Search results for "genomics"

showing 10 items of 1255 documents

Functional Metagenomics of the Bronchial Microbiome in COPD

2015

Altres ajuts: Sociedad Catalana de Neumología; Fundació Catalana de Neumología; Fundació Parc Tauli; Marató de TV3; Sociedad Española de Neumología y Cirugía Torácica; Fundación Menarini; Generalitat Valenciana (Spain) [Prometeo/2009/092] i Centro de Investigación Biomédica en Red de Enfermedades Respiratorias (CIBERES) The course of chronic obstructive pulmonary disease (COPD) is frequently aggravated by exacerbations, and changes in the composition and activity of the microbiome may be implicated in their appearance. The aim of this study was to analyse the composition and the gene content of the microbial community in bronchial secretions of COPD patients in both stability and exacerbati…

MaleExacerbationlcsh:MedicineCarbohydrate metabolismBiologyBioinformaticsPulmonary Disease Chronic ObstructiveRNA Ribosomal 16SmedicineHumansMicrobiomeKEGGlcsh:ScienceLungAgedCOPDMultidisciplinaryLungBacteriaMicrobiotaChronic obstructive pulmonary diseaselcsh:RSputumMiddle Agedmedicine.diseasemedicine.anatomical_structureRibosomal RNAMetagenomicsImmunologyDisease ProgressionMetagenomeSputumPyrosequencingFemalelcsh:QMicrobiomeSequence databasesMetagenomicsmedicine.symptomResearch ArticlePLOS ONE
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Comprehensive exploration of the effects of miRNA SNPs on monocyte gene expression.

2012

We aimed to assess whether pri-miRNA SNPs (miSNPs) could influence monocyte gene expression, either through marginal association or by interacting with polymorphisms located in 3'UTR regions (3utrSNPs). We then conducted a genome-wide search for marginal miSNPs effects and pairwise miSNPs × 3utrSNPs interactions in a sample of 1,467 individuals for which genome-wide monocyte expression and genotype data were available. Statistical associations that survived multiple testing correction were tested for replication in an independent sample of 758 individuals with both monocyte gene expression and genotype data. In both studies, the hsa-mir-1279 rs1463335 was found to modulate in cis the expres…

MaleGene Expressionlcsh:MedicineGenome-wide association studyCoronary Artery DiseaseLinkage DisequilibriumMonocytes0302 clinical medicineGene expressionGenotypelcsh:Science3' Untranslated RegionsOligonucleotide Array Sequence AnalysisGenetics0303 health sciencesMultidisciplinaryGenomicsMiddle Aged3. Good healthFemaleRNA InterferenceEpigeneticsResearch ArticleAdultmedicine.medical_specialtyImmune CellsImmunologyLocus (genetics)Single-nucleotide polymorphismBiologyPolymorphism Single Nucleotide03 medical and health sciencesMolecular geneticsmedicineGeneticsHumansGeneBiology030304 developmental biologyAgedPopulation BiologyHaplotypelcsh:RComputational BiologyMicroRNAsCase-Control StudiesLeukocytes MononuclearLinear ModelsGenetic Polymorphismlcsh:QTranscriptomeGenome Expression Analysis030217 neurology & neurosurgeryPopulation GeneticsGenome-Wide Association StudyPLoS ONE
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Transforming RNA-Seq Data to Improve the Performance of Prognostic Gene Signatures

2014

Gene expression measurements have successfully been used for building prognostic signatures, i.e for identifying a short list of important genes that can predict patient outcome. Mostly microarray measurements have been considered, and there is little advice available for building multivariable risk prediction models from RNA-Seq data. We specifically consider penalized regression techniques, such as the lasso and componentwise boosting, which can simultaneously consider all measurements and provide both, multivariable regression models for prediction and automated variable selection. However, they might be affected by the typical skewness, mean-variance-dependency or extreme values of RNA-…

MaleGene Expressionlcsh:Medicinecomputer.software_genreBioinformaticslcsh:ScienceExtreme value theoryMultidisciplinaryMultivariable calculusStatisticsRegression analysisGenomicsPrognosisKidney NeoplasmsNeoplasm ProteinsLeukemia Myeloid AcuteMedicineProbability distributionFemaleSequence AnalysisAlgorithmsResearch ArticleStatistical DistributionsRiskBoosting (machine learning)Clinical Research DesignFeature selectionBiostatisticsBiologyMachine learningMolecular GeneticsGenome Analysis ToolsCovariateHumansStatistical MethodsGene PredictionBiologyCarcinoma Renal CellProbabilityClinical GeneticsSequence Analysis RNAbusiness.industrylcsh:RPersonalized MedicineModelingComputational BiologyProbability TheorySurvival AnalysisSkewnessMultivariate AnalysisRNAlcsh:QArtificial intelligenceGenome Expression AnalysisTranscriptomebusinesscomputerMathematicsPLoS ONE
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Novel loci for childhood body mass index and shared heritability with adult cardiometabolic traits

