and single nucleotide changes. Transfection experiments of a fusion gene consisting of K6a and a yellow fluorescent reporter (YFP) resulted in normal keratin filament formation in transfected cells as assayed by fluorescence microscopy. Similar constructs containing a single nucleotide change (N171K) or a three-nucleotide deletion (N171del) showed keratin aggregate formation. Mutant-specific small inhibitory RNAs (siRNAs) effectively targeted these site…

Small interfering RNABiologymedicine.disease_causeTransfectionGeneral Biochemistry Genetics and Molecular BiologyFusion geneHistory and Philosophy of ScienceCell Line TumorKeratinmedicinePachyonychia congenitaHumansRNA MessengerRNA Small Interferingchemistry.chemical_classificationMutationKeratin Filamentintegumentary systemGeneral NeuroscienceGenetic Diseases InbornKeratin-6RNAKeratin 6Amedicine.diseaseMolecular biologychemistryPachyonychia CongenitaMutationMutagenesis Site-DirectedKeratinsDimerizationAnnals of the New York Academy of Sciences
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What’s the resolutive surgery for Pseudo-Ainhum in vohwinkel syndrome? A case report and review of the literature

2021

Vohwinkel Syndrome, also known as Keratoderma Hereditarium Mutilans, is an extremely rare dominant autosomal keratosis. It typically presents with “starfish” keratoses on the knuckles, palmoplantar keratoderma (PPK), hearing impairment and mutilating digital constriction bands (pseudoainhum) that cause strangulation, often leading to autoamputation of the affected digit. Both medical and surgical treatment haven’t shown to date consistent results, in the treatment of pseudoainhum. In this study we present the case of a woman with Vohwinkel syndrome who showed constriction bands causing ischemic changes of the 5th digit of the right hand for which she was treated with surgery. We also presen…

VOHWINKEL SYNDROMEOrthopedic surgerymedicine.medical_specialtyKeratosisbusiness.industryPseudoainhum Hand surgey Genetic disease Vohwinkel SyndromeGenetic diseaseKERATODERMA HEREDITARIUM MUTILANSCase Reportmedicine.diseasePseudo-ainhumNumerical digitSurgeryPalmoplantar keratodermamedicineHand surgeyOrthopedics and Sports MedicineVohwinkel SyndromeSurgical treatmentbusinessskin and connective tissue diseasesPseudoainhumAutoamputationRD701-811
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Su un caso di sindrome di Wolfram

2011

To aim of this paper was to offer a further contribution to the knowledge af the Wolfram Syndrome within the Italian population about hypotheses of its genetic trasmission and diagnostic problems.The study comprised the genetic investigation and the clinical ophthalmological examination of all the family members. Genetic investigation of family members showed the presence of a genetic disorder,consisting in a mutation of the gene wfs1, located on the short arm of chromosome 4, and which codifies for the protein Wolfranin. The gene wfs1 presents high penetrance and expression,since the mutation is recessive, it can only be clinicallly detected when it is present in the homozygous form. It sh…

Wolfram syndromeSettore MED/30 - Malattie Apparato Visivooptic atrophyGenetic diseases imborn
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No Difference in Penetrance between Truncating and Missense/Aberrant Splicing Pathogenic Variants in MLH1 and MSH2: A Prospective Lynch Syndrome Data…

2021

Background. Lynch syndrome is the most common genetic predisposition for hereditary cancer. Carriers of pathogenic changes in mismatch repair (MMR) genes have an increased risk of developing colorectal (CRC), endometrial, ovarian, urinary tract, prostate, and other cancers, depending on which gene is malfunctioning. In Lynch syndrome, differences in cancer incidence (penetrance) according to the gene involved have led to the stratification of cancer surveillance. By contrast, any differences in penetrance determined by the type of pathogenic variant remain unknown. Objective. To determine cumulative incidences of cancer in carriers of truncating and missense or aberrant splicing pathogenic …

