Search results for "inflammation"

showing 10 items of 2662 documents

dIvergEnt: How IgE Axis Contributes to the Continuum of Allergic Asthma and Anti-IgE Therapies

2017

[EN] Asthma is an airway disease characterised by chronic inflammation with intermittent or permanent symptoms including wheezing, shortness of breath, chest tightness, and cough, which vary in terms of their occurrence, frequency, and intensity. The most common associated feature in the airways of patients with asthma is airway inflammation. In recent decades, efforts have been made to characterise the heterogeneous clinical nature of asthma. The interest in improving the definitions of asthma phenotypes and endotypes is growing, although these classifications do not always correlate with prognosis nor are always appropriate therapeutic approaches. Attempts have been made to identify the m…

AllergyDiseaseOmalizumabReviewOmalizumabImmunoglobulin Eimmunomodulation0302 clinical medicine030212 general & internal medicineanti-IgEAnti-Asthmatic Agentsbiological treatmentSpectroscopyBiología molecularbiologyAntibodies MonoclonalAllergic asthmaGeneral MedicineComputer Science Applicationsmedicine.symptommedicine.drugBioquímicaimmunological mechanismsmedicine.drug_classInflammationMonoclonal antibodyCatalysisInorganic Chemistry03 medical and health sciencesmedicineImmunologic FactorsAnimalsHumansfactores inmunitariosPhysical and Theoretical ChemistryMolecular BiologyAsthmaimmunoglobulin E (IgE)Inflammationbusiness.industryOrganic ChemistrybiomarkersMicrobiología médicaasthmaImmunoglobulin Emedicine.diseaseallergyrespiratory tract diseases030228 respiratory systemImmunologybiology.proteinbusiness
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Mucosal immunoregulation: transcription factors as possible therapeutic targets.

2005

Much progress has been recently made with regard to our understanding of the mucosal immune system in health and disease. In particular, it has been shown that uncontrolled mucosal immune responses driven by lymphocytes or non-lymphoid cells may lead to immunological diseases such as allergy, hypersensitivity and inflammation. Thus, a more detailed understanding of mucosal immune regulation and decision making at mucosal surfaces is essential for a better understanding of mucosal immune responses in health and disease. Antigen presenting cells and T lymphocytes play a key role in controlling mucosal immune responses. To deal with this key task, T helper cells differentiate into functionally…

AllergyImmunologyInflammationApoptosisSuppressor of Cytokine Signaling ProteinsAllergic inflammationPathogenesisImmune systemImmunitymedicineHypersensitivityImmunology and AllergyAnimalsHumansIL-2 receptorMast CellsAntigen-presenting cellGlucocorticoidsImmunity MucosalPharmacologybusiness.industrymedicine.diseaseInflammatory Bowel DiseasesAsthmaIntestinesSuppressor of Cytokine Signaling 3 ProteinImmunologyCytokinesmedicine.symptombusinessTranscription FactorsCurrent drug targets. Inflammation and allergy
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The role of NO in contact hypersensitivity

2001

Contact dermatitis or contact hypersensitivity (CHS) is a common T lymphocyte-mediated allergic disease characterized by local inflammatory skin reactions following contact with small reactive compounds called haptens. In common with other allergic processes, the development of contact dermatitis proceeds in two phases: a sensitization phase which occurs on first exposure to allergen, and an elicitation phase which occurs on subsequent exposure when the clinical manifestations of the disease are observed. This process is hapten-specific. While the pathophysiology of the sensitization phase is well characterized, our understanding of the elicitation phase is still incomplete, including the r…

AllergyImmunologyInflammationDermatitis ContactNitric OxideNitric oxidechemistry.chemical_compoundImmune systemDownregulation and upregulationmedicineAnimalsHumansImmunology and Allergyskin and connective tissue diseasesSensitizationPharmacologyintegumentary systembiologymedicine.diseaseNitric oxide synthasemedicine.anatomical_structurechemistryImmunologybiology.proteinmedicine.symptomContact dermatitisInternational Immunopharmacology
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Epileptic seizures as a manifestation of cow's milk allergy: a studied relationship and description of our pediatric experience

2014

Adverse reactions after ingestion of cow's milk proteins can occur at any age, from birth and even amongst exclusively breast-fed infants, although not all of these are hypersensitivity reactions. The most common presentations related to cow's milk protein allergy are skin reactions, failure to thrive, anaphylaxis as well as gastrointestinal and respiratory disorders. In addition, several cases of cow's milk protein allergy in the literature have documented neurological involvement, manifesting with convulsive seizures in children. This may be due to CNS spread of a peripheral inflammatory response. Furthermore, there is evidence that pro-inflammatory cytokines are responsible for disruptin…

