Search results for "hypoxic"

showing 10 items of 42 documents

Wnt-Dependent Oligodendroglial-Endothelial Interactions Regulate White Matter Vascularization and Attenuate Injury

2020

Recent studies have indicated oligodendroglial-vascular crosstalk during brain development, but the underlying mechanisms are incompletely understood. We report that oligodendrocyte precursor cells (OPCs) contact sprouting endothelial tip cells in mouse, ferret and human neonatal white matter. Using transgenic mice, we show that increased or decreased OPC density results in cognate changes in white matter vascular investment. Hypoxia promoted both increased OPC numbers and higher white matter vessel density, and endothelial cell expression of the Wnt pathway targets Apcdd1 and Axin2, suggesting paracrine OPC-endothelial signaling. Conditional knockout of OPC Wntless resulted in diminished w…

0301 basic medicineGenetically modified mouseoligodendrocytesMice TransgenicBiologyArticleWhite matter03 medical and health sciencesParacrine signallingMice0302 clinical medicinetip cell angiogenesisAxin ProteinConditional gene knockoutmedicineAXIN2AnimalsHumanshypoxic-ischemic encephalopathyHypoxiaWnt Signaling PathwayGeneral NeuroscienceWnt signaling pathwayFerretsIntracellular Signaling Peptides and ProteinsEndothelial CellsMembrane ProteinsCell DifferentiationHypoxia (medical)Wnt signalingWhite Matter3. Good healthCell biologyEndothelial stem cellstomatognathic diseasesOligodendroglia030104 developmental biologymedicine.anatomical_structurenervous systemEndothelium Vascularmedicine.symptom030217 neurology & neurosurgeryNeuron
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Methods for assessing the severity of perinatal asphyxia and early prognostic tools in neonates with hypoxic-ischemic encephalopathy treated with the…

2020

Despite the progress in perinatal care, perinatal asphyxia (PA) remains a significant problem in neonatology. The development of therapeutic hypothermia (TH) has improved the prognosis, but it still remains uncertain in hypoxic neonates. The evaluation of the severity of ischemia/hypoxia after birth is crucial to the choice of treatment, and with accurate long-term prognosis, appropriate further patient care can be planned. This article presents various methods for the preliminary assessment of brain damage and prognosis in newborns with PA treated with TH. The importance of assessing the neurological condition and the usefulness of laboratory and electrophysiological testing and imaging ar…

030213 general clinical medicinemedicine.medical_specialtyEncephalopathyIschemiaprediction factorsMedicine (miscellaneous)therapeutic hypothermiaBrain damageGeneral Biochemistry Genetics and Molecular BiologyHypoxic Ischemic Encephalopathy03 medical and health sciencesAsphyxia0302 clinical medicineHypothermia InducedInternal MedicinemedicineHumansPharmacology (medical)NeonatologyIntensive care medicineperinatal asphyxiaGenetics (clinical)Asphyxia Neonatorumbusiness.industryInfant NewbornInfantHypoxia (medical)medicine.diseasePrognosishypoxic–ischemic encephalopathyPerinatal asphyxiaClinical trialReviews and References (medical)Hypoxia-Ischemia Brainneonatemedicine.symptombusinessAdvances in clinical and experimental medicine : official organ Wroclaw Medical University
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Role of microglia in blood pressure and respiratory responses to acute hypoxic exposure in rats

2021

AbstractMicroglia modulate cardiorespiratory activities during chronic hypoxia. It has not been clarified whether microglia are involved in the cardiorespiratory responses to acute hypoxia. Here we investigated this issue by comparing cardiorespiratory responses to two levels of acute hypoxia (13% O2 for 4 min and 7% O2 for 5 min) in conscious unrestrained rats before and after systemic injection of minocycline (MINO), an inhibitor of microglia activation. MINO increased blood pressure but not lung ventilation in the control normoxic condition. Acute hypoxia stimulated cardiorespiratory responses in MINO-untreated rats. MINO failed to significantly affect the magnitude of hypoxia-induced bl…

Acute hypoxiaPhysiologyHypoxic ventilatory responseBlood pressure telemetryAnimalsBlood PressureMinocyclineMicrogliaCardiorespiratory regulationHypoxiaLungRatsJournal of Physiological Sciences
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11,12-EET Stimulates the Association of BK Channel α and β1 Subunits in Mitochondria to Induce Pulmonary Vasoconstriction

2012

In the systemic circulation, 11,12-epoxyeicosatrienoic acid (11,12-EET) elicits nitric oxide (NO)- and prostacyclin-independent vascular relaxation, partially through the activation of large conductance Ca(2+)-activated potassium (BK) channels. However, in the lung 11,12-EET contributes to hypoxia-induced pulmonary vasoconstriction. Since pulmonary artery smooth muscle cells also express BK channels, we assessed the consequences of BKβ(1) subunit deletion on pulmonary responsiveness to 11,12-EET as well as to acute hypoxia. In buffer-perfused mouse lungs, hypoxia increased pulmonary artery pressure and this was significantly enhanced in the presence of NO synthase (NOS) and cyclooxygenase (…

