Search results for "interleukin 10"

showing 10 items of 146 documents

Autocrine production of interleukin-4 and interleukin-10 is required for survival and growth of thyroid cancer cells.

2006

AbstractAlthough CD95 and its ligand are expressed in thyroid cancer, the tumor cell mass does not seem to be affected by such expression. We have recently shown that thyroid carcinomas produce interleukin (IL)-4 and IL-10, which promote resistance to chemotherapy through the up-regulation of Bcl-xL. Here, we show that freshly purified thyroid cancer cells were completely refractory to CD95-induced apoptosis despite the consistent expression of Fas-associated death domain and caspase-8. The analysis of potential molecules able to prevent caspase-8 activation in thyroid cancer cells revealed a remarkable up-regulation of cellular FLIPL (cFLIPL) and PED/PEA-15, two antiapoptotic proteins whos…

Cancer Researchmedicine.medical_treatmentNF-KAPPA-BOligonucleotidesC-FLIPCASP8 and FADD-Like Apoptosis Regulating ProteinApoptosisSuppressor of Cytokine Signaling ProteinsSIGNALING COMPLEXThyroid cancerTumorCARCINOMA CELLSANDROGEN RECEPTORIntracellular Signaling Peptides and ProteinsInterleukinHASHIMOTOS-THYROIDITISMiddle AgedProtein-Tyrosine KinasesInterleukin-10Up-RegulationMALIGNANT GLIOMA-CELLSInterleukin 10CytokineOncologyAged; Antibodies; Apoptosis; CASP8 and FADD-Like Apoptosis Regulating Protein; Cell Growth Processes; Cell Line Tumor; Humans; Interleukin-10; Interleukin-4; Intracellular Signaling Peptides and Proteins; Janus Kinase 1; Middle Aged; Oligonucleotides Antisense; Phosphoproteins; Protein-Tyrosine Kinases; Repressor Proteins; STAT6 Transcription Factor; Suppressor of Cytokine Signaling 1 Protein; Suppressor of Cytokine Signaling Proteins; Thyroid Neoplasms; Up-Regulation; fas Receptor; Oncology; Cancer Researchmedicine.medical_specialtyANTIAPOPTOTIC PROTEINSCell Growth ProcessesAntibodiesCell LineThyroid carcinomaSuppressor of Cytokine Signaling 1 ProteinSettore MED/04 - PATOLOGIA GENERALEInternal medicineCell Line TumormedicineHumansThyroid Neoplasmsfas ReceptorAntisenseAutocrine signallingInterleukin 4AgedAPOPTOSIS-INDUCING LIGANDbusiness.industryJanus Kinase 1Oligonucleotides Antisensemedicine.diseasePhosphoproteinsRepressor ProteinsEndocrinologyCancer cellCancer researchInterleukin-4businessApoptosis Regulatory ProteinsSTAT6 Transcription FactorCancer research
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Anti-inflammatory effects of chemically modified tetracyclines by the inhibition of nitric oxide and interleukin-12 synthesis in J774 cell line

2001

We investigated the effects of chemically modified tetracyclines (CMTs) on the production of nitric oxide (NO) and on the synthesis of some cytokines: tumour necrosis factor alpha (TNF-alpha), interleukin(IL)-10 and IL-12 in lipopolysaccharide (LPS)-treated J774 cell line. Furthermore, we studied the ability of these drugs to modify the viability in LPS-stimulated J774 macrophages. CMTs decreased, in a dose-dependent manner, inducible NO synthase (iNOS) activity and, consequently, nitrite formation in J774 cultures. The CMT-induced decrease in NO production is due to the inhibition of enzyme activity rather than to a direct effect on enzyme expression. The absence of the inhibition in mRNA …

