Search results for "hypercalciuria"

showing 10 items of 10 documents

Predictive and prognostic value of magnesium serum level in FOLFIRI plus cetuximab or bevacizumab treated patients with stage IV colorectal cancer: r…

2020

Magnesium wasting is a frequent side effect of epidermal growth factor receptor (EGFR)-antibody treatment as magnesium-absorption mechanisms are dependent on EGFR signaling. EGFR-inhibition results in decreased renal reabsorption. There is evidence that hypomagnesemia during cetuximab treatment correlates with response. The prognostic role of hypomagnesemia during bevacizumab treatment has not been studied yet. Here, we evaluate the prognostic value of hypomagnesemia in patients with metastatic colorectal cancer treated with FOLFIRI plus cetuximab or bevacizumab as first-line therapy. A total of 391 of 752 patients of the firstline irinotecan study population had magnesium levels measured a…

0301 basic medicineMaleCancer Researchmedicine.medical_specialtyRenal Tubular Transport Inborn ErrorsBevacizumabSide effectColorectal cancerHypercalciuriaLeucovorinCetuximabIrinotecanGastroenterologyHypomagnesemia03 medical and health sciences0302 clinical medicineInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansPharmacology (medical)MagnesiumAgedRetrospective StudiesPharmacologyCetuximabbusiness.industryHazard ratiomedicine.diseasePrognosisIrinotecanBevacizumabSurvival RateNephrocalcinosis030104 developmental biologyOncology030220 oncology & carcinogenesisFOLFIRICamptothecinFemaleFluorouracilbusinessColorectal Neoplasmsmedicine.drugFollow-Up StudiesAnti-cancer drugs
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Increased Gastrin and Calcitonin Secretion after Oral Calcium or Peptones Administration in Patients with Hypercalciuria: A Clue to an Alteration in …

2005

The calcium-sensing receptor (CaSR) has been detected in human antral gastrin-secreting cells, where, upon calcium and/or amino acid allosteric activation, it stimulates gastrin secretion. Patients with absorptive hypercalciuria (AH) display an enhanced gastric acid output; therefore, we evaluated the secretion of gastrin in subjects with AH ( 30 subjects vs. 30 healthy female controls, all postmenopausal) after oral calcium administration ( 1 g calcium gluconate) and, on a separate occasion, after peptone loading test ( protein hydrolyzed, 10 g). Gastrin and monomeric calcitonin responses were higher in AH after both oral calcium administration ( P < 0.01) and peptone loading ( P< 0.01). B…

Calcitoninmedicine.medical_specialtyEndocrinology Diabetes and MetabolismClinical BiochemistryThyroid GlandAdministration Oralchemistry.chemical_element.CalciumBiochemistryKidney CalculiEndocrinologyOral administrationCalcium Metabolism DisordersInternal medicineGastrinsmedicineHumansGastrin-Secreting CellsHypercalciuriaAgedGastrinBiochemistry (medical)Middle AgedCalcitonin secretionmedicine.diseaseCalcium GluconateEndocrinologychemistryGastrointestinal hormoneParathyroid HormoneCalcitoninPeptonesFemaleCalcium-sensing receptorReceptors Calcium-Sensinghormones hormone substitutes and hormone antagonistsThe Journal of Clinical Endocrinology &amp; Metabolism
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Hypercalciuria and kidney calcifications in terminal 4q deletion syndrome: further evidence for a putative gene on 4q.

2004

We report a newborn girl with a de novo terminal 4q deletion (q31.3 --> qter) and a characteristic phenotype of minor facial anomalies, cleft palate, congenital heart defect, abnormalities of hands and feet, and postnatal onset of growth deficiency. Laboratory studies showed excessive urinary calcium excretion on standard milk formula and on oral calcium load. Blood measurements of parathyroid hormone, calcitonin, bicarbonate, calcium, phosphorus, magnesium, sodium, chlorine, potassium, and urinary measurements of phosphorus, magnesium, sodium, chlorine, potassium were normal for age. At 2 months of life, ultrasonography showed kidney calcifications. Clinical and laboratory data support the…

Infant NewbornIndiaSyndromecalcificationKidney CalculiKaryotypingHumansAbnormalities MultipleCalciumFemaledeletionChromosome DeletionChromosomes Human Pair 4hypercalciuriaUltrasonographyAmerican journal of medical genetics. Part A
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Enuresis Subtypes Based on Nocturnal Hypercalciuria: A Multicenter Study

