Search results for "immune system"

showing 10 items of 2885 documents

Tumor lymphocyte immune response to preoperative radiotherapy in locally advanced rectal cancer: The LYMPHOREC study

2017

IF 7.719; International audience; Introduction: Some studies have suggested that baseline tumor-infiltrating-lymphocytes (TILs), such as CD8+ and FoxP3+ T-cells, may be associated with a better prognosis in colorectal cancer. We sought to investigate modulation of the immune response by preoperative radiotherapy (preopRT) and its impact on survival in locally advanced rectal cancer (LARC). Materials & Methods: We analyzed data for 237 patients with LARC who received RT. Density of TILS (CD8+ and FoxP3+) in intraepithelial (iTILs) and stromal compartments (sTILs) were evaluated from surgery pathological specimens and biopsies performed at baseline. The primary endpoint was to assess the impa…

lcsh:Immunologic diseases. Allergy0301 basic medicineOncologymedicine.medical_specialtyColorectal cancermedicine.medical_treatmentImmunology[SDV.CAN]Life Sciences [q-bio]/Cancerchemical and pharmacologic phenomenalcsh:RC254-282radiation therapy[ SDV.CAN ] Life Sciences [q-bio]/Cancer03 medical and health sciences0302 clinical medicineInternal medicinemedicineClinical endpointImmunology and Allergyrectal cancertranslational studyOriginal ResearchUnivariate analysisbusiness.industryTumor-infiltrating lymphocytesDose fractionationFOXP3hemic and immune systemslcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.disease3. Good healthRadiation therapy030104 developmental biologyOncology030220 oncology & carcinogenesistumor-infiltrating lymphocyteslcsh:RC581-607businessAdjuvant
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The Metabolic Sensor GPR43 Receptor Plays a Role in the Control of Klebsiella pneumoniae Infection in the Lung

2018

Pneumonia is one of the leading causes of death and mortality worldwide. The inflammatory responses that follow respiratory infections are protective leading to pathogen clearance but can also be deleterious if unregulated. The microbiota is known to be an important protective barrier against infections, mediating both direct inhibitory effects against the potential pathogen and also regulating the immune responses contributing to a proper clearance of the pathogen and return to homeostasis. GPR43 is one receptor for acetate, a microbiota metabolite shown to induce and to regulate important immune functions. Here, we addressed the role of GPR43 signaling during pulmonary bacterial infection…

lcsh:Immunologic diseases. Allergy0301 basic medicinePhagocytosisshort-chain fatty acids030106 microbiologyImmunologyInflammationBiologyGut floraMicrobiology03 medical and health sciencesImmune systemmicrobiotamedicinepneumoniaImmunology and AllergyPathogenLunglung infectionGPR43biology.organism_classificationmedicine.disease030104 developmental biologymedicine.anatomical_structureinflammationIncreased inflammatory responsemedicine.symptomlcsh:RC581-607Pneumonia (non-human)Frontiers in Immunology
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Non-specific Effects of Vaccines Illustrated Through the BCG Example: From Observations to Demonstrations

2018

Epidemiological studies regarding many successful vaccines suggest that vaccination may lead to a reduction in child mortality and morbidity worldwide, on a grander scale than is attributable to protection against the specific target diseases of these vaccines. These non-specific effects (NSEs) of the Bacille Calmette-Guérin (BCG) vaccine, for instance, implicate adaptive and innate immune mechanisms, with recent evidence suggesting that trained immunity might be a key instrument at play. Collectively referring to the memory-like characteristics of innate immune cells, trained immunity stems from epigenetic reprogramming that these innate immune cells undergo following exposure to a primary…

lcsh:Immunologic diseases. Allergy0301 basic medicinemedicine.medical_treatmentImmunologyReviewImmunity HeterologousWorld Health OrganizationEpigenesis Genetictrained immunity03 medical and health sciences0302 clinical medicineImmunityHumansTuberculosisImmunology and AllergyMedicineBCG030212 general & internal medicineEpigeneticsImmunity CellularInnate immune systemepigeneticsbusiness.industryVaccinationvaccinesMycobacterium bovisImmunity InnateChild mortalityVaccination030104 developmental biologyCell metabolismCytokinenon-specific effectsImmunologyBCG Vaccineepidemiologylcsh:RC581-607businessImmunologic MemoryReprogrammingFrontiers in Immunology
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Role of the IL-23/IL-17 Pathway in Rheumatic Diseases: An Overview

