Search results for "inflammation"

showing 10 items of 2662 documents

Détection orosensorielle des lipides alimentaires chez la souris : mécanismes impliqués et altérations au cours de l'obésité

2015

Dietary lipids are detected by the gustatory system in rodents and humans. Two candidate lipid-receptors are found in taste buds: CD36, which is involved in the fat taste as shown by studies conducted in our laboratory, and GPR120. Our results show that GPR120 is not directly involved in the gustatory detection of lipids in mice, but could rather be involved in the modulation of the sensitivity for fat. When this gustatory system works properly, food choices can meet the organism’s energy needs. Besides, the pleasure brought by the consumption of palatable foods is satisfied. However, obese people often overconsume energy-dense food. In the central nervous system, perturbations of the rewar…

[SDV.MHEP.EM] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismInflammationSantéMicrobioteLPSGoût du grasGPR120Food behaviorMicrobiota[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionLipides[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismLipids[SDV.AEN] Life Sciences [q-bio]/Food and NutritionComportement alimentaireHealthObesityObésitéCD36[SDV.AEN]Life Sciences [q-bio]/Food and NutritionFat taste
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Rôle du récepteur nucléaire d'activation et de prolifération des péroxysomes (PPAR-alpha) dans la modulation de l'inflammation et l'activation des ce…

2010

We assessed, in this study, the effects of PPARα deficiency on the expression of mRNAencoding for insulin gene transcription factors in pancreatic β-cells along with thoseimplicated in inflammation in adipose tissues. On fasting, the adult PPARα-null mice werehypoglycemic. Serum insulin concentrations and its pancreatic mRNA transcripts weredownregulated in PPARα-null mice, suggesting that PPARα gene deletion contributes to lowinsulin gene transcription. The PPARα gene deletion downregulates the mRNA expression ofinsulin gene transcription factors, i.e., Pdx-1, Nkx6.1 and MafA. Besides, the pancreaticfunction was diminished by PPARα deficiency as PPARα-null mice expressed low pancreaticGlut…

[SDV.SA]Life Sciences [q-bio]/Agricultural sciencesInflammation[SDV.SA] Life Sciences [q-bio]/Agricultural sciences[SDV.MHEP] Life Sciences [q-bio]/Human health and pathology[SDV.BA] Life Sciences [q-bio]/Animal biology[SDV.BA]Life Sciences [q-bio]/Animal biologyInsulinorésistanceInsulin resistanceAGPI n-3[ SDV.BA ] Life Sciences [q-bio]/Animal biologyPPARα[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyN-3 fatty acids[ SDV.SA ] Life Sciences [q-bio]/Agricultural sciences[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyPPAR-α
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Microglia and α-synuclein implication in Parkinson's disease

2011

Age-related neurodegenerative disorders like Parkinson’s disease take an enormous toll on individuals and on society. Despite extensive efforts, Parkinson’s disease remains incurable and only very limited treatments exist. Indeed, Parkinson’s pathogenesis is still not clear and research on its molecular mechanisms is ongoing. In this study, we focused our interest on two abnormal events occurring in Parkinson’s patients, namely α-synuclein aggregation and microglial activation. We first investigated α-synuclein and its abnormal polymerisation. For this purpose, we developed novel methods, which allowed the in vitro production of different types of α-synuclein oligomers. Using highly sensiti…

[SDV.SA]Life Sciences [q-bio]/Agricultural sciencesΑ-synucléineAgingMaladie de ParkinsonDopamineCultures primairesBrain immunityNeuronesVieillissementImmunité du cerveauPotassium channelsNeuroinflammationToxicitéC8-B4 cell line[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyMéthode d’isolation in vitroElectrophysiologieCanaux potassiquesNeurodegeneration[ SDV.SA ] Life Sciences [q-bio]/Agricultural sciencesIn vitro isolation methodPrimary cultureKv1.3 - Kir2.1Neurons[SDV.SA] Life Sciences [q-bio]/Agricultural sciences[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyToxicityMaladies neurodégénérativesΑ-synucleinNitric oxideNeurodégénérationOligomèresElectrophysiologyMicrogliesBrain macrophagesOligomersOxyde nitriqueNeurodegenerative disordersParkinson’s diseaseCytokinesLignée cellulaire C8-B4MicrogliaPatch-clamp[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Time Course of Inflammation and Apoptosis in a Scopolamine–Induced Dry Eye Rat Model