Cell Survivalmedicine.medical_treatmentImmunologyNitric Oxide Synthase Type IIApoptosisEnzyme-Linked Immunosorbent AssayNitric OxideCell LineNitric oxideMicechemistry.chemical_compoundEthidiumIn Situ Nick-End LabelingmedicineAnimalsImmunology and AllergyRNA MessengerViability assayEnzyme InhibitorsFluorescent DyesPharmacologybiologyReverse Transcriptase Polymerase Chain ReactionAnti-Inflammatory Agents Non-SteroidalInterleukinBiological activityInterleukin-12Acridine OrangeCell biologyNitric oxide synthaseInterleukin 10CytokinechemistryBiochemistryTetracyclinesApoptosisbiology.proteinCytokinesElectrophoresis Polyacrylamide GelIndicators and ReagentsNitric Oxide SynthaseInternational Immunopharmacology
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Interleukin-10-treated dendritic cells do not inhibit Th2 immune responses in ovalbumin/alum-sensitized mice.

2005

<i>Background:</i> It is well known that the immunoregulatory cytokine interleukin (IL)-10 inhibits the accessory function of human dendritic cells (DC) in vitro. Recently, we have shown that these IL-10 DC inhibit the production of T helper cell 1 (Th1) and T helper cell 2 (Th2) cytokines by T cells from atopic individuals in vitro. The current study was set out to analyze whether IL-10 DC also exert inhibitory effects in vivo in a murine model of allergy to ovalbumin adsorbed to the adjuvant aluminium hydroxide (OVA/alum). <i>Methods:</i> OVA-pulsed or unpulsed bone marrow-derived DC, treated with IL-10 or left untreated during generation, were injected intravenous…

Cell TransplantationOvalbuminmedicine.medical_treatmentImmunologyLymphocyte ActivationMiceImmune systemTh2 CellsAdjuvants ImmunologicmedicineHypersensitivityImmune ToleranceImmunology and AllergyAnimalsAntigen-presenting cellCell ProliferationMice Inbred BALB CbiologyInterleukinGeneral MedicineDendritic cellDendritic CellsImmunoglobulin EFlow CytometryInterleukin-10OvalbuminInterleukin 10Disease Models AnimalCytokineImmunologybiology.proteinInterleukin 12Alum CompoundsCytokinesFemaleInternational archives of allergy and immunology
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Mast cells control the expansion and differentiation of IL-10-competent B cells

2014

Abstract The discovery of B cell subsets with regulatory properties, dependent on IL-10 production, has expanded our view on the mechanisms that control inflammation. Regulatory B cells acquire the ability to produce IL-10 in a stepwise process: first, they become IL-10 competent, a poised state in which B cells are sensitive to trigger signals but do not actually express the Il-10 gene; then, when exposed to appropriate stimuli, they start producing IL-10. Even if the existence of IL-10–competent B cells is now well established, it is not yet known how different immune cell types cross talk with B cells and affect IL-10–competent B cell differentiation and expansion. Mast cells (MCs) contr…

Cell typeRegulatory B cellsCellular differentiationImmunologyCD40 LigandB-Lymphocyte SubsetsRegulatory B cellsB-cellBiologyExosomesLymphocyte ActivationImmunophenotypingMast cellMiceImmunophenotypingImmune systemmedicineImmunology and AllergyAnimalsMast CellsB cell differentiationCD40 AntigensB cellmast cell; IL-10; B-cellMice KnockoutCD40Cell DifferentiationCell biologyInterleukin-10Gastrointestinal TractInterleukin 10medicine.anatomical_structurePhenotypeMast cell; Regulatory B cells; IL-10; B cell differentiationImmunologyIL-10biology.proteinFemaleJournal of immunology (Baltimore, Md.
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Impaired contact hypersensitivity to trinitrochlorobenzene in interleukin-4-deficient mice

