Search results for "hepatitis c virus"

showing 10 items of 403 documents

Hepatitis C virus and cardiovascular: A review

2017

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Systemic diseaseCirrhosisFibrosiHepatitis C virusReviewmedicine.disease_causeVirus03 medical and health sciences0302 clinical medicineInsulin resistanceFibrosismedicine030212 general & internal medicineGenerallcsh:Science (General)ComputingMethodologies_COMPUTERGRAPHICSlcsh:R5-920MultidisciplinaryHigh prevalencebusiness.industryvirus diseasesmedicine.diseaseCardiovascular riskAtherosclerosisFibrosisdigestive system diseasesDeathAtherosclerosiImmunologyHCV030211 gastroenterology & hepatologyHearthSteatosisbusinesslcsh:Medicine (General)lcsh:Q1-390Journal of Advanced Research
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Intracellular accumulation of incompletely processed transforming growth factor-alpha polypeptides in ground glass hepatocytes of chronic hepatitis B…

1996

Abstract Background: Transforming growth factor-alpha is an intracellularly processed and secreted polypeptide that induces a proliferative response in epithelial target cells and represents a potential regulatory factor in embryonic development, liver regeneration, and also hepatocarcinogenesis. We have observed focal transforming growth factor-alpha expression in liver tissues with chronic hepatitis B virus infection. Methods: To further elucidate the nature of this focal transforming growth factor-alpha accumulation were have analyzed overall 23 different liver tissues with chronic hepatitis B virus and hepatitis C virus infection as well as normal liver tissues by immunohistology, ELISA…

TGF alphaHepatitis B Surface AntigensHepatologybiologyHepatitis C virusGrowth factormedicine.medical_treatmentHepatitis BTransforming Growth Factor alphamedicine.disease_causemedicine.diseasebiology.organism_classificationHepatitis BVirologyMolecular biologyLiver regenerationHepadnaviridaeLiverTransforming growth factor beta 3Case-Control StudiesChronic DiseasemedicineHumansGDF15PeptidesJournal of hepatology
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Management of HCV-Related Liver Disease in Hemophilia and Thalassemia

2018

AbstractChronic infection with the hepatitis C virus (HCV) has long been the dominant complication of substitution therapy in patients with inherited blood disorders and the cause of anticipated death due to end-stage liver disease. In hemophilia, transmission of HCV with clotting factors concentrates started to be curbed in the mid-1980s following the adoption of procedures of virus inactivation of concentrates based on heat, whereas in the 1990s treatment of HCV infection with interferon monotherapy was attempted, however, with little success. The advent of combination therapy of interferon with ribavirin led to a substantial improvement of treatment outcome (40% rate of cure), that howev…

Time FactorsTime FactorThalassemiaHepatitis C virusPopulationAdministration Oral030204 cardiovascular system & hematologyHemophilia Amedicine.disease_causeAntiviral Agents03 medical and health scienceschemistry.chemical_compoundLiver disease0302 clinical medicinesickle cell anemiaRisk FactorshemophiliamedicineHumansBlood TransfusioneducationImmunodeficiencyAntiviral AgentClotting factoreducation.field_of_studydirect antiviral agentHepatologybusiness.industryRisk FactorRibavirinHepatitis CHepatitis C Chronicmedicine.diseaseTreatment OutcomechemistryImmunologyThalassemiaDrug Therapy Combination030211 gastroenterology & hepatologyhepatitis CbusinessHumanSeminars in Liver Disease
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Boceprevir is highly effective in treatment-experienced hepatitis C virus-positive genotype-1 menopausal women

2014

AIM: To investigate the safety/efficacy of Boceprevirbased triple therapy in hepatitis C virus (HCV)-G1 menopausal women who were historic relapsers, partial-responders and null-responders. METHODS: In this single-assignment, unblinded study, we treated fifty-six menopausal women with HCV-G1, 46% F3-F4, and previous PEG-α/RBV failure (7% null, 41% non-responder, and 52% relapser) with 4 wk lead-in with PEG-IFNα2b/RBV followed by PEGIFNα2b/RBV+Boceprevir for 32 wk, with an additional 12 wk of PEG-IFN-α-2b/RBV if patients were HCV-RNA-positive by week 8. In previous null-responders, 44 wk of triple therapy was used. The primary objective of retreatment was to verify whether a sustained virolo…

