Search results for "immunosenescence"

showing 10 items of 90 documents

Immunosenescence and Cytomegalovirus

2010

Since Looney at al. published their seminal paper a decade ago [1] it has become clear that many of the differences in T cell immunological parameters observed between young and old people are related to the age-associated increasing prevalence of infection with the persistent β-herpesvirus HHV-5 (Cytomegalovirus). Ten years later, studies suggest that hallmark age-associated changes in peripheral blood T cell subset distribution may not occur at all in people who are not infected with this virus [[2]; Derhovanessian et al., in press]. Whether the observed changes are actually caused by CMV is an open question, but very similar, rapid changes observed in uninfected patients receiving CMV-in…

lcsh:Immunologic diseases. AllergyAgingCMV ImmunosenescenceageingT cellImmunologyCongenital cytomegalovirus infectionYellow fever vaccine32 Biomedical and Clinical Scienceslcsh:GeriatricsVirusImmune systemMedicine3202 Clinical Sciencesbiologybusiness.industryvirus diseasesImmunosenescenceBiological Sciencesmedicine.disease3204 Immunologylcsh:RC952-954.6Ageingmedicine.anatomical_structureImmunologyT cell subsetQR180biology.proteinCommentaryAntibodylcsh:RC581-607businessmedicine.drugImmunity & ageing
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Immunosenescence and lymphomagenesis

2018

Abstract One of the most important determinants of aging-related changes is a complex biological process emerged recently and called “immunosenescence”. Immunosenescence refers to the inability of an aging immune system to produce an appropriate and effective response to challenge. This immune dysregulation may manifest as increased susceptibility to infection, cancer, autoimmune disease, and vaccine failure. At present, the relationship between immunosenescence and lymphoma in elderly patients is not defined in a satisfactory way. This review presents a brief overview of the interplay between aging, cancer and lymphoma, and the key topic of immunosenescence is addressed in the context of t…

lcsh:Immunologic diseases. AllergyAgingLymphomaImmunosenescenceImmunologyContext (language use)Diseaselcsh:Geriatricsmedicine.disease_cause03 medical and health sciences0302 clinical medicineImmune systemimmune system diseaseshemic and lymphatic diseasesmedicineCancerAutoimmune diseasebusiness.industryImmunosenescenceImmune dysregulationmedicine.diseaseLymphomagenesisEpstein–Barr virusLymphomaLymphomagenesiAgeinglcsh:RC952-954.6030220 oncology & carcinogenesisImmunologylcsh:RC581-607business030215 immunologyImmunity & Ageing
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Age and immunity

2006

Abstract Longitudinal studies are defining progressive alterations to the immune system associated with increased mortality in the very elderly. Many of these changes are exacerbated by or even caused by chronic T cell stimulation by persistent antigen, particularly from Cytomegalovirus. The composition of T cell subsets, their functional integrity and representation in the repertoire are all markedly influenced by age and by CMV. How these findings relate to epidemiological, functional, genetic, genomic and proteomic studies of human T cell immunosenescence was the subject of intense debate at an international conference held just before Christmas 2005 in the Black Forest.

lcsh:Immunologic diseases. AllergyAgingbiologyT cellRepertoireImmunologyShort ReportCongenital cytomegalovirus infectionImmunosenescencelcsh:Geriatricsmedicine.diseaseaged aging apoptosis article CD4+ CD25+ T lymphocytelcsh:RC952-954.6Immune systemmedicine.anatomical_structureAntigenImmunityImmunologybiology.proteinmedicineAntibodylcsh:RC581-607
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Role of persistent CMV infection in configuring T cell immunity in the elderly