cancer incidence0302 clinical medicineMalalties hereditàriesMissense mutation8Q23.3CàncerCancerGenetics0303 health sciencesmedicine.diagnostic_testFactors de risc en les malaltiesMISMATCH REPAIR GENESRMLH1General MedicinePenetranceLynch syndrome3. Good healthsyöpägeenit030220 oncology & carcinogenesisMedicinesyöpätauditilmaantuvuusGenetic diseasescongenital hereditary and neonatal diseases and abnormalitiesmissense11Q23.1Risk factors in diseasesCANCER-RISKMLH1Articleaberrant splicing03 medical and health sciencesAGEmedicineGenetic predispositionddc:610<i>MSH2</i>Lynchin oireyhtymäpenetrance030304 developmental biologyGenetic testingMLH1; MSH2; penetrance; cancer incidence; truncating; missense; aberrant splicing; Lynch syndromeperinnölliset tauditbusiness.industryMUTATIONSHMSH2Cancernutritional and metabolic diseasesmedicine.diseasedigestive system diseasesMSH2Lynch syndromeMSH23121 General medicine internal medicine and other clinical medicine<i>MLH1</i>businesstruncating
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Molecular Approaches Fighting Nonsense

2021

Nonsense mutations are the result of single nucleotide substitutions in the DNA that change a sense codon (coding for an amino acid) to a nonsense or premature termination codon (PTC) within the coding region of the mRNA [...]

endocrine system diseasesQH301-705.5media_common.quotation_subjectNonsenseNonsense mutationBiologyCatalysisInorganic ChemistrySense Codonchemistry.chemical_compoundHumansCoding regionNucleotideBiology (General)Physical and Theoretical ChemistryQD1-999Molecular BiologySpectroscopymedia_commonchemistry.chemical_classificationGeneticsMessenger RNAOrganic ChemistryGenetic Diseases InbornSettore CHIM/06 - Chimica OrganicaGeneral MedicineNonsense Mediated mRNA DecayComputer Science ApplicationsAmino acidChemistryn/aEditorialchemistryCodon NonsenseGenetic DiseasesProtein BiosynthesisMutationsense organsDNAInternational Journal of Molecular Sciences
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Introduzione

2019

Il volume indaga le radici storiche delle malattie che oggi definiamo genetiche e/o ereditarie, ponendo le premesse terminologiche e scientifiche che stanno a fondamento della riflessione moderna e contemporanea sulla malattia genetica, qui animata dal filo della continuità e della pur forte discontinuità con il pensiero antico. In tal senso, si presenta una retrospettiva storico-critica che, affondando le proprie radici nell’indagine lessicografica dei termini fondanti il pensiero embriogenetico moderno, interroga la validità storica ed epistemica di alcuni concetti e parole-chiave dell’era post-genomica, tra cui quello di causalità, o il rapporto tra natura e cultura, per toccare il nucle…

medical therapymalattia ereditariamedical epistemologygenetic diseasestoria della medicina antica e modernaTerminologia medicamalattia geneticahistory of ancient and modern medicineMedical terminologyhereditary diseaseterapia medicaepistemologia medicaSettore L-FIL-LET/02 - Lingua E Letteratura Greca
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The policy of public health genomics in Italy.

2013

Abstract Italy has a monitoring system for genetic testing, consisting in a periodic census of clinical and laboratory activities performed in the country. The experience is limited, however, concerning the translation of genomic testing for complex diseases into clinical practice. For the first time the Italian Ministry of Health has introduced a policy strategic plan on genomics and predictive medicine within the 2010–2012 National Prevention Plan. This achievement was supported by the Italian Network for Public Health Genomics (GENISAP) and will likely contribute to the integration of public health genomics into health care in the country. Our experience might be of interest not only in …

medicine.medical_specialtyEconomic growthpublic health genomicsSettore MED/03 - GENETICA MEDICASettore MED/42 - Igiene Generale E ApplicataPredictive medicinepredictive medicineEnvironmental healthHealth caremedicineNational PolicyHumansGenetic Testingpolicy genomicsHealth policyGenetic testingStrategic planningnational policyPublic health genomicsmedicine.diagnostic_testbusiness.industryPublic healthHealth PolicyGenetic Diseases InbornGenomicspublic health genomics public health policyHealth PlanningInbornItalyGenetic DiseasesPublic HealthbusinessDelivery of Health Carepolicypublic health genomics; predictive medicine; national policyHealth policy (Amsterdam, Netherlands)
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