Search results for "interleukin 10"

showing 10 items of 146 documents

Activation and methotrexate-mediated suppression of the TNF alpha promoter in T cells and macrophages.

1998

Transcriptional ActivationCD4-Positive T-LymphocytesRecombinant Fusion ProteinsT-LymphocytesLymphocyte ActivationTransfectionGeneral Biochemistry Genetics and Molecular BiologyCell LineText miningHistory and Philosophy of SciencemedicineHumansPromoter Regions GeneticCells Culturedbusiness.industryTumor Necrosis Factor-alphaGeneral NeuroscienceMacrophagesInterleukin 10MethotrexateGene Expression RegulationCancer researchMethotrexateTumor necrosis factor alphabusinessmedicine.drugAnnals of the New York Academy of Sciences
researchProduct

Proceedings of Réanimation 2017, the French Intensive Care Society International Congress

2017

biologybusiness.industry030208 emergency & critical care medicineLymphocyte proliferationCritical Care and Intensive Care MedicineMeeting Abstractslaw.invention03 medical and health sciencesInterleukin 100302 clinical medicine030228 respiratory systemlawPD-L1ImmunologyPostoperative infectionCardiopulmonary bypassbiology.proteinMedicine030212 general & internal medicinebusinessAnnals of Intensive Care
researchProduct

Donor and host B cell-derived IL-10 contributes to suppression of graft-versus-host disease

2014

Graft-versus-host disease (GvHD) is a frequent life-threatening complication following allogeneic HSC transplantation (HSCT). IL-10 is a regulatory cytokine with important roles during GvHD, yet its relevant sources, and mode of action, remain incompletely defined in this disease. Using IL-10-deficient donor or host mice (BALB/c or C57BL/6, respectively) in a MHC-mismatched model for acute GvHD, we found a strongly aggravated course of the disease with increased mortality when either donor or host cells could not produce this cytokine. A lack of IL-10 resulted in increased allogeneic T-cell responses and enhanced activation of host DCs in spleen and MLNs. Remarkably, IL-10 was prominently p…

biologymedicine.medical_treatmentImmunologychemical and pharmacologic phenomenaSpleenmedicine.diseaseInterleukin 10surgical procedures operativemedicine.anatomical_structureGraft-versus-host diseaseCytokineimmune system diseasesCD1DImmunologybiology.proteinmedicineImmunology and AllergyCD5Mode of actionB cellEuropean Journal of Immunology
researchProduct

Reciprocal influence of B cells and tumor macro and microenvironments in the ApcMin/+ model of colorectal cancer

2017

One of the most fascinating aspects of the immune system is its dynamism, meant as the ability to change and readapt according to the organism needs. Following an insult, we assist to the spontaneous organization of different immune cells which cooperate, locally and at distance, to build up an appropriate response. Throughout tumor progression, adaptations within the systemic tumor environment, or macroenvironment, result in the promotion of tumor growth, tumor invasion and metastasis to distal organs, but also to dramatic changes in the activity and composition of the immune system. In this work, we show the changes of the B-cell arm of the immune system following tumor progression in the…

lcsh:Immunologic diseases. Allergy0301 basic medicineColorectal cancerImmunologySpleenintestinal cancerBiologylcsh:RC254-282Metastasis03 medical and health sciencesImmune systemPeritoneummedicineImmunology and Allergyapcmin/+ miceApcMin/+mice; B lymphocytes; IgA; IL-10; intestinal cancer; Immunology and Allergy; Immunology; OncologyB lymphocyteApcMin/+micelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseasePhenotypeInterleukin 10030104 developmental biologymedicine.anatomical_structureOncologyTumor progressionIL-10Immunologyb lymphocyteslcsh:RC581-607IgAOncoImmunology
researchProduct

Interleukin 10 restores lipopolysaccharide-induced alterations in synaptic plasticity probed by repetitive magnetic stimulation

2020

Systemic inflammation is associated with alterations in complex brain functions such as learning and memory. However, diagnostic approaches to functionally assess and quantify inflammation-associated alterations in synaptic plasticity are not well-established. In previous work, we demonstrated that bacterial lipopolysaccharide (LPS)-induced systemic inflammation alters the ability of hippocampal neurons to express synaptic plasticity, i.e., the long-term potentiation (LTP) of excitatory neurotransmission. Here, we tested whether synaptic plasticity induced by repetitive magnetic stimulation (rMS), a non-invasive brain stimulation technique used in clinical practice, is affected by LPS-induc…

lcsh:Immunologic diseases. AllergyLipopolysaccharides0301 basic medicinenon-invasive brain stimulationInterleukin-1betaImmunologyTNFα-reporter mouseMice TransgenicStimulationNeurotransmissionHippocampusSynaptic TransmissionneuroinflammationInterferon-gammaMice03 medical and health sciences0302 clinical medicineGenes Reportertranscranial magnetic stimulationAnimalsImmunology and Allergyddc:610NeuroinflammationOriginal ResearchInflammationNeuronsNeuronal Plasticitysynaptic plasticityInterleukin-6Tumor Necrosis Factor-alphaChemistryLong-term potentiationInterleukin-10Mice Inbred C57BLOrganoids030104 developmental biologyBrain stimulationSynaptic plasticityExcitatory postsynaptic potentialTumor necrosis factor alphaMicrogliainterleukin 10lcsh:RC581-607Neuroscience030217 neurology & neurosurgery
researchProduct

Tolerogenic Dendritic Cells for Regulatory T Cell Induction in Man.

