Search results for "hyperhomocysteinemia"

showing 10 items of 33 documents

The risk of recurrent cardiovascular events in patients with increased plasma homocysteine levels is reduced by short but not long-term therapy with …

2006

Hyperhomocysteinemia is considered an independent risk factor for atherosclerosis, atherothrombosis and Venous ThromboEmbolism (VTE) [1], [2] and [3]. Normal total plasma homocysteine concentrations range from 5 to 15 μmol/L in the fasting state. Hyperhomocysteinemia is classified as moderate (homocysteine concentration, 15 to 30 μmol/L), intermediate (> 30 to 100 μmol/L), and severe (> 100 μmol/L) on the basis of concentrations measured during fasting. Although severe hyperhomocysteinemia is rare, mild hyperhomocysteinemia occurs in approximately 5% to 7% of the general population. In this case, patients are typically asymptomatic until the third or fourth decade of life when premature Cor…

Malecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyHyperhomocysteinemiaTime FactorsHomocysteinePopulationHyperhomocysteinemiaGastroenterologyAsymptomaticchemistry.chemical_compoundFolic AcidRecurrenceRisk FactorsInternal medicineMedicineHumansIn patientRisk factoreducationHomocysteineAgededucation.field_of_studybusiness.industrynutritional and metabolic diseasesHematologyMiddle Agedmedicine.diseaseB vitaminsEndocrinologyTreatment OutcomechemistryCardiovascular DiseasesVitamin B ComplexPlasma homocysteineFemalemedicine.symptombusinessThrombosis research
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Asymmetric dimethylarginine (ADMA) and hyperhomocysteinemia in patients with acute myocardial infarction

2006

We sought to investigate the association between increased levels of asymmetric dimethylarginine (ADMA), an endogenous nitric oxide synthase inhibitor, and total plasma homocysteinemia (tHcy) in patients with acute myocardial infarction (AMI).In 138 patients hospitalized for AMI24 h on admission, serum levels of ADMA, its symmetric stereoisomer (SDMA) and tHcy were measured.ADMA was positively associated with SDMA (p0.001) and tHcy (p=0.03) but not with estimated glomerular filtration rates (eGFR, p=0.96), while tHcy strongly correlated with eGFR (p=0.002) and SDMA (p0.001). By multiple linear regression, SDMA but not ADMA was independently associated with tHcy (p=0.005).Our findings sugges…

Malemedicine.medical_specialtyHyperhomocysteinemiaHomocysteineClinical BiochemistryCardiovascular risk factorsHyperhomocysteinemiaMyocardial InfarctionRenal functionArginineStatistics Nonparametricchemistry.chemical_compoundRisk FactorsInternal medicinemedicineHumansIn patientcardiovascular diseasesMyocardial infarctionHomocysteineAgedbusiness.industryStereoisomerismGeneral MedicineMiddle Agedmedicine.diseaseEndocrinologychemistryRisk indicatorCardiologyRegression AnalysisFemalebusinessAsymmetric dimethylarginineClinical Biochemistry
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The aminothiol redox status in haemodialysis patients does not improve with folate therapy.

2008

Patients on haemodialysis suffer from high cardiovascular morbidity and mortality, and oxidative stress may play a role in the pathophysiology of cardiovascular disease in these patients. Hyperhomocysteinemia is common in dialysis patients and may have pro-oxidant effects. Moreover, the redox status of the major plasma aminothiols (homocysteine [Hcy], cysteine and cysteinylglycine) may be regarded as a biomarker of oxidative stress. In the present study, we investigated the aminothiol redox status during a period of homocysteine-lowering therapy with folinic acid.In the first part of the study, 32 stable patients receiving maintenance haemodialysis were compared with 32 reference subjects. …

Malemedicine.medical_specialtyHyperhomocysteinemiaHomocysteineClinical Biochemistrymedicine.disease_causeGastroenterologychemistry.chemical_compoundFolinic acidFolic AcidRenal DialysisInternal medicinemedicineHumansSulfhydryl CompoundsAgedbusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseRedox statusPathophysiologySurgerychemistryBiomarker (medicine)FemalebusinessOxidation-ReductionOxidative stressCysteinemedicine.drugScandinavian journal of clinical and laboratory investigation
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Blood levels of homocysteine, cysteine, glutathione, folic acid, and vitamin B12 in the acute phase of atherothrombotic stroke.