2003

Purpose: Desmopressin may not be effective for nocturnal enuresis associated with polyuria and hypercalciuria. Nighttime hypercalciuria in an enuretic population from 5 centers and its correlation with nighttime polyuria were verified. Materials and Methods: A total of 450 enuretic patients (278 males, 172 females, mean age 9.7 years) were evaluated with 72-hour micturition charts, urinalysis, serum creatinine and osmolarity, diurnal and nocturnal electrolytes with fractional Na+ and K+ urinary excretion, and nocturnal (4 a.m.) plasma vasopressin. Creatinine electrolytes and osmolarity were measured in daytime (8 a.m. to 8 p.m.) and nighttime (8 p.m. to 8 a.m.) urine volumes. Patients were …

Malemedicine.medical_specialtyAdolescentendocrine system diseasesUrinalysisVasopressinsUrologymedia_common.quotation_subjectPopulationUrinary incontinenceUrologyUrinary incontinenceurologic and male genital diseasesUrinationDiagnosis DifferentialElectrolytesAdrenocorticotropic HormonePolyuriaEnuresisElectrolyteInternal medicineEnuresimedicineHumansEnuresis; Polyuria; Urinary incontinence; Vasopressin; Adolescent; Adrenocorticotropic Hormone; Calcium; Child; Circadian Rhythm; Creatinine; Deamino Arginine Vasopressin; Diagnosis Differential; Electrolytes; Enuresis; Female; Humans; Male; Polyuria; Vasopressins; UrologyDeamino Arginine VasopressinHypercalciuriaChildDesmopressineducationmedia_commoneducation.field_of_studymedicine.diagnostic_testPolyuriabusiness.industryEnuresismedicine.diseasefemale genital diseases and pregnancy complicationsCircadian RhythmEndocrinologyCreatinineCalciumFemalemedicine.symptombusinessVasopressinhormones hormone substitutes and hormone antagonistsHumanmedicine.drugJournal of Urology
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Vitamin D substrate-product relationship in idiopathic hypercalciuria.

2007

Absorptive hypercalciuria (AH) is associated with elevated levels of 1,25-dihydroxyvitamin D (1,25(OH)(2)D). While no increase of 1,25(OH)(2)D after oral administration of 25-hydroxyvitamin D (25OHD) at high doses has been claimed in normal subjects, a substrate-product relationship has been reported in normal children, young people after UV irradiation, older persons, postmenopausal women, primary hyperparathyroidism, renal failure, osteomalacia, and sarcoidosis. No data of this relationship in AH is available. To investigate 25OHD-1,25(OH)(2)D substrate-product relationship in AH, 161 AH patients (mean age 60.9+/-11.7 years) and 110 age- and sex-matched controls (mean age 61.5+/-12.4 year…

Malemedicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryHypercalciuriaAdministration OralBiochemistryBasal (phylogenetics)EndocrinologyOral administrationInternal medicinemedicineVitamin D and neurologyHumansHypercalciuriaVitamin DMolecular BiologyAgedKidneyOsteomalaciaChemistryCell BiologyMiddle Agedmedicine.diseaseSteroid hormoneEndocrinologymedicine.anatomical_structureCase-Control StudiesMolecular MedicineFemaleSeasonsPrimary hyperparathyroidismThe Journal of steroid biochemistry and molecular biology
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[Osteoporosis and diabetes].

2011

Diabetes mellitus and osteoporosis are chronic diseases with an elevated and growing incidence in the elderly. Recent epidemiological studies have demonstrated an elevated risk of hip, humerus and foot fractures in elder diabetic subjects. While type 1 diabetes is generally associated with a mild reduction in bone mineral density (BMD), type 2 diabetes, more prevalent in old subjects, is frequently linked to a normal or high BMD. Studies on experimental models of diabetes have suggested an altered bone structure that may help to explain the elevated risk of fractures observed in these animals and may as well help to explain the paradox of an incremented risk of fractures in type 2 diabetic …

Malemedicine.medical_specialtylcsh:Internal medicineTime FactorsBone densityOsteoporosisPopulationlcsh:MedicineType 2 diabetesDiabetes Mellitus ExperimentalFractures BoneSex FactorsRheumatologyBone DensityRisk FactorsInternal medicineDiabetes mellitusHyperinsulinemiaMedicineAnimalsHumansHypoglycemic AgentsInsulinHypercalciuriaProspective Studieseducationlcsh:RC31-1245AgedAged 80 and overType 1 diabeteseducation.field_of_studybusiness.industrylcsh:RAge FactorsMiddle Agedmedicine.diseaseRatsEndocrinologyDiabetes Mellitus Type 1Diabetes Mellitus Type 2OsteoporosisAccidental FallsFemalebusinessFollow-Up StudiesReumatismo
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VITAMIN D SUBSTRATE-PRODUCT RELATIONSHIP IN IDIOPATHIC HYPERCALCIURIA

