Search results for "genotype"

showing 10 items of 1725 documents

TheMAOA T941G polymorphism and short-term treatment response to mirtazapine and paroxetine in major depression

2006

This study investigated the possible association of the MAOA T941G gene variant with differential antidepressant response to mirtazapine and/or paroxetine in 102 patients with major depression (DSM-IV criteria) participating in a randomized double-blind controlled clinical trial. Female mirtazapine-treated patients homozygous for the T-allele had a significantly faster and better treatment response than TG/GG-patients. In males, we failed to show an association between MAOA T941G gene variant and mirtazapine response. In the paroxetine-treated group, there were no significant differences in treatment response between MAOA T941G genotype groups. Time course of response and antidepressant eff…

AdultMaleOncologymedicine.medical_specialtyTime FactorsGenotypeGenetic LinkageMirtazapineMirtazapineMianserinPolymorphism Single NucleotideCellular and Molecular NeuroscienceDouble-Blind MethodGene FrequencyInternal medicineGenotypemedicineHumansAlleleMonoamine OxidaseGenotypingGenetics (clinical)Depressive Disorder MajorSex Characteristicsbusiness.industryMiddle AgedParoxetineAntidepressive AgentsClinical trialParoxetinePsychiatry and Mental healthTreatment OutcomeEndocrinologyAntidepressantFemalebusinessReuptake inhibitormedicine.drugAmerican Journal of Medical Genetics Part B: Neuropsychiatric Genetics
researchProduct

Lack of efficacy of mitoxantrone in primary progressive Multiple Sclerosis irrespective of pharmacogenetic factors: A multi-center, retrospective ana…

2014

Abstract Background Mitoxantrone is used on an off-label basis in primary progressive MS (PPMS). ABC -transporter-genotypes are associated with therapeutic response in relapsing/secondary progressive MS (RP/SPMS). Objective To evaluate potential pharmacogenetic response markers for mitoxantrone in PPMS. Methods 41 mitoxantrone-treated PPMS-patients, 155 mitoxantrone-treated RP/SPMS-patients and 43 PPMS-controls were retrospectively assessed for clinical therapy-response and in correlation with four single-nucleotide-polymorphisms in ABCB1 - and ABCG2 -genes. Results 53.7% PPMS-patients were mitoxantrone-responders, in comparison to 78.1% of RP/SPMS-patients (p = 0.039). There was no associa…

AdultMaleOncologymedicine.medical_specialtyTreatment responseATP Binding Cassette Transporter Subfamily Bmedicine.medical_treatmentImmunologyPrimary Progressive Multiple SclerosisPharmacologyInternal medicineGenotypeLack of efficacymedicineRetrospective analysisATP Binding Cassette Transporter Subfamily G Member 2HumansImmunology and AllergyRetrospective StudiesAnalgesicsMitoxantronebusiness.industryImmunosuppressionMiddle AgedMultiple Sclerosis Chronic ProgressiveNeoplasm Proteins3. Good healthNeurologyPharmacogeneticsATP-Binding Cassette TransportersFemaleNeurology (clinical)MitoxantronebusinessPharmacogeneticsmedicine.drugJournal of Neuroimmunology
researchProduct

Interaction of genetic vulnerability to schizophrenia and Communication Deviance of adoptive parents associated with MMPI schizophrenia vulnerability…

2008

The aim of this study was to establish possible genotype-environment interaction in high-risk and low-risk adoptees' vulnerability to schizophrenia. The study population consisted of a subgroup of 41 adoptive families with a high genetic risk adoptee and 58 families with a low genetic risk adoptee from the Finnish Adoptive Family Study of Schizophrenia. Communication style was assessed based on the Communication Deviance (CD) of the adoptive parents, and the adoptees' vulnerability indicators were measured with the Minnesota Multiphasic Personality Inventory (MMPI). Taken separately, only the genetic liability to schizophrenia, but not the communication style of the adoptive parents, was si…

AdultMaleParentsmedicine.medical_specialtyAdolescentGenotypeSchizophrenia (object-oriented programming)VulnerabilityHostilityEnvironmentSeverity of Illness IndexDevelopmental psychologyMinnesota Multiphasic Personality InventoryCommunication devianceMMPIAdoptionmedicineHumansGenetic Predisposition to DiseaseGenetic riskPsychiatryGenetic vulnerabilityCommunicationPsychiatry and Mental healthSchizophreniaPopulation studyFemalemedicine.symptomPsychologyNordic journal of psychiatry
researchProduct

Gene-environment interaction as a predictor of early adjustment in first episode psychosis.