2009

Blood levels of total homocysteine (tHcy), cysteine (Cys), total and reduced glutathione (tGSH and rGSH), folic acid (FA), and vitamin B(12) (B(12) change during ischemic stroke as accompaniment of the tissue damage. The relationship between these changes remains scantly investigated. We evaluated the variation of these molecules in the 48 h after acute large artery atherothrombotic stroke (LAAS) and searched for the presence of matched variation of them. The study involved 50 subjects affected by acute LAAS and 49 healthy controls. Plasma levels of tHcy and Cys were significantly higher and serum levels of FA and B(12) and plasma levels of rGSH were significantly lower in the patients than…

Malemedicine.medical_specialtyLarge artery atherothrombotic strokeHomocysteineAtherothrombotic strokeHyperhomocysteinemiaDermatologyBrain Ischemiachemistry.chemical_compoundFolic AcidRisk FactorsSettore BIO/10 - BiochimicaInternal medicinemedicineHumansVitamin B12CysteineVitamin B(12)HomocysteineAgedAged 80 and overbusiness.industryLarge arteryGeneral MedicineGlutathionePlasma levelsVitaminsMiddle AgedGlutathioneReduced glutathioneStrokePsychiatry and Mental healthVitamin B 12EndocrinologychemistryFolic acidBiochemistryAcute DiseaseSettore MED/26 - NeurologiaFemaleNeurology (clinical)Intracranial ThrombosisbusinessCysteineNeurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology
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The methylenetetrahydrofolate reductase C677T polymorphism and the risk of congenital heart diseases: a literature review

2014

Congenital Heart Diseases (CHDs) are the most commonand serious developmental anomaly and the leading non-infectious cause of mortality in the first year of life. Despite the advances in diagnosis and treatment, understanding of the developmental causes and aetiologies of CHDs has been limited. The hyperhomocysteinemia is one of the proved risk factors related to the occurrence of CHDs. The connection between cardiac defects, folate and hyperhomocysteinemia could be explained by a mutation in the methylenetetrahydrofolate reductase (MTHFR) gene. Indeed, the C677T MTHFR mutation produces a thermolabile variant of MTHFR with reduced enzymatic action resulting in higher plasma levels of homocy…

Methylenetetrahydrofolato reductasebiologyFolic acidC677T MTHFR mutationbusiness.industryHyperhomocysteinemiaBioinformaticsSettore MED/40 - Ginecologia E OstetriciaBirth defectMTHFR polymorphismMethylenetetrahydrofolate reductaseMTHFRbiology.proteinMthfr c677tMedicinebusinessCongenital heart disease
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A simple high-performance liquid chromatography (HPLC) method for the measurement of pyridoxal-5-phosphate and 4-pyridoxic acid in human plasma.

2014

Abstract Background Low concentration of plasma pyridoxal-5-phosphate (PLP) is associated with hyperhomocysteinemia and inflammation. Most methods for the measurement of plasma PLP require large specimen volume and involve the use of toxic reagents. Methods We have developed a HPLC method for the measurement of PLP and 4-pyridoxic acid (4-PA) in plasma, which requires small specimen volume. The samples are prepared without adding any toxic reagents. Furthermore, we have examined whether intake of vitamin B 6 affects the concentration of plasma PLP and 4-PA. Results The coefficient of variation of the method was 6% and the recovery of the added vitamin in plasma was about 100%. The concentra…

VitaminVitamin bAdultMaleHyperhomocysteinemiaPyridoxal 5-PhosphatePyridoxic AcidAdolescentCoefficient of variationClinical BiochemistryBiochemistryHigh-performance liquid chromatographychemistry.chemical_compoundYoung AdultmedicineHumansHplc methodChromatography High Pressure LiquidAgedChromatographyBiochemistry (medical)General MedicineMiddle Agedmedicine.diseaseHealthy VolunteerschemistryPyridoxal PhosphateCalibrationlipids (amino acids peptides and proteins)FemaleBlood Chemical AnalysisPyridoxic AcidClinica chimica acta; international journal of clinical chemistry
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Folate deficiencies and cardiovascular pathologies.

1998

Abstract Although folates are widely distributed in foods, folate deficiencies may be more frequent than expected because their true availability may be impaired due to their lability under various food cooking and processing conditions. Folate deficiency is frequently observed in elderly people, smokers, alcoholics and oral contraceptive users. It is also associated with the mutation leading to the thermolabile variant of N5, 10-methylenetetrahydrofolate reductase which is observed in about 10 % of the population. In addition to the essential role of the intracellular pool of polyglutamates in de novo biosynthesis of deoxyribonucleotides which allow cell growth and division, the reduced an…

Vitaminmedicine.medical_specialtyHyperhomocysteinemiaHomocysteineClinical BiochemistryPopulationDiseaseReductaseFolic Acid Deficiencychemistry.chemical_compoundFolic AcidInternal medicineMedicineHumansRisk factoreducationHomocysteineeducation.field_of_studyMethionineMolecular Structurebusiness.industryBiochemistry (medical)General Medicinemedicine.diseaseEndocrinologychemistryCardiovascular DiseasesbusinessClinical chemistry and laboratory medicine
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Safety of Dietary Guanidinoacetic Acid: A Villain of a Good Guy?

