Search results for "glia"

showing 10 items of 1274 documents

The Amino Acid Transporter JhI-21 Coevolves with Glutamate Receptors, Impacts NMJ Physiology, and Influences Locomotor Activity in Drosophila Larvae

2015

AbstractChanges in synaptic physiology underlie neuronal network plasticity and behavioral phenomena, which are adjusted during development. The Drosophila larval glutamatergic neuromuscular junction (NMJ) represents a powerful synaptic model to investigate factors impacting these processes. Amino acids such as glutamate have been shown to regulate Drosophila NMJ physiology by modulating the clustering of postsynaptic glutamate receptors and thereby regulating the strength of signal transmission from the motor neuron to the muscle cell. To identify amino acid transporters impacting glutmatergic signal transmission, we used Evolutionary Rate Covariation (ERC), a recently developed bioinforma…

0301 basic medicinejuvenile-hormonemelanogasterAmino Acid Transport Systemsextracellular glutamateprotein-protein interactionsPhysiology[ SDV.BA ] Life Sciences [q-bio]/Animal biologySynaptic Transmissionin-vivo0302 clinical medicinePostsynaptic potentialDrosophila Proteinsgenesglial xctMotor NeuronsAnimal biologyMultidisciplinary[SDV.BA]Life Sciences [q-bio]/Animal biologyGlutamate receptorBiological Evolutiondrosophilemedicine.anatomical_structureReceptors GlutamateLarvaExcitatory postsynaptic potentialDrosophila[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Drosophila ProteinSignal Transductionevolutionary rate covariationNeuromuscular JunctionPresynaptic TerminalsNeurotransmissionBiologyMotor ActivityArticlesynaptic vesicle03 medical and health sciencesGlutamatergicneuromuscular-junctionBiologie animalemedicineAnimalsAmino acid transporterevolutionary rate covariation;protein-protein interactions;juvenile-hormone;neuromuscular-junction;synaptic vesicle;in-vivo;extracellular glutamate;glial xct;melanogaster;genesfungiNeurosciencesExcitatory Postsynaptic PotentialsMotor neuron030104 developmental biology[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neurons and CognitionMutation030217 neurology & neurosurgeryScientific Reports
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Commensal Bacterium Rothia aeria Degrades and Detoxifies Gluten via a Highly Effective Subtilisin Enzyme

2020

Celiac disease is characterized by a chronic immune-mediated inflammation of the small intestine, triggered by gluten contained in wheat, barley, and rye. Rothia aeria, a gram-positive natural colonizer of the oral cavity and the upper digestive tract is able to degrade and detoxify gluten in vitro. The objective of this study was to assess gluten-degrading activity of live and dead R. aeria bacteria in vitro, and to isolate the R. aeria gluten-degrading enzyme. Methods: After an overnight fast, Balb/c mouse were fed a 1 g pellet of standard chow containing 50% wheat (and 4% gliadin) with or without 1.6 &times

0301 basic medicinelcsh:TX341-641detoxifymedicine.disease_causedigestive systemBacterial cell structure<i>Rothia</i>Microbiology03 medical and health sciences0302 clinical medicinemedicineZymographydegradationchemistry.chemical_classificationepitopeNutrition and DieteticsbiologyRothia aeria<i>Bacillus</i>food and beveragesnutritional and metabolic diseasesbiology.organism_classificationGlutenneutralizecuredigestive system diseasesEnzyme assay030104 developmental biologychemistryglutencommensalbiology.proteinsubtilisin030211 gastroenterology & hepatologyDigestionGliadinlcsh:Nutrition. Foods and food supplyceliac diseaseBacteriaFood ScienceNutrients
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Exosomes as mediators of neuron-glia communication in neuroinflammation

2019

In recent years, a type of extracellular vesicles named exosomes has emerged that play an important role in intercellular communication under physiological and pathological conditions. These nanovesicles (30–150 nm) contain proteins, RNAs and lipids, and their internalization by bystander cells could alter their normal functions. This review focuses on recent knowledge about exosomes as messengers of neuron-glia communication and their participation in the physiological and pathological functions in the central nervous system. Special emphasis is placed on the role of exosomes under toxic or pathological stimuli within the brain, in which the glial exosomes containing inflammatory molecules…

