Search results for "hepadnaviridae"

showing 10 items of 57 documents

Duck Hepatitis B Virus Requires Cholesterol for Endosomal Escape during Virus Entry

2008

ABSTRACT The identity and functionality of biological membranes are determined by cooperative interaction between their lipid and protein constituents. Cholesterol is an important structural lipid that modulates fluidity of biological membranes favoring the formation of detergent-resistant microdomains. In the present study, we evaluated the functional role of cholesterol and lipid rafts for entry of hepatitis B viruses into hepatocytes. We show that the duck hepatitis B virus (DHBV) attaches predominantly to detergent-soluble domains on the plasma membrane. Cholesterol depletion from host membranes and thus disruption of rafts does not affect DHBV infection. In contrast, depletion of chole…

AvihepadnavirusbiologyvirusesImmunologyDuck hepatitis B virusBiological membraneEndosomesVirus Internalizationbiology.organism_classificationMicrobiologyVirologyVirusHepatitis B Virus DuckVirus-Cell InteractionsCholesterolViral envelopeHepadnaviridaeViral entryCell Line TumorVirologyInsect ScienceHepatocytesHumanslipids (amino acids peptides and proteins)Lipid raftJournal of Virology
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Proliferative response of CD4+ T cells and hepatitis B virus clearance in chronic hepatitis with or without hepatitis B e-minus hepatitis B virus mut…

1995

To assess the significance of cell-mediated immunity, T cells were derived from the peripheral blood and liver tissue of hepatitis B virus (HBV)-infected patients and controls. The analysis of the 3H-thymidine-uptake in response to a panel of recombinant HBV antigens revealed that peripheral blood mononuclear cells (PBMC) of the 25 viremic patients with inflammatory active, chronic hepatitis B, 16 with wild-type and nine with HBe-minus HBV mutant infection, showed stronger proliferative responses to HBc and HBe antigens than 16 asymptomatic nonviremic HBsAg carriers with normal aminotransferase levels (HBc: SI 19.3 +/- 3.9 vs. 13.0 +/- 3.2 vs. 8.0 +/- 1.2; P.01 and HBe: SI 16.6 +/- 4.0 vs. …

CD4-Positive T-LymphocytesHBsAgHepatitis B virusmedicine.disease_causeVirusAntigenmedicineHumansHepatitis B e AntigensHepatitis B virusHepatologybiologybusiness.industryvirus diseasesInterferon-alphaHepatitis Bmedicine.diseasebiology.organism_classificationHepatitis BVirologyHepatitis B Core Antigensdigestive system diseasesHBcAgHBeAgHepadnaviridaeImmunologyChronic DiseaseMutationbusinessCell DivisionHepatology (Baltimore, Md.)
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Low frequency of cytotoxic liver-infiltrating T lymphocytes specific for endogenous processed surface and core proteins in chronic hepatitis B.

1993

To investigate the role of hepatitis B virus (HBV)-specific CD8+ T cells in chronic hepatitis B, the lytic activity of peripheral blood mononuclear cells (PBMC) and liver-infiltrating T cell clones and cytotoxic T cell (CTL) lines stimulated by recombinant vaccinia virus-infected cells were analyzed. Autologous and allogeneic Epstein-Barr virus-transformed B cells infected with vaccinia vectors (VAC) that contain sequences of the surface (S), secretory core (E), cytoplasmatic core (C) VAC antigen of HBV, or the wild-type (WT) VAC served as target cells. ELISA and immunoblotting showed HBV antigen expression in infected cells. Neither PBMC nor C- or E-VAC-stimulated CTL lines showed specific…

Cytotoxicity ImmunologicHerpesvirus 4 HumanT cellGenes MHC Class IVaccinia virusBiologymedicine.disease_causeHepatitis B AntigensAntigenCell MovementmedicineImmunology and AllergyCytotoxic T cellHumansHepatitis B e AntigensHepatitis ChronicHepatitis B virusHepatitisB-LymphocytesHepatitis B Surface AntigensHepatitis Bmedicine.diseasebiology.organism_classificationCell Transformation ViralHepatitis BVirologyHepatitis B Core AntigensRecombinant ProteinsCTL*Infectious Diseasesmedicine.anatomical_structureHepadnaviridaeLiverProtein Processing Post-TranslationalT-Lymphocytes CytotoxicThe Journal of infectious diseases
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Development and characterization of a 293 cell line with regulatable expression of the hepatitis B virus large envelope protein

2004

During the life cycle of hepatitis B virus (HBV) the large L envelope protein plays a pivotal role that is related to its peculiar dual transmembrane topology. To study the complex structure and diverse functions of L under regulated conditions of production, a human 293 cell line stably expressing L under the control of the ecdysone-inducible promoter was generated. Cells demonstrated stringent dose- and time-dependent kinetics of induction with undetectable background expression in the absence of the inducer. Temporal control of L expression allowed to trace (i) its posttranslational reorientation resulting in the mixed topology; (ii) its spatial redistribution from the endoplasmic reticu…