AllergyImmunologyPro-inflammatory cytokinesAtypical clinical featuresAtypical clinical features; CNS inflammation; Cow's milk allergy; Pro-inflammatory cytokines; Seizures;Cns inflammationCow's milk allergyProinflammatory cytokineNOAtopyCow's milk allergySeizuresmedicineImmunology and AllergyIngestionAnimalsHumansChildbusiness.industryfood and beveragesmedicine.diseaseCNS inflammationMilkSettore MED/38 - PEDIATRIA GENERALE E SPECIALISTICAImmunologyFailure to thrivemedicine.symptomMilk HypersensitivitybusinessAnaphylaxis
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Higher serum levels of periostin and the risk of exacerbations in moderate asthmatics

2016

BACKGROUND: In asthma, exacerbations and poor disease control are linked to airway allergic inflammation. Serum periostin has been proposed as a systemic biomarker of eosinophilic inflammation. This pilot study aims at evaluating whether in patients with moderate asthma, higher baseline levels of serum periostin are associated with a greater risk of exacerbation. METHODS: Fifteen outpatients with moderate allergic asthma were recruited. Serum concentrations of periostin were assessed (ELISA) at baseline, and the frequency of asthma exacerbations was recorded during a one-year follow-up. RESULTS: Patients (M/F: 10/5, mean age of 47.6 ± 11.0 years) had mean ACQ score of 5.5 ± 4.2 and FEV1%pre…

Allergymedicine.medical_specialtyExacerbationSerum biomarkerPeriostinSettore MED/10 - Malattie Dell'Apparato RespiratorioBiochemistryAllergic inflammation03 medical and health sciences0302 clinical medicineInternal medicinemedicineAsthmatic patient030212 general & internal medicineAirway inflammation; Asthma; Exacerbation; Periostin; Serum biomarkerAsthmabusiness.industryResearchOrganic ChemistryExacerbationmedicine.diseaseSerum biomarker Asthma Periostin Exacerbation Airway inflammationAsthmaPeriostin030228 respiratory systemImmunologyBiomarker (medicine)businessAirwayAirway inflammation
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Cat Allergy as a Source Intensification of Atopic Dermatitis in Adult Patients

2020

Atopic dermatitis (AD) is characterized by exacerbations and remissions of eczematous skin, underlain by impaired skin barrier and aberrant Th2-type and Th-22 cytokine production. A number of allergens, in particular contact with fur animals, may aggravate the disease. This study seeks to define the influence of having a regular contact with a pet cat at home on the severity of symptoms and signs of AD. We addressed the issue using the SCORing Atopic Dermatitis (SCORAD) and visual analog (VAS) scores to assess the intensity of pruritus and by measuring the blood content of specific IgE and IL-4, IL-13, and IL-22 cytokines. The study group consisted of 47 adult patients suffering from AD sin…

Allergy; Animal fur allergen; Atopic dermatitis; Inflammation; Interleukins; Pet cat; Skin pruritus.
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Synthesis of polyfluoroalkyl sp2-iminosugar glycolipids and evaluation of their immunomodulatory properties towards anti-tumor, anti-leishmanial and …

2019

Immunomodulatory glycolipids, among which α-galactosylceramide (KRN7000) is an iconic example, have shown strong therapeutic potential in a variety of conditions ranging from cancer and infection to autoimmune or neurodegenerative diseases. A main difficulty for those channels is that they often provoke a cytokine storm comprising both pro- and anti-inflammatory mediators that antagonize each other and negatively affect the immune response. The synthesis of analogues with narrower cytokine secretion-inducing capabilities is hampered by the intrinsic difficulty at controlling the stereochemical outcome in glycosidation reactions, particularly if targeting the α-anomer, which seriously hamper…

Allosteric regulationIminosugar01 natural sciencesImmunomodulation03 medical and health sciencesGlycolipidGlycomimeticDrug DiscoverymedicineLeishmaniasisp38α MAPKCancer030304 developmental biologyInflammationPharmacology0303 health sciences010405 organic chemistryChemistryOrganic ChemistryAutophosphorylationBiological activityGeneral Medicinemedicine.disease0104 chemical sciencessp2-Iminosugar glycolipidsBiochemistryMechanism of actionPolyfluoroalkyl compoundsmedicine.symptomCytokine stormEuropean Journal of Medicinal Chemistry
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Emerging contributions of formyl peptide receptors to neurodegenerative diseases.

