Search results for "glycolysi"

showing 10 items of 155 documents

Lactate: mirror and motor of tumor malignancy

2004

A number of studies have shown that malignant transformation is associated with an increase in glycolytic flux and in anaerobic and aerobic cellular lactate excretion. Using quantitative bioluminescence imaging in various primary carcinomas in patients (uterine cervix, head and neck, colorectal region) at first diagnosis of the disease, we showed that lactate concentrations in tumors in vivo can be relatively low or extremely high (up to 40 micromol/g) in different individual tumors or within the same lesion. In all tumor entities investigated, high concentrations of lactate were correlated with a high incidence of distant metastasis already in an early stage of the disease. Low lactate tum…

MaleVascular Endothelial Growth Factor ACancer ResearchPathologymedicine.medical_specialtyMalignancyMalignant transformationchemistry.chemical_compoundNeoplasmsLactate dehydrogenasemedicineHumansBioluminescence imagingRadiology Nuclear Medicine and imagingGlycolysisLactic AcidHyaluronic AcidNeoplasm MetastasisL-Lactate Dehydrogenasebusiness.industryHypoxia-Inducible Factor 1 alpha Subunitmedicine.diseaseCell HypoxiaUp-RegulationVascular endothelial growth factorCell Transformation NeoplasticOncologychemistryCancer cellFemalebusinessAnaerobic exerciseTranscription FactorsSeminars in Radiation Oncology
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Expression of transketolase TKTL1 predicts colon and urothelial cancer patient survival: Warburg effect reinterpreted

2006

Abstract Tumours ferment glucose to lactate even in the presence of oxygen (aerobic glycolysis; Warburg effect). The pentose phosphate pathway (PPP) allows glucose conversion to ribose for nucleic acid synthesis and glucose degradation to lactate. The nonoxidative part of the PPP is controlled by transketolase enzyme reactions. We have detected upregulation of a mutated transketolase transcript (TKTL1) in human malignancies, whereas transketolase (TKT) and transketolase-like-2 (TKTL2) transcripts were not upregulated. Strong TKTL1 protein expression was correlated to invasive colon and urothelial tumours and to poor patients outcome. TKTL1 encodes a transketolase with unusual enzymatic prop…

Maleaerobic glycolysiCancer ResearchAdenocarcinomanPentose phosphate pathwayTransketolaseBiologyMetastasispentose phosphate pathway (PPP)Downregulation and upregulationPredictive Value of TestsmedicineHumansNeoplasm InvasivenessGlycolysisNeoplasm MetastasisMolecular Diagnosticsaerobic glycolysisAgedtransketolase-like-1 (TKTL1)transketolase (TKT)Gene Expression ProfilingCancerMiddle AgedPrognosismedicine.diseaseSurvival AnalysisWarburg effectUp-RegulationUrinary Bladder NeoplasmsOncologyBiochemistryAnaerobic glycolysispharmacodiagnostic markerColonic NeoplasmsCancer researchFemaleWarburg effectTransketolaseGlycolysisBritish Journal of Cancer
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Striated muscle-specific serine/threonine-protein kinase beta (SPEGβ) segregates with high- versus low-responsiveness to endurance exercise training

2019

Bidirectional selection for either high or low responsiveness to endurance running has created divergent rat phenotypes of high-response trainers (HRT) and low-response trainers (LRT). We conducted proteome profiling of HRT and LRT gastrocnemius of 10 female rats (body weight 279 ± 35 g; n = 5 LRT and n = 5 HRT) from generation 8 of selection. Differential analysis of soluble proteins from gastrocnemius was conducted by label-free quantitation. Genetic association studies were conducted in 384 Russian international-level athletes (age 23.8 ± 3.4 yr; 202 men and 182 women) stratified to endurance or power disciplines. Proteomic analysis encompassed 1,024 proteins, 76 of which exhibited stat…

MaleentsyymitPhysiologykestävyysharjoitteluliquid chromatography mass spectrometryMuscle ProteinslihaksetSerine threonine protein kinaseRC1200Gene Frequencyendurance trainingProtein Interaction MapsliikuntafysiologiaExercise capacityPhenotypeexercise capacitymedicine.anatomical_structureOrgan SpecificityFemalesportsGlycolysisResearch Articlekinaasitmedicine.medical_specialtymassaspektrometriaresponsiveness to exerciseBiologyProtein Serine-Threonine KinasesPolymorphism Single NucleotideYoung Adultartificial selection modelEndurance trainingInternal medicinePhysical Conditioning AnimalGeneticsmedicineAnimalsHumansco‐immunopreciptiationskeletal muscleBeta (finance)Muscle SkeletalQH426Skeletal musclelabel‐free quantitationMuscle StriatedRatsharjoitusvasteEndocrinologyProtein Kinases
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Antagonistic effects of hypertrehalosemic neuropeptide on the activities of 6-phosphofructo-1-kinase and fructose-1,6-bisphosphatase in cockroach fat…

