Search results for "hypoxia-inducible factor"

showing 10 items of 83 documents

Hypoxia-induced epithelial VEGF-C/VEGFR-3 upregulation in carcinoma cell lines

2008

Adaptation to hypoxia, a universal hallmark of carcinomas, is a critical step for tumor cell survival and growth. One of the principal regulators of hypoxia-responsive pathways is the transcription factor hypoxia-inducible factor-1 alpha (HIF-1 alpha). Currently, it is known that tumoral production of members of the vascular endothelial growth factor (VEGF)-family (VEGFs) may promote tumor growth and progression by acting on carcinoma cells that express the cognate receptors (VEGFRs). However, the influence of hypoxia in the formation of such a tumoral VEGF/VEGFR loop is not completely understood. In the present study we examined the potential existence of a HIF-1 alpha/VEGF/VEGFR autocrine…

Cancer Researchmedicine.medical_specialtyLung NeoplasmsVascular Endothelial Growth Factor CCellBreast NeoplasmsBiologychemistry.chemical_compoundDownregulation and upregulationCell Line TumorInternal medicinemedicineHumansAutocrine signallingVascular Endothelial Growth Factor Receptor-1CarcinomaKinase insert domain receptorCell cycleHypoxia-Inducible Factor 1 alpha SubunitVascular Endothelial Growth Factor Receptor-3Vascular Endothelial Growth Factor Receptor-2Cell HypoxiaUp-RegulationGene Expression Regulation NeoplasticVascular endothelial growth factorAutocrine CommunicationHIF1AEndocrinologymedicine.anatomical_structureOncologyVascular endothelial growth factor CchemistryCancer researchColorectal NeoplasmsInternational Journal of Oncology
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Haem oxygenase-1 induction reverses the actions of interleukin-1β on hypoxia-inducible transcription factors and human chondrocyte metabolism in hypo…

2013

HO-1 (haem oxygenase-1) catalyses the degradation of haem and possesses anti-inflammatory and cytoprotective properties. The role of inflammatory mediators in the pathogenesis of OA (osteoarthritis) is becoming increasingly appreciated. In the present study, we investigated the effects of HO-1 induction in OA and healthy HACs (human articular chondrocytes) in response to inflammatory cytokine IL-1 β (interleukin-1β) under hypoxic conditions. Hypoxia was investigated as it is a more physiological condition of the avascular cartilage. Hypoxic signalling is mediated by HIFs (hypoxia-inducible factors), of which there are two main isoforms, HIF-1α and HIF-2α. Normal and OA chondrocytes were sti…

Cartilage ArticularMaleSmall interfering RNAmedicine.medical_treatmentInterleukin-1betaBiologyMatrix metalloproteinaseChondrocytesOsteoarthritisBasic Helix-Loop-Helix Transcription FactorsmedicineHumansHypoxiaCollagen Type IITranscription factorAgedTumor Necrosis Factor-alphaCatabolismSOX9 Transcription FactorGeneral MedicineMiddle AgedHypoxia (medical)Hypoxia-Inducible Factor 1 alpha SubunitCOPPMatrix MetalloproteinasesCell biologyCytokineBiochemistryFemaleTumor necrosis factor alphamedicine.symptomHeme Oxygenase-1Clinical Science
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Hypoxia and HIF Signaling: One Axis with Divergent Effects

2020

The correct concentration of oxygen in all tissues is a hallmark of cellular wellness, and the negative regulation of oxygen homeostasis is able to affect the cells and tissues of the whole organism. The cellular response to hypoxia is characterized by the activation of multiple genes involved in many biological processes. Among them, hypoxia-inducible factor (HIF) represents the master regulator of the hypoxia response. The active heterodimeric complex HIF α/β, binding to hypoxia-responsive elements (HREs), determines the induction of at least 100 target genes to restore tissue homeostasis. A growing body of evidence demonstrates that hypoxia signaling can act by generating contrasting res…

CellInflammationReviewBiologyCatalysislcsh:ChemistryInorganic ChemistryImmune systemSettore BIO/13 - Biologia ApplicataOxygen homeostasisBasic Helix-Loop-Helix Transcription FactorsmedicineHumansRNA MessengerAcute and chronic diseasesPhysical and Theoretical ChemistryHypoxialcsh:QH301-705.5Molecular BiologySpectroscopyTissue homeostasisInflammationKidneyImmune cellsOrganic ChemistryHIF-αNuclear ProteinsGeneral MedicineHypoxia (medical)Cell HypoxiaComputer Science ApplicationsCell biologyDNA-Binding ProteinsOxygenmedicine.anatomical_structurelcsh:Biology (General)lcsh:QD1-999Hypoxia-Inducible Factor 1medicine.symptomSignal transductionSignal TransductionTranscription FactorsInternational Journal of Molecular Sciences
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Hypoxia: Importance in tumor biology, noninvasive measurement by imaging, and value of its measurement in the management of cancer therapy