0301 basic medicinemedia_common.quotation_subjectCentral nervous systemneuronsReviewexosomesBiologyExosomelcsh:RC346-429neuroinflammationPathogenesis03 medical and health sciences0302 clinical medicineDevelopmental NeuroscienceBystander effectmedicineInternalizationNeuroinflammationlcsh:Neurology. Diseases of the nervous systemmedia_commonbiomarkers; exosomes; glial cells; neuroinflammation; neuron-glia commuication; neurons; neuropathology; therapyneuropathologytherapyneuron-glia commuicationbiomarkersMicrovesiclesglial cells030104 developmental biologymedicine.anatomical_structureNeuronNeuroscience030217 neurology & neurosurgeryNeural Regeneration Research
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Early increased density of cyclooxygenase-2 (COX-2) immunoreactive neurons in Down syndrome

2017

iNeuroinflammation is one of the hallmarks of Alzheimer's disease. One of the enzymes involved in neuroinflammation, even in early stages of the disease, is COX-2, an inducible cyclooxygenase responsible for the generation of eicosanoids and for the generation of free radicals. Individuals with Down syndrome develop Alzheimer's disease early in life. Previous studies pointed to the possible overexpression of COX-2 and correlated it to brain regions affected by the disease. We analysed the COX-2 expression levels in individuals with Down syndrome and in young, adult and old mice of the Ts65Dn mouse model for Down syndrome. We have observed an overexpression of COX-2 in both, Down syndrome in…

0301 basic medicinemedicine.medical_specialtyDown syndromelcsh:MedicineMice TransgenicDiseasePathology and Forensic Medicineneuroinflammationmicroglia</i>Mice03 medical and health sciences0302 clinical medicineInternal medicinemedicineAnimalsHumansPostnatal dayNeuroinflammationNeuronschemistry.chemical_classificationMicrogliabiologybusiness.industryNeurodegenerationlcsh:RBrainmedicine.disease<i>Ts65Dn030104 developmental biologyEndocrinologymedicine.anatomical_structureEnzymechemistryCyclooxygenase 2biology.proteinNeurology (clinical)CyclooxygenaseDown SyndromebusinessNeuroscienceAlzheimer’s disease030217 neurology & neurosurgeryFolia Neuropathologica
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Current disease modifying approaches to treat Parkinson's disease

2015

Parkinson's disease (PD is a progressive neurological disorder characterized by the degeneration and death of midbrain dopamine and non-dopamine neurons in the brain leading to motor dysfunctions and other symptoms, which seriously influence the quality of life of PD patients. The drug L-dopa can alleviate the motor symptoms in PD, but so far there are no rational therapies targeting the underlying neurodegenerative processes. Despite intensive research, the molecular mechanisms causing neuronal loss are not fully understood which has hampered the development of new drugs and disease-modifying therapies. Neurotrophic factors are by virtue of their survival promoting activities attract candi…

0301 basic medicinemedicine.medical_specialtyParkinson's diseaseNeurturinNeurotrophic factorBiologySettore BIO/09 - Fisiologia03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineNeuroinflammationDopamineNeurotrophic factorsInternal medicineα-SynucleinmedicineGlial cell line-derived neurotrophic factorMolecular BiologyCerebral dopamine neurotrophic factorDopamine neuronPharmacologyDopaminergicCell Biologymedicine.diseaseDopamine neurons; ER stress; Mitochondria; Neuroinflammation; Neuropeptides; Neurotrophic factors; Protein aggregation; α-Synuclein; Molecular Medicine; Molecular Biology; Pharmacology; Cellular and Molecular Neuroscience; Cell Biology3. Good healthMitochondriaNeuropeptide030104 developmental biologyNerve growth factorEndocrinologybiology.proteinER streMolecular MedicineProtein aggregationNeuroscience030217 neurology & neurosurgerymedicine.drug
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Glucose and hypothalamic astrocytes: More than a fueling role?