BK channelAnatomy and PhysiologyLarge-Conductance Calcium-Activated Potassium Channel beta SubunitsRespiratory Systemlcsh:MedicineCardiovascularCardiovascular SystemBiochemistryIon ChannelsMembrane PotentialsMice81114-Eicosatrienoic AcidHypoxic pulmonary vasoconstrictionHypoxiaLarge-Conductance Calcium-Activated Potassium Channel alpha Subunitslcsh:ScienceLungEnergy-Producing OrganellesEpoxide HydrolasesMembrane Potential MitochondrialMembrane potentialMultidisciplinarybiologyChemistryDepolarizationHyperpolarization (biology)IberiotoxinMitochondriaBiochemistryCirculatory Physiologycardiovascular systemMedicinelipids (amino acids peptides and proteins)medicine.symptomResearch ArticleCell Physiologymedicine.medical_specialtyPulmonary ArteryBioenergeticsCardiovascular PharmacologyInternal medicinemedicineAnimalsHumansArterial Pressureddc:610Protein InteractionsBiologylcsh:RProteinsCalcium-activated potassium channelMice Inbred C57BLHEK293 CellsEndocrinologyVasoconstrictionbiology.proteinlcsh:QGene DeletionVasoconstrictionPLoS ONE
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Antioxidant treatment for impaired hypoxic ventilatory responses in experimental diabetes in the rat

2018

Inflammation, tissue hypoxia, and impaired hypoxic ventilatory response (HVR) are the intricately entwined features of diabetes which perpetuate the disease and its sequelae. Hyperglycemia, notably, is an oxygen consuming process due to enhanced cellular metabolism. Oxidative stress underlies diabetic pathogenesis and also is a crucial modulator of the hypoxic chemoreflex. The present study seeks to determine if suppressed ventilation in diabetes could be improved by antioxidant treatment. The study was performed in streptozotocin-induced diabetes in awake rats. Two weeks into full-fledged diabetes, the rats were divided into mangiferin (potent natural antioxidant)-treated and untreated, wi…

Blood GlucoseMale0301 basic medicinePulmonary and Respiratory MedicineAntioxidantPhysiologyXanthonesmedicine.medical_treatmentInflammationHypoxic ventilatory responsePharmacologymedicine.disease_causeThiobarbituric Acid Reactive SubstancesAntioxidantsDiabetes Mellitus ExperimentalSuperoxide dismutase03 medical and health scienceschemistry.chemical_compoundAntioxidant treatment; Diabetes; Hypoxic ventilatory response; Inflammation; Mangiferin; Oxidative stress.Diabetes mellitusmedicineAnimalsRats WistarHypoxiaMangiferinInflammationbiologySuperoxide DismutaseTumor Necrosis Factor-alphabusiness.industryRespirationGeneral NeuroscienceBrainmedicine.diseaseOxidative Stress030104 developmental biologychemistrybiology.proteinTumor necrosis factor alphaLipid Peroxidationmedicine.symptombusinessOxidative stressRespiratory Physiology & Neurobiology
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Ceramide Mediates Acute Oxygen Sensing in Vascular Tissues

2014

AbstractAims: A variety of vessels, such as resistance pulmonary arteries (PA) and fetoplacental arteries and the ductus arteriosus (DA) are specialized in sensing and responding to changes in oxygen tension. Despite opposite stimuli, normoxic DA contraction and hypoxic fetoplacental and PA vasoconstriction share some mechanistic features. Activation of neutral sphingomyelinase (nSMase) and subsequent ceramide production has been involved in hypoxic pulmonary vasoconstriction (HPV). Herein we aimed to study the possible role of nSMase-derived ceramide as a common factor in the acute oxygen-sensing function of specialized vascular tissues. Results: The nSMase inhibitor GW4869 and an anticera…

CeramideContraction (grammar)PhysiologyClinical BiochemistryGene ExpressionChick EmbryoPulmonary ArterySphingomyelin phosphodiesteraseBiologyCeramidesBenzylidene CompoundsBiochemistrychemistry.chemical_compoundHypoxic pulmonary vasoconstrictionmedicineAnimalsHumansHypoxiaMolecular BiologyGeneral Environmental Sciencechemistry.chemical_classificationReactive oxygen speciesAniline CompoundsDuctus ArteriosusCell BiologyCell biologyOxygen tensionOxygenOriginal Research CommunicationsSphingomyelin PhosphodiesteraseShaw Potassium ChannelsBiochemistrychemistryVasoconstrictionBlood VesselsGeneral Earth and Planetary Sciencesmedicine.symptomReactive Oxygen SpeciesSphingomyelinVasoconstrictionAntioxidants & Redox Signaling
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Measurements of Tumor Blood Flow Using Intraperitoneal Deuterium and 2H-NMR Spectroscopy

