Search results for "granulocyte"

showing 10 items of 244 documents

IL-17A and IL-17F do not contribute vitally to autoimmune neuro-inflammation in mice

2009

The clear association of Th17 cells with autoimmune pathogenicity implicates Th17 cytokines as critical mediators of chronic autoimmune diseases such as EAE. To study the impact of IL-17A on CNS inflammation, we generated transgenic mice in which high levels of expression of IL-17A could be initiated after Cre-mediated recombination. Although ubiquitous overexpression of IL-17A led to skin inflammation and granulocytosis, T cell–specific IL-17A overexpression did not have a perceptible impact on the development and health of the mice. In the context of EAE, neither the T cell–driven overexpression of IL-17A nor its complete loss had a major impact on the development of clinical disease. Sin…

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentT cellEncephalomyelitisPopulation610 Medicine & healthMice TransgenicInflammation2700 General Medicine10263 Institute of Experimental ImmunologyMyelin oligodendrocyte glycoproteinMicemedicineAnimalseducationCells CulturedGlycoproteinseducation.field_of_studybiologybusiness.industryInterleukin-17General MedicineTh1 Cellsmedicine.diseasePeptide FragmentsMice Inbred C57BLCytokinemedicine.anatomical_structureImmunologybiology.protein570 Life sciences; biologyExperimental pathologyFemaleMyelin-Oligodendrocyte GlycoproteinInterleukin 17medicine.symptombusinessGranulocytesResearch Article
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Characterization of a novel population of low-density granulocytes associated with disease severity in HIV-1 infection

2012

The mechanisms resulting in progressive immune dysfunction during the chronic phase of HIV infection are not fully understood. We have previously shown that arginase, an enzyme with potent immunosuppressive properties, is increased in HIV seropositive (HIV+) patients with low CD4(+) T cell counts. Here we show that the cells expressing arginase in peripheral blood mononuclear cells of HIV+ patients are low-density granulocytes (LDGs) and that whereas these cells have a similar morphology to normal-density granulocyte, they are phenotypically different. Importantly, our results reveal that increased frequencies of LDGs correlate with disease severity in HIV+ patients.

Enzyme Metabolismlcsh:MedicineHIV InfectionsBiochemistryACTIVATION0302 clinical medicineImmunophenotypingImmunodeficiency VirusesRENAL-CELL CARCINOMAHIV SeropositivityMedicineSUPPRESSOR-CELLSlcsh:ScienceImmune ResponseCD180303 health scienceseducation.field_of_studyMultidisciplinarymedicine.diagnostic_testT Cellsvirus diseasesMiddle Aged3. Good healthEnzymesSEROPOSITIVE PATIENTSArginasemedicine.anatomical_structurePhenotypeHIV epidemiologyDisease ProgressionMedicineInfectious diseasesScience & Technology - Other TopicsNEUTROPHILResearch ArticleAdultGeneral Science & TechnologyT cellImmune CellsPopulationImmunologyCD18Viral diseasesGranulocytePeripheral blood mononuclear cellMicrobiologyFlow cytometryImmunophenotyping03 medical and health sciencesADHERENCEVirologyMD MultidisciplinaryHumanseducationBiology030304 developmental biologyScience & TechnologyArginasebusiness.industryTetraspanin 30MULTIDISCIPLINARY SCIENCESlcsh:RARGINASE-IHIVVirologyENDOTHELIAL-CELLSAntigens CD63ImmunologyLeukocytes Mononuclearlcsh:Qbusiness030215 immunologyGranulocytes
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Binding of Escherichia coli hemolysin and activation of the target cells is not receptor-dependent.

2005

Abstract Production of a single cysteine substitution mutant, S177C, allowed Escherichia coli hemolysin (HlyA) to be radioactively labeled with tritiated N-ethylmaleimide without affecting biological activity. It thus became possible to study the binding characteristics of HlyA as well as of toxin mutants in which one or both acylation sites were deleted. All toxins bound to erythrocytes and granulocytes in a nonsaturable manner. Only wild-type toxin and the lytic monoacylated mutant stimulated production of superoxide anions in granulocytes. An oxidative burst coincided with elevation of intracellular Ca2+, which was likely because of passive influx of Ca2+ through the toxin pores. Competi…

ErythrocytesAcylationMutantBacterial ToxinsBiologymedicine.disease_causeBiochemistryHemolysin ProteinsSuperoxidesmedicineEscherichia coliHumansReceptorMolecular BiologyEscherichia coliRespiratory BurstSequence DeletionBinding SitesToxinHemolysinBiological activityCell BiologyMolecular biologyLymphocyte Function-Associated Antigen-1Respiratory burstBiochemistryAmino Acid SubstitutionMutationMutagenesis Site-DirectedbacteriaCalciumK562 CellsIntracellularGranulocytesThe Journal of biological chemistry
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TTAS a New Stilbene Derivative that Induces Apoptosis in Leishmania Infantum

