Search results for "interleukin 10"

showing 10 items of 146 documents

Neuroimmune and Mu-Opioid Receptor Alterations in the Mesocorticolimbic System in a Sex-Dependent Inflammatory Pain-Induced Alcohol Relapse-Like Rat …

2021

Evidence concerning the role of alcohol-induced neuroinflammation in alcohol intake and relapse has increased in the last few years. It is also proven that mu-opioid receptors (MORs) mediate the reinforcing properties of alcohol and, interestingly, previous research suggests that neuroinflammation and MORs could be related. Our objective is to study neuroinflammatory states and microglial activation, together with adaptations on MOR expression in the mesocorticolimbic system (MCLS) during the abstinence and relapse phases. To do so, we have used a sex-dependent rat model of complete Freund’s adjuvant (CFA)-induced alcohol deprivation effect (ADE). Firstly, our results confirm that only CFA-…

Malemedicine.medical_treatmentFreund's AdjuvantReceptors Opioid mualcohol deprivation effectNitric Oxide Synthase Type IImicroglianeuroinflammationRats Sprague-DawleyRecurrenceLimbic SystemImmunology and AllergypainPhosphorylationReceptormedia_commonMicrogliaAlcohol AbstinencealcoholMicrofilament ProteinsNF-kappa BBrief Research ReportInterleukin 10AlcoholismCytokinemedicine.anatomical_structureCytokinesFemaleμ-opioid receptorInflammation Mediatorsmedicine.medical_specialtyNeuroimmunomodulationmedia_common.quotation_subjectImmunologyPrefrontal CortexSex FactorsDownregulation and upregulationInternal medicinemedicineAnimalsNeuroinflammationbusiness.industryCalcium-Binding ProteinsAbstinenceRC581-607EndocrinologyCyclooxygenase 2mu-opioid receptorImmunologic diseases. AllergybusinessFrontiers in Immunology
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Echinostoma caproni (Trematoda): differential in vivo cytokine responses in high and low compatible hosts.

2011

In order to investigate the factors determining the expulsion of intestinal trematodes, we have analyzed the in vivo cytokine responses at several levels and the local responses against Echinostoma caproni (Trematoda) in two host species displaying different compatibility with the parasite. The response of the high compatible host (mice) is characterized by a mixed Th1/Th2 phenotype in the spleen, Peyer's patches and mesenteric lymph nodes. At the intestine, a marked Th1 response with a marked increase of IFN-γ together with elevated number of mucosal neutrophils and expression of induced nitric oxide synthase were observed. The responses in the host of low compatibility (rats) with the par…

Malemedicine.medical_treatmentImmunologyNitric Oxide Synthase Type IISpleenPolymerase Chain ReactionHost-Parasite InteractionsMicePeyer's PatchesRandom AllocationSpecies SpecificityIn vivoEchinostomamedicineMesenteric lymph nodesAnimalsMesenteryRNA MessengerRats WistarInterleukin 5Analysis of VarianceEchinostomiasisMice Inbred ICRbiologyGeneral Medicinebiology.organism_classificationRatsIntestinesInterleukin 10Infectious Diseasesmedicine.anatomical_structureCytokineImmunologyInterleukin 13CytokinesParasitologyLymph NodesTrematodaRNA HelminthSpleenExperimental parasitology
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β-Catenin in dendritic cells exerts opposite functions in cross-priming and maintenance of CD8+ T cells through regulation of IL-10

2015

Recent studies have demonstrated that β-catenin in DCs serves as a key mediator in promoting both CD4(+) and CD8(+) T-cell tolerance, although how β-catenin exerts its functions remains incompletely understood. Here we report that activation of β-catenin in DCs inhibits cross-priming of CD8(+) T cells by up-regulating mTOR-dependent IL-10, suggesting blocking β-catenin/mTOR/IL-10 signaling as a viable approach to augment CD8(+) T-cell immunity. However, vaccination of DC-β-catenin(-/-) (CD11c-specific deletion of β-catenin) mice surprisingly failed to protect them against tumor challenge. Further studies revealed that DC-β-catenin(-/-) mice were deficient in generating CD8(+) T-cell immunit…

Mice KnockoutImmunity CellularMultidisciplinaryTOR Serine-Threonine KinasesPriming (immunology)Dendritic CellsBiologyBiological SciencesCD8-Positive T-LymphocytesCancer VaccinesCell biologyInterleukin-10Interleukin 10MiceMediatorImmunityCateninNeoplasmsImmunologyCytotoxic T cellAnimalsPI3K/AKT/mTOR pathwayCD8beta Catenin
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Human T helper cells reactive with somatic bacterial antigens belong to the Th1 subset

