Search results for "helium"

showing 10 items of 1689 documents

Alkaline phosphatase dual-binding sites for collagen dictate cell migration and microvessel assembly in vitro

2020

Interactions between cell types, growth factors, and extracellular matrix components involved in angiogenesis are crucial for new vessel formation leading to tissue regeneration. This study investigated whether cocultures of fibroblasts and endothelial cells (ECs; from macro- or microvasculature) play a role in the formation of microvessel-like structures by ECs, as well as modulate fibroblast differentiation and growth factors production (vascular endothelial cell growth factor, basic fibroblast growth factor, active transforming growth factor-beta 1, and interleukin-8), which are important for vessel sprouting and maturation. Data obtained revealed that in vitro coculture systems of fibro…

0301 basic medicineCell typeAngiogenesisProtein ConformationBasic fibroblast growth factorNeovascularization PhysiologicIn Vitro TechniquesBiochemistryExtracellular matrix03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCell MovementmedicineHumansFibroblastMolecular BiologyMicrovesselCells CulturedCell ProliferationBinding SitesChemistryHealth sciences Medical and Health sciencesCiências médicas e da saúdeCell migrationCell DifferentiationCell BiologyFibroblastsAlkaline PhosphataseCell biology030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisMicrovesselsMedical and Health sciencesAlkaline phosphataseCollagenEndothelium VascularCiências da Saúde Ciências médicas e da saúde
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Epithelium‐specific MyD88 signaling, but not DCs or macrophages, control acute intestinal infection with Clostridium difficile

2019

Infection with Clostridium difficile is one of the major causes of health care acquired diarrhea and colitis. Signaling though MyD88 downstream of TLRs is critical for initiating the early protective host response in mouse models of C. difficile infection (CDI). In the intestine, MyD88 is expressed in various tissues and cell types, such as the intestinal epithelium and mononuclear phagocytes (MNP), including DC or macrophages. Using a genetic gain-of-function system, we demonstrate here that restricting functional MyD88 signaling to the intestinal epithelium, but also to MNPs is sufficient to protect mice during acute CDI by upregulation of the intestinal barrier function and recruitment o…

0301 basic medicineCell typeImmunologyBiologyMice03 medical and health sciences0302 clinical medicineDownregulation and upregulationmedicineAnimalsImmunology and AllergyIntestinal MucosaColitisEnterocolitis PseudomembranousBarrier functionClostridioides difficileMacrophagesDendritic CellsClostridium difficilemedicine.diseaseIntestinal epitheliumPhenotypeEpitheliumDisease Models Animal030104 developmental biologymedicine.anatomical_structureHost-Pathogen InteractionsMyeloid Differentiation Factor 88ImmunologySignal Transduction030215 immunologyEuropean Journal of Immunology
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Single cell RNAseq provides a molecular and cellular cartography of changes to the human endometrium through the menstrual cycle

2018

In a human menstrual cycle, the endometrium undergoes remodeling, shedding, and regeneration which are driven by substantial gene expression changes in the underlying cellular hierarchy. Despite its importance in human fertility and regenerative biology, mechanistic understanding of this unique type of tissue homeostasis remains rudimentary. Here, we characterized the transcriptomic transformation of human endometrium at single cell resolution, dissecting multidimensional cellular heterogeneity of the tissue across the entire natural menstrual cycle. We analyzed 6 endometrial cell types, including a previously uncharacterized ciliated epithelial cell type, during four major phases of endome…

0301 basic medicineCell typeRegeneration (biology)CellObstetrics and GynecologyBiologyEndometriumEpitheliumCell biologyTranscriptome03 medical and health sciences030104 developmental biologymedicine.anatomical_structureReproductive MedicineStromamedicineTissue homeostasisFertility and Sterility
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Candida albicansis able to use M cells as a portal of entry across the intestinal barrierin vitro

2015

Candida albicans is the most frequent yeast responsible for systemic infections in humans. These infections mainly originate from the gastrointestinal tract where C. albicans can invade the gut epithelial barrier to gain access to the bloodstream. Along the gut, pathogens can use Microfold (M) cells as a portal of entry to cross the epithelial barrier. M cells are specialized cells mainly located in the follicule-associated epithelium of Peyer patches. In this study, we used scanning electron and fluorescence microscopy, adhesion and invasion assays and fungal mutants to investigate the interactions of C. albicans with M cells obtained in an established in vitro model whereby enterocyte-lik…

0301 basic medicineCellular differentiationImmunologyBiologyEndocytosisbiology.organism_classificationMicrobiologyIn vitroEpitheliumCorpus albicansMicrobiology03 medical and health sciences030104 developmental biologymedicine.anatomical_structureTranscytosisVirologyImmunologymedicineCandida albicansMicrofold cellCellular Microbiology
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IL-17 controls central nervous system autoimmunity through the intestinal microbiome