1999

We have examined the role of endogenously produced interleukin-4 (IL-4) in the contact hypersensitivity (CH) reaction to the haptene trinitrochlorobenzene (TNCB). The CH reaction was abolished in IL-4 genetically deficient mice (IL-4 KO), when compared to wild-type (wt) mice. The CH reaction was restored by treatment with IL-4 and further analysis revealed that IL-4 exerted its action both at the induction and effector stages of the CH reaction. Despite failure to develop a CH reaction, IL-4 KO mice developed a T helper type 1 (Th1) response to TNCB, in terms of lymphokine production in vitro. Furthermore, the number of Vgamma3+ cells accumulating in the lymph nodes of TNCB-immune IL-4 KO m…

ChemistryImmunologyLymphokineInflammationMolecular biologyProinflammatory cytokineOxazolonePicryl chlorideInterleukin 10chemistry.chemical_compoundInterleukin 13ImmunologymedicineImmunology and Allergymedicine.symptomInterleukin 4Immunology
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Tolerance towards resident intestinal flora in mice is abrogated in experimental colitis and restored by treatment with interleukin-10 or antibodies …

1996

There is now increasing evidence that hyperresponsiveness towards intestinal flora is a crucial event in the pathogenesis of inflammatory bowel disease (IBD). In support of this hypothesis, we recently described in humans that tolerance exists towards indigenous intestinal flora but is broken in active IBD lesions. In the present study, we have attempted to transfer this model into mice from different genetic backgrounds (BALB/c, SJL/J, C3H/HeJ). We found that mononuclear cells from spleen, small bowel and large bowel of mice do not proliferate, i.e. are tolerant when exposed to bacterial sonicates derived from autologous intestine (BsA) but do proliferate, i.e. are immune when exposed to b…

ColonImmunologySpleenBiologyLymphocyte ActivationInflammatory bowel diseaseMicrobiologyMicePeyer's PatchesImmune systemCrohn DiseaseSpecies SpecificityImmunityIntestine SmallImmune TolerancemedicineAnimalsHumansImmunologic FactorsImmunology and AllergyColitisMice Inbred BALB CMice Inbred C3HBacteriaAntibodies MonoclonalInterleukinColitismedicine.diseaseInterleukin-12Recombinant ProteinsInterleukin-10RatsSpecific Pathogen-Free OrganismsIntestinesDisease Models AnimalInterleukin 10medicine.anatomical_structureTrinitrobenzenesulfonic AcidImmunologyLeukocytes MononuclearInterleukin 12SpleenEuropean Journal of Immunology
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P051 Macrophages as a source of Notch Ligands in Crohn’s disease: implications in fibrosis

2021

Abstract Background Fibrosis constitute the main complications associated to Crohn’s disease (CD). Notch signalling has been implicated in lung, kidney, liver and cardiac fibrosis. Macrophages contribute to fibrosis through the release of different mediators and the pattern of secretion may vary according to their microenvironment. The aim of the present study is to analyze the role of Notch ligands derived from macrophages in the complications of CD. Methods We have analyzed: the mRNA expression of cytokines and Notch ligands in CD patients with fistulizing and stenting pattern, the mRNA and protein expression of macrophage markers and Notch ligands in macrophages treated with the main cyt…

Crohn's diseasebiologybusiness.industrymedicine.medical_treatmentGastroenterologyVimentinGeneral Medicinemedicine.diseaseInterleukin 10CytokineFibrosismedicinebiology.proteinCancer researchSignal transductionInterleukin 6businessInterleukin 4Journal of Crohn's and Colitis
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The phosphodiesterase 4 inhibitor roflumilast augments the Th17-promoting capability of dendritic cells by enhancing IL-23 production, and impairs th…

2016

Phosphodiesterase 4 (PDE4) inhibitors serve to prevent degradation of the intracellular second messenger cAMP, resulting in broad anti-inflammatory effects on different cell types including immune cells. Agents that elevate cAMP levels via activation of adenylate cyclase have been shown to imprint a Th17-promoting capacity in dendritic cells (DCs). Therefore, we studied the potential of therapeutically relevant PDE inhibitors to induce a pronounced Th17-skewing capacity in DCs. Here we show that mouse bone marrow-derived (BM-) DCs when treated with the PDE4 inhibitor roflumilast (ROF, trade name: Daxas) in the course of stimulation with LPS (ROF-DCs) evoked elevated IL-17 levels in cocultur…