2020

The genetic background of childhood body mass index (BMI), and the extent to which the well-known associations of childhood BMI with adult diseases are explained by shared genetic factors, are largely unknown. We performed a genome-wide association study meta-analysis of BMI in 61,111 children aged between 2 and 10 years. Twenty-five independent loci reached genome-wide significance in the combined discovery and replication analyses. Two of these, located nearNEDD4LandSLC45A3, have not previously been reported in relation to either childhood or adult BMI. Positive genetic correlations of childhood BMI with birth weight and adult BMI, waist-to-hip ratio, diastolic blood pressure and type 2 d…

MaleGenetic-variationNedd4 Ubiquitin Protein LigasesPooled AnalysisBlood Pressure0302 clinical medicineHuman geneticsMendelian RandomizationYoung adultChildhealth care economics and organizationsBody mass indexAdiposityGenetics & Heredity0303 health sciencesStatistics1184 Genetics developmental biology physiologyGenomicsadulto3. Good healthCardiovascular DiseasesChild PreschoolPhysical SciencesMenarchegenetic-variationpresión sanguíneaMonosaccharide Transport ProteinsGenetic locieducationenfermedades cardiovascularesProstate-specific AntigenGenetic correlation03 medical and health sciencesSDG 3 - Good Health and Well-beingDiabetes MellitusGeneticsHumansprostate-specific antigenStatistical MethodsMolecular BiologyEcology Evolution Behavior and Systematicschildhood0604 Genetics[SDV.GEN]Life Sciences [q-bio]/GeneticsScience & TechnologyEarly Growth Genetics ConsortiumBiology and Life SciencesComputational Biologynutritional and metabolic diseasesSingle nucleotide polymorphismsMendelian Randomization AnalysisBiological TissueDiabetes Mellitus Type 2estudio de asociación genómica completagenetic factorsmendelian randomizationanálisis de la aleatorización mendelianaproteínas de transporte de monosacáridosBody mass index030217 neurology & neurosurgeryMathematicsDemographyDevelopmental BiologyCardiovascular RiskCancer ResearchobesityPhysiologyhumanosadolescenteOverweightQH426-470Genome-wide association studiesWaist–hip ratioMathematical and Statistical TechniquesMedicine and Health Sciencesbody mass index (BMI)Genetics of diseaseGenetics (clinical)2. Zero hungeradiposityMetaanalysisPhysiological ParametersConnective Tissue/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFemalemedicine.symptomAnatomypooled analysisLife Sciences & BiomedicineResearch ArticleAdultcardiovascular riskAdolescentBirth weightmenarquiaAdipose tissueBiology3121 Internal medicineResearch and Analysis MethodsmedicineoverweightGenetic Predisposition to DiseaseObesity030304 developmental biologyMenarcheWaist-Hip Ratioíndice de masa corporalBody WeightCardiometabolic Risk Factorspredisposición genética a la enfermedadHeritabilityOverweightGenome Analysisyoung-adultsGenome-wide Associationíndice cintura-caderaYoung-adultsgenome-wide associationGenome-Wide Association StudyPLoS genetics
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A high-density SNP linkage scan with 142 combined subtype ADHD sib pairs identifies linkage regions on chromosomes 9 and 16

2008

As part of the International Multi-centre ADHD Gene (IMAGE) project we have completed an affected sibling pair study of 142 narrowly defined DSM-IV combined type ADHD proband-sibling pairs. We found suggestive linkage on chromosomes 9 and 16 with non-parametric multipoint peak LOD scores of 2.13 and 3.1 respectively. There have been several previous ADHD linkage scans. The UCLA study (Fisher et al. 2002; Ogdie et al. 2004; Ogdie et al. 2003), the Dutch study (Bakker et al. 2003), the German study (Hebebrand et al. 2006) and the MGH Study (Faraone et al., submitted) applied the affected sib pair (ASP) strategy; the Columbian study used extended pedigrees ascertained from a population isolate…