2021

Interleukin-17A- (IL-17A) and IL-17F-producing CD4(+) T helper cells (T(H)17 cells) are implicated in the development of chronic inflammatory diseases, such as multiple sclerosis and its animal model, experimental autoimmune encephalomyelitis (EAE). T-H 17 cells also orchestrate leukocyte invasion of the central nervous system (CNS) and subsequent tissue damage. However, the role of IL-17A and IL-17F as effector cytokines is still confused with the encephalitogenic function of the cells that produce these cytokines, namely, T-H 17 cells, fueling a long-standing debate in the neuroimmunology field. Here, we demonstrated that mice deficient for IL-17A/F lose their susceptibility to EAE, which…

0301 basic medicineCentral Nervous SystemMaleEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisreceptorImmunologyCentral nervous system610 Medicine & healthGut flora10263 Institute of Experimental Immunologymedicine.disease_causeAutoimmunityinterleukin-1703 medical and health sciencesMice0302 clinical medicinemedicinecytokineAnimalsHumanscnst-cellsMice Knockout2403 Immunologybiologygut microbiotaMultiple sclerosisExperimental autoimmune encephalomyelitisGeneral MedicineFecal Microbiota Transplantationneutralizationmedicine.diseasebiology.organism_classificationAdoptive Transfer3. Good healthGut EpitheliumGastrointestinal Microbiome030104 developmental biologyNeuroimmunologymedicine.anatomical_structureImmunology2723 Immunology and Allergy570 Life sciences; biologyTh17 CellssequencesFemaleInterleukin 17030217 neurology & neurosurgery
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Angiotensin II and leukocyte trafficking: New insights for an old vascular mediator. Role of redox-signaling pathways.

2019

Abstract Inflammation and activation of the immune system are key molecular and cellular events in the pathogenesis of cardiovascular diseases, including atherosclerosis, hypertension-induced target-organ damage, and abdominal aortic aneurysm. Angiotensin II (Ang-II) is the main effector peptide hormone of the renin-angiotensin system. Beyond its role as a potent vasoconstrictor and regulator of blood pressure and fluid homeostasis, Ang-II is intimately involved in the development of vascular lesions in cardiovascular diseases through the activation of different immune cells. The migration of leukocytes from circulation to the arterial subendothelial space is a crucial immune response in le…

0301 basic medicineChemokineEndotheliumInflammationBiochemistry03 medical and health sciences0302 clinical medicineMediatorImmune systemPhysiology (medical)Leukocyte TraffickingLeukocytesMedicinebiologybusiness.industryCell adhesion moleculeAngiotensin IIEndothelial CellsAngiotensin IICell biology030104 developmental biologymedicine.anatomical_structurebiology.proteinmedicine.symptombusinessOxidation-Reduction030217 neurology & neurosurgerySignal TransductionFree radical biologymedicine
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Effects of gaseous and solid constituents of air pollution on endothelial function

2018

Abstract Ambient air pollution is a leading cause of non-communicable disease globally. The largest proportion of deaths and morbidity due to air pollution is now known to be due to cardiovascular disorders. Several particulate and gaseous air pollutants can trigger acute events (e.g. myocardial infarction, stroke, heart failure). While the mechanisms by which air pollutants cause cardiovascular events is undergoing continual refinement, the preponderant evidence support rapid effects of a diversity of pollutants including all particulate pollutants (e.g. course, fine, ultrafine particles) and gaseous pollutants such as ozone, on vascular function. Indeed alterations in endothelial function…

0301 basic medicineClinical ReviewFine particulateAir pollutionAir pollution030204 cardiovascular system & hematologymedicine.disease_cause03 medical and health sciencesGreenhouse Gases0302 clinical medicineOzoneRisk FactorsEnvironmental healthMedicineAnimalsHumansEndothelial dysfunctionNitrogen dioxidePollutantInflammationAir PollutantsHuman studiesAmbient air pollutionbusiness.industryEnvironmental ExposureParticulates3. Good healthEditor's Choice030104 developmental biology13. Climate actionCardiovascular DiseasesOxidative stressEndothelium VascularCurrent (fluid)Cardiology and Cardiovascular MedicinebusinessVascular functionParticulate matterTranslational MedicineEuropean Heart Journal
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Enterocyte Purge and Rapid Recovery Is a Resilience Reaction of the Gut Epithelium to Pore-Forming Toxin Attack.