2007

Abstract Ageing is associated with declines in many physiological parameters, including multiple immune system functions. The rate of acceleration of the frequency of death due to cardiovascular disease or cancer seems to increase with age from middle age up to around 80 years, plateauing thereafter. Mortality due to infectious disease, however, does not plateau, but continues to accelerate indefinitely. The elderly commonly possess oligoclonal expansions of T cells, especially of CD8 cells, which, surprisingly, are often associated with cytomegalovirus (CMV) seropositivity. This in turn is associated with many of the same phenotypic and functional alterations to T cell immunity that have b…

lcsh:Immunologic diseases. AllergyAgingbiologybusiness.industryImmunologyMembrane raftReviewDiseaseImmunosenescencelcsh:GeriatricsBioinformaticsVaccinationlcsh:RC952-954.6aged aging antigen expression apoptosis cancer incidence CD4+ T lymphocyte CD8+ T lymphocyte cellular immunityAgeingImmune systemInfectious disease (medical specialty)ImmunityImmunologybiology.proteinMedicineAntibodybusinesslcsh:RC581-607Immunity & Ageing
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Report from the second cytomegalovirus and immunosenescence workshop

2011

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.; International audience; The Second International Workshop on CMV & Immunosenescence was held in Cambridge, UK, 2-4th December, 2010. The presentations covered four separate sessions: cytomegalovirus and T cell phenotypes; T cell memory frequency, inflation and immunosenescence; cytomegalovirus in aging, mortality and disease states; and the immunobiology of cytomegalovirus-specific T cells and effects of the virus on vacc…

lcsh:Immunologic diseases. AllergyGerontologyAging[SDV.IMM] Life Sciences [q-bio]/Immunology[SDV]Life Sciences [q-bio]ImmunologyCongenital cytomegalovirus infectionDiseaseAgeing CMV immunitylcsh:Geriatrics0601 Biochemistry and Cell BiologyVaccine Related03 medical and health sciences0302 clinical medicineSDG 3 - Good Health and Well-beingMedicinecytomegalovirusComputingMilieux_MISCELLANEOUS030304 developmental biologyimmunosenescence0303 health sciencesbusiness.industryGeriatrics gerontologyImmunosenescencemedicine.disease3. Good healthlcsh:RC952-954.6AgeingHCMV InfectionInfectious DiseasesCommentary/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_being[SDV.IMM]Life Sciences [q-bio]/ImmunologyImmunizationlcsh:RC581-607business030215 immunology
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Pathophysiology of ageing, longevity and age related diseases

2007

Abstract On April 18, 2007 an international meeting on Pathophysiology of Ageing, Longevity and Age-Related Diseases was held in Palermo, Italy. Several interesting topics on Cancer, Immunosenescence, Age-related inflammatory diseases and longevity were discussed. In this report we summarize the most important issues. However, ageing must be considered an unavoidable end point of the life history of each individual, nevertheless the increasing knowledge on ageing mechanisms, allows envisaging many different strategies to cope with, and delay it. So, a better understanding of pathophysiology of ageing and age-related disease is essential for giving everybody a reasonable chance for living a …

lcsh:Immunologic diseases. AllergyGerontologyAgingmedia_common.quotation_subjectImmunologyReviewDiseaselcsh:Geriatricslongevity in Sicily and Sardiniaddc:570Age relatedMedicinecancerLife historymedia_commonimmunosenescenceEnd pointGeriatrics gerontologybusiness.industryLongevityImmunosenescencelcsh:RC952-954.6AgeingageingAging T-Lymphocytes aged micelcsh:RC581-607business
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Mechanisms of immunosenescence

2009

Abstract On April 7,8, 2009 a Symposium entitled "Pathophysiology of Successful and Unsuccessful Ageing" took place in Palermo, Italy. Here, the lectures of G. Pawelec, D. Dunn-Walters and. G. Colonna-Romano on T and B immunosenescence are summarized. In the elderly, many alterations of both innate and acquired immunity have been described. Alterations to the immune system in the older person are generally viewed as a deterioration of immunity, leading to the use of the catch-all term immunosenescence. Indeed, many immunological parameters are often markedly different in elderly compared to young people, and some, mostly circumstantial, evidence suggests that retained function of both innat…

lcsh:Immunologic diseases. AllergyOlder personAgingbusiness.industryGeriatrics gerontologyImmunologyShort ReportImmunosenescencelcsh:GeriatricsAcquired immune systemImmune Dysfunctionhumanitieslcsh:RC952-954.6AgeingImmune systemCMV IMMUNOSENESCENCEAGEINGImmunityImmunologyMedicinelcsh:RC581-607businessImmunity & Ageing
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Models of Immune Aging

