Search results for "harm"

showing 10 items of 13866 documents

Host-Derived CD8+ Dendritic Cells Protect Against Acute Graft-versus-Host Disease after Experimental Allogeneic Bone Marrow Transplantation

2014

Graft-versus-host disease (GVHD) is a frequent life-threatening complication after allogeneic hematopoietic stem cell transplantation (HSCT) and induced by donor-derived T cells that become activated by host antigen-presenting cells. To address the relevance of host dendritic cell (DC) populations in this disease, we used mouse strains deficient in CD11c(+) or CD8α(+) DC populations in a model of acute GVHD where bone marrow and T cells from BALB/c donors were transplanted into C57BL/6 hosts. Surprisingly, a strong increase in GVHD-related mortality was observed in the absence of CD11c(+) cells. Likewise, Batf3-deficient (Batf3(-/-)) mice that lack CD8α(+) DCs also displayed a strongly incr…

medicine.medical_treatmentGraft vs Host DiseasePriming (immunology)CD11cHematopoietic stem cell transplantationchemical and pharmacologic phenomenaHematopoietic stem cell transplantationCD8-Positive T-LymphocytesBiologyGraft-versus-host diseaseDendritic cellsMiceimmune system diseasesBATF3medicineAnimalsTransplantation HomologousBone Marrow TransplantationMice Inbred BALB CTransplantationPeripheral toleranceHematologyDendritic cellmedicine.diseaseMice Inbred C57BLsurgical procedures operativeGraft-versus-host diseasemedicine.anatomical_structureImmunologyBone marrowCD8Biology of Blood and Marrow Transplantation
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Neuroinflammatory and behavioral susceptibility profile of mice exposed to social stress towards cocaine effects.

2021

Using the social defeat (SD) model, numerous studies have shown that stressed mice display an enhanced response to the motivational effects of cocaine in the self-administration (SA) and conditioned-place preference (CPP) paradigms. However, not all subjects exposed to stress express its harmful effects. Some are particularly susceptible to the deleterious effects of repeated SD, while resilient mice successfully cope with stressful experiences and display adjusted psychological functioning after stress. Vulnerability to develop stress-related disorders, such as depression, has been linked to coping strategies and more recently to individual differences in the immune system. However, no stu…

medicine.medical_treatmentHippocampusStriatumHippocampusSocial defeat03 medical and health sciences0302 clinical medicineImmune systemNeuroinflammationCocaineDopamine Uptake InhibitorsSocial defeatConditioning PsychologicalMedicineAnimalsCX3CL1CytokineBiological PsychiatryNeuroinflammationPharmacologySocial stressResilienceBehavior Animalbusiness.industryChemokine CX3CL1Corpus Striatum030227 psychiatryPsicobiologiaCytokineChemokineSusceptibilityImmunologyCytokinesbusinessStress PsychologicalProgress in neuro-psychopharmacologybiological psychiatry
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POLYPHENOLS FROM RED WINE MODULATE IMMUNE RESPONSIVENESS: BIOLOGICAL AND CLINICAL SIGNIFICANCE.

2008

Many studies have been conducted on the effects of red wine polyphenols on certain diseases, primarily, coronary heart disease (CHD) and, in this respect, evidence has been demonstrated that intake of red wine is associated with a reduction of CHD symptomatology. In this framework, the purpose of this review is to illustrate the effects of polyphenols on immune cells from human healthy peripheral blood. Data will show that polyphenols are able to stimulate both innate and adaptive immune responses. In particular, the release of cytokines such as interleukin (IL)-12, interferon (IFN)-gamma, and IL-10 as well as immunoglobulins may be important for host protection in different immune related …

medicine.medical_treatmentImmunoglobulinsCoronary DiseaseWineImmunoglobulin ENitric OxidePeripheral blood mononuclear cellp38 Mitogen-Activated Protein KinasesNitric oxidePOLYPHENOLSIMMUNE SYSTEMCYTOKINESIMMUNOGLOBULINSNITRIC OXIDEATHEROSCLEROSISRED WINEchemistry.chemical_compoundImmune systemPhenolsInterferonDrug DiscoverymedicineAnimalsHumansPharmacologyFlavonoidsSettore MED/04 - Patologia Generalebiologybusiness.industryImmunityfood and beveragesInterleukinPolyphenolsCytokinechemistryImmunologyChronic Diseasebiology.proteinLeukocytes MononuclearCytokinesAntibodybusinessmedicine.drug
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Peripherally Circulating CD4+ FOXP3+ CXCR3+ T Regulatory Cells Correlate with Renal Allograft Function