2017

Abstract Background This study aims to explore the gene-environment interaction hypothesis applied to pre-symptomatic neurodevelopmental phenotypes of first episode psychosis (FEP), that is, genetic factors might increase vulnerability to the effects of environmental adverse conditions occurring at later stages of development. Methods We constructed a schematic ‘two-hit’ model, with Val/Val homozygosity for the catechol- O -methyltransferase ( COMT ) Val158Met polymorphism as the ‘first hit’ and history of obstetric complications and parental socioeconomic status as ‘second hits’. Early adjustment, measured using the Premorbid Adjustment Scale, was considered the main outcome. The study pop…

AdultMaleParentsmedicine.medical_specialtyGenotypePremorbid Adjustment ScaleCatechol O-MethyltransferasePolymorphism Single NucleotideStatistics Nonparametric03 medical and health sciencesYoung Adult0302 clinical medicineMethioninePolymorphism (computer science)First episode psychosismedicineHumansGenetic Predisposition to DiseaseGene–environment interactionPsychiatrySocioeconomic statusBiological PsychiatryRetrospective StudiesAdverse conditionsValinemedicine.disease030227 psychiatryPsychiatry and Mental healthPsychotic DisordersSchizophreniaPopulation studyFemaleGene-Environment InteractionPsychology030217 neurology & neurosurgeryDemographySchizophrenia research
researchProduct

Human papillomavirus DNA in oral mucosal lesions

2002

This study determined the presence of human papillomavirus (HPV) DNA in oral mucosa cells from 121 patients with different types of oral mucosal lesions (13 squamous cell carcinomas, 59 potentially malignant lesions, 49 benign erosive ulcerative lesions) and from 90 control subjects. HPV DNA was detected by nested polymerase chain reaction, and genotype was determined by DNA sequencing. HPV prevalence was 61.5% in carcinomas, 27.1% in potentially malignant lesions, 26.5% in erosive ulcerative lesions, and 5.5% in control subjects. The risk of malignant or potentially malignant lesions was associated with HPV and was statistically significant. HPV-18 was found in 86.5% of HPV-positive lesion…

AdultMalePathologymedicine.medical_specialtyAdolescentBiologyViruslaw.inventionLesionlawGenotypemedicineImmunology and AllergyHumansOral mucosaOral UlcerPapillomaviridaePolymerase chain reactionAgedAged 80 and overHPV infectionMouth Mucosavirus diseasesMiddle Agedmedicine.diseasefemale genital diseases and pregnancy complicationsInfectious Diseasesmedicine.anatomical_structureEpidermoid carcinomaDNA ViralCarcinoma Squamous CellFemaleMouth Neoplasmsmedicine.symptomNested polymerase chain reaction
researchProduct

Genotype-phenotype correlations in nemaline myopathy caused by mutations in the genes for nebulin and skeletal muscle alpha-actin.

2003

We present comparisons of the clinical pictures in a series of 60 patients with nemaline myopathy in whom mutations had been identified in the genes for nebulin or skeletal muscle alpha-actin. In the patients with nebulin mutations, the typical form of nemaline myopathy predominated, while severe, mild or intermediate forms were less frequent. Autosomal recessive inheritance had been verified or appeared likely in all nebulin cases. In the patients with actin mutations, the severe form of nemaline myopathy was the most common, but some had the mild or typical form, and a few showed other associated features such as intranuclear rods or actin accumulation. Most cases were sporadic, but in ad…

AdultMalePathologymedicine.medical_specialtyAdolescentGenotypeNonsense mutationDNA Mutational AnalysisMuscle ProteinsBiologymedicine.disease_causeMyopathies Nemaline03 medical and health sciencesNebulin0302 clinical medicineNemaline myopathyGenotypemedicineHumansChildMuscle SkeletalGenetics (clinical)Actin030304 developmental biologyGenetics0303 health sciencesMutationInfantmedicine.diseaseCongenital myopathyPhenotypeActinsPhenotypeNeurologyChild PreschoolPediatrics Perinatology and Child HealthMutationbiology.proteinFemaleNeurology (clinical)030217 neurology & neurosurgeryNeuromuscular disorders : NMD
researchProduct

Low SPINK5 expression in chronic rhinosinusitis

2012

Objectives/Hypothesis: Chronic rhinosinusitis (CRS) is a multifactorial disease that probably arises as a result of genetic diversity and environmental factors. SPINK5 is a serine protease inhibitor, which is supposed to be an important regulator of epithelial barrier maintenance. The role of SPINK5 polymorphisms and expression in CRS, especially in individuals with aspirin intolerance, is unclear. Study Design: SPINK5 single-nucleotide polymorphisms (SNPs) and SPINK5 expression levels were correlated with CRS without (CRSsNP) and with nasal polyps (CRSwNP), aspirin intolerance, asthma, and allergies. Methods: One hundred four nasal tissue samples, 15 from patients with CRSsNP, 59 from pati…

AdultMalePathologymedicine.medical_specialtyAllergyAdolescentGenotypeProteinase Inhibitory Proteins SecretorySingle-nucleotide polymorphismPolymerase Chain ReactionPolymorphism Single NucleotideSensitivity and SpecificitySampling Studieslaw.inventionDrug HypersensitivityTissue Culture TechniquesYoung AdultNasal PolypsReference ValueslawGenotypemedicineHumansSNPNasal polypsRNA MessengerSinusitisPolymerase chain reactionAgedRhinitisAsthmaAged 80 and overAspirinbusiness.industryMiddle Agedmedicine.diseaseAsthmaPathophysiologyNasal MucosaGene Expression RegulationOtorhinolaryngologyChronic DiseaseImmunologySerine Peptidase Inhibitor Kazal-Type 5FemalebusinessThe Laryngoscope
researchProduct

Reproductive outcome of women with 21-hydroxylase-deficient nonclassic adrenal hyperplasia.