2018

Abstract Biochemical changes, impaired immune responses to new antigens, and inflammation-based disorders are commonly found in aged individuals. Thus, many studies have addressed the immune system of healthy elderly, including centenarians, since a well-preserved immune system appears to be a major factor of longevity. Longitudinal studies in humans are complicated, as most immune changes associated with aging develop slowly. Human models of accelerated immune aging in clinical conditions allow exploring the age-related changes in the human immune system and the mechanisms of accelerated aging in chronic infections and autoimmunity. Even if they do not perfectly mimic immune function and i…

media_common.quotation_subjectSystems biologyLongevityInflammationImmunosenescencebiochemical phenomena metabolism and nutritionBiologymedicine.disease_causeT cell cloningAutoimmunityImmune systemAntigenImmunologymedicinemedicine.symptommedia_common
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Centenarians overexpress BCL-xL, which confers them a protection against apoptosis, oxidative stress and immunosenescence

2015

Centenarians not only have an extraordinary longevity, but also show a compression of morbidity. They preserve the capacity of maintaining homeostasis, and this is the reason for them to reach such a long life. We studied their mRNA expression profile and identified 1721 mRNAs differentially expressed by centenarians when compared with septuagenarians and young people. A sub-network analysis showed six common genes: interferon, T-cell receptor, tumor necrosis factor, SP1 transcription factor, transforming growth factor and IL-32.These six centenarian-specific genes are related to Bcl-xL, Fas, and Fas ligand all of them involved in the control of apoptosis. RT-PCR analysis confirmed that cen…

medicine.medical_specialtyBcl-xLImmunosenescenceBiologyMitochondrionmedicine.disease_causeBiochemistryFas ligandEndocrinologyApoptosisPhysiology (medical)Internal medicineImmunologymedicinebiology.proteinmedia_common.cataloged_instanceEuropean unionLeukocyte chemotaxisOxidative stressmedia_commonFree Radical Biology and Medicine
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Bcl-xL as a Modulator of Senescence and Aging

2021

Many features of aging result from the incapacity of cells to adapt to stress conditions. When cells are overwhelmed by stress, they can undergo senescence to avoid unrestricted growth of damaged cells. Recent findings have proven that cellular senescence is more than that. A specific grade of senescence promotes embryo development, tissue remodeling and wound healing. However, constant stresses and a weakening immune system can lead to senescence chronicity with aging. The accumulation of senescent cells is directly related to tissue dysfunction and age-related pathologies. Centenarians, the most aged individuals, should accumulate senescent cells and suffer from their deleterious effects,…

senescenceReviewmedicine.disease_causelcsh:Chemistry0302 clinical medicineImmunologic Surveillancelcsh:QH301-705.5SpectroscopyCellular Senescenceimmunosenescence0303 health sciencesapoptosisGeneral MedicineImmunosenescenceComputer Science ApplicationsCell biologyOrgan Specificity030220 oncology & carcinogenesisDisease SusceptibilitycentenariansProtein BindingSignal TransductionSenescencebcl-X ProteinBcl-xLBiologyCatalysisInorganic Chemistry03 medical and health sciencesImmune systemStress PhysiologicalmedicineAnimalsHumansPhysical and Theoretical ChemistrySenolyticMolecular Biology030304 developmental biologyBcl-xLOrganic ChemistryIntrinsic apoptosisagingGene Expression Regulationlcsh:Biology (General)lcsh:QD1-999senolyticsbiology.proteinWound healingOxidative stressBiomarkersDNA DamageInternational Journal of Molecular Sciences
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