2016

International audience; Besides digesting nutrients, the gut protects the host against invasion by pathogens. Enterocytes may be subjected to damage by both microbial and host defensive responses, causing their death. Here, we report a rapid epithelial response that alleviates infection stress and protects the enterocytes from the action of microbial virulence factors. Intestinal epithelia exposed to hemolysin, a pore-forming toxin secreted by Serratia marcescens, undergo an evolutionarily conserved process of thinning followed by the recovery of their initial thickness within a few hours. In response to hemolysin attack, Drosophila melanogaster enterocytes extrude most of their apical cyto…

0301 basic medicineCytoplasmDisease toleranceSurvivalApoptosismedicine.disease_causeOral infectionHemolysin ProteinsLipid droplet[SDV.IDA]Life Sciences [q-bio]/Food engineeringMitochondrial extrusionIntestinal MucosaSerratia marcescensBacterial-infectionPore-forming toxinbiologyCell DeathMicrovilliPlasma-membrane[ SDV.IDA ] Life Sciences [q-bio]/Food engineeringGut EpitheliumMitochondriamedicine.anatomical_structureDrosophila melanogasterEnterocyteVirulence FactorsVarroidaeSerratia-marcescensBacterial ToxinsVirulenceMicrobiologyMicrobiologySerratia Infections03 medical and health sciencesVirologymedicineAnimalsApical cytoplasmDefense strategyDrosophila cyclin jToxinbiology.organism_classificationLipid dropletsDisease Models AnimalIntestinal Diseases030104 developmental biologyEnterocytesSerratia marcescensParasitologyDigestive SystemCell hostmicrobe
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Angiogenic response in an in vitro model of dog microvascular endothelial cells stimulated with antigenic extracts from Dirofilaria immitis adult wor…

2019

Abstract Background Angiogenesis can occur under pathological conditions when stimuli such as inflammation, vascular obstruction or hypoxia exist. These stimuli are present in cardiopulmonary dirofilariosis (Dirofilaria immitis). The aim of this study was to analyze the capacity of D. immitis antigens to modify the expression of angiogenic factors and trigger the formation of pseudocapillaries (tube-like structures) in an in vitro model of endothelial cells. Methods The expression of VEGF-A, sFlt, mEndoglin and sEndoglin in cultures of canine microvascular endothelial cells stimulated with extract of adult worms of D. immitis obtained from an untreated dog (DiSA) and from a dog treated for …

0301 basic medicineDirofilaria immitis antigenic extractsEndotheliumAngiogenesisCell SurvivalDirofilaria immitis030231 tropical medicineCellNeovascularization PhysiologicCanine microvascular endothelial cellsDirofilaria immitisBiologylcsh:Infectious and parasitic diseasesAndrologyWolbachia amount03 medical and health sciences0302 clinical medicineDogsAntigenmedicineAnimalslcsh:RC109-216Cells CulturedInflammationMatrigelAntigens BacterialAngiogenic factorsResearchEndothelial CellsParasitologia veterinàriabiology.organism_classificationIn vitroCapillariesAngiogènesi030104 developmental biologyInfectious Diseasesmedicine.anatomical_structureCell cultureAntigens HelminthParasitologyPseudocapillaries formationWolbachia
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Usefulness of Caco-2/HT29-MTX and Caco-2/HT29-MTX/Raji B Coculture Models To Predict Intestinal and Colonic Permeability Compared to Caco-2 Monocultu…

2017

The Caco-2 cellular monolayer is a widely accepted in vitro model to predict human permeability but suffering from several and critical limitations. Therefore, some alternative cell cultures to mimic the human intestinal epithelium, as closely as possible, have been developed to achieve more physiological conditions, as the Caco-2/HT29-MTX coculture and the triple Caco-2/HT29-MTX/Raji B models. In this work the permeability of 12 model drugs of different Biopharmaceutical Classification System (BCS) characteristics, in the coculture and triple coculture models was assessed. Additionally, the utility of both models to classify compounds according to the BCS criteria was scrutinized. The obta…

0301 basic medicineDrugColonmedia_common.quotation_subjectPharmaceutical Science02 engineering and technologyBiologydigestive systemPermeability03 medical and health sciencesCell Line TumorDrug DiscoverymedicineLow permeabilityHumansIntestinal Mucosamedia_commonHt29 mtxIntestinal permeability021001 nanoscience & nanotechnologymedicine.diseaseIntestinal epitheliumCoculture Techniques030104 developmental biologyIntestinal AbsorptionCaco-2Cell culturePermeability (electromagnetism)ImmunologyCancer researchMolecular MedicineCaco-2 Cells0210 nano-technologyHT29 CellsMolecular Pharmaceutics
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