1994

The aim of this study was to characterize the cytokine secretion patterns of human T helper cells from healthy donors reactive with somatic antigens from various bacteria, the nematode Anisakis and tetanus toxoid. From the peripheral blood of four healthy donors we have established 70 T cell lines reactive with antigens from Yersinia, Salmonella, Morganella, Klebsiella, Serratia, Escherichia, Chlamydia, Shigella, Streptococcus, tetanus toxoid and Anisakis, respectively. Our results show that all T cells reactive with bacteria produce interferon-gamma (IFN-gamma) and tumor necrosis factor-alpha (TNF-alpha), but no interleukin (IL)-4 and no or very little IL-2 and IL-10 and, thus, belong to t…

Microbiology (medical)Interleukin 2T cellImmunologyBiologyCell LineMicrobiologyAntigenTetanus ToxoidmedicineAnimalsHumansImmunology and AllergyCells CulturedInterleukin 4Antigens BacterialToxoidGeneral MedicineT lymphocyteTh1 CellsAnisakisInterleukin 10medicine.anatomical_structureAntigens HelminthImmunologyCytokinesCytokine secretionmedicine.drugMedical Microbiology and Immunology
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Regulatory T Cells and IL-10 Independently Counterregulate Cytotoxic T Lymphocyte Responses Induced by Transcutaneous Immunization

2011

Background: The imidazoquinoline derivate imiquimod induces inflammatory responses and protection against transplanted tumors when applied to the skin in combination with a cognate peptide epitope (transcutaneous immunization, TCI). Here we investigated the role of regulatory T cells (Treg) and the suppressive cytokine IL-10 in restricting TCI-induced cytotoxic T lymphocyte (CTL) responses. Methodology/Principal Findings: TCI was performed with an ointment containing the TLR7 agonist imiquimod and a CTL epitope was applied to the depilated back skin of C57BL/6 mice. Using specific antibodies and FoxP3-diphteria toxin receptor transgenic (DEREG) mice, we interrogated inhibiting factors after…

Mouselcsh:MedicineEpitopes T-LymphocyteAdaptive ImmunityT-Lymphocytes RegulatoryImmune toleranceMiceMedicineCytotoxic T celllcsh:ScienceImmune ResponseSkinMice KnockoutB-LymphocytesMultidisciplinaryImiquimodFOXP3hemic and immune systemsForkhead Transcription FactorsAnimal ModelsFlow CytometryInterleukin-10Interleukin 10medicine.anatomical_structureAminoquinolinesCytokinesIntercellular Signaling Peptides and ProteinsImmunotherapyResearch ArticleHeparin-binding EGF-like Growth FactorT cellImmune CellsImmunologychemical and pharmacologic phenomenaImmune SuppressionImmunomodulationImmune systemModel OrganismsImmune ToleranceAnimalsBiologyB cellbusiness.industrylcsh:RImmunityMice Inbred C57BLCTL*Immune SystemImmunologyImmunologic Techniqueslcsh:QImmunizationbusinessT-Lymphocytes CytotoxicPLoS ONE
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Excessive CpG 1668 stimulation triggers IL-10 production by cDC that inhibits IFN-alpha responses by pDC.

2008

Upon stimulation with a wide range of concentrations of CpG oligodeoxynucleotide 2216 (CpG 2216), plasmacytoid DC are induced to produce type I IFN (IFN-alpha/beta). In contrast, CpG 1668 shows a bell-shaped dose-response correlation, i.e. only intermediate but not high doses of CpG 1668 induce IFN-alpha/beta. Interestingly, high-dose CpG 1668 completely inhibited IFN-alpha responses induced by CpG 2216. Experiments using supernatant of high-dose CpG-1668-treated cells indicated that secreted inhibitor(s) mediated the IFN-alpha shut-off. Among modulating cytokines, IL-10 turned out to be one important negative regulator. In line with this, supernatants of IL-10-deficient DC cultures stimula…

MuromegalovirusCpG OligodeoxynucleotideImmunologyStimulationmedicine.disease_causeNegative regulatorAutoimmunityMiceAdjuvants ImmunologicmedicineImmunology and AllergyAnimalsCells CulturedbiologyTLR9Interferon-alphaDendritic Cellsbiology.organism_classificationMolecular biologyInterleukin-10Interleukin 10CpG siteOligodeoxyribonucleotidesVesicular stomatitis virusToll-Like Receptor 9ImmunologyCytokinesEuropean journal of immunology
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IL-10 Controls Ultraviolet-Induced Carcinogenesis in Mice

2007

Abstract UV radiation-induced immunosuppression contributes significantly to the development of UV-induced skin cancer by inhibiting protective immune responses. IL-10 has been shown to be a key mediator of UV-induced immunosuppression. To investigate the role of IL-10 during photocarcinogenesis, groups of IL-10+/+, IL-10+/−, and IL-10−/− mice were chronically irradiated with UV. IL-10+/+ and IL-10+/− mice developed skin cancer to similar extents, whereas IL-10−/− mice were protected against the induction of skin malignancies by UV. Because UV is able to induce regulatory T cells, which play a role in the suppression of protective immunity, UV-induced regulatory T cell function was analyzed…