2015

Dendritic cells are (DC) highly specialized professional antigen-presenting cells (APC) that regulate immune responses, maintaining the balance between tolerance and immunity. Mechanisms via which they can promote central and peripheral tolerance include clonal deletion, inhibition of memory T cell responses, T cell anergy and induction of regulatory T cells. These properties have led to the analysis of human tolerogenic DC as a therapeutic strategy for induction or re-establishment of tolerance. In the recent years, numerous protocols for the generation of human tolerogenic DC have been developed and their tolerogenic mechanisms, including induction of regulatory T cells, are relatively we…

lcsh:Immunologic diseases. AllergyRegulatory T celldendritic cellmedicine.medical_treatmentImmunologychemical and pharmacologic phenomenaReviewClonal deletionregulatory T cellsImmune systemmedicineImmunology and Allergystudyhumanstolerancebusiness.industryPeripheral tolerancehemic and immune systemsImmunotherapyDendritic cellvaccinationInterleukin 10medicine.anatomical_structureImmunologyIL-10lcsh:RC581-607businessMemory T cellFrontiers in immunology
researchProduct

Evaluation of cytokine polymorphisms (TNFalpha, IFNgamma and IL-10) in Down patients with coeliac disease.

2005

Abstract Background In Down syndrome there is an increased prevalence of coeliac disease, but the reasons for this association are yet unknown. Aims To evaluate a possible correlation between TNFα, IFNγ and IL-10 genotype polymorphisms with the susceptibility to coeliac disease in Down syndrome patients. Methods Single nucleotide polymorphisms of TNFα (−308G → A promoter region), IFNγ (+874T → A promoter region) and IL-10 (−1082G → A promoter region) have been studied in 10 Down patients with coeliac disease, in 40 Down patients without coeliac disease and in 220 healthy controls. Clinical features were also studied in coeliac disease–Down syndrome patients. Results The 10 coeliac disease–D…

medicine.medical_specialtyDown syndromeAdolescentSingle-nucleotide polymorphismGastroenterologyPolymorphism Single NucleotideCoeliac diseaseSerologyInterferon-gammaInternal medicineGenotypeBiopsymedicineHumansGenetic Predisposition to DiseaseChildPolymorphism GeneticHepatologymedicine.diagnostic_testbusiness.industryTumor Necrosis Factor-alphaGastroenterologynutritional and metabolic diseasesInfantmedicine.diseasesyndromedigestive system diseasesSurgeryInterleukin-10Interleukin 10Celiac DiseaseChild PreschoolCytokinesDownDown SyndromebusinessTrisomyDigestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
researchProduct

Regulation of endotoxin-induced IL-6 production in liver sinusoidal endothelial cells and Kupffer cells by IL-10

1997

SUMMARY Sinusoidal endothelial cells and Kupffer cells are the first cell populations in the liver that come into contact with gut-derived endotoxin in portal blood. Although endotoxin concentrations as high as 1 ng/ml are physiologically present in portal blood, no local inflammation is seen. We show that the proinflammatory cytokine IL-6, which is central to the development of inflammatory reactions in the liver, is produced by sinusoidal endothelial cells and Kupffer cells in response to low concentrations of endotoxin (100 pg/ml to 1 ng/ml). The anti-inflammatory cytokine IL-10 down-regulated endotoxin-induced IL-6 release in endothelial and Kupffer cells. Importantly, Kupffer cells sec…

medicine.medical_specialtyEndotheliumKupffer Cellsmedicine.medical_treatmentImmunologyInflammationBiologyProinflammatory cytokineMiceInternal medicinemedicineAnimalsImmunology and AllergyInterleukin 4Mice Inbred BALB CInterleukin-6MicrocirculationKupffer cellOriginal ArticlesInterleukin-10EndotoxinsEndothelial stem cellInterleukin 10medicine.anatomical_structureCytokineEndocrinologyLiverImmunologyEndothelium Vascularmedicine.symptomClinical and Experimental Immunology
researchProduct

Estrogen regulates cytokine production and apoptosis in PMA-differentiated, macrophage-like U937 cells

2003

We have investigated the effects of sex steroids, estradiol (E2), and testosterone (T) on the synthesis of tumor necrosis factor alpha (TNF-alpha) and interleukin-10 (IL-10) in phorbol-myristate-acetate (PMA)-differentiated human monoblastic U937 cells. The ability of both hormones to modulate the viability and programmed cell death of macrophage-like PMA-differentiated U937 cells was also inspected. E2 increased TNF-alpha synthesis, whereas T had no effect on the production of this cytokine. The combination of E2 and its antagonist tamoxifen or ICI-182,789 completely abolished the induction of TNF-alpha, while combination of T and its antagonist Casodex (CSDX) did not significantly affect …

medicine.medical_specialtyProgrammed cell deathmedicine.drug_classmedicine.medical_treatmentCell BiologyBiologyBiochemistryCell biologyInterleukin 10CytokineEndocrinologyEstrogenApoptosisInternal medicinemedicineMacrophageTumor necrosis factor alphaIL-2 receptorMolecular BiologyJournal of Cellular Biochemistry
researchProduct

Responses to self and non-self intestinal microflora in health and inflammatory bowel disease.

1997

medicine.medical_specialtyT-LymphocytesImmunologyBiologyCross ReactionsInflammatory bowel diseaseGastroenterologyAntibodiesImmune tolerancePathogenesisMiceImmune systemCrohn DiseaseInternal medicinemedicineAnimalsHumansCrohn diseaseCross reactionsmedicine.diseaseInflammatory Bowel DiseasesUlcerative colitisInterleukin-12Interleukin-10IntestinesInterleukin 10Disease Models AnimalImmunologyForecastingResearch in immunology
researchProduct