2006

The Cancer Imaging Program of the National Cancer Institute convened a workshop to assess the current status of hypoxia imaging, to assess what is known about the biology of hypoxia as it relates to cancer and cancer therapy, and to define clinical scenarios in which in vivo hypoxia imaging could prove valuable.Hypoxia, or low oxygenation, has emerged as an important factor in tumor biology and response to cancer treatment. It has been correlated with angiogenesis, tumor aggressiveness, local recurrence, and metastasis, and it appears to be a prognostic factor for several cancers, including those of the cervix, head and neck, prostate, pancreas, and brain. The relationship between tumor oxy…

Diagnostic ImagingOncologymedicine.medical_specialtyCancer therapyBiologyAntigens NeoplasmNeoplasmsInternal medicineAntineoplastic Combined Chemotherapy ProtocolsBiomarkers TumormedicineHumansRadiology Nuclear Medicine and imagingMedical physicsCarbonic Anhydrase IXHypoxiaCarbonic AnhydrasesCancer Imaging ProgramRadiological and Ultrasound TechnologyTumor biologyReproducibility of ResultsHypoxia (medical)PrognosisUnited StatesCancer treatmentIsoenzymesOxygenRadiographyNational Institutes of Health (U.S.)Hypoxia-Inducible Factor 1medicine.symptomInternational Journal of Radiation Biology
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HIF-1α induces MXI1 by alternate promoter usage in human neuroblastoma cells

2009

Adaptation to low oxygen conditions is essential for maintaining homeostasis and viability in oxygen-consuming multi-cellular tissues, including solid tumors. Central in these processes are the hypoxia-inducible transcription factors, HIF-1 and HIF-2, controlling genes involved in e.g. glucose metabolism and neovascularization. Tumor hypoxia and HIF expression have also been associated with a dedifferentiated phenotype and increased aggressiveness. In this report we show that the MAX interactor-1 (MXI1) gene is directly regulated by HIF proteins in neuroblastoma and breast cancer cells. HIF-binding and transactivation were detected within MXI1 gene regulatory sequences in the vicinity of th…

Gene isoformGenes mycBreast NeoplasmsBiologyTransfectionNeuroblastomaTransactivationCell Line TumorNeuroblastomaBasic Helix-Loop-Helix Transcription FactorsmedicineHumansGenes Tumor SuppressorRNA Small InterferingPromoter Regions GeneticGeneTranscription factorOligonucleotide Array Sequence AnalysisBase SequenceTumor hypoxiaTumor Suppressor ProteinsCell BiologyHypoxia-Inducible Factor 1 alpha Subunitmedicine.diseaseCell HypoxiaUp-RegulationGene Expression Regulation NeoplasticHIF1ARegulatory sequenceCancer researchFemaleExperimental Cell Research
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Hypoxia and anemia: effects on tumor biology and treatment resistance

2004

In locally advanced solid tumors, oxygen (O2) delivery is frequently reduced or even abolished. This is due to abnormalities of the tumor microvasculature, adverse diffusion geometries, and tumor-associated and/or therapy-induced anemia. Up to 50-60% of locally advanced solid tumors may exhibit hypoxic and/or anoxic tissue areas that are heterogeneously distributed within the tumor mass. In approximately 30% of pretreatment patients, a decreased O2 transport capacity of the blood as a result of tumor-associated anemia can greatly contribute to the development of tumor hypoxia. While normal tissues can compensate for this O2 deficiency status by a rise in blood flow rate, locally advanced tu…

Genome instabilityAnemiaClinical BiochemistryDrug resistanceBiologyRadiation ToleranceNeoplasmsmedicineHumansHypoxiaRegulation of gene expressionTumor hypoxiaBiochemistry (medical)NF-kappa BNuclear ProteinsAnemiaHematologyHypoxia (medical)Hypoxia-Inducible Factor 1 alpha SubunitPrognosismedicine.diseaseNeoplasm ProteinsDNA-Binding ProteinsGene Expression Regulation NeoplasticOxygenHypoxia-inducible factorsDrug Resistance NeoplasmTumor progressionImmunologyDisease ProgressionCancer researchHypoxia-Inducible Factor 1medicine.symptomCell DivisionTranscription FactorsTransfusion Clinique et Biologique
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Hypoxia inducible factor-1 alpha expression is increased in infected positive HPV16 DNA oral squamous cell carcinoma and positively associated with H…

2011

Abstract Background There is increasing evidence for the role of High Risk (HR) Human PapillomaVirus (HPV) in the pathogenesis of Oral Squamous Cell Carcinoma (OSCC). The E6 and E7 oncogenes from HR HPVs are responsible for the deregulation of p53 and pRB proteins involved in cell cycle and apoptotic pathways. In cell lines experiments, the HPV E7 protein seems to be able to enhance Hypoxia Inducible Factor-1 alpha (HIF-1α) activity, normally involved in the response to hypoxia and able to enhance angiogenesis. Results We studied tumor specimens from 62 OSCC; a higher prevalence of tumors in TNM stage II and also in pT2 class between OSCC infected positive HPV16 DNA than non-infected ones w…