Cyclopropanes0301 basic medicineT cellImmunologyAnti-Inflammatory AgentsAminopyridinesStimulationBiologyLymphocyte ActivationInterleukin-23Mice03 medical and health sciencesTh2 Cells0302 clinical medicineImmune systemHypersensitivitymedicineAnimalsImmunology and AllergyNeutralizing antibodyProtein kinase ACells CulturedRoflumilastPharmacologyMice Inbred BALB CDendritic CellsInterleukin-10Cell biologyMice Inbred C57BLInterleukin 10030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisBenzamidesImmunologybiology.proteinTh17 CellsPhosphodiesterase 4 InhibitorsInterleukin 17medicine.drugInternational Immunopharmacology
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Administration of a Synbiotic to Free-Living Elderly and Evaluation of Serum Cytokines. A Pilot Study

2010

Ten free-living elderly were administered with a synbiotic [fermented milk containing Lactobacillus rhamnosus Gorbach and Goldin (LGG)] and oligofructose as a prebiotic for one month. Serum cytokines were evaluated before (T(0)) and after (T(1)) synbiotic administration. At T(0), values of Interleukin (IL)-12, IL-6, IL-10, IL-1beta and Tumor Necrosis Factor (TNF)-alpha were lower than normal controls, with the exception of IL-8, thus confirming previous results on the impairment of both innate and adaptive responses in elderly. At T(1), the synbiotic was able to significantly increase, depressed values of IL-1, IL-6 and IL-8 with a trend to a modest increase for the restant cytokines. In co…

Cytokines immunosenescence Lactobacillus rhamnosus Gorbach and Goldin (LGG) oligofructose probiotics synbiotics elderly.medicine.medical_specialtySynbioticsmedicine.medical_treatmentPilot ProjectsGastroenterologyImmune systemLactobacillus rhamnosusInternal medicineDrug DiscoverymedicineHumansAgedAged 80 and overPharmacologybiologyLacticaseibacillus rhamnosusbusiness.industryProbioticsInterleukinImmunosenescenceFlow Cytometrybiology.organism_classificationInterleukin 10CytokineImmune SystemImmunologyInterleukin 12Cytokinesbusiness
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Comparison of salivary cytokines levels among individuals with Down syndrome, cerebral palsy and normoactive

2019

Background Individuals with Down syndrome (DS) present increased susceptibility to infections and high prevalence of periodontal disease. The objective of this study is to evaluate the salivary concentrations of IL-1β, IL-6, IL-8, IL-10, TNFα and IL-12p70 of DS individuals and compare to cerebral palsy (CP) and normoactive patients (all with gingivitis). Material and Methods Twenty-two individuals with DS, 24 with CP and 22 normoactive participated in this cross-sectional study. Salivary flow rate, osmolality rate, Oral Hygiene Index, Gingival Index (GI) and salivary inflammatory markers IL-1β, IL-6, IL-8, IL-10, TNFα and IL-12p70 were evaluated. Shapiro-Wilks, Chi-square, ANOVA One-Way and…

Down syndromemedicine.medical_specialtySalivamedicine.medical_treatmentGastroenterologyCerebral palsy03 medical and health sciencesGingivitis0302 clinical medicineStatistical significanceInternal medicinemedicineGeneral Dentistrybusiness.industryResearch030206 dentistrymedicine.disease:CIENCIAS MÉDICAS [UNESCO]Interleukin 10Cytokine030220 oncology & carcinogenesisUNESCO::CIENCIAS MÉDICASOdontostomatology for the Disabled or Special PatientsAnalysis of variancemedicine.symptombusiness
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