Gene Expression Regulation ViralHepatitis B virusEcdysoneProtein ConformationEndoplasmic reticulumLiver cellCell MembraneCellGolgi ApparatusBiologyEndoplasmic Reticulummedicine.disease_causebiology.organism_classificationMolecular biologymedicine.anatomical_structureViral Envelope ProteinsHepadnaviridaeCell cultureVirologyMembrane topologymedicineHumansSecretionPromoter Regions GeneticCell Line TransformedJournal of Virological Methods
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Detection of Hepatitis B Virus DNA in the Liver of Children with Chronic Hepatitis B by In Situ Hybridization and Its Relation to Other Viral Markers

1992

The aim of the study was to detect hepatitis B virus (HBV) DNA by in situ hybridization (ISH) with a 35S-labeled radioactive probe in frozen liver biopsy tissue sections of 63 hepatitis B virus surface antigen (HBsAg)-positive children. The results were compared to other markers of viral replication. HBV DNA was detected in 48 children. Of the 15 negative cases, four had hepatitis B envelope antigen (HBeAg), 10 anti-HBe, and one neither HBeAg nor anti-HBe. Free HBV DNA in serum and liver was positive in one patient. Forty of the positive children were HBeAg- and six anti-HBe-positive; two were negative for both. Of 45 36 had HBV DNA in serum. In 38 of 47 HBV DNA and in 31 of 42 HBcAg could …

Genetic MarkersMaleHepatitis B virusHBsAgAdolescentHepatitis B virus DNA polymerasemedicine.disease_causemedicineHumansChildHepatitis B virusbiologymedicine.diagnostic_testGastroenterologyInfantNucleic Acid Hybridizationvirus diseasesHepatitis BHepatitis Bbiology.organism_classificationmedicine.diseaseHepatitis B Core AntigensVirologydigestive system diseasesBlotting SouthernHBcAgLiverHepadnaviridaeHBeAgChild PreschoolLiver biopsyChronic DiseaseDNA ViralPediatrics Perinatology and Child HealthFemaleJournal of Pediatric Gastroenterology and Nutrition
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Translation of hepatitis B virus (HBV) surface proteins from the HBV pregenome and precore RNAs in Semliki Forest virus-driven expression.

2004

Hepatitis B virus (HBV) pregenome RNA (pgRNA) serves as a translation template for the HBV core (HBc) protein and viral polymerase (Pol). HBV precore RNA (pcRNA) directs the synthesis of the precore (preC) protein, a precursor of the hepatitis B e antigen (HBeAg). pgRNA and pcRNA were expressed in the Semliki Forest virus (SFV) expression system. Besides the HBc and preC proteins, there was revealed the synthesis of all three forms of HBV surface (HBs) proteins: long (LHBs), middle (MHBs) and short (SHBs), the start codons of which are located more than 1000 nt downstream of the HBc and preC start codons. Moreover, other HBV templates, such as 3′-truncated pgRNA lacking 3′ direct repeat and…

HBV RNA encapsidation signal epsilonHepatitis B virusvirusesGene ExpressionLeaky scanningDNA-Directed DNA Polymerasemedicine.disease_causeSemliki Forest virusVirus ReplicationCell LineViral Envelope ProteinsVirologymedicineAnimalsHepatitis B e AntigensRNA MessengerCloning MolecularProtein PrecursorsHepatitis B virusHepatitis B Surface Antigensbiologyvirus diseasesRNA virusTemplates Geneticbiology.organism_classificationVirologyMolecular biologyHepatitis B Core AntigensImmunohistochemistrySemliki forest virusdigestive system diseasesGenetic translationHBeAgHepadnaviridaeProtein BiosynthesisRNA ViralThe Journal of general virology
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Relationship of pre-S encoded antigens in liver and clinical manifestations of chronic hepatitis B infection.

2008

Pre-S1 and pre-S2 encoded antigens of hepatitis B virus were localized in liver tissue using monoclonal antibodies. They were found to be exclusively expressed in the cytoplasm of liver cells. Cell bound pre-S1 encoded protein was often detected in patients with chronic liver disease and viremia. Only a small number of the HBsAg positive cells also contained pre-S1 antigen. There was no correlation with nuclear HBcAg. Livers of non-viremic HBsAg carriers contained many HBsAg expressing liver cells, that were frequently also positive for pre-S2 encoded protein but contained no detectable pre-S1 encoded protein at all. It remains open whether cell bound pre-S2 containing proteins of middle si…

HBsAgHepatitis B virusBiopsyRadioimmunoassayViremiaBiologyChronic liver diseaseImmunoenzyme Techniques03 medical and health sciencesLiver disease0302 clinical medicineAntigenmedicineHumans030304 developmental biologyHepatitis0303 health sciencesHepatitis B Surface AntigensHepatologyvirus diseasesmedicine.diseasebiology.organism_classificationHepatitis BVirology3. Good healthHBcAgHepadnaviridaeLiverImmunologyCarrier State030211 gastroenterology & hepatologyLiver
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Hepatic expression patterns of the large and middle hepatitis B virus surface proteins in viremic and nonviremic chronic hepatitis B.