2015

Brain plays a central role in energy homeostasis continuously integrating numerous peripheral signals such as circulating nutrients, and in particular blood glucose level, a variable that must be highly regulated. Then, the brain orchestrates adaptive responses to modulate food intake and peripheral organs activity in order to achieve the fine tuning of glycemia. More than fifty years ago, the presence of glucose-sensitive neurons was discovered in the hypothalamus, but what makes them specific and identifiable still remains disconnected from their electrophysiological signature. On the other hand, astrocytes represent the major class of macroglial cells and are now recognized to support an…

0301 basic medicinemedicine.medical_specialty[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionHypothalamusNutrient sensingEnergy homeostasis03 medical and health sciences0302 clinical medicineInternal medicinemedicineAnimalsHumansastroglial hemichannelsglucoselactateArc (protein)biologyastroglial gap junctionsMechanism (biology)GlucokinaseGeneral NeuroscienceGlucose transporterGap Junctionsconnexins 30 and 43030104 developmental biologyEndocrinologyHypothalamushypothalamic glucose sensing[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Astrocytesbiology.proteinGLUT2[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neuroscience[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgery
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Immunosuppression, peripheral inflammation and invasive infection from endogenous gut microbiota activate retinal microglia in mouse models

2016

Although its actual role in the progression of degenerative processes is not fully known, the persistent activated state of retinal microglia and the concurrent secretion of inflammatory mediators may contribute to neuronal death and permanent vision loss. Our objective was to determine whether non-ocular conditions (immunosuppression and peripheral inflammation) could lead to activation of retinal microglia. Mouse models of immunosuppression induced by cyclophosphamide and/or peripheral inflammation by chemically induced sublethal colitis in C57BL/6J mice were used. Retinal microglia morphology, spatial distribution and complexity, as well as MHCII and CD11b expression levels were determin…

0301 basic medicinemedicine.medical_treatmentImmunologyInflammationMicrobiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDownregulation and upregulationVirologymedicineColitisMicrogliabiologyImmunosuppressionRetinalmedicine.diseaseTLR2030104 developmental biologymedicine.anatomical_structureIntegrin alpha MchemistryImmunologybiology.proteinmedicine.symptom030217 neurology & neurosurgeryMicrobiology and Immunology
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The Multiple Sclerosis Genomic Map: Role of peripheral immune cells and resident microglia in susceptibility

2017

Abstract:We assembled and analyzed genetic data of 47,351 multiple sclerosis (MS) subjects and 68,284 control subjects and establish a reference map of the genetic architecture of MS that includes 200 autosomal susceptibility variants outside the major histocompatibility complex (MHC), one chromosome X variant, and 32 independent associations within the extended MHC. We used an ensemble of methods to prioritize up to 551 potentially associated MS susceptibility genes, that implicate multiple innate and adaptive pathways distributed across the cellular components of the immune system. Using expression profiles from purified human microglia, we do find enrichment for MS genes in these brain -…

0303 health sciencesMicrogliaMultiple sclerosisCentral nervous systemBiologymedicine.diseaseMajor histocompatibility complex03 medical and health sciences0302 clinical medicineImmune systemmedicine.anatomical_structureAutoimmune ProcessImmunologymedicinebiology.proteinGene030217 neurology & neurosurgeryX chromosome030304 developmental biology
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Immunohistopathology in Diagnostic Neuropathology

1983

Within the field of surgical pathology, immunohistochemistry is now frequently applied to the morphological diagnosis of lymphomas (11), certain carcinomas and other selected types of tumor (12). In neuropathology, the study of neuro-oncological and non-neoplastic diseases may also receive diagnostic support from performing immunohistological techniques, which encompass immunofluorescent and immunoperoxidase methods. The following report represents a survey of our experience in this recently developed field of diagnostic neuropathology.

0303 health sciencesPathologymedicine.medical_specialtyGlial fibrillary acidic proteinbiologyImmunoperoxidaseNeuropathologymedicine.disease3. Good healthSurgical pathology03 medical and health sciences0302 clinical medicinePituitary adenoma030220 oncology & carcinogenesisbiology.proteinmedicineImmunohistochemistry030304 developmental biology
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Modulation of lipid membrane organization of Müller Glial Cells (MGC) by 24-(S)-hydroxycholesterol (24-S-OHC) in glaucoma

2016

Modulation of lipid membrane organization of Müller Glial Cells (MGC) by 24-(S)-hydroxycholesterol (24-S-OHC) in glaucoma. Session 3: Nutrition and oxysterols biological activities. ENOR Symposium

24 s hydroxycholesterolraft[SDV.AEN] Life Sciences [q-bio]/Food and Nutritionglaucomamuller glial cell
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