Search results for "hydrogen peroxide"

showing 10 items of 321 documents

Effect of cooking on oxalate content of pulses using an enzymatic procedure.

2003

An enzymatic method proposed for the determination of oxalate in urine is adapted for the estimation of soluble oxalate content in beans, chickpeas and lentils. Oxalates were extracted in water by refluxing for 2 h. The method is based on the oxidation of oxalate by the oxidase and the determination of the resulting hydrogen peroxide, which in presence of peroxidase, 3-methyl-2 benzotiazinolone and 3-dimethylamino benzoic, gives an indamine compound with an absorption maximum at 590 nm. The linearity (from 0.015 to 0.6 mM) of the method is adequate to the analysis of oxalate contents in pulses, and the inter-day precision of the method expressed as relative standard deviation was good (3.01…

Absorption (pharmacology)chemistry.chemical_classificationOxalatesChromatographyHot TemperaturebiologyChemistryFood HandlingInorganic chemistryRelative standard deviationFabaceaeSoluble oxalateOxalateFood handlingchemistry.chemical_compoundEnzymebiology.proteinHydrogen peroxideMicrowavesNutritive ValueFood SciencePeroxidaseInternational journal of food sciences and nutrition
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Diverse stress signals activate the C1 subgroup MAP kinases ofArabidopsis

2007

AbstractMitogen-activated protein kinase (MAPK) cascades play an important role in mediating stress responses in plants. In Arabidopsis, 20 MAPKs have been identified and classified into four major groups (A–D). Little is known about the role of group C MAPKs. We have studied the activation of Arabidopsis subgroup C1 MAPKs (AtMPK1/AtMPK2) in response to mechanical injury. An increase in their kinase activity was detected in response to wounding that was blocked by cycloheximide. Jasmonic acid (JA) activated AtMPK1/AtMPK2 in the absence of wounding. Wound and JA-induction of AtMPK1/2 kinase activity was not prevented in the JA-insensitive coi1 mutant. Other stress signals, such as abscisic a…

AcclimatizationArabidopsisBiophysicsBiochemistryGene Expression Regulation Enzymologicchemistry.chemical_compoundGene Expression Regulation PlantStructural BiologyArabidopsisGeneticsASK1Kinase activityProtein kinase AMolecular BiologyJasmonic acidMAP kinase kinase kinasebiologyArabidopsis ProteinsKinaseJasmonic acidWoundHydrogen PeroxideCell Biologybiology.organism_classificationBiochemistrychemistryMitogen-activated protein kinasebiology.proteinMAP kinaseStress MechanicalMitogen-Activated Protein KinasesAbscisic AcidSignal TransductionFEBS Letters
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Redox and autonomic responses to acute exercise-post recovery following Opuntia ficus-indica juice intake in physically active women

2021

Background: The aim of this study was to investigate if the supplementation with Opuntia ficus-indica (OFI) juice may affect plasma redox balance and heart rate variability (HRV) parameters following a maximal effort test, in young physically active women. Methods: A randomized, double blind, placebo controlled and crossover study comprising eight women (23.25 ± 2.95 years, 54.13 ± 9.05 kg, 157.75 ± 0.66 cm and BMI of 21.69 ± 0.66 kg/m2) was carried out. A juice containing OFI diluted in water and a Placebo solution were supplied (170 ml; OFI = 50 ml of OFI juice + 120 ml of water; Placebo = 170 ml beverage without Vitamin C and indicaxanthin). Participants consumed the OFI juice or Placebo…

Adult0301 basic medicineCactus pear juice supplementationOpuntia ficusTotal antioxidant capacityClinical nutritionmedicine.disease_causePlaceboYoung Adult03 medical and health scienceschemistry.chemical_compoundRedox balanceOxygen Consumption0302 clinical medicineAnimal scienceDouble-Blind MethodHigh frequencyHeart RatemedicineHumansHeart rate variabilityAutonomic nervous systemExerciseCross-Over StudiesNutrition and DieteticsSettore M-EDF/02 - Metodi E Didattiche Delle Attivita' SportiveVitamin Cbusiness.industryOpuntiaHydrogen Peroxide030229 sport sciencesCrossover studyFruit and Vegetable JuicesHydroperoxideHydroperoxides030104 developmental biologychemistryOxidative stressOxidative streFemalebusinessLow frequencyOxidation-ReductionIndicaxanthinSettore M-EDF/01 - Metodi E Didattiche Delle Attivita' MotorieOxidative stressResearch ArticleFood Science
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Involvement of hydrogen and lipid peroxides in acute tobacco smoking-induced platelet hyperactivity

1995

Previous studies have established that cigarette smoking results in acute platelet hyperaggregability. We investigated whether changes in plasma oxidative properties could occur after smoking and whether such changes could be responsible for this enhanced platelet activity. In the present work, we report that platelets from nonsmokers become hyperactive after incubation with plasma prepared from blood of smokers obtained 10 min after smoking. This effect was not observed with presmoking plasma and could be inhibited in vitro by adding either catalase or reduced glutathione plus peroxidase to plasma or 2,6-di-tert-butyl-p-cresol (BHT) to platelets before incubation. Comparison of pre- and p…