2012

Leishmania parasites are able to undergo apoptosis (programmed cell death), similarly to mammalian cells. Recently it was demonstrated in vitro the anti-leishmanial effect of some natural and synthetic stilbenoids including resveratrol and piceatannol. In this study we evaluated the Leishmanicidal activity of a pool of stilbene derivatives which had previously shown high apoptotic efficacy against neoplastic cells. All the compounds tested were capable to decrease the parasite viability in a dose-dependent manner. Trans-stilbenes proved to be markedly more effective than cis-isomers. This was different from that observed in tumor cells in which cis-stilbenes were more potent cytotoxic agent…

G2 PhaseProgrammed cell deathLeishmaniasiSettore MED/17 - Malattie InfettiveImmunologyAntiprotozoal AgentsTUBULINApoptosisResveratrolChromatography AffinityLethal Dose 50chemistry.chemical_compoundGranulocyte-Macrophage Progenitor CellsAnnexin A5Leishmania infantumCytotoxicityCells CulturedMembrane Potential MitochondrialPiceatannolDose-Response Relationship DrugbiologyGeneral MedicineFlow CytometryHematopoietic Stem Cellsbiology.organism_classificationLeishmaniaPROGRAMMED CELL DEATHIn vitroInfectious DiseaseschemistryBiochemistrySTILBENESAntimony Sodium GluconateApoptosisStilbeneElectrophoresis Polyacrylamide GelParasitologyLeishmania infantumCell DivisionLEISHMANIASIS
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Proliferation of gastrointestinal carcinoma cells by T lymphocyte factors interleukin-3 and granulocyte-macrophage colony-stimulating factor

1991

Hematopoietic growth factors have been well characterized by cDNA cloning in recent years. In order to determine the influence of rhGM-CSF and rhIL-3 on epithelial cells of the gastrointestinal tract, their influence on in vitro cultured gastric and pancreas cancer cells was determined. A more than two-fold enhancement of proliferation was observed by IL-3 and GM-CSF in Mz-Sto-1 gastric and 818-4 pancreas carcinoma cells, applying a sensitive microculture system which allows precise quantification. The highest growth rates were obtained adding 1-10 ng/ml of the growth factors, but even picogram amounts were effective. Expression of mRNA for GM-CSF and IL-3 remained undetectable in the cell …

Gastrointestinal tractT-LymphocytesImmunologyGranulocyte-Macrophage Colony-Stimulating FactorBiologyLymphocyte ActivationMolecular biologyIn vitroPancreatic NeoplasmsHaematopoiesisGranulocyte macrophage colony-stimulating factorStomach NeoplasmsCell cultureCancer cellTumor Cells CulturedmedicineHumansInterleukin-3ReceptorCell DivisionInterleukin 3medicine.drugImmunologic Research
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Current insights into neutrophil homeostasis

2012

Neutrophil granulocytes represent the first immunologic barrier against invading pathogens, and neutropenia predisposes to infection. However, neutrophils may also cause significant collateral inflammatory damage. Therefore, neutrophil numbers are tightly regulated by an incompletely understood homeostatic feedback loop adjusting the marrow's supply to peripheral needs. Granulocyte colony-stimulating factor (G-CSF) is accepted to be the major determinant of neutrophil production, and G-CSF levels have, soon after its discovery, been described to be inversely correlated with neutrophil counts. A neutrophil sensor, or "neutrostat," has, therefore, been postulated. The prevailing feedback hypo…

General NeuroscienceCellular differentiationGranulocyteNeutropeniaBiologymedicine.diseaseGranulopoiesisGeneral Biochemistry Genetics and Molecular Biologymedicine.anatomical_structureHistory and Philosophy of ScienceImmunologymedicineMyelopoiesisReceptorNeutrophil homeostasisHomeostasisAnnals of the New York Academy of Sciences
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Antigens and Cytokine Genes in Antitumor Vaccines

2006

Studies against cancer, including clinical trials, have shown that a correct activation of the immune system can lead to tumor rejection whereas incorrect signaling results in no positive effects or even anergy. We have worked assuming that two signals, GM-CSF (granulocyte and macrophage colony-stimulating factor) and tumor antigens are necessary to mediate an antitumor effective response. To study which is the ideal temporal sequence for their administration, we have used a murine model of antimelanoma vaccine employing whole B16 tumor cells or their membrane protein antigens (TMPs) in combination with gm-csf transfer before or after the antigen delivery. Our results show that: (i) When gm…

General NeuroscienceMelanomaCancerTransfectionGranulocyteBiologymedicine.diseaseGeneral Biochemistry Genetics and Molecular BiologyTumor antigenmedicine.anatomical_structureImmune systemHistory and Philosophy of ScienceAntigenImmunologymedicineMacrophageAnnals of the New York Academy of Sciences
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Lentivirus-induced dendritic cells for immunization against high-risk WT1(+) acute myeloid leukemia.