MaleGenetics and epigenetic pathways of disease [NCMLS 6]GENOMEWIDE SCANMedizin2804 Cellular and Molecular NeuroscienceCHILDRENComorbidityNeuroinformatics [DCN 3]Severity of Illness IndexDevelopmental psychology2738 Psychiatry and Mental Health0302 clinical medicinePerception and Action [DCN 1]HETEROGENEITYIsraelChildGeneticsObserver Variation0303 health sciencesATTENTION-DEFICIT/HYPERACTIVITY DISORDERPSYCHIATRIC-DISORDERSDOPAMINE TRANSPORTER GENEASSOCIATION10058 Department of Child and Adolescent PsychiatryEuropePsychiatry and Mental healthFemalePsychologyChromosomes Human Pair 9linkageFunctional Neurogenomics [DCN 2]GenotypeDEFICIT HYPERACTIVITY DISORDER610 Medicine & healthPolymorphism Single NucleotideMental health [NCEBP 9]Genetic determinismWhite PeopleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceCognitive neurosciences [UMCN 3.2]Genetic linkage1312 Molecular BiologymedicineSNPAttention deficit hyperactivity disorderHumansADHDSiblingMolecular Biology030304 developmental biologyLinkage (software)SiblingsChromosomemedicine.diseaseSib pairsUnited Statesaffected sib pairsGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with HyperactivityCONDUCT DISORDERLod ScoreDISEQUILIBRIUM030217 neurology & neurosurgeryChromosomes Human Pair 16
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A novel DFNB1 deletion allele supports the existence of a distant cis-regulatory region that controls GJB2 and GJB6 expression

2010

Contains fulltext : 87760_1.pdf (author's version ) (Open Access) Contains fulltext : 87760_2.pdf (Publisher’s version ) (Closed access) Eleven affected members of a large German-American family segregating recessively inherited, congenital, non-syndromic sensorineural hearing loss (SNHL) were found to be homozygous for the common 35delG mutation of GJB2, the gene encoding the gap junction protein Connexin 26. Surprisingly, four additional family members with bilateral profound SNHL carried only a single 35delG mutation. Previously, we demonstrated reduced expression of both GJB2 and GJB6 mRNA from the allele carried in trans with that bearing the 35delG mutation in these four persons. Usin…

MaleGenetics and epigenetic pathways of disease [NCMLS 6][SDV]Life Sciences [q-bio]PenetranceMESH: Base SequenceRegulatory Sequences Nucleic Acidsensorineural hearing lossConnexinsMESH: GenotypeMESH: Hearing Loss Sensorineural/diagnosisMESH: PenetranceGenotypeCopy-number variationGenetics (clinical)Sequence DeletionGeneticsComparative Genomic Hybridization0303 health sciencesMESH: Genetic TestingMESH: Gene Expression Regulation*030305 genetics & heredityPenetranceGJB2PedigreeConnexin 26MESH: Sequence Deletion*MESH: Hearing Loss Sensorineural/geneticsFemaleChromosome DeletionFunctional Neurogenomics [DCN 2]GJB6GenotypeMESH: PedigreeMESH: Chromosome DeletionHearing Loss SensorineuralMolecular Sequence Dataconnexin 26connexin 30DFNB1gene expression regulationGJB2GJB6sensorineural hearing losssequence deletionBiologyMESH: Connexin 30MESH: Connexins/genetics*MESH: Sequence Homology Nucleic AcidArticleGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesMonoallelic MutationGJB6MESH: Connexin 26Sequence Homology Nucleic AcidConnexin 30otorhinolaryngologic diseasesGeneticsHumansGenetic TestingAlleleGeneMESH: Regulatory Sequences Nucleic Acid/genetics*AllelesDFNB1030304 developmental biologyFamily HealthMESH: HumansMESH: Molecular Sequence DataBase SequenceChromosomes Human Pair 13MESH: AllelesBreakpointMESH: MaleMESH: Comparative Genomic HybridizationGene Expression RegulationMESH: Family Healthbiology.proteinHuman medicineMESH: Chromosomes Human Pair 13/geneticsMESH: FemaleClinical Genetics
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Role of cyclooxygenae-2 and 5-lypoxygenase polymorphisms in Alzheimer's disease in a population from northern Italy:implications for pharmacogenomics

2010

Alzheimer's disease (AD) is a neurodegenerative disorder clinically characterized by cognitive deficit with progressive worsening of memory. Recent data indicate that neurons, as well as other brain cells, can express enzymes such as cyclooxygenases (COXs) and 5-lipoxygenase (5-LO) which are considered important in inflammatory cells. Moreover, it has been demonstrated that COX-2 and 5-LO enzymes play a considerable role in the pathophysiology of AD. In order to assess the possible role of COX-2 and 5-LO single nucleotide polymorphisms (SNPs) in AD, we examined their distribution in 341 AD patients and 190 controls from Northern Italy. A significant difference was observed in the distributi…

MaleGenotypePopulationSingle-nucleotide polymorphismDiseaseBiologyBioinformaticsPolymorphism Single NucleotideAlzheimer's diseaseCOX-2 5-LO pharmacogenomicsGene FrequencyPopulation GroupsAlzheimer DiseaseGenetic variationGenotypeSNPHumansSettore MED/05 - Patologia ClinicaGenetic Predisposition to DiseaseAlleleAge of OnseteducationAgedAged 80 and overSettore MED/04 - Patologia Generaleeducation.field_of_studyArachidonate 5-LipoxygenaseGeneral NeuroscienceGeneral MedicineMiddle AgedPsychiatry and Mental healthClinical PsychologyItalyCyclooxygenase 2PharmacogenomicsFemaleGeriatrics and Gerontology
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No Major Host Genetic Risk Factor Contributed to A(H1N1)2009 Influenza Severity

2015

Garcia-Etxebarria, Koldo et al.