AdultBlood PlateletsMaleLipid Peroxidesmedicine.medical_specialtyPlatelet AggregationPhysiologymedicine.medical_treatmentFatty Acids NonesterifiedAntioxidantschemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineHumansPlateletPlatelet activationIncubationchemistry.chemical_classificationbiologyVitamin ESmokingThrombinFatty acidHydrogen PeroxideGlutathioneButylated HydroxytolueneMiddle AgedBlood Physiological PhenomenaAdenosine DiphosphateEndocrinologychemistryBiochemistryCatalasebiology.proteinCardiology and Cardiovascular MedicinePeroxidaseAmerican Journal of Physiology-Heart and Circulatory Physiology
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Induction of heme oxygenase-1 and adaptive protection against the induction of DNA damage after hyperbaric oxygen treatment.

2000

Hyperbaric oxygen (HBO) treatment of human subjects (i.e. exposure to 100% oxygen at a pressure of 2.5 ATA for a total period of 3 x 20 min) caused clear and reproducible DNA damage in lymphocytes, as detected with the comet assay (single cell gel electrophoresis). Induction of DNA damage was found only after the first HBO exposure and not after further treatments of the same individuals. Furthermore, blood taken 24 h after HBO treatment was significantly protected against the induction of DNA damage by hydrogen peroxide (H(2)O(2)) in vitro, indicating that adaptation occurred due to induction of antioxidant defenses. The cells were not significantly protected against the genotoxic effects …

AdultCancer ResearchDNA RepairDNA repairDNA damageCarbon-Oxygen LyasesBiologymedicine.disease_causeSuperoxide dismutasemedicineDNA-(Apurinic or Apyrimidinic Site) LyaseHumansLymphocytesDNA Polymerase betachemistry.chemical_classificationReactive oxygen speciesHyperbaric OxygenationSuperoxide DismutaseMembrane ProteinsGeneral MedicineHydrogen PeroxideCatalaseMolecular biologyDNA-(apurinic or apyrimidinic site) lyaseAdaptation PhysiologicalDeoxyribonuclease IV (Phage T4-Induced)Comet assayOxidative StresschemistryBiochemistryCatalaseEnzyme InductionHeme Oxygenase (Decyclizing)biology.proteinOxidative stressHeme Oxygenase-1DNA DamageCarcinogenesis
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3D molecular phenotyping of cleared human brain tissues with light-sheet fluorescence microscopy

2022

AbstractThe combination of optical tissue transparency with immunofluorescence allows the molecular characterization of biological tissues in 3D. However, adult human organs are particularly challenging to become transparent because of the autofluorescence contributions of aged tissues. To meet this challenge, we optimized SHORT (SWITCH—H2O2—antigen Retrieval—TDE), a procedure based on standard histological treatments in combination with a refined clearing procedure to clear and label portions of the human brain. 3D histological characterization with multiple molecules is performed on cleared samples with a combination of multi-colors and multi-rounds labeling. By performing fast 3D imaging…

AdultImaging Three-DimensionalMicroscopy FluorescenceBrainFluorescent Antibody TechniqueHumansMedicine (miscellaneous)Hydrogen PeroxideGeneral Agricultural and Biological SciencesSettore FIS/07 - Fisica Applicata(Beni Culturali Ambientali Biol.e Medicin)General Biochemistry Genetics and Molecular BiologyAgedCommunications Biology
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Comparison of diffusion, cytotoxicity and tissue inflammatory reactions of four commercial bleaching products against human dental pulp stem cells

2018

AbstractMultiple side effects related to bleaching were found to occur in the dental pulp tissue, including decreased cell metabolism and viability. In this work we evaluated the in vitro diffusion capacity, cytotoxicity and biocompatibility of four commercial bleaching products on stem cells from human dental pulp (hDPSCs). Two commercial bleaching gels hydrogen peroxide-based (HP), Norblanc Office 37.5% (Nor-HP) and Opalescence Boost 40% (Opal-HP) were applied for 30 min to enamel/dentine discs. Another two gels from the same manufacturers, 16% carbamide peroxide-based (CP), Norblanc Home (Nor-CP) and Opalescence CP 16% (Opal-CP), were applied for 90 min. The diffusion of HP was analysed …

AdultMale0301 basic medicineNecrosisBiocompatibilityScienceCarbamide PeroxideArticleDiffusionTooth whiteningYoung Adult03 medical and health scienceschemistry.chemical_compoundDental biomaterials0302 clinical medicineAnti-Infective Agentsstomatognathic systemDental pulp stem cellsTooth BleachingmedicineAnimalsHumansRats WistarTooth Bleaching AgentsHydrogen peroxideCytotoxicityDental PulpInflammationMultidisciplinaryEnamel paintStem CellsQRHydrogen PeroxideMolecular biologyPeroxidesRatsstomatognathic diseases030104 developmental biologychemistryvisual_artToxicityMicroscopy Electron Scanningvisual_art.visual_art_mediumMedicinePulp (tooth)Femalemedicine.symptom030217 neurology & neurosurgeryScientific Reports
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Mitochondrial dysfunction, persistent oxidative damage, and catalase inhibition in immune cells of naïve and treated Crohnʼs disease