2013

Wilms' tumor 1 antigen (WT1) is overexpressed in acute myeloid leukemia (AML), a high-risk neoplasm warranting development of novel immunotherapeutic approaches. Unfortunately, clinical immunotherapeutic use of WT1 peptides against AML has been inconclusive. With the rationale of stimulating multiantigenic responses against WT1, we genetically programmed long-lasting dendritic cells capable of producing and processing endogenous WT1 epitopes. A tricistronic lentiviral vector co-expressing a truncated form of WT1 (lacking the DNA-binding domain), granulocyte-macrophage colony-stimulating factor (GM-CSF), and interleukin-4 (IL-4) was used to transduce human monocytes ex vivo. Overnight transd…

Genes Wilms TumorCell SurvivalGenetic VectorsAntineoplastic AgentsBiologyCD8-Positive T-LymphocytesLymphocyte ActivationPeripheral blood mononuclear cellEpitopeMonocytesViral vectorMiceAntigenRisk FactorsGeneticsmedicineNeoplasmAnimalsHumansMolecular BiologyResearch ArticlesOligonucleotide Array Sequence AnalysisCD86LentivirusGene Transfer TechniquesMyeloid leukemiaGranulocyte-Macrophage Colony-Stimulating FactorCell DifferentiationDendritic CellsGenetic Therapymedicine.diseaseAdoptive TransferLeukemia Myeloid AcuteGene Expression RegulationCancer researchLeukocytes MononuclearMolecular MedicineInterleukin-4Ex vivoHuman gene therapy
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The designer cytokine hyper-IL-6 mediates growth inhibition and GM-CSF-dependent rejection of B16 melanoma cells.

2000

The low immunogenic B16 melanoma cell line was transfected with a mammalian expression vector containing the complementary DNA for a sIL-6R/IL-6 fusion protein, termed Hyper-IL-6 (H-IL-6), which was shown to have biological activities at 100-1000-fold lower concentrations than IL-6 in combination with sIL-6R. The secreted p84 glycoprotein was detected in the supernatant of transfected cells and was fully active on BAF3/gp130 cells, which respond to IL-6/sIL-6R but not to IL-6 alone. Administration of recombinant H-IL-6 to C57BL/6 mice resulted in a prolonged acute phase protein gene expression indicating long systemic persistence of the fusion protein. Transfected B16 cells (B16/H-IL6 cells…

Graft RejectionCancer ResearchTumor suppressor geneRatónmedicine.medical_treatmentRecombinant Fusion ProteinsMelanoma ExperimentalMice TransgenicTransfectionchemistry.chemical_compoundMiceGene expressionGeneticsmedicineAnimalsDrug InteractionsInterleukin 6neoplasmsMolecular BiologybiologyInterleukin-6MelanomaGranulocyte-Macrophage Colony-Stimulating FactorReceptors Interleukinmedicine.diseaseReceptors Interleukin-6Growth Inhibitorsrespiratory tract diseasesCytokinechemistryCell cultureReceptors Granulocyte-Macrophage Colony-Stimulating FactorImmunologybiology.proteinCancer researchGrowth inhibitionImmunosuppressive AgentsOncogene
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GM-CSF restores innate, but not adaptive, immune responses in glucocorticoid-immunosuppressed human blood in vitro.

2003

Abstract Infection remains the major complication of immunosuppressive therapy in organ transplantation. Therefore, reconstitution of the innate immunity against infections, without activation of the adaptive immune responses, to prevent graft rejection is a clinically desirable status in transplant recipients. We found that GM-CSF restored TNF mRNA and protein expression without inducing IL-2 production and T cell proliferation in glucocorticoid-immunosuppressed blood from either healthy donors or liver transplant patients. Gene array experiments indicated that GM-CSF selectively restored a variety of dexamethasone-suppressed, LPS-inducible genes relevant for innate immunity. A possible ex…

Graft RejectionLipopolysaccharidesT-LymphocytesCell Cycle ProteinsCell SeparationOrgan transplantationDexamethasoneMiceCDC2-CDC28 KinasesConcanavalin ATumor Cells CulturedImmunology and AllergySkin TransplantationMiddle AgedCyclin-Dependent KinasesUp-RegulationSurvival Ratemedicine.anatomical_structureImmunity ActiveTumor necrosis factor alphaGlucocorticoidCell DivisionCyclin-Dependent Kinase Inhibitor p27Immunosuppressive Agentsmedicine.drugAdultmedicine.medical_specialtyT cellImmunologyDown-RegulationBiologyProtein Serine-Threonine KinasesImmune systemAdjuvants ImmunologicIn vivomedicineAnimalsHumansDexamethasoneAgedSalmonella Infections AnimalInnate immune systemTumor Suppressor ProteinsCyclin-Dependent Kinase 2Granulocyte-Macrophage Colony-Stimulating FactorImmunity InnateGene Expression RegulationImmunologyLeukocytes MononuclearMice Inbred CBAInterleukin-2Interleukin-1Journal of immunology (Baltimore, Md. : 1950)
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