2021

Guanidinoacetic acid (GAA) is a natural amino acid derivative that is well-recognized for its central role in the biosynthesis of creatine, an essential compound involved in cellular energy metabolism. GAA (also known as glycocyamine or betacyamine) has been investigated as an energy-boosting dietary supplement in humans for more than 70 years. GAA is suggested to effectively increase low levels of tissue creatine and improve clinical features of cardiometabolic and neurological diseases, with GAA often outcompeting traditional bioenergetics agents in maintaining ATP status during stress. This perhaps happens due to a favorable delivery of GAA through specific membrane transporters (such as…

congenital hereditary and neonatal diseases and abnormalitiesNutrition and DieteticsNutrition. Foods and food supplyGlycinetoxicitynutritional and metabolic diseasesReviewMCDARisk AssessmentVDP::Medisinske Fag: 700::Helsefag: 800::Ernæring: 811creatineDietary SupplementsneuromodulationAnimalsHumansTX341-641methylationEnergy MetabolismhyperhomocysteinemiaHomocysteineAgedFood ScienceNutrients
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Genetic variants in the MTHFR are not associated with fatty liver disease.

2020

The common missense sequence variants of methylenetetrahydrofolate reductase (MTHFR), rs1801131 (c.A1298C) and rs1801133 (c.C677T), favour the development of hyperhomocysteinemia and diminished DNA methylation. Previous studies, carried out in small series and with suboptimal characterization of the hepatic phenotype, tested the association of these genetic variants with fatty liver disease (FLD), with conflicting results. Here, we assessed the association of rs1801131 and rs1801133 with hepatic phenotype in the Liver Biopsy Cross-Sectional Cohort, a large cohort (n=1375 from Italy and 411 from Finland) of European individuals with suspect FLD associated with dysmetabolism. A total of 1786 …

medicine.medical_specialtyHyperhomocysteinemiaGenotypeGastroenterologyPolymorphism Single Nucleotide03 medical and health sciences0302 clinical medicineNAFLDInternal medicinesteatosisMedicineMissense mutationHumansGenetic Predisposition to DiseaseFinlandMethylenetetrahydrofolate Reductase (NADPH2)Hepatologymedicine.diagnostic_testbiologybusiness.industryFatty liverNASHmedicine.diseaseFatty LiverCross-Sectional StudiesItaly030220 oncology & carcinogenesisLiver biopsyMethylenetetrahydrofolate reductaseCase-Control StudiesMTHFRDNA methylationCohortbiology.proteinfatty liver disease030211 gastroenterology & hepatologySteatosisfibrosibusinessLiver international : official journal of the International Association for the Study of the LiverREFERENCES
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Therapeutical approach to plasma homocysteine and cardiovascular risk reduction

2008

Marcello Ciaccio, Giulia Bivona, Chiara BelliaDepartment of Medical Biotechnologies and Forensic Medicine, Faculty of Medicine, University of Palermo, ItalyAbstract: Homocysteine is a sulfur-containing aminoacid produced during metabolism of methionine. Since 1969 the relationship between altered homocysteine metabolism and both coronary and peripheral atherotrombosis is known; in recent years experimental evidences have shown that elevated plasma levels of homocysteine are associated with an increased risk of atherosclerosis and cardiovascular ischemic events. Several mechanisms by which elevated homocysteine impairs vascular function have been proposed, including impairment of endothelial…

medicine.medical_specialtyHyperhomocysteinemiaHomocysteineReviewRM1-950folatehomocysteine MTHFR cardiovascular disease folate B vitaminchemistry.chemical_compoundcardiovascular diseaseInternal medicineMedicinePharmacology (medical)General Pharmacology Toxicology and PharmaceuticsEndothelial dysfunctionchemistry.chemical_classificationReactive oxygen speciesChemical Health and SafetyMethioninebiologybusiness.industryhomocysteineGeneral MedicineMetabolismmedicine.diseaseB vitaminB vitaminsEndocrinologychemistryMethylenetetrahydrofolate reductaseMTHFRbiology.proteinTherapeutics. PharmacologybusinessSafety ResearchTherapeutics and Clinical Risk Management
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