2009

Absorptive hypercalciuria (AH) is associated with elevated levels of 1,25-dihydroxyvitamin D (1,25(OH)(2)D). While no increase of 1,25(OH)(2)D after oral administration of 25-hydroxyvitamin D (25OHD) at high doses has been claimed in normal subjects, a substrate-product relationship has been reported in normal children, young people after UV irradiation, older persons, postmenopausal women, primary hyperparathyroidism, renal failure, osteomalacia, and sarcoidosis. No data of this relationship in AH is available. To investigate 25OHD-1,25(OH)(2)D substrate-product relationship in AH, 161 AH patients (mean age 60.9+/-11.7 years) and 110 age- and sex-matched controls (mean age 61.5+/-12.4 year…

Settore MED/09 - Medicina Internavitamin d hypercalciuria kidney stones
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The effect of ions at the surface of calcium oxalate monohydrate crystals on cell-crystal interactions

2003

Magnesium is an abundant ion in biologic systems, including renal tubular fluid; however, the precise role of magnesium during the interaction of calcium oxalate crystals with cells has not been previously defined. In addition, the respective roles of calcium and hydrogen ions during the cell-crystal bonding interaction remain poorly defined. Here we report an atomic level three-dimensional study of a single crystal of calcium oxalate monohydrate (COM; whewellite) which was bathed in a solution of magnesium hexahydrate for 1 year. Magnesium was not incorporated into the structure of whewellite to any significant degree. Instead, COM accepted magnesium primarily as an adsorbate in a binding …

Time FactorsCell SurvivalUrologyInorganic chemistryCalcium oxalatechemistry.chemical_elementCrystal structureCalciumengineering.materialKidneyCell Linechemistry.chemical_compoundDogsImaging Three-DimensionalCell AdhesionmedicineAnimalsMagnesiumHypercalciuriaCarbon RadioisotopesIonsCalcium OxalateChemistryMagnesiumWhewelliteEpithelial CellsAdhesionHydrogen-Ion Concentrationmedicine.diseaseMicroscopy FluorescenceMicroscopy Electron ScanningengineeringCalciumCrystallizationSingle crystalUrological Research
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PATHOPHYSIOLOGY AND CLINICAL ASPECTS OF URINARY LITHIASIS

2007

Urine is a complex balanced solution containing dissociated and non-dissociated solutes. Any variation in urine saturation grade (number of crystals dissolved in a volume of urine), urinary pH and the concentration of crystallization inhibitors can break the normal existing balance and lead to urolithiasis. In the present article we analyze the principal mechanisms (absorptive, renal, resorptive) of hypercalciuria. It will be also shown how heredity directly influences the clinical aspects of cystine, xanthine and oxalate lithiasis and how diet, in association with metabolic disorders, interferes in uric acid and oxalate stone formation. Finally, we report on the roles of urinary tract malf…

medicine.medical_specialtyDrug-Related Side Effects and Adverse ReactionsUrologyUrinary systemHypercalciuriaCystineUrologyHyperuricemiaUrinenKidneyXanthineOxalatechemistry.chemical_compoundUrolithiasisHumansMedicineHypercalciuriaCalcium metabolismHyperoxaluriaCystinuriabusiness.industryOxalic AcidXanthinemedicine.diseaseUric AcidchemistryUrogenital AbnormalitiesUrinary Tract InfectionsCystineUric acidCalciumbusiness
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Hypercalciuria and kidney calcifications in terminal 4q deletion syndrome: Further evidence for a putative gene on 4q

2003

We report a newborn girl with a de novo terminal 4q deletion (q31.3 → qter) and a characteristic phenotype of minor facial anomalies, cleft palate, congenital heart defect, abnormalities of hands and feet, and postnatal onset of growth deficiency. Laboratory studies showed excessive urinary calcium excretion on standard milk formula and on oral calcium load. Blood measurements of parathyroid hormone, calcitonin, bicarbonate, calcium, phosphorus, magnesium, sodium, chlorine, potassium, and urinary measurements of phosphorus, magnesium, sodium, chlorine, potassium were normal for age. At 2 months of life, ultrasonography showed kidney calcifications. Clinical and laboratory data support the d…

medicine.medical_specialtyKidneybusiness.industryUrinary systemParathyroid hormonechemistry.chemical_elementCalciummedicine.diseaseUrinary calciumEndocrinologymedicine.anatomical_structurechemistryCalcitoninInternal medicineGeneticsmedicineHypercalciuriabusinessGenetics (clinical)CalcificationAmerican Journal of Medical Genetics Part A
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