2021

Abstract Inflammation is a central element of many neurodegenerative diseases. Formyl peptide receptors (FPRs) can trigger several receptor-dependent signal transduction pathways that play a key role in neuroinflammation and neurodegeneration. They are chemotactic receptors that help to regulate pro- and anti-inflammatory responses in most mammals. FPRs are primarily expressed in the immune and nervous systems where they interact with a complex pattern of pathogen-derived and host-endogenous molecules. Mounting evidence points towards a contribution of FPRs – via neuropathological ligands such as Amyloid beta, and neuroprotective ligands such as Humanin, Lipoxin A4, and Annexin A1 – to mult…

Amyloid beta-PeptidesClinical BiochemistryNeurodegenerationChemotaxisNeurodegenerative DiseasesBiologymedicine.diseaseLigandsBiochemistryNeuroprotectionReceptors Formyl PeptideNeuroinflammatory DiseasesmedicineFunctional selectivityAnimalsHumansSignal transductionMolecular BiologyCentral elementNeuroscienceNeuroinflammationHumaninBiological chemistryReferences
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Oxidative stress in Alzheimer’s Disease: Implications for Prevention and Therapy

2006

Oxidative stress is a marker of neurodegeneration and has been recently shown to be also involved in the early stages of the pathogenesis of various neurodegenerative disorders. In general, all biomolecules of the cell can be oxidized and thereby damaged. Consequently, the concept of neuroprotection by antioxidants has been developed. In many cases the direct scavanging of free radicals have been used as a strategy to prevent oxidative stress damage and a variety of physiological and synthetic antioxidant molecules have been identified and synthesized including the female sex homone estrogen. In Alzheimer’s Disease amyloid-β protein on its way to brain deposition can also induce oxidative c…

Amyloidbusiness.industryNeurodegenerationInflammationDNA oxidationBlood–brain barrierBioinformaticsmedicine.disease_causemedicine.diseaseProtein oxidationNeuroprotectionmedicine.anatomical_structuremedicinemedicine.symptombusinessOxidative stress
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Safety profile of the interleukin-1 inhibitors anakinra and canakinumab in real life clinical practice: a nationwide multicenter retrospective observ…

2018

A few studies have reported the safety profile of interleukin (IL)-1 blockers from real life. The aim of this study is to describe anakinra (ANA) and canakinumab (CAN) safety profile in children and adults, based on data from a real-life setting. Demographic, clinical, and therapeutic data from patients treated with ANA and CAN were retrospectively collected and analyzed. Four hundred and seventy five patients were enrolled; ANA and CAN were prescribed in 421 and 105 treatment courses, respectively. During a mean follow-up of 24.39 ± 27.04 months, 89 adverse events (AE) were recorded; 13 (14.61%) were classified as serious AE (sAE). The overall estimated rate of AE and sAE was 8.4 per 100 p…

Anakinra; Autoinflammatory disorders; Canakinumab; Interleukin-1; Safety profile0301 basic medicineMaleSettore MED/16 - REUMATOLOGIAAutoinflammatory disorders0302 clinical medicineRetrospective StudieAnakinra; Autoinflammatory disorders; Canakinumab; Interleukin-1; Safety profile; RheumatologyChildAntibodies MonoclonalGeneral MedicineMiddle AgedAnakinraTreatment OutcomeAutoinflammationFemaleCohort studymedicine.drugHumanAdultmedicine.medical_specialtyAdolescentLogistic ModelCanakinumabNeutropeniaAntibodies Monoclonal HumanizedAutoimmune DiseaseAutoimmune Diseases03 medical and health sciencesYoung AdultRheumatologyInternal medicineInjection site reactionmedicineHumansAnakinra Autoinflammatory disorders Canakinumab Interleukin-1 Safety profile Adolescent Adult Antibodies Monoclonal Antibodies Monoclonal Humanized Autoimmune Diseases Child Female Humans Interleukin 1 Receptor Antagonist Protein Logistic Models Male Middle Aged Retrospective Studies Treatment Outcome Young AdultAdverse effectRetrospective Studies030203 arthritis & rheumatologyAnakinrabusiness.industryRetrospective cohort studymedicine.diseaseCanakinumabInterleukin 1 Receptor Antagonist ProteinLogistic Models030104 developmental biologyAutoinflammatory disorderSafety profileObservational studybusinessInterleukin-1
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