Time FactorsViral HepatitisClinical Trials StudyHepacivirusViral hepatitiPolyethylene GlycolPolyethylene Glycolschemistry.chemical_compoundPegylated interferonOdds RatioMultivariate AnalysiPegylated InterferonGastroenterologyGeneral MedicineHepatitis CHepatitis c virus treatmentMiddle AgedRecombinant ProteinViral LoadGenotype 1Recombinant ProteinsMenopauseTreatment OutcomeItalyRNA ViralDrug Therapy CombinationFemaleMenopauseViral hepatitisViral loadPegylated interferonHumanmedicine.drugmedicine.medical_specialtyGenotypeLogistic ModelProlineTime FactorInterferon alpha-2Hepatitis C virus treatmentAntiviral AgentsPharmacotherapyInternal medicineBoceprevirRibavirinmental disordersmedicineHumansAntiviral AgentHepacivirubusiness.industryInterferon-alphaBiomarkerGenotype 1; Hepatitis c virus treatment; Menopause; Pegylated interferon; Viral hepatitis; Antiviral Agents; Biomarkers; Drug Therapy Combination; Female; Genotype; Hepacivirus; Hepatitis C Chronic; Humans; Interferon-alpha; Italy; Logistic Models; Middle Aged; Multivariate Analysis; Odds Ratio; Polyethylene Glycols; Proline; RNA Viral; Recombinant Proteins; Ribavirin; Time Factors; Treatment Outcome; Viral Load; Menopause; GastroenterologyHepatitis C Chronicmedicine.diseaseVirologyLogistic ModelschemistryHepatitis C Virus PositiveMultivariate AnalysisGenotype 1; Hepatitis C virus treatment; Menopause; Pegylated Interferon; Viral HepatitisbusinessBiomarkers
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Last gasps of the hepatitis C virus dragon: Direct acting antiviral failures and hepatitis C virus-positive donors.

2016

TransplantationHepatologybusiness.industrymedicine.medical_treatmentHepatitis C virusLiver transplantationmedicine.disease_causeVirologyAntiviral AgentsHepatitis CTreatment failureLiver Transplantation03 medical and health sciences0302 clinical medicineHepatitis C Virus PositiveMedicineHumans030211 gastroenterology & hepatologySurgery030212 general & internal medicineTreatment FailurebusinessDirect actingLiver transplantation : official publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society
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Efficacy of the retreatment of hepatitis C virus infections after liver transplantation: Role of an aggressive approach

2012

A sustained virological response (SVR) is achieved by 30% of naive liver transplantation (LT) recipients treated with pegylated interferon (PEG-IFN) and ribavirin (RBV). Almost no data are available about retreatment. The aim of this study was to assess the efficacy, tolerability, and SVR predictors of retreatment. Data were collected from 4 centers on the retreatment of prior nonresponders to standard therapy or PEG-IFN (with or without RBV) and relapsers. Seventy-nine of 301 treatment-experienced LT patients (26%), who had a median age of 59 years (range = 35-77 years) and were mostly male (72%) and infected with genotype 1 (87%), were retreated with PEG-IFN and RBV at a median of 6.9 yea…

Transplantationmedicine.medical_specialtyCirrhosisHepatologybusiness.industryRibavirinmedicine.medical_treatmentHepatitis C virusLiver transplantationmedicine.diseasemedicine.disease_causeGastroenterologyTacrolimusSurgerychemistry.chemical_compoundchemistryTolerabilityErythropoietinPegylated interferonInternal medicinemedicineSurgerybusinessmedicine.drugLiver Transplantation
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Anemia is not predictive of sustained virological response in liver transplant recipients with hepatitis C virus who are treated with pegylated inter…