1990

The envelope of hepatitis B virus consists of large, middle, and small hepatitis B surface proteins. Recent data from in vitro studies suggest that intracellular expression and distribution of the three polypeptides may be variable. These observations in artificial expression systems prompted this analysis of the occurrence and distribution of the three hepatitis B surface proteins in the liver tissue of substantial viremic (hepatitis B virus DNA- and hepatitis B e antigen-positive) and low-viremic or nonviremic (hepatitis B virus DNA-negative, anti-hepatitis B e antigen-positive) carriers by specific monoclonal antibodies against large, middle, and small proteins. Patients with an active f…

HBsAgHepatitis B virusHepatitis B virus DNA polymerasemedicine.disease_causeHepatitis B virus PRE betaImmunoenzyme Techniques03 medical and health sciences0302 clinical medicineViral Envelope ProteinsmedicineHumansViremiaProtein Precursors030304 developmental biologyHepatitis ChronicHepatitisHepatitis B virus0303 health sciencesHepatitis B Surface AntigensHepatologybiologyGastroenterologyAntibodies MonoclonalHepatitis Bmedicine.diseasebiology.organism_classificationHepatitis BVirologyMolecular biology3. Good healthHBeAgHepadnaviridaeLiverDNA Viral030211 gastroenterology & hepatologyGastroenterology
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HBV-specific immune defect in chronic hepatitis B (CHB) is correlated with a dysregulation of pro- and anti-inflammatory cytokines.

1999

SUMMARY The aim of this study was to examine the immunomodulating effects of rhIL-12 on the immune response induced by hepatitis B virus (HBV) antigens in clinical subgroups of patients with HBV infection. Peripheral blood mononuclear cells (PBMC) of 80 patients were stimulated with HBsAg, HBcAg, pre-S1Ag and tetanus toxoid in the absence or presence of IL-12 (0.01, 0.1 and 1 ng/ml). Stimulation by anti-CD3 + anti-CD28 and lipopolysaccharide (LPS) were used as controls. Proliferation and cytokine production were determined by 3H-thymidine uptake and ELISA after 72 h. After stimulation with HBV antigens only, production of tumour necrosis factor-alpha (TNF-α) or IL-10 was observed in all pat…

HBsAgHepatitis B virusImmunologyAntigen-Presenting CellsIn Vitro Techniquesmedicine.disease_causeLymphocyte ActivationHepatitis B AntigensInterferon-gammaHepatitis B ChronicOrthohepadnavirusmedicineImmunology and AllergyHumansHepatitis B AntibodiesHepatitisHepatitis B virusbiologybusiness.industryTumor Necrosis Factor-alphavirus diseasesOriginal ArticlesHepatitis Bmedicine.diseasebiology.organism_classificationVirologyInterleukin-12digestive system diseasesRecombinant ProteinsInterleukin-10HBcAgHBeAgHepadnaviridaeImmunologyDNA ViralLeukocytes MononuclearCytokinesInflammation MediatorsbusinessClinical and experimental immunology
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Long-term efficacy of hepatitis B vaccine, booster policy, and impact of hepatitis B virus mutants

2005

The long-term efficacy of hepatitis B vaccine, long-term effectiveness of hepatitis B immunisation programmes, immune memory induced by hepatitis B vaccine, current booster policies, and impact of hepatitis B virus mutants on immunisation programmes were reviewed at the Viral Hepatitis Prevention Board (VHPB) meeting in Sevilla, Spain, March 2004. The main focus was on universal vaccination programmes with data being presented from Italy, Saudi Arabia, Singapore, Spain, Taiwan, Thailand, The Gambia, and USA (Alaska).

Hepatitis B vaccineSettore MED/42 - Igiene Generale e ApplicataImmunization Secondarymedicine.disease_causeTimeOrthohepadnavirusparasitic diseasesHumanshepatitis B vaccination long-term efficacy virus mutantsMedicineHepatitis B VaccinesHepatitis B AntibodiesHepatitis B virusGeneral VeterinaryGeneral Immunology and MicrobiologybiologyImmunization Programsbusiness.industryPublic Health Environmental and Occupational HealthHepatitis BHepatitis Bbiology.organism_classificationmedicine.diseaseVirologyVaccinationInfectious DiseasesHepadnaviridaeDNA ViralMutationMolecular MedicineViral diseasebusinessViral hepatitisImmunologic Memorygeographic locationsVaccine
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