1992

Tumors usually have a sparse, disorganized, and inefficient vascular network that leaves a large fraction of the tumor cells in an oxygen deprived and hostile metabolic microenvironment. Hence tumor blood flow, or more correctly nutritive perfusion, has important interactions with treatment efficacy. For example, hypoxic tumor cells, which occur in tumors with low blood flow, are less susceptible to radiation and are probably responsible for most radiation treatment failures (Adams, 1981). Similarly, cytotoxic drug delivery could be predicted by blood flow measurements, with clear implications regarding the expected success of chemotherapy. Thus, the ability to conveniently measure tumor bl…

ChemotherapyHypoxic tumorDeuteriumVascular networkCytotoxic drugChemistrymedicine.medical_treatmentmedicineAnalytical chemistryCancer researchNuclear magnetic resonance spectroscopyBlood flowPerfusion
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Influence of Training Load on Mood Disturbance at Sea Level and 3900 m Altitude: A Case Study of a Wheelchair Athlete

2018

The purpose of this case study was to investigate the influence of a training load (TL), oxygen saturation (SO2) and blood pressure (BP) on mood states in a wheelchair marathoner during (7 weeks at sea level (SL), 5 weeks at 3860 m altitude, 1 week returning to SL). TL was obtained with Foster’s equation while mood states were obtained with the Profile of Mood States Questionnaire (POMS). Furthermore, SO2 and BP were assessed upon wakening. SO2 (%) decreased at altitude, compared to SL (88.31 ± 2.46 vs. 98.52 ± 0.11) and increased until the last week at altitude (92.64 ± 1.12). Systolic pressure (SP) increased at altitude compared to pre-altitude (126.0 ± 5…

Esportslcsh:Sportshypoxic environmentbusiness.industryparalympicathleticsPhysical Therapy Sports Therapy and RehabilitationCase ReportProfile of mood statesbaroreflex sensitivitylcsh:GV557-1198.995AltitudeWheelchairBlood pressureAnimal scienceMoodAtletisme EntrenamentMedicineOrthopedics and Sports MedicinePOMSTraining loadbusinessSea levelOxygen saturation (medicine)Sports
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Failure to Sustain Recovery of Na,K ATPase Function Is a Possible Mechanism for Striatal Neuron Necrosis in Hypoxic-Ischemic Newborn Piglets

1999

Failure to Sustain Recovery of Na,K ATPase Function Is a Possible Mechanism for Striatal Neuron Necrosis in Hypoxic-Ischemic Newborn Piglets

Hypoxic ischemicmedicine.medical_specialtyNecrosisintegumentary systembiologyStriatal neuronChemistryMechanism (biology)animal diseasesATPasefluids and secretionsEndocrinologynervous systemAnesthesiaInternal medicinePediatrics Perinatology and Child Healthmedicinebiology.proteinNa+/K+-ATPasemedicine.symptomFunction (biology)Pediatric Research
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Telmisartan cardioprotects from the ischaemic/hypoxic damage through a miR‐1‐dependent pathway

2019

The aim of this study was to investigate whether telmisartan protects the heart from the ischaemia/reperfusion damage through a local microRNA-1 modulation. Studies on the myocardial ischaemia/reperfusion injury in vivo and on the cardiomyocyte hypoxia/reoxygenation damage in vitro were done. In vivo, male Sprague-Dawley rats administered for 3 weeks with telmisartan 12 mg/kg/d by gastric gavage underwent ischaemia/reperfusion of the left descending coronary artery. In these rats, infarct size measurement, ELISA, immunohistochemistry (IHC) and reverse transcriptase real-time polymerase chain reaction showed that expressions of connexin 43, potassium voltage-gated channel subfamily Q member …

Male0301 basic medicineCell SurvivalMyocardial InfarctionIschemiaConnexinMyocardial Reperfusion InjuryPharmacologymiR‐1telmisartanCell Lineconnexin 43Rats Sprague-Dawleyhypoxic H9c2 cells03 medical and health sciences0302 clinical medicineIn vivomedicineAnimalsBcl-2Myocytes CardiacKCNQ1ChemistryBcl‐2Original ArticlesCell BiologyTransfectionHypoxia (medical)medicine.diseasemiR-1Cell HypoxiaIn vitroRatsMicroRNAsmyocardial ischaemia/reperfusion030104 developmental biologyProto-Oncogene Proteins c-bcl-2030220 oncology & carcinogenesisKCNQ1 Potassium ChannelMolecular Medicinehypoxic H9c2 cellOriginal Articlemedicine.symptomTelmisartanReperfusion injurymedicine.drugJournal of Cellular and Molecular Medicine
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