Neoplasms Radiation-InducedSkin NeoplasmsUltraviolet RaysRegulatory T cellImmunologyMice NudeBiologymedicine.disease_causeT-Lymphocytes RegulatoryMiceImmune systemImmunityImmune TolerancemedicineAnimalsImmunology and AllergyIL-2 receptorMice KnockoutMolecular biologyInterleukin-10Mice Inbred C57BLInterleukin 10medicine.anatomical_structureImmunologyGranzyme ACytokinesCarcinogenesisCD8The Journal of Immunology
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IL-17 promotes progression of cutaneous leishmaniasis in susceptible mice.

2009

Abstract Resistance to leishmaniasis in C57BL/6 mice depends on Th1/Tc1 cells. BALB/c mice preferentially develop Th2 immunity and succumb to infection. We now assessed the role of IL-17 in cutaneous leishmaniasis. During the course of Leishmania major infection, BALB/c CD4 cells and neutrophils produced increased amounts of IL-17 as compared with cells from C57BL/6 mice. This increase was associated with significantly increased IL-23 release from L. major-infected BALB/c dendritic cells (DC), whereas IL-6 and TGF-β1 production by BALB/c and C57BL/6 DC were comparable. Interestingly, lesion sizes in infected IL-17-deficient BALB/c mice were dramatically smaller and failed to progress as com…

NeutrophilsImmunologyLeishmaniasis CutaneousBiologyInterleukin-23ArticleLesionMiceImmune systemTh2 CellsCutaneous leishmaniasisSpecies SpecificityImmunitymedicineImmunology and AllergyAnimalsLeishmania majorGenetic Predisposition to DiseaseInterleukin 4Cells CulturedLeishmania majorMice KnockoutImmunity CellularMice Inbred BALB CInterleukin-17Cell DifferentiationDendritic Cellsmedicine.diseasebiology.organism_classificationUp-RegulationMice Inbred C57BLInterleukin 10ImmunologyDisease ProgressionInterleukin 17medicine.symptomJournal of immunology (Baltimore, Md. : 1950)
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Signals involved in the early TH1/TH2 polarization of an immune response depending on the type of antigen.

1999

Abstract Background: The early production of distinct cytokines by epidermal cells (ECs) in response to antigen exposure may govern the development of T H1 -like immune responses, such as contact sensitivity, or T H2 -like immune responses, such as IgE-dependent allergies of the immediate type, depending on the type of antigen. Objective: The aim of this study was to compare the signals induced by protein allergens with those induced by haptens in ECs and subsequently in local draining lymph node cells (LNCs) or splenocytes. Methods: BALB/c mice were primed in vivo with the protein allergens ovalbumin or birch pollen or the haptens 2,4-dinitrofluorobenzene or trinitrochlorbenzene, respectiv…

Ovalbuminmedicine.medical_treatmentImmunologyImmunoglobulinsEnzyme-Linked Immunosorbent AssayPicryl ChlorideBiologyMiceImmune systemTh2 CellsAntigenmedicineDinitrochlorobenzeneImmunology and AllergyAnimalsRNA MessengerCells CulturedMice Inbred BALB CReverse Transcriptase Polymerase Chain ReactionCell PolarityEpithelial CellsT lymphocyteAllergensTh1 CellsInterleukin-10Interleukin 10OvalbuminBlotting SouthernKineticsCytokineImmunologybiology.proteinCytokinesPollenFemaleLymph NodesAntibodyHaptenHaptensSpleenSignal TransductionThe Journal of allergy and clinical immunology
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P80Cannabinoid receptor CB2 prevents development of heart failure in a murine model of pressure overload

2014

Purpose: Cardiac adaptation to pressure overload is associated with inflammatory reaction, which untreated leads to myocardial fibrosis and heart failure. We have recently demonstrated that endogenous cannabinoids and the cannabinoid receptor 2 (CB2) are activated and associated with persistent inflammation in hypertrophic myocardium of patients with aortic valve stenosis. Therefore, we investigated the role of the CB2 in a mouse model of pressure overload. Methods: Transverse aortic constriction was performed in CB2-/--mice and their wildtype littermates (CB2+/+; n=8-12/group). Taqman® RT-qPCR analysis was performed after 3 and 7 days. After M-mode echocardiography and Millar® pressure-vol…

Pressure overloadmedicine.medical_specialtyPhysiologybusiness.industryInflammationmedicine.diseaseMuscle hypertrophyInterleukin 10EndocrinologyPhysiology (medical)Heart failureInternal medicineAortic valve stenosismedicineCardiologylipids (amino acids peptides and proteins)Myocardial fibrosismedicine.symptomCardiology and Cardiovascular MedicinebusinessReceptorCardiovascular Research
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