2006

International audience; Purpose: : Dry eye syndrome is due to tear secretion deficiency or excessive tear evaporation leading to tear film instability. The aim of this study was to evaluate the time course of inflammation and apoptosis in a scopolamine–induced dry eye rat model. Methods: : Osmotic pumps continuously delivering scopolamine (12.5mg/d) were implanted subcutaneously in female Lewis rats for 1, 2, 3, 7, 10, 17 and 28 days. During the treatment, the corneal status was assessed by fluorescein impregnation and tears were collected for prostaglandin E2 (PGE2) immunoassay. At the end of the treatment period, the animals were sacrificed and the ocular tissues and the lacrimal glands w…

[SDV] Life Sciences [q-bio]conjunctiva[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process Engineeringfilm/dry eyeinflammation[SDV]Life Sciences [q-bio][SDV.IDA]Life Sciences [q-bio]/Food engineering[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process Engineeringsense organs[SDV.IDA] Life Sciences [q-bio]/Food engineeringcornea: tears/teareye diseases
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Lipid composition of lacrimal glands in rats: comparison with human lacrimal glands and possible nutritional modulation

2007

International audience; Purpose:: Prostaglandins (PGs) have been found in tears of patients with ocular surface disorders, and are powerful markers of inflammation. The lacrimal gland has been reported to synthesize PGs. Polyunsaturated fatty acids esterified in phospholipids are the precursors of pro- and anti-inflammatory PGs. The aim of this study was to compare the phospholipid content of the lacrimal gland in human and in different rat species. Moreover the changes in fatty acids of the lacrimal glands of Lewis rats were studied after a nutritional intervention. Methods:: Lacrimal glands were excised from Wistar, Sprague-Dawley and Lewis rats (3 month-old) and from Humans 3 to 36 hours…

[SDV] Life Sciences [q-bio]lipids[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process Engineeringinflammation[SDV]Life Sciences [q-bio][SDV.IDA]Life Sciences [q-bio]/Food engineeringlipids (amino acids peptides and proteins)[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process Engineering[SDV.IDA] Life Sciences [q-bio]/Food engineeringlacrimal gland
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Modelling Keloids Dynamics: A Brief Review and New Mathematical Perspectives

2022

The keloids are fibroproliferative disorders described by an excessive growth of fibrotic tissue, which also invades adjacent areas (beyond the original wound borders). Since these disorders are specific to humans (no other animal species naturally develop keloid-like tissue), the experimental in vivo/in vitro research has not lead to significant advances in this field. One possible approach could be to combine in vitro human models with calibrated in silico mathematical approaches (i.e., models and simulations) to generate new testable biological hypotheses related to biological mechanisms and improved treatments. Since these combined approaches do not really exist for keloid disorders, in…

[SDV] Life Sciences [q-bio]modelling cancer and wound healinginflammationkeloid disordersreviewmathematical modelling and computational approaches[MATH] Mathematics [math]biomechanics
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Regulation of protein cysteinylation and the inflammatory cascade by thioredoxin-related protein of 14 kDa in pancreas

2020

Although under physiological conditions ROS are essential for normal cell signaling, ROS overproduction under oxidative stress conditions may cause damage to biomolecules. Cysteine residues within proteins are often easily oxidized. Reversible cysteine oxidation is used as a mechanism of redox signaling and control of protein function. However, oxidative stress promotes oxidation of protein thiols, leading to the formation of mixed disulfides between proteins and low-molecular-weight thiols. Disulfide reductases such as thioredoxins can reduce disulfide bonds back to free thiols. Acute pancreatitis is currently one of the leading causes of hospital admission for gastrointestinal disorders, …

acute inflammationcysteinylationUNESCO::CIENCIAS MÉDICASdisulfide stresspancreatitisoxidative stressTRP14thioredoxin:CIENCIAS MÉDICAS [UNESCO]
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Role of Chemerin in Cardiovascular Diseases.