2001

Hypertrehalosemic neuropeptides from the corpora cardiaca such as the decapeptide Bld HrTH bring about a profound switch in the metabolic activity of cockroach fat body during which production of the blood sugar trehalose is stimulated while the catabolism of carbohydrate (glycolysis) is inhibited. The mechanisms of the metabolic switch are not fully understood. Incubation of isolated fat body from the cockroach Blaptica dubia with 10(-8) M Bld HrTH, for 10-60 min, stimulated glycogen breakdown and increased the content of the substrates of both the glycolytic enzyme 6-phosphofructo-1-kinase (PFK, EC 2.7.1.11) and the gluconeogenic enzyme fructose-1,6-bisphosphatase (FBPase, EC 3.1.3.11) in…

Malemedicine.medical_specialtyBlaptica dubiaPhosphofructokinase-1Fat BodyFructose 16-bisphosphataseCockroachesIn Vitro TechniquesBiologyBiochemistryGene Expression Regulation Enzymologicchemistry.chemical_compoundInternal medicineFructosediphosphatesmedicineAnimalsGlycolysisPhosphofructokinase 1Molecular BiologyCatabolismNeuropeptidesTrehaloseFructosebiology.organism_classificationAdenosine MonophosphateFructose-BisphosphataseKineticsEndocrinologyFructose 26-bisphosphatechemistryBiochemistryInsect HormonesInsect Sciencebiology.proteinGlycogenPhosphofructokinaseInsect Biochemistry and Molecular Biology
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Comparative study of the effects of chloral hydrate and trichloroethanol on cerebral metabolism

1973

The isolated perfused rat brain was used for a comparative study of the effects of chloral hydrate and trichloroethanol on cerebral energy metabolism. After a perfusion period of 30 min the brain levels of the following substrates and metabolites were measured spectrophotometrically: P-creatine, creatine, ATP, ADP, AMP, glycogen, glucose, glucose-6-P, fructose diphosphate, α-glycero-P, dihydroxyacetone-P, pyruvate, lactate, glutamate, α-ketoglutarate and ammonia. Furthermore, the concentration of chloral hydrate and trichloroethanol in the isolated brain and in the perfusion medium was measured colorimetrically. Little more than 10% of chloral hydrate in the isolated brain and in the perfus…

Malemedicine.medical_specialtyChloral hydrateIn Vitro TechniquesCreatinechemistry.chemical_compoundAdenosine TriphosphateInternal medicinemedicineAnimalsGlycolysisChloral HydrateBrain ChemistryPharmacologyEthanolEthanolGlycogenHydrocarbons HalogenatedBrainFructoseGeneral MedicineIsolated brainCreatineAdenosine MonophosphateRatsAdenosine DiphosphatePerfusionAdenosine diphosphateGlucoseEndocrinologyBiochemistrychemistryCattleChlorineGlycolysisGlycogenmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Thimerosal induces calcium mobilization, fructose 2,6-bisphosphate synthesis and cytoplasmic alkalinization in rat thymus lymphocytes

1991

The effect of thimerosal on intracellular calcium ([Ca2+]i), pH (pHi) and fructose 2,6-bisphosphate (Fru 2,6-P2) in thymus lymphocytes was investigated. The effect of thimerosal on cell growth was also examined. Thimerosal produced a dose-dependent increase in [Ca2+]i, pHi and in the level of fructose 2,6-bisphosphate. Thimerosal was, however, unable to produce cell proliferation and inhibited [3H]thymidine incorporation when cells were challenged with PHA and costimulator. In the absence of external calcium, thimerosal produced only a slight increase in [Ca2+]i. In Na(+)-containing buffer, thimerosal induced an initial acidification (0.05 +/- 0.01 pH units), followed by an alkalinization o…

Malemedicine.medical_specialtyCytoplasmT-LymphocytesFluorescence spectrometrychemistry.chemical_elementBiologyCalciumLymphocyte ActivationTritiumCalcium in biologychemistry.chemical_compoundInternal medicinemedicineFructosediphosphatesAnimalsLactic AcidMolecular BiologyIon transporterProtein kinase CThimerosalSodiumThimerosalFructoseRats Inbred StrainsCell BiologyHydrogen-Ion ConcentrationRatsEndocrinologyFructose 26-bisphosphatechemistryLactatesCalciumGlycolysisHydrogenThymidine
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Endogenous nitric oxide is responsible for the early loss of P450 in cultured rat hepatocytes

1998

AbstractLoss of P450 during the early hours of monolayer formation is known to be the more serious limitation of primary cultured hepatocytes as an adequate model for the study of drug metabolism, toxicity and P450 induction. This study reports that endogenous nitric oxide (NO) formation is activated shortly after isolation by the classical collagenase-based liver perfusion methods. Both rapid P450 loss and aerobic mitochondrial energy metabolism impairment – with subsequent changes on glucose metabolism – are directly related to the high local generation of the radical at this stage. These effects can be reverted by the sole addition of NO biosynthesis inhibitors during liver perfusion and…

Malemedicine.medical_specialtyGlycogenolysisGlycogenolysisBiophysicsMitochondria LiverCarbohydrate metabolismHepatocyte primary cultureBiochemistryNitric oxideRats Sprague-DawleyP450 contentchemistry.chemical_compoundCytochrome P-450 Enzyme SystemBiosynthesisStructural BiologyInternal medicineGeneticsmedicineAnimalsGlycolysisMolecular BiologyCells CulturedAMPDrug metabolismAdenine NucleotidesNitric oxideCell BiologyLiver GlycogenRatsKineticsNG-Nitroarginine Methyl EsterEndocrinologyLiverchemistryToxicityMicrosomes LiverCollagenaseGlycolysisDrug metabolismmedicine.drugFEBS Letters
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Effects of insulin-like growth factor I on the rates of glucose transport and utilization in rat skeletal muscle in vitro.