MaleGenotypeRisk factors in diseaseslcsh:MedicineGenome-wide association studySingle-nucleotide polymorphismDiseaseBiologyBioinformaticsmedicine.disease_causePolymorphism Single NucleotideSeverity of Illness IndexGripInfluenza A Virus H1N1 SubtypeGene FrequencyRisk FactorsGenotypeSeverity of illnessInfluenza HumanInfluenza A virusmedicineSNPHumanslcsh:ScienceAllele frequencyMultidisciplinaryFactors de risc en les malaltieslcsh:RGenomicsInfluenzaGenòmicaEstudi de casosRNA ViralRNAFemalelcsh:QCase studiesResearch Article
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Signatures of selection in the genome of Swedish warmblood horses selected for sport performance

2019

Abstract Background A growing demand for improved physical skills and mental attitude in modern sport horses has led to strong selection for performance in many warmblood studbooks. The aim of this study was to detect genomic regions with low diversity, and therefore potentially under selection, in Swedish Warmblood horses (SWB) by analysing high-density SNP data. To investigate if such signatures could be the result of selection for equestrian sport performance, we compared our SWB SNP data with those from Exmoor ponies, a horse breed not selected for sport performance traits. Results The genomic scan for homozygous regions identified long runs of homozygosity (ROH) shared by more than 85%…

MaleGenotyping TechniquesPerformanceDIVERSITYBreedingRuns of HomozygosityHorseGenomeHISTORYInbreedingHOMOZYGOSITYGenetics & Heredity0303 health sciencesSnp dataeducation.field_of_studyHomozygoteRUNSASSOCIATIONGenomics04 agricultural and veterinary sciencesBreedDIFFERENTIATIONWarmbloodFemaleLife Sciences & BiomedicineTRAITSResearch ArticleSportsBiotechnologyGENESlcsh:QH426-470lcsh:BiotechnologyPopulationBiologyRuns of homozygosityPolymorphism Single Nucleotide03 medical and health scienceslcsh:TP248.13-248.65GeneticsAnimalsHorsesFunctional studieseducationSelection (genetic algorithm)030304 developmental biologyScience & Technology0402 animal and dairy scienceSelection signature040201 dairy & animal sciencelcsh:GeneticsBiotechnology & Applied MicrobiologyEvolutionary biologyWIDE SCANHaplotype homozygosityBMC Genomics
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Telomere length and physical performance at older ages:an individual participant meta-analysis

2013

<p>Background: Telomeres are involved in cellular ageing and shorten with increasing age. If telomere length is a valuable biomarker of ageing, then telomere shortening should be associated with worse physical performance, an ageing trait, but evidence for such an association is lacking. The purpose of this study was to examine whether change in telomere length is associated with physical performance.</p>\ud \ud <p>Methods: Using data from four UK adult cohorts (ages 53–80 years at baseline), we undertook cross-sectional and longitudinal analyses. We analysed each study separately and then used meta-analytic methods to pool the results. Physical performance was measured us…

MaleGerontologyAnatomy and PhysiologyEpidemiologyPhysical fitnesslcsh:MedicineWalkingCohort StudiesGrip strength0302 clinical medicineLongitudinal StudiesWellcome Trustlcsh:ScienceMusculoskeletal SystemEpidemiological MethodsAged 80 and overMolecular Epidemiology0303 health sciencesMultidisciplinaryHand StrengthChromosome BiologyStatistics15/SAG09977Epidemiology of AgingGenomicsMiddle AgedEPSRCMRCTelomeresBBSRCMedicineFemalePublic HealthCell agingResearch ArticleAdultClinical Research DesignPostureBiostatisticsBiology03 medical and health sciencesTelomere HomeostasisHand strengthGeneticsHumansESRCStatistical MethodsBiologyAged030304 developmental biologyBalance (ability)business.industrylcsh:RTelomere HomeostasisRCUKHuman GeneticsPreferred walking speedBiomarker EpidemiologyCross-Sectional StudiesGeriatricsPhysical FitnessAgeinglcsh:QbusinessMathematics030217 neurology & neurosurgeryDemography
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