2022

(1) Background: Obesity is closely connected to the pathophysiology of cardiovascular diseases (CVDs). Excess fat accumulation is associated with metabolic malfunctions that disrupt cardiovascular homeostasis by activating inflammatory processes that recruit immune cells to the site of injury and reduce nitric oxide levels, resulting in increased blood pressure, endothelial cell migration, proliferation, and apoptosis. Adipose tissue produces adipokines, such as chemerin, that may alter immune responses, lipid metabolism, vascular homeostasis, and angiogenesis. (2) Methods: We performed PubMed and MEDLINE searches for articles with English abstracts published between 1997 (when the first re…

adipokinechemerin receptorsMedicine (miscellaneous)610 Medicine & health2701 Medicine (miscellaneous)endothelial dysfunctionGeneral Biochemistry Genetics and Molecular Biologyadipokine cardiovascular disease chemerin chemerin receptors chemerin-targeting therapeutic agents endothelial dysfunction inflammation1300 General Biochemistry Genetics and Molecular Biologycardiovascular diseaseinflammation10209 Clinic for Cardiologychemerin-targeting therapeutic agentschemerinBiomedicines
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Advances in immunopathogenesis of macrophage activation syndrome during rheumatic inflammatory diseases: toward new therapeutic targets?

2017

Introduction: Macrophage activation syndrome (MAS) is a severe, hyperinflammatory life-threatening syndrome, generally complicating different rheumatic diseases. Despite the severity of the disease, little is known about the pathogenic mechanisms and, thus, possible targeted therapies in the management of these patients. Areas covered: In this review, we aimed to update the current pathogenic knowledge of MAS, during rheumatic diseases, focusing mainly on immunologic abnormalities and on new possible therapeutic strategies. Expert commentary: The difficult pathogenic scenario of MAS, in which genetic defects, predisposing diseases, and triggers are mixed together with the high mortality rat…

adult onset Still’s disease0301 basic medicineAdultAdult-onset Still's diseasemedicine.medical_treatmentImmunologyInflammationmacrophagemacromolecular substancesDisease03 medical and health sciences0302 clinical medicineRheumatic DiseasescytokinemedicineImmunology and AllergyMacrophageAnimalsHumanssystemic juvenile idiopathic arthritiMolecular Targeted TherapyChildExpert Testimony030203 arthritis & rheumatologyInflammationbusiness.industrymusculoskeletal neural and ocular physiologyMacrophage Activation Syndromehyperferritinemic syndromeMacrophage Activationmedicine.diseaseSettore MED/16 - Reumatologia030104 developmental biologyCytokinenervous systemMacrophage activation syndromeImmunologymedicine.symptombusinessExpert review of clinical immunology
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Ambient Air Pollution Increases the Risk of Cerebrovascular and Neuropsychiatric Disorders through Induction of Inflammation and Oxidative Stress

2020

Exposure to ambient air pollution is a well-established determinant of health and disease. The Lancet Commission on pollution and health concludes that air pollution is the leading environmental cause of global disease and premature death. Indeed, there is a growing body of evidence that links air pollution not only to adverse cardiorespiratory effects but also to increased risk of cerebrovascular and neuropsychiatric disorders. Despite being a relatively new area of investigation, overall, there is mounting recent evidence showing that exposure to multiple air pollutants, in particular to fine particles, may affect the central nervous system (CNS) and brain health, thereby contributing to …

air pollutionneurological disordersAir pollutionReviewDisease010501 environmental sciencesGlobal Healthmedicine.disease_cause01 natural scienceslcsh:Chemistry0302 clinical medicineMedicineStrokelcsh:QH301-705.5SpectroscopyDepression (differential diagnoses)media_commonAir Pollutantscerebrovascular disordersGeneral MedicinestrokeComputer Science ApplicationsDisease Susceptibilitycerebrovascular disorders ; dementia ; oxidative stress ; mental disorders ; air pollution ; particulate matter ; inflammation ; neurological disorders ; strokePollutionmedicine.medical_specialtymedia_common.quotation_subjectCatalysisInorganic Chemistry03 medical and health sciencesEnvironmental healthAnimalsHumansDementiaPhysical and Theoretical ChemistryMolecular Biology0105 earth and related environmental sciencesInflammationparticulate matterbusiness.industryPublic healthOrganic ChemistryCOVID-19medicine.diseasemental disordersDisease Models AnimalOxidative Stresslcsh:Biology (General)lcsh:QD1-999business030217 neurology & neurosurgeryOxidative stressdementiaInternational Journal of Molecular Sciences
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