Search results for "hydroxynorketamine"

showing 4 items of 4 documents

Sexually Dimorphic Behavioral Profile in a Transgenic Model Enabling Targeted Recombination in Active Neurons in Response to Ketamine and (2R,6R)-Hyd…

2020

Background: Rapid-acting antidepressants ketamine and (2R,6R)-hydroxynorketamine ((2R,6R)-HNK) have overcome some of the major limitations of classical antidepressants. However, little is known about sex-specific differences in the behavioral and molecular effects of ketamine and (2R,6R)-HNK in rodents. Methods: We treated mice with an intraperitoneal injection of either saline, ketamine (30 mg kg&minus

Male0301 basic medicineHydroxynorketaminemedicine.medical_treatmentAntidepressantAnxietyHippocampuslcsh:Chemistry0302 clinical medicinelcsh:QH301-705.5Salineactivated neuronsSpectroscopyNeuronsRecombination GeneticSex CharacteristicsBehavior AnimalhydroxynorketamineGeneral MedicineComputer Science ApplicationsActivated neuronsAntidepressantFemaleKetaminemedicine.drugmedicine.medical_specialtyketamineMemory Episodicsex differenceGreen Fluorescent ProteinsIntraperitoneal injectionMice TransgenicIn situ hybridizationBiologyHydroxynorketamineArticleCatalysisInorganic Chemistry03 medical and health sciencesInternal medicineketamine ; sex difference ; activated neurons ; antidepressant ; behavior ; BDNF ; rapid-acting ; hydroxynorketaminemedicineAnimalsKetamineRapid-actingPhysical and Theoretical ChemistrySocial BehaviorMolecular BiologyCell NucleusBehaviorantidepressantbehaviorBrain-Derived Neurotrophic FactorOrganic ChemistrySex differencerapid-actingSexual dimorphismDisease Models AnimalBDNF030104 developmental biologyEndocrinologylcsh:Biology (General)lcsh:QD1-999030217 neurology & neurosurgeryBehavioural despair test
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Longitudinal CSF proteome profiling in mice to uncover the acute and sustained mechanisms of action of rapid acting antidepressant (2R,6R)-hydroxynor…

2021

Delayed onset of antidepressant action is a shortcoming in depression treatment. Ketamine and its metabolite (2R,6R)-hydroxynorketamine (HNK) have emerged as promising rapid-acting antidepressants. However, their mechanism of action remains unknown. In this study, we first described the anxious and depression-prone inbred mouse strain, DBA/2J, as an animal model to assess the antidepressant-like effects of ketamine and HNK in vivo. To decode the molecular mechanisms mediating HNK's rapid antidepressant effects, a longitudinal cerebrospinal fluid (CSF) proteome profiling of its acute and sustained effects was conducted using an unbiased, hypothesis-free mass spectrometry-based proteomics app…

ProteomicsNeurophysiology and neuropsychologyanimal structuresHydroxynorketaminePhysiologyGlucocorticoid receptor signalingAntidepressantCSFNeurosciences. Biological psychiatry. NeuropsychiatryBiologyPharmacologyProteomicsBiochemistryCellular and Molecular NeuroscienceEndocrinologyGlucocorticoid receptorNeurotrophic factorsmedicineOriginal Research ArticleKetamine ; CSF ; Antidepressant ; (2R6R)-Hydroxynorketamine ; Glucocorticoid receptor signaling ; ProteomicsRC346-429Molecular BiologyPI3K/AKT/mTOR pathwayEndocrine and Autonomic SystemsQP351-495Mechanism of action(2R6R)-Hydroxynorketamineembryonic structuresAntidepressantKetamineNeurology. Diseases of the nervous systemmedicine.symptomSignal transductionRC321-571Neurobiology of Stress
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Decoding the Mechanism of Action of Rapid-Acting Antidepressant Treatment Strategies: Does Gender Matter?

2019

Gender differences play a pivotal role in the pathophysiology and treatment of major depressive disorder. This is strongly supported by a mean 2:1 female-male ratio of depression consistently observed throughout studies in developed nations. Considering the urgent need to tailor individualized treatment strategies to fight depression more efficiently, a more precise understanding of gender-specific aspects in the pathophysiology and treatment of depressive disorders is fundamental. However, current treatment guidelines almost entirely neglect gender as a potentially relevant factor. Similarly, the vast majority of animal experiments analysing antidepressant treatment in rodent models exclus…

MalePsychotherapistsex differencemedia_common.quotation_subjectmedicine.medical_treatmentReviewelectroconvulsive therapyCatalysisNeglectlcsh:ChemistryInorganic Chemistryendocrinology03 medical and health sciences0302 clinical medicineElectroconvulsive therapygendermedicineAnimalsHumansPhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopyDepression (differential diagnoses)media_commonDepressive Disorder Majorantidepressantbusiness.industryOrganic ChemistryGeneral MedicineRapid-acting antidepressantrapid-actingmedicine.diseaseAntidepressive Agents030227 psychiatryComputer Science ApplicationsReview articleTreatment Outcomelcsh:Biology (General)lcsh:QD1-999(2R6R)-HydroxynorketaminedepressionTreatment strategyAntidepressantMajor depressive disorderFemaleKetaminebusiness030217 neurology & neurosurgery
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Rapid acting antidepressant (2R,6R)-hydroxynorketamine (HNK) targets glucocorticoid receptor signaling: a longitudinal cerebrospinal fluid proteome s…

2020

AbstractDelayed onset of antidepressant action is a shortcoming in depression treatment. Ketamine and its metabolite (2R,6R)-hydroxynorketamine (HNK) have emerged as promising rapidacting antidepressants. However, their mechanism of action remains unknown. In this study, we first described the anxious and depression-prone inbred mouse strain, DBA/2J, as a animal model to assess the antidepressant-like effects of ketamine and HNK in vivo. To decode the molecular mechanisms mediating HNK’s rapid antidepressant effects, a longitudinal cerebrospinal fluid (CSF) proteome profiling of its acute and sustained effects was conducted using an unbiased, hypothesis-free mass spectrometry-based proteomi…

Glucocorticoid receptorHydroxynorketamineMechanism of actionNeurotrophic factorsProteomemedicineSignal transductionPharmacologyBiologymedicine.symptomProteomicsPI3K/AKT/mTOR pathway
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