2011

In the immunocompetent setting, antiviral therapy–related anemia has recently been shown to be associated with a sustained virological response (SVR). Our goal was to assess whether this is also true for liver transplantation (LT). We included 160 LT patients with recurrent hepatitis C virus (HCV) who were treated with pegylated interferon and ribavirin (RBV) between 2002 and 2010; 76% of the patients were men, the median age of the patients was 56 years (range = 33-75 years), 63% had advanced fibrosis, and 86% were infected with HCV genotype 1a or 1b. The baseline immunosuppression was tacrolimus in 56% of the patients. Mycophenolate mofetil (MMF) was used in 15%. Anemia was defined as a h…

Transplantationmedicine.medical_specialtyHepatologybusiness.industryAnemiamedicine.medical_treatmentRibavirinHepatitis C virusRenal functionImmunosuppressionLiver transplantationmedicine.disease_causemedicine.diseaseGastroenterologychemistry.chemical_compoundchemistryErythropoietinPegylated interferonInternal medicineImmunologymedicineSurgerybusinessmedicine.drugLiver Transplantation
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Prospective validation of a noninvasive index for predicting liver fibrosis in hepatitis C virus-infected liver transplant recipients

2009

We previously developed a mathematical model, the Hospital Universitario La Fe (HULF) index, as an alternative to protocol liver biopsy (PLB) to estimate significant fibrosis (SF) in patients who underwent liver transplantation (LT) for liver damage caused by chronic HCV infection. In the present study, we sought to validate this noninvasive index. The commonly derived clinical and laboratory data for calculating the HULF index were prospectively collected over 2.7 years from patients undergoing LT and PLB. The sensitivity, specificity, positive and negative predictive values, and diagnostic capacity were evaluated with receiver operating characteristic curve analysis. Biopsy was performed …

Transplantationmedicine.medical_specialtyHepatologymedicine.diagnostic_testReceiver operating characteristicbusiness.industryHepatitis C virusmedicine.medical_treatmentConcordanceLiver transplantationmedicine.disease_causeGastroenterologySurgeryPositive predicative valueLiver biopsyInternal medicineBiopsymedicineSurgerybusinessBody mass indexLiver Transplantation
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Extrahepatic Morbidity and Mortality of Chronic Hepatitis C

2015

Chronic hepatitis C virus (HCV) infection is associated with several extra-hepatic manifestations. Patients with HCV may develop mixed cryoglobulinemia and its sequelae, ranging from cutaneous and visceral vasculitis to glomerulonephritis and B cell non-Hodgkin’s lymphoma. HCV-infected patients have increased rates of insulin resistance, diabetes and atherosclerosis, which may lead to increased cardiovascular morbidity and mortality. Neurologic manifestations of HCV infection include fatigue and cognitive impairment. The mechanisms causing the extra-hepatic effects of HCV infection are likely multifactorial and may include endocrine effects, HCV replication in extra-hepatic cells, or a heig…

VasculitisLymphomaGlomerulonephritis/epidemiology/virologyLymphoma/epidemiology/virologyHepatitis C virusAlpha interferonHepacivirusddc:616.07Cryoglobulinemia/epidemiology/virologymedicine.disease_causeAntiviral AgentsAntiviral Agents/administration & dosage/pharmacology/therapeutic useHepacivirus/drug effects/pathogenicityLiver diseasechemistry.chemical_compoundGlomerulonephritisDiabetes mellitusRibavirinmedicineHumansGlucose Metabolism Disorders/epidemiology/virologyInterferon alfaGlucose Metabolism Disordersddc:616Hepatologybusiness.industryHepatitis C Chronic/drug therapy/epidemiology/immunology/mortality/virologyRibavirinVasculitis/epidemiology/virologyGastroenterologyInterferon-alphavirus diseasesHepatitis C Chronicmedicine.diseaseCryoglobulinemiadigestive system diseasesCryoglobulinemiachemistryRibavirin/pharmacology/therapeutic useHCVImmunologyMorbiditybusinessVasculitisInterferon-alpha/pharmacology/therapeutic usemedicine.drugGastroenterology
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Replication of hepatitis C virus

2000

Viral pathogenesisHepatitis C virusVirus AssemblyViral transformationHepatitis CHepacivirusBiologymedicine.diseasemedicine.disease_causeTransfectionVirus ReplicationMicrobiologyVirologyPathogenesisViral ProteinsViral replicationReplication (statistics)medicineAnimalsRNA ViralOncovirusCells Cultured
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