1992

1. The effects of insulin-like growth factor I (IGF-I) on the rates of glucose transport and utilization and its interaction with insulin were investigated in rat soleus muscle in vitro. IGF-I increased the rates of glucose transport, lactate formation, glycogen synthesis and the flux of glucose to hexose monophosphate, but it had no effect on the rate of glucose oxidation or glycogenolysis. 2. In the absence of insulin, low levels of IGF-I (0-30 ng/ml) increased the rate of glycolysis and the content of fructose 2,6-bisphosphate, but the content of glucose 6-phosphate remained unaltered; at higher levels of IGF-I (300-3000 ng/ml) the rate of glycolysis and the content of fructose 2,6-bisph…

Malemedicine.medical_specialtyGlycogenolysismedicine.medical_treatmentGlucose-6-PhosphateBiologyIn Vitro TechniquesBiochemistrychemistry.chemical_compoundInternal medicinemedicineFructosediphosphatesAnimalsInsulinGlycolysisInsulin-Like Growth Factor IPhosphorylationGlycogen synthaseMolecular BiologyGlycogenInsulinMusclesGlucose transporterGlucosephosphatesFructoseBiological TransportRats Inbred StrainsCell BiologyRatsKineticsEndocrinologyGlucosechemistryFructolysisbiology.proteinGlycolysisOxidation-ReductionGlycogenResearch Article
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Influence of therapeutic and toxic doses of neuroleptics and antidepressants on energy metabolism of the isolated perfused rat brain.

1973

The isolated perfused rat brain was used for a comparative study of the effects of promazine, imipramine, monodesmethyl promazine and desipramine on cerebral energy metabolism. After perfusion for 30 min or 1 h the brain levels of the following substrates and metabolites were estimated: P-creatine, creatine, ATP, ADP, AMP, glycogen, glucose, glucose-6-P, fructose diphosphate, dihydroxyacetone-P, pyruvate, lactate, α-ketoglutarate, and ammonia. Drug concentrations of 5·10−6 M and 10−5 M in the perfusion medium caused a significant decrease of glucose-6-P alone. When the drug concentration was raised to a toxic range (10−4 M), reflected in the EEG by the pattern of secondary discharges, an ac…

Malemedicine.medical_specialtyImipraminePhosphocreatineBiologyPharmacologyCreatineImipramineAcetonechemistry.chemical_compoundOrganophosphorus CompoundsAmmoniaInternal medicineDesipramineTriosesmedicineAnimalsGlycolysisPyruvatesPromazinePromazinePharmacologyGlycogenDose-Response Relationship DrugDesipramineFructosephosphatesGlucosephosphatesBrainFructoseElectroencephalographyGeneral MedicineRibonucleotidesCreatineAntidepressive AgentsRatsPerfusionEndocrinologyGlucoseTranquilizing AgentschemistryLactatesKetoglutaric AcidsEnergy MetabolismPerfusionGlycolysismedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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The neuroprotective effect of lactate is not due to improved glutamate uptake after controlled cortical impact in rats.

2012

For many years lactate was considered to be a waste product of glycolysis. Data are accumulating that suggest that lactate is an important energy substrate for neurons during activation. In severe traumatic brain injury (TBI) glutamate release and ischemic cerebral blood flow (CBF) are major factors for a mismatch between energy demand and supply and for neuronal cell death. Although ATP and behavior could be improved by lactate treatment after TBI, no histological correlate nor any linkage to better astrocytic glutamate uptake or CBF as possible mechanisms have been described. We subjected male rats to a controlled cortical impact (CCI; 5 m/sec, 2.5 mm). To study the effects of lactate tre…

Malemedicine.medical_specialtyMicrodialysisCoumaric AcidsMicrodialysisGlutamic AcidNeuroprotectionRats Sprague-DawleyStereotaxic TechniquesOxygen ConsumptionInternal medicinemedicineAnimalsGlycolysisLactic AcidChromatography High Pressure LiquidBrain ChemistryCerebral CortexSkull FracturesChemistryGlutamate receptorGlutamic acidRatsmedicine.anatomical_structureEndocrinologyNeuroprotective AgentsCerebral blood flowCerebral cortexAnesthesiaBrain InjuriesCerebrovascular CirculationStereotaxic techniqueNeurology (clinical)Extracellular SpaceJournal of neurotrauma
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