2021

Interleukin-23 (IL-23) is a pro-inflammatory cytokine composed of two subunits, IL-23A (p19) and IL-12/23B (p40), the latter shared with Interleukin-12 (IL-12). IL-23 is mainly produced by macrophages and dendritic cells, in response to exogenous or endogenous signals, and drives the differentiation and activation of T helper 17 (Th17) cells with subsequent production of IL-17A, IL-17F, IL-6, IL-22, and tumor necrosis factor α (TNF-α). Although IL-23 plays a pivotal role in the protective immune response to bacterial and fungal infections, its dysregulation has been shown to exacerbate chronic immune-mediated inflammation. Well-established experimental data support the concept that IL-23/IL…

lcsh:Immunologic diseases. Allergy0301 basic medicinemedicine.medical_treatmentImmunologyinflammatory diseasesInflammationautoimmune diseaseAutoimmunityReviewInflammatory bowel diseaseInterleukin-23Th17 CellRheumatic Disease03 medical and health sciencesPsoriatic arthritis0302 clinical medicineImmune systemIL-23PsoriasisRheumatic DiseasesInterleukin 23medicineAnimalsHumansImmunology and Allergyautoimmune diseasesMolecular Targeted TherapyIL-23/IL-17 axi030203 arthritis & rheumatologyInflammationbusiness.industryIL-23/IL-17 axisAnimalInterleukin-17medicine.diseaseinflammatory diseaseIL-17030104 developmental biologyCytokineImmunologyTh17 CellsInterleukin 17medicine.symptomlcsh:RC581-607businessHuman
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Deciphering the Roles of Innate Lymphoid Cells in Cancer

2019

Cancer is a complex disease and the role played by innate lymphoid cells (ILCs) in cancer development has begun to be uncovered over recent years. We aim to provide an exhaustive summary of the knowledge acquired on the role of ILCs in cancer. ILCs are classified into 3 different categories, ILC1s, ILC2s, and ILC3s, each encompassing specific and unique functions. ILC1s exhibit NK cells characteristics and can exert anti-tumor functions, but surprisingly their IFNγ production is not associated with a better immune response. In response to TGF-β or IL-12, ILC1s were shown to exert pro-tumor functions and to favor tumor growth. ILC2s role in cancer immune response is dependent on cytokine con…

lcsh:Immunologic diseases. Allergy0301 basic medicinemedicine.medical_treatmentMDSCImmunologyinnate lymphoid cellsContext (language use)ReviewBiology03 medical and health sciences0302 clinical medicineImmune systemImmunityNeoplasmsEosinophil activationcytokinemedicinecancerAnimalsHumansImmunology and AllergyLymphocytesInnate lymphoid cellCancerImmunotherapymedicine.diseaseImmunity Innate030104 developmental biologyCytokineCancer researchimmunotherapylcsh:RC581-607030215 immunologyFrontiers in Immunology
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Is the Complement Protein C1q a Pro- or Anti-tumorigenic Factor? Bioinformatics Analysis Involving Human Carcinomas

2019

C1q is the first subcomponent of the classical pathway of the complement system and belongs to the C1q/Tumor Necrosis Factor superfamily. C1q can perform a diverse range of immune and non-immune functions in a complement-dependent as well as -independent manner. Being a pattern recognition molecule of the innate immunity, C1q can recognize a number of self, non-self and altered-self ligands and bring about effector mechanisms designed to clear pathogens via opsonisation and inflammatory response. C1q is locally synthesized by macrophages and dendritic cells, and thus, can get involved in a range of biological processes, such as angiogenesis and tissue remodeling, immune modulation, and immu…

lcsh:Immunologic diseases. Allergy0301 basic medicinetumorLung NeoplasmsMicroenvironmentPrognosiImmunologyComplementBreast Neoplasmschemical and pharmacologic phenomenaKaplan-Meier EstimateBiology03 medical and health sciencesClassical complement pathway0302 clinical medicineImmune systemimmune system diseasesmedicineHumansImmunology and Allergycomplementclassical pathwayskin and connective tissue diseasesC1qOriginal ResearchTumorInnate immune systemEffectorComplement C1qComputational BiologyCancerPrognosismedicine.diseasemicroenvironmentKidney NeoplasmsComplement systemClear cell renal cell carcinomaC1q; Classical pathway; Complement; Microenvironment; Prognosis; Tumor030104 developmental biologyClassical pathwayCancer researchAdenocarcinomaprognosislcsh:RC581-607030215 immunologyFrontiers in Immunology
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TLR7 controls VSV replication in CD169(+) SCS macrophages and associated viral neuroinvasion

2019

Vesicular stomatitis virus (VSV) is an insect-transmitted rhabdovirus that is neurovirulent in mice. Upon peripheral VSV infection, CD169+ subcapsular sinus (SCS) macrophages capture VSV in the lymph, support viral replication, and prevent CNS neuroinvasion. To date, the precise mechanisms controlling VSV infection in SCS macrophages remain incompletely understood. Here, we show that Toll-like receptor-7 (TLR7), the main sensing receptor for VSV, is central in controlling lymph-borne VSV infection. Following VSV skin infection, TLR7−/− mice display significantly less VSV titers in the draining lymph nodes (dLN) and viral replication is attenuated in SCS macrophages. In contrast to effects o…