2006

Many women with 21-hydroxylase (21-OH)-deficient nonclassic adrenal hyperplasia (NCAH) carry at least one allele containing a severe mutation of CYP21, and as such are at risk for giving birth to an infant having classic adrenal hyperplasia (CAH). Infants with NCAH typically are asymptomatic at birth, in contrast to those with CAH, but they do develop symptoms of hyperandrogenism later in childhood or as adults. This international multicenter study, conducted both retrospectively and prospectively, was an attempt to determine how often mothers with 21-OH-deficient NCAH bear infants having CAH or NCAH. The 101 women entering the study had a total of 203 pregnancies that could be evaluated. F…

AdultMalePediatricsmedicine.medical_specialtyReferralGenotypeOffspringEndocrinology Diabetes and MetabolismClinical BiochemistryContext (language use)AsymptomaticBiochemistryEndocrinologyPregnancyInternal medicinePrevalenceMedicineHumansProspective StudiesProspective cohort studyAdrenal HyperplasiaRetrospective StudiesPregnancybiologyAdrenal Hyperplasia Congenitalbusiness.industryIncidence (epidemiology)HyperandrogenismBiochemistry (medical)21-HydroxylaseInfant NewbornObstetrics and GynecologyInfantRetrospective cohort studyGeneral MedicineHyperplasiamedicine.diseaseEndocrinologyGlucocorticoid therapyChild Preschoolbiology.proteinFemaleSteroid 21-Hydroxylasemedicine.symptomLive birthbusinessThe Journal of clinical endocrinology and metabolism
researchProduct

Interest of genotyping and phenotyping of drug-metabolizing enzymes for the interpretation of biological monitoring of exposure to styrene

2002

In the field of occupational and/or environmental toxicology, the measurement of specific metabolites in urine may serve to assess exposure to the parent compounds (biological monitoring of exposure). Styrene is one of the chemicals for which biological monitoring programs have been validated and implemented in environmental and occupational medicine. However, inter-individual differences in the urinary excretion exist both for the main end-products (mandelic acid and phenylglyoxylic acid) and for its specific mercapturic acids (phenylhydroxyethylmercapturic acids, PHEMA). This limits to a certain extent the use of these metabolites for an accurate assessment of styrene exposure. In a group…

AdultMalePhenylglyoxylic acidGenotypeMetaboliteUrinary systemPopulation10050 Institute of Pharmacology and Toxicology610 Medicine & healthUrinePharmacologyBiologyPolymerase Chain Reaction3000 General Pharmacology Toxicology and PharmaceuticsExcretionchemistry.chemical_compound1311 GeneticsGeneticsHumansLymphocytesGeneral Pharmacology Toxicology and PharmaceuticseducationGenotypingStyreneGlutathione TransferaseEpoxide Hydrolaseseducation.field_of_studyPolymorphism GeneticGlyoxylatesCytochrome P-450 CYP2E1Environmental ExposureCYP2E1AcetylcysteineIsoenzymesPhenotypeGlutathione S-Transferase piBiochemistrychemistry570 Life sciences; biologyMandelic AcidsBiomarkersPolymorphism Restriction Fragment LengthEnvironmental Monitoring
researchProduct

Genotyping and antifungal susceptibility of human subgingival Candida albicans isolates.

2002

Subgingival colonization by Candida albicans has been described in human immunodeficiency virus (HIV)-infected individuals, but subgingival isolates have scarcely been characterized, particularly with respect to genotype and antifungal susceptibility. A series of 29 subgingival strains of C. albicans isolated from nine HIV-infected individuals was typed by electrophoretic karyotyping and tested for susceptibility to fluconazole, itraconazole, the new investigational triazole posaconazole and amphotericin B. DNA typing showed genetic heterogeneity within subgingival isolates, as almost every individual harbored his/her own specific isolate. Genetic identity was usually demonstrated within or…

AdultMalePosaconazoleAntifungal AgentsItraconazoleDental PlaqueHIV InfectionsMicrobial Sensitivity TestsMicrobiologyPeriodontal pathogenGenetic HeterogeneityDrug Resistance FungalAmphotericin BGenotypeCandida albicansmedicineHumansCandida albicansDNA FungalMycological Typing TechniquesGeneral DentistryGenotypingFluconazolebiologyCell BiologyGeneral MedicineTriazolesbiology.organism_classificationVirologyCorpus albicansOtorhinolaryngologyKaryotypingFemaleItraconazoleFluconazolemedicine.drugArchives of oral biology
researchProduct