HPV16Cancer ResearchPathologymedicine.medical_specialtyHPVNecrosisAngiogenesisEpidemiologyAlpha (ethology)BiologyOral Squamous Cell Carcinomalcsh:RC254-282lcsh:Infectious and parasitic diseasesPathogenesisHypoxia-Inducible Factor 1-Alphachemistry.chemical_compoundmedicinelcsh:RC109-216E7Oral Squamous Cell Carcinoma Hif-1α HPV HPV16 E7virus diseasesHypoxia (medical)lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensfemale genital diseases and pregnancy complicationsstomatognathic diseasesInfectious DiseaseschemistryOncologyApoptosisCancer researchmedicine.symptomHif-1αDNAResearch ArticleInfectious agents and cancer
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Respiratory plasticity in response to changes in oxygen supply and demand

2011

Aerobic organisms maintain O(2) homeostasis by responding to changes in O(2) supply and demand in both short and long time domains. In this review, we introduce several specific examples of respiratory plasticity induced by chronic changes in O(2) supply (environmental hypoxia or hyperoxia) and demand (exercise-induced and temperature-induced changes in aerobic metabolism). These studies reveal that plasticity occurs throughout the respiratory system, including modifications to the gas exchanger, respiratory pigments, respiratory muscles, and the neural control systems responsible for ventilating the gas exchanger. While some of these responses appear appropriate (e.g., increases in lung su…

Hyperoxia0303 health sciencesOxygen supplyCellular respirationPlant SciencePlasticityBiologyLung surface03 medical and health sciences0302 clinical medicineHypoxia-inducible factorsImmunologymedicineAnimal Science and Zoologymedicine.symptomRespiratory systemNeuroscience030217 neurology & neurosurgeryHomeostasis030304 developmental biologyIntegrative and Comparative Biology
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IS HYPOXIA-INDUCIBLE FACTOR (HIF-1) ALPHA INVOLVED IN TARAVANA SYNDROME?

2021

Breath holding diving (BH) is a discipline practiced by an increasing number of people. BH-divers are exposed to extreme environmental conditions such as: increased hyperbaric pressure and low temperature that caused change in arterial blood gases (1-2) and induced an human diving response which includes bradycardia, reduced cardiac output, increased arterial blood pressure and peripheral vasoconstriction (3). Some of these adaptations are still not fully understood, so we decided to investigate some biomarkers that should be involved in this disorders starting to analyse Hypoxia-inducible factor 1 alpha (HIF-1α). We enrolled in our study 31 healthy free divers from different part of Sicily…

Hypoxia-inducible factor 1Breath-holding divingsporthyperbaric pressure
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Increased expression of leptin and the leptin receptor as a marker of breast cancer progression: possible role of obesity-related stimuli.

2006

Abstract Purpose: Recent in vitro studies suggested that the autocrine leptin loop might contribute to breast cancer development by enhancing cell growth and survival. To evaluate whether the leptin system could become a target in breast cancer therapy, we examined the expression of leptin and its receptor (ObR) in primary and metastatic breast cancer and noncancer mammary epithelium. We also studied whether the expression of leptin/ObR in breast cancer can be induced by obesity-related stimuli, such as elevated levels of insulin, insulin-like growth factor-I (IGF-I), estradiol, or hypoxic conditions. Experimental Design: The expression of leptin and ObR was examined by immunohistochemistry…

LeptinCancer ResearchER-BETAmedicine.medical_treatmentHYPOXIA-INDUCIBLE FACTOR-1NeoplasmsTumor Cells CulturedBreastInsulin-Like Growth Factor Iskin and connective tissue diseasesReceptorAged 80 and overEstradiolIGF-I RECEPTORCELL-LINEReverse Transcriptase Polymerase Chain ReactionLeptindigestive oral and skin physiologyCarcinoma Ductal BreastMiddle AgedMetastatic breast cancerINSULINCell HypoxiaESTROGENOncologyDisease ProgressionIGF-I RECEPTOR; HYPOXIA-INDUCIBLE FACTOR-1; OB GENE; GROWTH-FACTOR; CELL-LINE; ER-BETA; ESTROGEN; ALPHA; INSULIN; MCF-7Receptors LeptinFemaleOB GENEhormones hormone substitutes and hormone antagonistsAdultmedicine.medical_specialtyGROWTH-FACTORBreast NeoplasmsReceptors Cell SurfaceBreast cancerInternal medicinemedicineBiomarkers TumorHumansObesityAutocrine signallingAgedLeptin receptorbusiness.industryInsulinmedicine.diseaseALPHAEndocrinologyTumor progressionCase-Control StudiesMCF-7business
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