2012

Peripheral immunoregulation depends on T regulatory cell trafficking into the allograft to modulate the local alloresponse. Little is known about the relevance of trafficking receptors for Tregs after solid organ transplantation in humans. In this study, expression of the peripheral chemokine receptors CXCR3 and CCR5 on CD4+ FOXP3+ Treg cells was analysed and correlated with allograft function in renal transplant recipients. Flow cytometry analysis of peripheral blood mononuclear cells of 54 renal transplant recipients receiving a calcineurin inhibitor-based immunosuppression was performed for CD4, CD25, FOXP3, CXCR3 and CCR5 within the first 18 months post-transplantation. Correlation anal…

medicine.medical_treatmentImmunologyFOXP3hemic and immune systemschemical and pharmacologic phenomenaImmunosuppressionGeneral MedicineBiologyCXCR3Peripheral blood mononuclear cellCalcineurinChemokine receptorImmunologymedicineIL-2 receptorReceptorScandinavian Journal of Immunology
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IL-27 controls the development of inducible regulatory T cells and Th17 cells via differential effects on STAT1

2007

IL-27 is an IL-12-related cytokine frequently present at sites of inflammation that can promote both anti- and pro-inflammatory immune responses. Here, we have analyzed the mechanisms how IL-27 may drive such divergent immune responses. While IL-27 suppressed the development of proinflammatory Th17 cells, a novel role for this cytokine in inhibiting the development of anti-inflammatory, inducible regulatory T cells (iTreg) was identified. In fact, IL-27 suppressed the development of adaptive, TGF-beta-induced Forkhead box transcription factor p3-positive (Foxp3(+)) Treg. Whereas the blockade of Th17 development was dependent on the transcription factor STAT1, the suppression of iTreg develo…

medicine.medical_treatmentImmunologyMice Transgenicchemical and pharmacologic phenomenaInflammationBiologyT-Lymphocytes RegulatoryProinflammatory cytokineMiceImmune systemT-Lymphocyte SubsetsmedicineAnimalsImmunology and AllergySTAT1IL-2 receptorTranscription factorInterleukinsFOXP3Forkhead Transcription FactorsFlow CytometryCoculture TechniquesCell biologySTAT1 Transcription FactorCytokineImmunologybiology.proteinmedicine.symptomEuropean Journal of Immunology
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Circumventing tolerance to a human MDM2-derived tumor antigen by TCR gene transfer

2001

We identified a tumor-associated cytotoxic T lymphocyte (CTL) epitope derived from the widely expressed human MDM2 oncoprotein and were able to bypass self-tolerance to this tumor antigen in HLA-A*0201 (A2.1) transgenic mice and by generating A2.1-negative, allo-A2.1-restricted human T lymphocytes. A broad range of malignant, as opposed to nontransformed cells, were killed by high-avidity transgenic mouse and allogeneic human CTLs specific for the A2.1-presented MDM2 epitope. Whereas the self-A2.1-restricted human T cell repertoire gave rise only to low-avidity CTLs unable to recognize the natural MDM2 peptide, human A2.1+ T lymphocytes were turned into efficient MDM2-specific CTLs upon exp…

medicine.medical_treatmentImmunologyT-cell receptorchemical and pharmacologic phenomenaImmunotherapyBiologyMajor histocompatibility complexMolecular biologyTumor antigenEpitopeCTL*Antigenmedicinebiology.proteinImmunology and AllergyCytotoxic T cell
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An unconventional TRAIL to cancer therapy

2013

Cellular immunotherapy offers novel, safe, and effective routes to treating cancer. However, approaches utilizing cytotoxic CD8+ T cells are hampered by the need to identify suitable target antigens that are expressed by tumor cells but not healthy tissues, and that are recognized with sufficient affinity. Most importantly, the applicability of CD8+ T-cell-based therapies is governed by the MHC restriction of tumor-specific epitopes, thereby limiting the potential benefit to patients carrying the appropriate MHC haplotype. Alternative approaches to harness the immune system against tumors exploit non-MHC-restricted γδ T cells that recognize stress-induced changes in transformed cells. A new…

medicine.medical_treatmentImmunologychemical and pharmacologic phenomenaImmunotherapyMHC restrictionBiologyNKG2DMajor histocompatibility complexEpitopeImmune systemAntigenImmunologymedicinebiology.proteinImmunology and AllergyCytotoxic T cellEuropean Journal of Immunology
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Major histocompatibility complex regulation of cytokine production.