lcsh:Immunologic diseases. Allergy0301 basic medicinevirusesImmunologyMedizinDENDRITIC CELLSRIG-IACTIVATION03 medical and health sciences0302 clinical medicinesubcapsular sinus macrophagesSUBCAPSULAR SINUS MACROPHAGESImmunitySIMULIUM-VITTATUM DIPTERAINFECTIONImmunology and Allergyinnate immunityvirus replicationHost factorconditional knock-out miceInnate immune systemScience & TechnologyLYMPH-NODESbiologysubcutaneous infectionPattern recognition receptorpattern recognition receptorsvirus diseasesTLR7VESICULAR STOMATITIS-VIRUSbiology.organism_classificationVirologyddc:Toll-like receptor 7stomatognathic diseases030104 developmental biologyViral replicationVesicular stomatitis virusNEW-JERSEY SEROTYPEINNATE IMMUNITYvesicular stomatitis viruslcsh:RC581-607Viral loadLife Sciences & Biomedicine030215 immunology
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Defective Induction of COX-2 Expression by Psoriatic Fibroblasts Promotes Pro-inflammatory Activation of Macrophages

2019

Fibroblasts play an important role as members of the innate immune system through the secretion of COX-2-derived inflammatory mediators such as prostaglandin E-2 (PGE(2)). However, it has been described that dermal fibroblasts behave like mesenchymal stem cells reducing lymphocyte recruitment and dendritic cell activation through PGE(2) release. As the role of fibroblasts in psoriasis remains poorly characterized, in the present study we have evaluated the possible influence of PGE(2) derived from dermal fibroblasts as modulator of the immune response in psoriatic skin. Our results indicate that under inflammatory conditions, psoriatic fibroblasts showed defective induction of COX-2, which …

lcsh:Immunologic diseases. AllergyAdultMale0301 basic medicineTHP-1 CellsLymphocyteImmunologyInflammationDinoprostoneYoung Adult03 medical and health sciences0302 clinical medicineImmune systemPsoriasismedicineHumansImmunology and AllergyPsoriasisProstaglandin E2SkinOriginal ResearchInflammationInnate immune systemChemistryMacrophagesMesenchymal stem cellpsoriasisDendritic cellMiddle AgedFibroblastsmedicine.diseaseCyclooxygenasecyclooxygenase030104 developmental biologymedicine.anatomical_structureCyclooxygenase 2inflammationCancer researchFemalemedicine.symptomlcsh:RC581-607030215 immunologymedicine.drug
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Immunosenescence and Cytomegalovirus

2010

Since Looney at al. published their seminal paper a decade ago [1] it has become clear that many of the differences in T cell immunological parameters observed between young and old people are related to the age-associated increasing prevalence of infection with the persistent β-herpesvirus HHV-5 (Cytomegalovirus). Ten years later, studies suggest that hallmark age-associated changes in peripheral blood T cell subset distribution may not occur at all in people who are not infected with this virus [[2]; Derhovanessian et al., in press]. Whether the observed changes are actually caused by CMV is an open question, but very similar, rapid changes observed in uninfected patients receiving CMV-in…

lcsh:Immunologic diseases. AllergyAgingCMV ImmunosenescenceageingT cellImmunologyCongenital cytomegalovirus infectionYellow fever vaccine32 Biomedical and Clinical Scienceslcsh:GeriatricsVirusImmune systemMedicine3202 Clinical Sciencesbiologybusiness.industryvirus diseasesImmunosenescenceBiological Sciencesmedicine.disease3204 Immunologylcsh:RC952-954.6Ageingmedicine.anatomical_structureImmunologyT cell subsetQR180biology.proteinCommentaryAntibodylcsh:RC581-607businessmedicine.drugImmunity & ageing
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Immunosenescence and lymphomagenesis

2018

Abstract One of the most important determinants of aging-related changes is a complex biological process emerged recently and called “immunosenescence”. Immunosenescence refers to the inability of an aging immune system to produce an appropriate and effective response to challenge. This immune dysregulation may manifest as increased susceptibility to infection, cancer, autoimmune disease, and vaccine failure. At present, the relationship between immunosenescence and lymphoma in elderly patients is not defined in a satisfactory way. This review presents a brief overview of the interplay between aging, cancer and lymphoma, and the key topic of immunosenescence is addressed in the context of t…

lcsh:Immunologic diseases. AllergyAgingLymphomaImmunosenescenceImmunologyContext (language use)Diseaselcsh:Geriatricsmedicine.disease_cause03 medical and health sciences0302 clinical medicineImmune systemimmune system diseaseshemic and lymphatic diseasesmedicineCancerAutoimmune diseasebusiness.industryImmunosenescenceImmune dysregulationmedicine.diseaseLymphomagenesisEpstein–Barr virusLymphomaLymphomagenesiAgeinglcsh:RC952-954.6030220 oncology & carcinogenesisImmunologylcsh:RC581-607business030215 immunologyImmunity & Ageing
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