2009

Background: Oxidative stress is considered a potential etiological factor for Crohn's disease (CD). We characterized the reactive oxygen species (ROS) generated in immune peripheral cells of CD patients, as well as their antioxidant enzyme status and the presence of oxidative damage. In addition, mitochondrial function (ΔΨm) was analyzed to detect the possible origin of ROS. Methods: Cells were obtained from patients at the onset of disease, prior to any treatment. Experiments were repeated when patients were in clinical remission. A set of experiments was carried out in a group of CD patients in persistent morphological remission. Controls were healthy volunteers who were not receiving any…

AdultMaleMitochondrial DiseasesMitochondrionBiologyPharmacologymedicine.disease_causeInflammatory bowel diseaseAntioxidantsMonocytesNitric oxideSuperoxide dismutasechemistry.chemical_compoundCrohn DiseaseMalondialdehydemedicineHumansImmunology and AllergyLymphocytesMembrane Potential Mitochondrialchemistry.chemical_classificationReactive oxygen speciesSuperoxide DismutaseSuperoxideGastroenterologyDeoxyguanosineHydrogen PeroxideCatalaseOxidantsmedicine.diseaseOxidative Stresschemistry8-Hydroxy-2'-DeoxyguanosineCatalaseCase-Control StudiesImmunologybiology.proteinFemaleReactive Oxygen SpeciesOxidative stressGranulocytesInflammatory Bowel Diseases
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Asthmatic Bronchial Epithelium Is More Susceptible to Oxidant-Induced Apoptosis

2002

Abnormal apoptotic mechanisms are associated with disease pathogenesis. Because the asthmatic bronchial epithelium is characteristically damaged with loss of columnar epithelial cells, we postulated that this is due to unscheduled apoptosis. Using an antibody directed toward the caspase cleavage product of poly(ADP-ribose) polymerase, immunohistochemistry applied to endobronchial biopsies showed higher levels of staining in the bronchial epithelium of subjects with asthma as compared with normal control subjects (% epithelial staining [median (range) = 10.5 (1.4-24.5) versus 0.4 (0.0-9.7)]; P < 0.001). Because we were unable to determine whether this difference was due to ongoing inflammati…

AdultMalePulmonary and Respiratory MedicinePathologymedicine.medical_specialtyCell Biology; Molecular Biology; Pulmonary and Respiratory MedicineBiopsyClinical BiochemistryCellApoptosisBronchiInflammationRespiratory MucosaBiologyIn vivomedicineHumansMolecular BiologyCells CulturedAgedTumor Necrosis Factor-alphaEpithelial CellsHydrogen PeroxideCell BiologyMiddle AgedFlow CytometryOxidantsAsthmaIn vitroStainingmedicine.anatomical_structureApoptosisbiology.proteinImmunohistochemistryFemalePoly(ADP-ribose) Polymerasesmedicine.symptomAntibodyAmerican Journal of Respiratory Cell and Molecular Biology
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AZT treatment induces molecular and ultrastructural oxidative damage to muscle mitochondria. Prevention by antioxidant vitamins.

1998

AIDS patients who receive zidovudine (AZT) frequently suffer from myopathy. This has been attributed to mitochondrial (mt) damage, and specifically to the loss of mtDNA. This study examines whether AZT causes oxidative damage to DNA in patients and to skeletal muscle mitochondria in mice, and whether this damage may be prevented by supranutritional doses of antioxidant vitamins. Asymptomatic HIV-infected patients treated with AZT have a higher urinary excretion (355+/-100 pmol/kg/d) of 8-oxo-7, 8-dihydro-2'-deoxyguanosine (8-oxo-dG) (a marker of oxidative damage to DNA) than untreated controls (asymptomatic HIV-infected patients) (182+/-29 pmol/kg/d). This was prevented (110+/-79 pmol/kg/d)…

AdultMalemedicine.medical_specialtyDNA damageAnti-HIV Agentsmedicine.medical_treatmentAscorbic AcidBiologyDNA MitochondrialAntioxidantsZidovudinechemistry.chemical_compoundMiceInternal medicinemedicineDeoxyguanosineAnimalsHumansVitamin Eheterocyclic compoundsMyopathyVitamin ESkeletal musclevirus diseasesDeoxyguanosineGeneral MedicineGlutathioneHydrogen PeroxideAscorbic acidMitochondria Musclemedicine.anatomical_structureEndocrinologychemistryBiochemistry8-Hydroxy-2'-Deoxyguanosinemedicine.symptomZidovudinemedicine.drugDNA DamageResearch ArticleThe Journal of clinical investigation
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