1996

This review describes the phenomenon of the major histocompatibility complex (MHC) control of cytokine production both in experimental animals and in humans. H-2 (mouse MHC) regulates which type of cytokine is selectively produced in response to the hapten trinitrophenyl (TNP). T cells from TNP-immune H-2k mice produce interferon-gamma (IFN-gamma), interleukin-2 (IL-2), IL-3, IL-5, tumor necrosis factor-alpha (TNF-alpha), IL-10, and very low levels of IL-4 on reexposure to the specific antigen in vitro. By contrast, T cells from H-2d mice produce IL-3, TNF-alpha, IL-10, and IL-4 but very low levels of IL-2, IL-5 and IFN-gamma. As MHC-congenic matched strains (BALB/k and BALB/c) are used, th…

medicine.medical_treatmentImmunologychemical and pharmacologic phenomenaMajor histocompatibility complexPeripheral blood mononuclear cellMajor Histocompatibility ComplexInterferon-gammaMiceImmune systemAntigenVirologyImmunopathologymedicineAnimalsHumansbiologyTumor Necrosis Factor-alphaInterleukinsH-2 AntigensCell BiologyCytokineImmunologyAntibody Formationbiology.proteinCytokinesTumor necrosis factor alphaHapten
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Nitric oxide and sensory afferent neurones modulate the protective effects of low-dose endotoxin on rat gastric mucosal damage

1995

Pretreatment (1 h) with low doses (5-40 micrograms/kg i.p.) of Escherichia coli endotoxin dose dependently reduced the gastric mucosal damage induced by a 10 min challenge with 1 ml ethanol (50% and 100%) in conscious rats. Treatment with the nitric oxide synthesis inhibitor, NG-nitro-L-arginine methyl ester (L-NAME, 5 and 10 mg/kg i.p.), significantly inhibited the protective effects of endotoxin (40 micrograms/kg i.p.). The actions of L-NAME were reversed by the prior administration of L-arginine (100 mg/kg i.p.). The protective effects of endotoxin were not influenced by pretreatment with dexamethasone (5 mg/kg s.c. twice) or indomethacin (5 mg/kg s.c.). However, ablation of sensory affe…

medicine.medical_treatmentIndomethacinPharmacologyArginineDexamethasoneNitric oxideRats Sprague-Dawleychemistry.chemical_compoundEscherichia colimedicineAnimalsNeurons AfferentEnzyme InhibitorsAntidoteDexamethasonePharmacologyAnalysis of VarianceEthanolEthanolSensory neuronRatsEndotoxinsNG-Nitroarginine Methyl Estermedicine.anatomical_structureMechanism of actionchemistryGastric MucosaCapsaicinAnesthesiaToxicityFemaleCapsaicinNitric Oxide Synthasemedicine.symptomInjections Intraperitonealmedicine.drugEuropean Journal of Pharmacology
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INFLAMMATORY CYTOKINES IN ACUTE ISCHEMIC STROKE.

2008

Three major cytokines, namely, tumor necrosis factor (TNF-alpha), interleukin (IL)-1, and IL-6 are produced by cultured brain cells after various stimuli such as ischemia. Neurones, astrocytes, microglia and oligodendrocytes can produce inflammatory mediators, and cytokine receptors are expressed constitutionally throughout the Central Nervous System (CNS), albeit at low levels. Cytokines are involved in virtually every facet of stroke and they have numerous pro-inflammatory and pro-coagulant effects on endothelium. TNF-alpha expression after stroke stimulates expression of tissue factor and adhesion molecules for leukocytes, release of interleukin-1 (IL-1), nitric oxide, factor VIII/von Wi…

medicine.medical_treatmentInflammationProinflammatory cytokineBrain IschemiaBrain ischemiaTissue factorDrug DiscoverymedicineHumansStrokePharmacologyInflammationMicrogliabusiness.industrymedicine.diseasePrognosisStrokeCytokinemedicine.anatomical_structureImmunologyAcute DiseaseCytokinesTumor necrosis factor alphamedicine.symptomStroke cytokinesbusinessBiomarkers
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